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Photodiagnosis and Photodynamic Therapy, ISSN 1572-1000, 12/2017, Volume 20, pp. 21 - 27
Ultraviolet (UV) radiation may induce skin alterations as observed in photoaging. Some recognized modifications are epidermal hyperplasia, amorphous deposition... 
Steady-state fluorescence spectroscopy | Hairless mice | Skin endogenous fluorescence | Photoaging | Protoporphyrin IX | AMINOLEVULINIC ACID | PHOTODYNAMIC THERAPY | CANCER | AUTOFLUORESCENCE | ONCOLOGY | PROTOPORPHYRIN-IX | IN-VIVO | COMPLICATIONS | MOUSE SKIN | PSORIASIS | ACTINIC KERATOSIS | Fluorescence spectroscopy | Fluorescence | Skin | Collagen | Radiation | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, pp. e0159998 - e0159998
Recent research found that Tiron was an effective antioxidant that could act as the intracellular reactive oxygen species (ROS) scavenger or alleviate the... 
IRRADIATION | OXIDATIVE STRESS | HUMAN SKIN | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | MECHANISMS | SODIUM 4,5-DIHYDROXYBENZENE-1,3-DISULFONATE | INDUCTION | C-JUN | NF-KAPPA-B | PROTEIN-KINASES | Promoter Regions, Genetic - radiation effects | Binding Sites - radiation effects | Humans | Transcriptional Activation - drug effects | Skin Aging - radiation effects | 1,2-Dihydroxybenzene-3,5-Disulfonic Acid Disodium Salt - pharmacology | Promoter Regions, Genetic - drug effects | Transcription Factor AP-1 - metabolism | Dermis - drug effects | Cytoprotection - drug effects | Signal Transduction - radiation effects | Dermis - cytology | Matrix Metalloproteinase 1 - genetics | Fibroblasts - metabolism | MAP Kinase Signaling System - radiation effects | Dermis - metabolism | Skin Aging - drug effects | Transcriptional Activation - radiation effects | Cells, Cultured | Dermis - radiation effects | Matrix Metalloproteinase 3 - metabolism | Antioxidants - pharmacology | Ultraviolet Rays - adverse effects | MAP Kinase Signaling System - drug effects | Fibroblasts - radiation effects | Signal Transduction - drug effects | Fibroblasts - drug effects | Transcription Factor AP-1 - radiation effects | Cytoprotection - genetics | Matrix Metalloproteinase 1 - metabolism | Cytoprotection - radiation effects | Matrix Metalloproteinase 3 - genetics | Genetic aspects | Skin | Genetic transcription | RNA | Analysis | Enzyme-linked immunosorbent assay | Oxidative stress | Reactive oxygen species | Transcription factors | Senescence | Oncology | Activation | Matrix metalloproteinase | Kinases | Proteins | Antioxidants | Signal transduction | Consent | Transcription activation | Fibroblasts | Aging | Biocompatibility | Metalloproteinase | Interstitial collagenase | Stromelysin 1 | Wound healing | U.V. radiation | Activator protein 1 | MAP kinase | Gene expression | Medicine | Signaling | Collagen | In vivo methods and tests | Binding sites | Apoptosis | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 6, pp. e11222 - e11222
Radiotherapy is a widely used treatment option in cancer. However, recent evidence suggests that doses of ionizing radiation (IR) delivered inside the tumor... 
SURVIVAL | APOPTOSIS | CELLS | THERAPY | VEGFR-2 | BIOLOGY | RECURRENCE | RADIOTHERAPY | Apoptosis - drug effects | Apoptosis - radiation effects | Humans | Microvessels - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Zebrafish - embryology | Regeneration - radiation effects | Bevacizumab | Neovascularization, Pathologic - pathology | Cell Hypoxia | Antibodies, Monoclonal, Humanized | Neoplasms - genetics | Neoplasms - radiotherapy | Proto-Oncogene Proteins c-akt - metabolism | Microvessels - radiation effects | Radiotherapy Dosage | MAP Kinase Signaling System - radiation effects | Antibodies, Monoclonal - pharmacology | Microvessels - drug effects | Proteoglycans - metabolism | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Endothelial Cells - radiation effects | Neoplasms - blood supply | Cell Movement - radiation effects | Collagen - metabolism | Animals | Neoplasm Metastasis - pathology | Cell Line, Tumor | Zebrafish - physiology | Gene Expression Regulation, Neoplastic - radiation effects | Mice | Laminin - metabolism | Endothelial Cells - pathology | Neoplasms - pathology | Dose-Response Relationship, Radiation | Drug Combinations | Proteins | Tyrosine | Care and treatment | Ionizing radiation | Growth | Cell death | Metastasis | Radiotherapy | Vascular endothelial growth factor | Endothelium | Cancer | Cell proliferation | Beams (radiation) | Animal models | Phosphorylation | Leukocyte migration | Laboratories | Leukemia | Oncology | Biology | Kinases | Tissues | Damage prevention | Cancer therapies | Mimicry | Cell adhesion & migration | Metastases | Angiogenesis | Embryogenesis | Biological effects | Cell cycle | Protein-tyrosine kinase | Drug dosages | Wound healing | Cell survival | Organs | Zebrafish | I.R. radiation | Breast cancer | Radiation therapy | Endothelial cells | Urban regeneration | Medical prognosis | Hypoxia | Radiation damage | Cell migration | Apoptosis | Index Medicus
Journal Article
Investigative Ophthalmology & Visual Science, ISSN 0146-0404, 08/2008, Volume 49, Issue 8, pp. 3402 - 3408
Journal Article
International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 2012, Volume 82, Issue 5, pp. 1559 - 1566
Purpose Blood-brain barrier (BBB) disruption is one of the major consequences of radiation-induced normal tissue injury in the central nervous system. We... 
