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Arthritis & Rheumatology, ISSN 2326-5191, 01/2016, Volume 68, Issue 1, pp. 210 - 217
ObjectiveTo examine whether lung endothelial cells (ECs) from patients with systemic sclerosis (SSc)-associated interstitial lung disease (ILD) express... 
PATHOGENESIS | FIBROSIS | TRANSITION CONTRIBUTES | ENDOMT | MYOFIBROBLAST | INDUCTION | RHEUMATOLOGY | FIBROBLASTS | MOLECULAR-MECHANISMS | Immunohistochemistry | Lung Diseases, Interstitial - etiology | Lung Diseases, Interstitial - pathology | Humans | Middle Aged | Transforming Growth Factor beta1 - metabolism | Actins - metabolism | Collagen Type III - metabolism | Scleroderma, Systemic - pathology | Male | Epithelial-Mesenchymal Transition - genetics | Actins - genetics | Neovascularization, Pathologic - pathology | Collagen Type I - genetics | Scleroderma, Systemic - complications | Adult | Female | Lung - metabolism | Nuclear Proteins - genetics | Snail Family Transcription Factors | Lung - pathology | Collagen Type I - metabolism | Endothelial Cells - metabolism | Scleroderma, Systemic - genetics | Nuclear Proteins - metabolism | Transforming Growth Factor beta1 - genetics | Collagen Type III - genetics | Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Fibronectins - metabolism | Transcription Factors - metabolism | Microscopy, Confocal | Lung Diseases, Interstitial - genetics | Twist-Related Protein 1 - genetics | Neovascularization, Pathologic - genetics | Aged | Connective Tissue Growth Factor - genetics | Fibronectins - genetics | Twist-Related Protein 1 - metabolism | Connective Tissue Growth Factor - metabolism | Lungs | Microscopy | Growth factors | Lung diseases | Collagen | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2010, Volume 285, Issue 4, pp. 2580 - 2590
Collagen triple helices are stabilized by 4-hydroxyproline residues. No function is known for the much less common 3-hydroxyproline (3Hyp), although genetic... 
PROLYL 3-HYDROXYLATION | AMINO-ACID-SEQUENCE | ARTICULAR-CARTILAGE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CYANOGEN-BROMIDE PEPTIDES | RECESSIVE OSTEOGENESIS IMPERFECTA | TRIPLE-HELIX | BASEMENT-MEMBRANE COLLAGEN | CARTILAGE COLLAGEN | HIGHER-ORDER STRUCTURES | COVALENT STRUCTURE | Collagen Type V - genetics | Humans | Collagen Type I - chemistry | Collagen Type III - metabolism | Molecular Sequence Data | Collagen - chemistry | Collagen Type XI - chemistry | Collagen Type XI - metabolism | Collagen Type II - metabolism | Young Adult | Tandem Mass Spectrometry | Collagen Type I - genetics | Collagen Type XI - genetics | Cattle | Bone and Bones - metabolism | Adult | Collagen - genetics | Collagen Type V - metabolism | Hydroxyproline - chemistry | Extracellular Matrix Proteins - metabolism | Amino Acid Sequence | Extracellular Matrix Proteins - chemistry | Collagen Type I - metabolism | Collagen Type V - chemistry | Bone and Bones - chemistry | Extracellular Matrix Proteins - genetics | Hydroxyproline - metabolism | Collagen Type III - chemistry | Collagen Type III - genetics | Cartilage - metabolism | Collagen Type II - genetics | Collagen - metabolism | Hydroxyproline - genetics | Animals | Chickens | Cartilage - chemistry | Collagen Type II - chemistry | Protein Processing, Post-Translational | Index Medicus | Mammal | Protein Structure and Folding | Cartilage | Extracellular Matrix | Post-translational Modification | Collagen | Hydroxyproline | Organisms | 3-hydroxyproline | Bone | Protein
Journal Article
Cancer Science, ISSN 1347-9032, 02/2014, Volume 105, Issue 2, pp. 228 - 235
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2011, Volume 108, Issue 31, pp. 12925 - 12930
GPR56, an orphan G protein-coupled receptor (GPCR) from the family of adhesion GPCRs, plays an indispensable role in cortical development and lamination.... 
