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Journal of Biological Chemistry, ISSN 0021-9258, 01/2010, Volume 285, Issue 4, pp. 2580 - 2590
Collagen triple helices are stabilized by 4-hydroxyproline residues. No function is known for the much less common 3-hydroxyproline (3Hyp), although genetic... 
PROLYL 3-HYDROXYLATION | AMINO-ACID-SEQUENCE | ARTICULAR-CARTILAGE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CYANOGEN-BROMIDE PEPTIDES | RECESSIVE OSTEOGENESIS IMPERFECTA | TRIPLE-HELIX | BASEMENT-MEMBRANE COLLAGEN | CARTILAGE COLLAGEN | HIGHER-ORDER STRUCTURES | COVALENT STRUCTURE | Collagen Type V - genetics | Humans | Collagen Type I - chemistry | Collagen Type III - metabolism | Molecular Sequence Data | Collagen - chemistry | Collagen Type XI - chemistry | Collagen Type XI - metabolism | Collagen Type II - metabolism | Young Adult | Tandem Mass Spectrometry | Collagen Type I - genetics | Collagen Type XI - genetics | Cattle | Bone and Bones - metabolism | Adult | Collagen - genetics | Collagen Type V - metabolism | Hydroxyproline - chemistry | Extracellular Matrix Proteins - metabolism | Amino Acid Sequence | Extracellular Matrix Proteins - chemistry | Collagen Type I - metabolism | Collagen Type V - chemistry | Bone and Bones - chemistry | Extracellular Matrix Proteins - genetics | Hydroxyproline - metabolism | Collagen Type III - chemistry | Collagen Type III - genetics | Cartilage - metabolism | Collagen Type II - genetics | Collagen - metabolism | Hydroxyproline - genetics | Animals | Chickens | Cartilage - chemistry | Collagen Type II - chemistry | Protein Processing, Post-Translational | Index Medicus | Mammal | Protein Structure and Folding | Cartilage | Extracellular Matrix | Post-translational Modification | Collagen | Hydroxyproline | Organisms | 3-hydroxyproline | Bone | Protein
Journal Article
Journal Article
Journal Article
British Journal of Cancer, ISSN 0007-0920, 12/2013, Volume 109, Issue 12, pp. 3049 - 3056
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 2008, Volume 229, Issue 1, pp. 33 - 43
Phytochemicals show promise as potential chemopreventive or chemotherapeutic agents against various cancers. Here we report the chemotherapeutic effects of... 
Berberine | Xanthine oxidase | Reactive oxygen species | Mitochondrial disruption | Apoptosis | RAT-LIVER | PATHWAYS | apoptosis | DEATH | CHEMOPREVENTION | INDUCTION | mitochondrial disruption | berberine | CYTOCHROME-C | PHYTOCHEMICALS | MITOCHONDRIAL PERMEABILITY TRANSITION | INHIBITION | reactive oxygen species | PORE | PHARMACOLOGY & PHARMACY | TOXICOLOGY | xanthine oxidase | Berberine - administration & dosage | Caspase 9 - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | Intracellular Signaling Peptides and Proteins - drug effects | Caspase 3 - metabolism | Male | Intracellular Signaling Peptides and Proteins - metabolism | Caspase 9 - drug effects | Collagen Type XI - metabolism | Dose-Response Relationship, Drug | Mitochondrial Proteins - drug effects | Antineoplastic Agents, Phytogenic - administration & dosage | Mitochondrial Proteins - metabolism | Caspase 3 - drug effects | Prostatic Neoplasms - drug therapy | Cytochromes c - drug effects | Membrane Potentials - drug effects | Collagen Type XI - drug effects | Cytochromes c - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Berberine - pharmacology | Cell Line, Tumor | Oxidative Stress - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | Oxidases | Care and treatment | Molybdenum compounds | Cancer cells | Xanthine | Prostate cancer | Cancer | Index Medicus | OXIDATION | PROSTATE | APOPTOSIS | ANTIOXIDANTS | NEOPLASMS | MITOCHONDRIA | RIBOSE | PWR TYPE REACTORS | 60 APPLIED LIFE SCIENCES | ADP | PHOSPHORS | OXIDASES | POLYMERASES | ANDROGENS
Journal Article
Histology and Histopathology, ISSN 0213-3911, 05/2010, Volume 25, Issue 5, pp. 599 - 608
Journal Article
International Journal of Cancer, ISSN 0020-7136, 12/2017, Volume 141, Issue 11, pp. 2305 - 2317
Journal Article
British Journal of Cancer, ISSN 0007-0920, 05/2009, Volume 100, Issue 9, pp. 1425 - 1433
Curcumin has been shown to inhibit the growth of various types of cancer cells; however, at concentrations much above the clinically achievable levels in... 