Radiology | Hematology, Oncology and Palliative Medicine | Tissue inhibitor of metalloproteinases | Extracellular matrix | Matrix metalloproteinases | Whole-brain irradiation | Collagen type IV | LUNG | ACTIVATION | BARRIER | INJURY | RADIATION-THERAPY | ONCOLOGY | EXTRACELLULAR-MATRIX | RAT-BRAIN | CEREBRAL-ISCHEMIA | EXPRESSION | MMP-9 | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Matrix Metalloproteinase 2 - radiation effects | Rats, Inbred F344 | Tissue Inhibitor of Metalloproteinase-2 - radiation effects | Extracellular Matrix - metabolism | Male | RNA, Messenger - metabolism | Cerebral Cortex - metabolism | Cranial Irradiation - methods | Tissue Inhibitor of Metalloproteinase-2 - genetics | Brain - metabolism | Tissue Inhibitor of Metalloproteinase-2 - metabolism | Matrix Metalloproteinase 9 - metabolism | Radiation Injuries, Experimental - etiology | Time Factors | Extracellular Matrix - radiation effects | Matrix Metalloproteinase 9 - genetics | Gelatinases - metabolism | Female | Real-Time Polymerase Chain Reaction | Radiation Injuries, Experimental - metabolism | Collagen Type IV - metabolism | Matrix Metalloproteinase 2 - metabolism | Fluorescent Antibody Technique - methods | Mice, Inbred C57BL | Rats | Matrix Metalloproteinase 9 - radiation effects | Brain - radiation effects | Cranial Irradiation - adverse effects | Gelatinases - radiation effects | Cerebral Cortex - radiation effects | Hippocampus - metabolism | Matrix Metalloproteinase 2 - genetics | Animals | Collagen Type IV - radiation effects | Hippocampus - radiation effects | Collagen Type IV - genetics | Mice | Nervous system diseases | Collagen | Radiation | Brain | Central nervous system | Antibodies | Basement membranes | Matrix metalloproteinase | Tissue inhibitor of metalloproteinase 2 | Gene expression | Tissue inhibitor of metalloproteinase 1 | Gelatinase B | Gelatinase A | Polymerase chain reaction | gamma Radiation | Blood-brain barrier | Collagen (type IV) | Immunoreactivity | Injuries | Index Medicus | ANTIBODIES | PATHOGENESIS | IRRADIATION | COLLAGEN | POLYMERASE CHAIN REACTION | MESSENGER-RNA | MEMBRANES | RADIOLOGY AND NUCLEAR MEDICINE | BLOOD-BRAIN BARRIER | RADIATION DOSES | BRAIN | RADIATION INJURIES | Whole brain irradiation
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2012, Volume 7, Issue 7, pp. e39969 - e39969
Fibroblasts residing in connective tissues throughout the body are responsible for extracellular matrix (ECM) homeostasis and repair. In response to tissue... 
SMOOTH MUSCLE ACTIN | GROWTH-FACTOR-BETA | HEPATIC STELLATE CELLS | PHOTODEGRADABLE HYDROGELS | LUNG MYOFIBROBLASTS | GRANULATION-TISSUE | MULTIDISCIPLINARY SCIENCES | TRANSFORMING GROWTH-FACTOR-BETA-1 | EXTRACELLULAR-MATRIX | IDIOPATHIC PULMONARY-FIBROSIS | AORTIC-VALVE DISEASE | Aortic Valve - radiation effects | Gene Expression - drug effects | Hydrogels | Actins - metabolism | Aortic Valve - drug effects | Polyethylene Glycols - chemistry | Actins - genetics | Myofibroblasts - metabolism | Collagen Type I - genetics | Swine | Light | Myofibroblasts - radiation effects | Biomimetic Materials - chemistry | Transforming Growth Factor beta1 - pharmacology | Biomarkers - metabolism | Cell Differentiation - radiation effects | Oligopeptides - chemical synthesis | Collagen Type I - metabolism | Acrylates - chemistry | Cell Cycle - radiation effects | Aortic Valve - cytology | Myofibroblasts - drug effects | Aortic Valve - metabolism | Gene Expression - radiation effects | Fibronectins - metabolism | Elastic Modulus - radiation effects | Myofibroblasts - cytology | Animals | Cell Differentiation - drug effects | Cell Proliferation - drug effects | Connective Tissue Growth Factor - genetics | Fibronectins - genetics | Primary Cell Culture | Cell Cycle - drug effects | Connective Tissue Growth Factor - metabolism | Cell Proliferation - radiation effects | Fibronectins | Collagen | Genes | Bone morphogenetic proteins | Transforming growth factors | Intermediate filament proteins | Apoptosis | Vimentin | Bioengineering | Collagens | Homeostasis | Smooth muscle | Kinases | Tissues | Fibronectin | Mimicry | Connective tissues | Proteins | Engineering | Genotype & phenotype | Reduction | Cell fate | Actin | Topography | Rodents | Cell cycle | Fibroblasts | Extracellular matrix | Stiffness | Repair | Growth factors | Heart diseases | Phenotypes | Developmental biology | Contractility | Organs | Muscles | Mechanical properties | Connective tissue growth factor | Modulus of elasticity | Stiffening | Muscle contraction | Substrates | Fibrosis | Stem cells | Molecular biology | Index Medicus
Journal Article
Cancer Cell, ISSN 1535-6108, 12/2004, Volume 6, Issue 6, pp. 553 - 563
The recent landmark Phase III clinical trial with a VEGF-specific antibody suggests that antiangiogenic therapy must be combined with cytotoxic therapy for the... 