Brain | Receptors | Neurons | Neuroglia | Collagens | Cell lines | Ligands | Mice | Gene expression regulation | Laminates | Brain malformation | Meningeal fibroblasts | NEURONAL MIGRATION | BILATERAL FRONTOPARIETAL POLYMICROGYRIA | GPR56 | MULTIDISCIPLINARY SCIENCES | CELL-ADHESION | DEVELOPING CORTEX | brain malformation | CEREBRAL-CORTEX | meningeal fibroblasts | ALPHA-6 INTEGRINS | EHLERS-DANLOS-SYNDROME | SYNDROME TYPE-IV | BASEMENT-MEMBRANE | Immunohistochemistry | NIH 3T3 Cells | Basement Membrane - ultrastructure | Brain - embryology | Immunoprecipitation | Receptors, G-Protein-Coupled - metabolism | Humans | Collagen Type III - metabolism | Immunoblotting | Neurons - cytology | rhoA GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - genetics | Cerebral Cortex - metabolism | GTP-Binding Protein alpha Subunits, G12-G13 - metabolism | Microscopy, Immunoelectron | Brain - metabolism | Neurons - metabolism | Cobblestone Lissencephaly - genetics | Neurons - drug effects | Cells, Cultured | Cobblestone Lissencephaly - metabolism | Collagen Type III - genetics | Recombinant Proteins - pharmacology | Basement Membrane - metabolism | Mice, Knockout | Cell Movement - drug effects | Animals | Basement Membrane - embryology | Cerebral Cortex - embryology | Protein Binding | Receptors, G-Protein-Coupled - genetics | Physiological aspects | Genetic aspects | G proteins | Collagen | Membranes | Glycoproteins | Mutation | Rodents | Index Medicus | Biological Sciences
Journal Article
Nature Communications, ISSN 2041-1723, 04/2015, Volume 6, Issue 1, pp. 6774 - 6774
Journal Article
Nature Biotechnology, ISSN 1087-0156, 01/2003, Volume 21, Issue 1, pp. 52 - 56
Journal Article
Hypertension, ISSN 0194-911X, 07/2008, Volume 52, Issue 1, pp. 130 - 136
textabstractThe aim of this study was to explore the effects of the renin inhibitor aliskiren in streptozotocin-diabetic TG(mRen-2)27 rats. Furthermore, we... 
Aliskiren | Diabetic nephropathy | Renin inhibitor | (pro)renin receptor | TG(mRen-2)rat | RENIN/PRORENIN RECEPTOR | NONPROTEOLYTIC ACTIVATION | aliskiren | TRANSGENIC RATS | ANGIOTENSIN-II | NEPHROPATHY | MESANGIAL CELLS | PERIPHERAL VASCULAR DISEASE | renin inhibitor | HEART-RATE | PRORENIN | HYPERTENSION | diabetic nephropathy | Renin - metabolism | Antihypertensive Agents - pharmacology | Gene Expression - drug effects | Diabetic Nephropathies - etiology | Humans | Collagen Type III - metabolism | Male | Antihypertensive Agents - metabolism | Receptors, Cell Surface - antagonists & inhibitors | Albuminuria - physiopathology | Collagen Type I - genetics | Diabetic Nephropathies - physiopathology | Amides - pharmacokinetics | Albuminuria - metabolism | Blood Pressure - drug effects | Diabetes Mellitus, Experimental - complications | Diabetes Mellitus, Experimental - metabolism | Diabetes Mellitus, Experimental - physiopathology | Amides - pharmacology | Collagen Type IV - metabolism | Collagen Type I - metabolism | Albuminuria - etiology | Diabetic Nephropathies - metabolism | Rats | Receptors, Cell Surface - metabolism | Antihypertensive Agents - pharmacokinetics | Collagen Type III - genetics | Rats, Sprague-Dawley | Animals | Renin - antagonists & inhibitors | Transforming Growth Factor beta - genetics | Fumarates - pharmacokinetics | Fumarates - pharmacology | Collagen Type IV - genetics | Transforming Growth Factor beta - metabolism | Receptors, Cell Surface - genetics | Index Medicus
Journal Article
Journal of Nutrition, ISSN 0022-3166, 10/2012, Volume 142, Issue 10, pp. 1821 - 1828
We investigated whether quercetin protects from steatosis and limits the expression of proinflammatory and fibrogenic genes in C57BL/6J mice with nonalcoholic... 
