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Stroke, ISSN 0039-2499, 04/2008, Volume 39, Issue 4, pp. 1121 - 1126
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, pp. e84934 - e84934
Objectives: Fibrosis is characterized by excessive tissue remodeling resulting from altered expression of various growth factors, cytokines and proteases. We... 
ENZYME-IMMUNOASSAY | GELATINASE-A | MARKER | LAMININ | MULTIDISCIPLINARY SCIENCES | LIVER FIBROSIS | SEQUENCE | UNIQUE | EXTRACELLULAR-MATRIX | SERUM | PEPTIDE | Collagen Type IV - metabolism | Autoantigens - metabolism | Enzyme-Linked Immunosorbent Assay | Humans | Rats | Matrix Metalloproteinase 12 - metabolism | Carbon Tetrachloride - toxicity | Liver Cirrhosis - chemically induced | Basement Membrane - physiopathology | Animals | Analysis of Variance | Mass Spectrometry | Liver Cirrhosis - metabolism | Pulmonary Fibrosis - metabolism | Pulmonary Disease, Chronic Obstructive - metabolism | Lung diseases, Obstructive | Liver diseases | Cytokines | Proteases | Collagen | Liver | Carbon tetrachloride | Fibrosis | Monoclonal antibodies | Mass spectrometry | Enzyme-linked immunosorbent assay | Antigenic determinants | Matrix metalloproteinases | Pathogenesis | Critical care | Chains | Biology | Matrix metalloproteinase | Assaying | Remodeling | Fragmentation | Degradation | Proteins | Ethics | Extracellular matrix | Tumor necrosis factor-TNF | Chronic obstructive pulmonary disease | Collagen (type IV) | Bioindicators | Metalloproteinase | Growth factors | Enzymes | Lung diseases | Organs | Fragments | Mass spectroscopy | Epitopes | Patients | Pulmonary fibrosis | Obstructive lung disease | Biomarkers | Laboratory animals | Immunoassays | Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 08/2011, Volume 80, Issue 4, pp. 358 - 368
Enhanced transforming growth factor-β1 (TGF-β1) expression in renal cells promotes fibrosis and hypertrophy during the progression of diabetic nephropathy. The... 
TGF-β | fibrosis | diabetic nephropathy | gene expression | cell signaling | Up-Regulation | Diabetes Mellitus, Type 2 - genetics | Homeodomain Proteins - metabolism | Transforming Growth Factor beta1 - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Homeostasis | MicroRNAs - metabolism | Diabetes Mellitus, Type 2 - metabolism | Transfection | Time Factors | Kruppel-Like Transcription Factors - metabolism | Upstream Stimulatory Factors - metabolism | Diabetes Mellitus, Type 1 - chemically induced | Diabetes Mellitus, Experimental - chemically induced | 3' Untranslated Regions | Diabetes Mellitus, Experimental - metabolism | Binding Sites | Collagen Type IV - metabolism | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Collagen Type I - metabolism | Diabetic Nephropathies - metabolism | Cells, Cultured | Diabetes Mellitus, Type 1 - pathology | Diabetes Mellitus, Type 1 - genetics | Diabetic Nephropathies - genetics | Transforming Growth Factor beta1 - genetics | Mesangial Cells - metabolism | Oligonucleotides - metabolism | Animals | Fibrosis | Diabetes Mellitus, Experimental - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Mutation | Zinc Finger E-box-Binding Homeobox 1 | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, pp. e24568 - e24568
Background: In chronic liver disease, hepatic stellate cells (HSC) transdifferentiate into myofibroblasts, promoting extracellular matrix (ECM) synthesis and... 
FAT-STORING CELLS | RAT-LIVER | SIGNAL-TRANSDUCTION | FIBROSIS | FACTOR SUPPRESSES | TGF-BETA | BIOLOGY | MICRORNA TARGET PREDICTIONS | BETA GENE-EXPRESSION | MYOFIBROBLASTS | HUMAN LIVER-DISEASE | Collagen Type IV - metabolism | Proto-Oncogene Proteins c-met - metabolism | Computational Biology - methods | Collagen Type I - metabolism | Extracellular Matrix - metabolism | Rats | Male | MicroRNAs - metabolism | RNA, Messenger - metabolism | Hepatic Stellate Cells - cytology | Hepatocyte Growth Factor - metabolism | Animals | Cell Differentiation | Fibrosis - pathology | 3' Untranslated Regions | Transforming Growth Factor beta - metabolism | Bile Ducts - pathology | Fibroblasts - metabolism | Liver diseases | MicroRNA | Collagen | Analysis | Liver | Bone morphogenetic proteins | Transforming growth factors | Cell culture | Collagen (type I) | Target recognition | Transforming growth factor-a | Immunoblotting | Stimulation | mRNA | Kinases | Western blotting | c-Met protein | Proteins | Signal transduction | Hepatitis | Reduction | Cell growth | Synthesis | Rodents | Hepatology | Gastroenterology | Fibroblasts | Extracellular matrix | Bioinformatics | Growth factors | Stellate cells | Internal medicine | Hepatocyte growth factor | MiRNA | Gene expression | Real time | Pathology | Hepatocytes | MicroRNAs | Fibrosis | Bile | Index Medicus
Journal Article
Hypertension, ISSN 0194-911X, 07/2008, Volume 52, Issue 1, pp. 130 - 136
textabstractThe aim of this study was to explore the effects of the renin inhibitor aliskiren in streptozotocin-diabetic TG(mRen-2)27 rats. Furthermore, we... 
Aliskiren | Diabetic nephropathy | Renin inhibitor | (pro)renin receptor | TG(mRen-2)rat | RENIN/PRORENIN RECEPTOR | NONPROTEOLYTIC ACTIVATION | aliskiren | TRANSGENIC RATS | ANGIOTENSIN-II | NEPHROPATHY | MESANGIAL CELLS | PERIPHERAL VASCULAR DISEASE | renin inhibitor | HEART-RATE | PRORENIN | HYPERTENSION | diabetic nephropathy | Renin - metabolism | Antihypertensive Agents - pharmacology | Gene Expression - drug effects | Diabetic Nephropathies - etiology | Humans | Collagen Type III - metabolism | Male | Antihypertensive Agents - metabolism | Receptors, Cell Surface - antagonists & inhibitors | Albuminuria - physiopathology | Collagen Type I - genetics | Diabetic Nephropathies - physiopathology | Amides - pharmacokinetics | Albuminuria - metabolism | Blood Pressure - drug effects | Diabetes Mellitus, Experimental - complications | Diabetes Mellitus, Experimental - metabolism | Diabetes Mellitus, Experimental - physiopathology | Amides - pharmacology | Collagen Type IV - metabolism | Collagen Type I - metabolism | Albuminuria - etiology | Diabetic Nephropathies - metabolism | Rats | Receptors, Cell Surface - metabolism | Antihypertensive Agents - pharmacokinetics | Collagen Type III - genetics | Rats, Sprague-Dawley | Animals | Renin - antagonists & inhibitors | Transforming Growth Factor beta - genetics | Fumarates - pharmacokinetics | Fumarates - pharmacology | Collagen Type IV - genetics | Transforming Growth Factor beta - metabolism | Receptors, Cell Surface - genetics | Index Medicus
Journal Article