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Journal of Cell Biology, ISSN 0021-9525, 06/2017, Volume 216, Issue 6, pp. 1761 - 1774
The unfolded protein response (UPR) handles unfolded/misfolded proteins accumulated in the endoplasmic reticulum (ER). However, it is unclear how vertebrates... 
UNFOLDED PROTEIN RESPONSE | NOTOCHORD | MOUSE T-GENE | TRANSCRIPTIONAL INDUCTION | RETICULUM EXIT SITES | ENDOPLASMIC-RETICULUM | MEDAKA FISH | STRESS | TRANSMEMBRANE PROTEIN | CELL BIOLOGY | Basic-Leucine Zipper Transcription Factors - metabolism | Notochord - secretion | Humans | Embryo, Nonmammalian - metabolism | Endoplasmic Reticulum - metabolism | Activating Transcription Factor 6 - genetics | Collagen Type II - metabolism | Oryzias - metabolism | Fish Proteins - genetics | Notochord - metabolism | Transfection | Time Factors | Gene Expression Regulation, Developmental | Collagen Type II - secretion | Transcription, Genetic | Oryzias - genetics | Animals, Genetically Modified | HCT116 Cells | COP-Coated Vesicles - metabolism | Fish Proteins - metabolism | Genotype | Basic-Leucine Zipper Transcription Factors - genetics | Unfolded Protein Response | Basement Membrane - metabolism | Protein Transport | Activating Transcription Factor 6 - metabolism | Phenotype | Animals | COP-Coated Vesicles - secretion | Endoplasmic Reticulum Stress | Vacuoles - metabolism | Oryzias - embryology | Physiological aspects | Embryonic development | Collagen | Exports | Pattern formation | Transducers | Collagen (type II) | Secretion | Chains | Chaperones | Cells | Proteins | Embryonic growth stage | Signal transduction | Vertebrates | Embryogenesis | Protein folding | Fish | Stress response | Endoplasmic reticulum | Notochord | Enlargement | Cargo handling | Index Medicus
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2010, Volume 285, Issue 4, pp. 2580 - 2590
Collagen triple helices are stabilized by 4-hydroxyproline residues. No function is known for the much less common 3-hydroxyproline (3Hyp), although genetic... 
PROLYL 3-HYDROXYLATION | AMINO-ACID-SEQUENCE | ARTICULAR-CARTILAGE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CYANOGEN-BROMIDE PEPTIDES | RECESSIVE OSTEOGENESIS IMPERFECTA | TRIPLE-HELIX | BASEMENT-MEMBRANE COLLAGEN | CARTILAGE COLLAGEN | HIGHER-ORDER STRUCTURES | COVALENT STRUCTURE | Collagen Type V - genetics | Humans | Collagen Type I - chemistry | Collagen Type III - metabolism | Molecular Sequence Data | Collagen - chemistry | Collagen Type XI - chemistry | Collagen Type XI - metabolism | Collagen Type II - metabolism | Young Adult | Tandem Mass Spectrometry | Collagen Type I - genetics | Collagen Type XI - genetics | Cattle | Bone and Bones - metabolism | Adult | Collagen - genetics | Collagen Type V - metabolism | Hydroxyproline - chemistry | Extracellular Matrix Proteins - metabolism | Amino Acid Sequence | Extracellular Matrix Proteins - chemistry | Collagen Type I - metabolism | Collagen Type V - chemistry | Bone and Bones - chemistry | Extracellular Matrix Proteins - genetics | Hydroxyproline - metabolism | Collagen Type III - chemistry | Collagen Type III - genetics | Cartilage - metabolism | Collagen Type II - genetics | Collagen - metabolism | Hydroxyproline - genetics | Animals | Chickens | Cartilage - chemistry | Collagen Type II - chemistry | Protein Processing, Post-Translational | Index Medicus | Mammal | Protein Structure and Folding | Cartilage | Extracellular Matrix | Post-translational Modification | Collagen | Hydroxyproline | Organisms | 3-hydroxyproline | Bone | Protein
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 09/2015, Volume 309, Issue 5, pp. H906 - H917
Macrophages accumulate in blood vessels during hypertension. However, their contribution to vessel remodeling is unknown. In the present study, we examined the... 
Hypertension | Vessel remodeling | Blood pressure | Macrophage polarization | Angiotensin II | Chemokine (C-C motif) receptor 2 | blood pressure | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | vessel remodeling | CCR2 ANTAGONIST | MONOCYTES | ATHEROSCLEROSIS | macrophage polarization | MECHANISMS | INDUCED HYPERTENSION | HYPERTROPHY | INTERLEUKIN-17 | angiotensin II | ARTERIAL STIFFNESS | VASCULAR INFLAMMATION | PERIPHERAL VASCULAR DISEASE | hypertension | chemokine (C-C motif) receptor 2 | ALTERNATIVE ACTIVATION | Blood Pressure | Receptors, CCR2 - genetics | Antigens, Ly - genetics | Arginase - metabolism | Aorta - metabolism | Fibrosis - metabolism | Antigens, CD - genetics | Antigens, CD - metabolism | Hypertension - etiology | Antigens, Ly - metabolism | Collagen - genetics | Angiotensin II - toxicity | Elastin - metabolism | Macrophages - classification | Aorta - drug effects | Mice, Inbred C57BL | Arginase - genetics | Hypertension - pathology | Hypertension - metabolism | Aorta - pathology | Collagen - metabolism | Macrophages - metabolism | Receptors, CCR2 - metabolism | Animals | Elastin - genetics | Nitric Oxide Synthase Type II - genetics | Macrophages - drug effects | Mice | Fibrosis - pathology | Nitric Oxide Synthase Type II - metabolism | Coronary vessels | Rodents | Gene expression | ACE inhibitors | Chemokines | Index Medicus
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6028, pp. 478 - 484
The growth factor progranulin (PGRN) has been implicated in embryonic development, tissue repair, tumorigenesis, and inflammation, but its receptors remain... 