GREEN TEA | CELLS | KAPPA-B ACTIVATION | NUTRITION & DIETETICS | REACTIVE OXYGEN | FATTY LIVER-DISEASE | GENE-EXPRESSION | RATS | FLAVONOIDS | MODEL | RECEPTOR 4 | Inflammation - pathology | Up-Regulation | Fibrosis - drug therapy | Liver - pathology | Fatty Liver - pathology | Collagen Type III - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Male | NF-kappa B - metabolism | RNA, Messenger - metabolism | HMGB1 Protein - genetics | Collagen Type I - genetics | Cyclooxygenase 2 - genetics | Inflammation - drug therapy | HMGB1 Protein - metabolism | Lipid Peroxidation - drug effects | Non-alcoholic Fatty Liver Disease | Thiobarbituric Acid Reactive Substances - analysis | JNK Mitogen-Activated Protein Kinases - genetics | Interleukin-6 - metabolism | Disease Models, Animal | Methionine - deficiency | Collagen Type I - metabolism | Interleukin-6 - genetics | Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Toll-Like Receptor 4 - genetics | Smad Proteins - genetics | Thiobarbituric Acid Reactive Substances - metabolism | Methionine - administration & dosage | Biomarkers - blood | Choline Deficiency - pathology | Collagen Type III - genetics | Toll-Like Receptor 4 - metabolism | Fatty Liver - drug therapy | Animals | Choline - administration & dosage | Diet | NF-kappa B - genetics | Quercetin - pharmacology | Cyclooxygenase 2 - metabolism | Mice | Fibrosis - pathology | Oxidative Stress - drug effects | Smad Proteins - metabolism | Prevention | Hepatitis | Fibrosis | Quercetin | Physiological aspects | Development and progression | Inflammation | Health aspects | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2012, Volume 287, Issue 27, pp. 22988 - 22997
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 01/2014, Volume 50, Issue 1, pp. 158 - 169
Idiopathic pulmonary fibrosis is a chronic progressive disease of increasing prevalence for which there is no effective therapy. Increased oxidative stress... 
Fibroblasts | Pulmonary fibrosis | NADPH oxidase 4 (Nox4) | Transforming growth factor-β | LUNG FIBROSIS | TGF-BETA | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUBUNIT NOX4 | PLASMINOGEN-ACTIVATOR INHIBITOR-1 | RECEPTOR | N-ACETYLCYSTEINE | PROLIFERATION | CELL BIOLOGY | transforming growth factor-beta | fibroblasts | RESPIRATORY SYSTEM | pulmonary fibrosis | IN-VIVO | GROWTH | SMOOTH-MUSCLE-CELLS | Small Molecule Libraries - pharmacology | Transcription, Genetic - drug effects | Idiopathic Pulmonary Fibrosis - genetics | Humans | Transforming Growth Factor beta1 - metabolism | Actins - metabolism | Collagen Type III - metabolism | NADPH Oxidases - metabolism | Male | Idiopathic Pulmonary Fibrosis - metabolism | Actins - genetics | Myofibroblasts - metabolism | Cell Differentiation - genetics | Muscle, Smooth - drug effects | Rodent Diseases - genetics | Collagen Type I - genetics | NADPH Oxidases - genetics | Rodent Diseases - pathology | Lung - metabolism | Extracellular Matrix Proteins - metabolism | Fibroblasts - metabolism | Myofibroblasts - pathology | Lung - pathology | Collagen Type I - metabolism | Extracellular Matrix Proteins - genetics | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Up-Regulation - genetics | Transforming Growth Factor beta1 - genetics | Collagen Type III - genetics | Muscle, Smooth - metabolism | NADPH Oxidase 4 | Fibroblasts - pathology | Myofibroblasts - drug effects | Rats, Sprague-Dawley | Fibronectins - metabolism | Up-Regulation - drug effects | Animals | Cell Differentiation - drug effects | Fibroblasts - drug effects | Lung - drug effects | Rodent Diseases - metabolism | Idiopathic Pulmonary Fibrosis - pathology | Fibronectins - genetics | Transcription, Genetic - genetics | Muscle, Smooth - pathology | Disease | Pulmonary arteries | Pathogenesis | Mortality | Kinases | Gene expression | Proteins | Studies | Pathology | Genotype & phenotype | Rodents | Medical prognosis | Collagen | Hypoxia | Blood pressure | Laboratory animals | Acquisitions & mergers | Apoptosis | Index Medicus
Journal Article