Immunohistochemistry | Basement Membrane - drug effects | Oligonucleotide Array Sequence Analysis | Apoptosis - drug effects | Humans | Gamma Rays - therapeutic use | Antibodies, Monoclonal - therapeutic use | Male | Vascular Endothelial Growth Factor Receptor-2 - immunology | Pericytes - physiology | Blood Vessels - radiation effects | Pericytes - chemistry | Time Factors | Angiopoietin-1 - physiology | Neovascularization, Pathologic - radiotherapy | Fluorescein Angiography | Receptor, TIE-2 - antagonists & inhibitors | Antibodies, Monoclonal - pharmacology | Brain Neoplasms - drug therapy | Basement Membrane - metabolism | Blotting, Western | Ephrin-B2 - genetics | Cell Movement - drug effects | Mice, Nude | Models, Biological | Neovascularization, Pathologic - drug therapy | Cell Line, Tumor | Collagen Type IV - genetics | Mice | Glioma - drug therapy | Apoptosis - radiation effects | Pericytes - cytology | Basement Membrane - pathology | Glioma - radiotherapy | Brain Neoplasms - metabolism | Oxygen - metabolism | Glioma - metabolism | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Transfection | Antigens - analysis | Angiogenesis Inhibitors - therapeutic use | Gene Expression Regulation, Neoplastic - drug effects | Brain Neoplasms - radiotherapy | Collagen Type IV - metabolism | Blood Vessels - chemistry | Dipeptides - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Proteoglycans - analysis | Up-Regulation - genetics | Receptor, TIE-2 - immunology | Collagen Type IV - analysis | Xenograft Model Antitumor Assays | Animals | Blood Vessels - drug effects | Matrix Metalloproteinase Inhibitors | Combined Modality Therapy - methods | Matrix Metalloproteinases - metabolism | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, pp. e0119334 - e0119334
While human mesenchymal stem cells (hMSCs), either in the bone marrow or in tumour microenvironment could be targeted by radiotherapy, their response is poorly... 
CARTILAGE REPAIR | MATRIX SYNTHESIS | IRRADIATION | MARROW | BONE MORPHOGENETIC PROTEIN-2 | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | IONIZING-RADIATION | STROMAL CELLS | HYPOXIA | CANCER-THERAPY | Mesenchymal Stromal Cells - radiation effects | Humans | Male | Young Adult | G2 Phase Cell Cycle Checkpoints - radiation effects | Mesenchymal Stromal Cells - cytology | Cellular Senescence - radiation effects | Adult | Collagen - genetics | Osteoblasts - cytology | Chondrogenesis | Cell Differentiation - radiation effects | Cell Line | Gene Expression | Osteoblasts - radiation effects | Cell Cycle - radiation effects | Oxygen Consumption | Mesenchymal Stromal Cells - metabolism | Cell Survival - radiation effects | Collagen - metabolism | X-Rays | Adolescent | Osteoblasts - metabolism | Osteogenesis | Cell Proliferation - radiation effects | Cell culture | Alkaline phosphatase | Collagen (type I) | Radiosensitivity | Mesenchyme | Sox9 protein | Genes | Radiation | Impairment | Collagens | Osteoblasts | Mimicry | Defects | Degradation | Proteins | Cell cycle | Bone marrow | Biocompatibility | Radiation effects | Cbfa-1 protein | Osteopontin | Radiation therapy | Gene expression | Collagenase 3 | Osteoblastogenesis | S phase | Stem cells | Irradiation | Hypoxia | Bone | Differentiation | Viability | Three dimensional models | α Radiation | Tumors | Index Medicus | Biomedical materials
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2013, Volume 8, Issue 9, pp. e74141 - e74141
Journal Article