Cartilage | Immunization | Antiinflammatories | Medical treatment | REPORTS | Collagens | Antibodies | Bones | Arthritis | Mice | Inflammation | TUMOR-NECROSIS-FACTOR | GENE | MULTIDISCIPLINARY SCIENCES | HOST-DEFENSE | DEGRADATION | OLIGOMERIC MATRIX PROTEIN | GRANULIN-EPITHELIN PRECURSOR | ADAMTS-7 | MUTATIONS | TRANSCRIPTION FACTOR | PSEUDOACHONDROPLASIA | Recombinant Proteins - therapeutic use | Tumor Necrosis Factor-alpha - metabolism | Arthritis, Experimental - drug therapy | Recombinant Fusion Proteins - pharmacology | Humans | Middle Aged | Recombinant Fusion Proteins - therapeutic use | Intercellular Signaling Peptides and Proteins - chemistry | Male | Receptors, Tumor Necrosis Factor, Type I - metabolism | Recombinant Fusion Proteins - metabolism | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | T-Lymphocytes, Regulatory - immunology | Arthritis, Experimental - pathology | Young Adult | Intercellular Signaling Peptides and Proteins - metabolism | Cartilage, Articular - metabolism | Receptors, Tumor Necrosis Factor, Type II - metabolism | Adult | Female | Protein Interaction Domains and Motifs | Anti-Inflammatory Agents, Non-Steroidal - metabolism | T-Lymphocytes, Regulatory - physiology | Arthritis, Experimental - physiopathology | Signal Transduction | Intercellular Signaling Peptides and Proteins - genetics | Mice, Transgenic | Arthritis, Experimental - immunology | Mice, Inbred Strains | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Cartilage, Articular - pathology | Animals | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Adolescent | Ligands | Aged | Intercellular Signaling Peptides and Proteins - therapeutic use | Care and treatment | Genetic aspects | Properties | Tumor necrosis factor | Growth factors | Index Medicus | Binding | Pathology | Receptors | Pathogenesis
Journal Article
Journal Article
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 01/2018, Volume 93, Issue 1, pp. 147 - 158
We examined activin receptor type IIA (ActRIIA) activation in chronic kidney disease (CKD) by signal analysis and inhibition in mice with Alport syndrome using... 
cardiovascular disease | chronic kidney disease | bone | fibrosis | mineral metabolism | vascular calcification | TGF-BETA | CARDIOVASCULAR EVENTS | RENAL FIBROSIS | ARTERIAL CALCIFICATION | BONE MORPHOGENETIC PROTEIN-7 | GROWTH-FACTOR 23 | UROLOGY & NEPHROLOGY | SMOOTH-MUSCLE-CELLS | LINKED ALPORT-SYNDROME | OSTEOCLAST DIFFERENTIATION | ATHEROSCLEROTIC LESION | Kidney - pathology | Recombinant Fusion Proteins - pharmacology | Blood Vessels - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - genetics | Vascular Calcification - metabolism | Kidney - metabolism | Bone Resorption - genetics | Bone and Bones - metabolism | Myocardium - metabolism | Renal Insufficiency, Chronic - genetics | Bone Resorption - metabolism | Disease Models, Animal | Bone Resorption - physiopathology | Glomerular Filtration Rate | Signal Transduction | Smad2 Protein - metabolism | Cardiomegaly - physiopathology | Nephritis, Hereditary - genetics | Renal Insufficiency, Chronic - physiopathology | Mice, Knockout | Renal Insufficiency, Chronic - prevention & control | Cardiomegaly - prevention & control | Nephritis, Hereditary - metabolism | Collagen Type IV - deficiency | Activin Receptors, Type II - genetics | Fibrosis | Sp7 Transcription Factor - metabolism | Collagen Type IV - genetics | Cardiomegaly - metabolism | Activin Receptors, Type II - metabolism | Bone and Bones - pathology | Phosphorylation | Vascular Remodeling | Blood Vessels - pathology | Vascular Calcification - genetics | Actins - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - physiopathology | Activin Receptors, Type II - antagonists & inhibitors | Blood Vessels - physiopathology | Renal Insufficiency, Chronic - metabolism | Ventricular Remodeling | Chronic Kidney Disease-Mineral and Bone Disorder - prevention & control | Muscle Proteins - metabolism | Vascular Calcification - physiopathology | Microfilament Proteins - metabolism | Nephritis, Hereditary - physiopathology | Kidney - physiopathology | Vascular Calcification - prevention & control | Myocardium - pathology | Bone Resorption - prevention & control | Bone and Bones - physiopathology | Animals | Nephritis, Hereditary - drug therapy | Cardiomegaly - genetics | Bone Remodeling | Index Medicus | Chronic kidney disease
Journal Article
Acta Biomaterialia, ISSN 1742-7061, 05/2014, Volume 10, Issue 5, pp. 2005 - 2013
Scaffold pore size is an important factor affecting tissue regeneration efficiency. The effect of pore size on cartilage tissue regeneration was compared by... 
Ice particulate | Cartilage regeneration | Pore size | Porogen | Collagen porous scaffold | DESIGN | MATERIALS SCIENCE, BIOMATERIALS | ENGINEERING, BIOMEDICAL | POLY(L-LACTIC ACID) FOAMS | CHONDROCYTE DISTRIBUTION | IN-VITRO | ARCHITECTURE | CELL-CULTURE | TISSUE-ENGINEERED CARTILAGE | DIFFERENTIATION | BONE | GAG SCAFFOLDS |