Curcumin | Chk1 | G2/M arrest | DNA damage | Pancreatic cancer | CARCINOMA-CELLS | DNA-DAMAGE | DOWN-REGULATION | REGULATED GENE-PRODUCTS | FACTOR-KAPPA-B | BENZYL ISOTHIOCYANATE | I CLINICAL-TRIAL | pancreatic cancer | ONCOLOGY | PROSTATE-CANCER | CHEMOPREVENTIVE AGENT | curcumin | Cell Cycle Proteins - drug effects | Protein Kinases - metabolism | Gene Expression Regulation, Enzymologic - drug effects | Protein Kinases - genetics | Gene Silencing - drug effects | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Collagen Type XI - metabolism | DNA-Binding Proteins - metabolism | Pancreatic Neoplasms - drug therapy | Transfection | Tumor Suppressor Proteins - genetics | Caspase 3 - drug effects | Cell Cycle Proteins - genetics | Gene Expression Regulation, Neoplastic - drug effects | Protein-Serine-Threonine Kinases - metabolism | DNA Damage - drug effects | Protein Kinases - drug effects | Tumor Suppressor Proteins - metabolism | Collagen Type XI - drug effects | Pancreatic Neoplasms - pathology | Cell Cycle Proteins - metabolism | Curcumin - pharmacology | DNA-Binding Proteins - drug effects | Pancreatic Neoplasms - enzymology | Protein-Serine-Threonine Kinases - genetics | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | Cell Division - drug effects | Protein-Serine-Threonine Kinases - drug effects | Cell Line, Tumor | Checkpoint Kinase 1 | Tumor Suppressor Proteins - drug effects | Cell Cycle - drug effects | Index Medicus | Translational Therapeutics | M arrest
Journal Article
Journal Article
Breast cancer research : BCR, ISSN 1465-5411, 2007, Volume 9, Issue 5, pp. R63 - R63
Background De-regulation of the wingless and integration site growth factor (WNT) signaling pathway via mutations in APC and Axin, proteins that target... 
ONCOLOGY | WNT/BETA-CATENIN | CYCLIN D1 EXPRESSION | PROTEIN-COUPLED RECEPTORS | TYROSINE KINASE INHIBITOR | MONOCLONAL-ANTIBODY | GROWTH-FACTOR | BETA-CATENIN | MAMMARY EPITHELIAL-CELLS | HUMAN COLORECTAL-CANCER | FACTOR-RELATED PEPTIDES | Receptors, Estrogen - metabolism | Cell Proliferation | Immunoprecipitation | Humans | Transcriptional Activation | Estrogen Antagonists - pharmacology | Phosphoproteins - antagonists & inhibitors | Wnt1 Protein - metabolism | Collagen Type XI - metabolism | Phosphoproteins - metabolism | Breast Neoplasms - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Receptor, Epidermal Growth Factor - metabolism | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | src-Family Kinases - metabolism | Membrane Proteins - metabolism | Dishevelled Proteins | Autocrine Communication | Signal Transduction | RNA, Small Interfering - pharmacology | Cell Cycle Proteins - metabolism | Phosphoproteins - genetics | Proto-Oncogene Proteins c-myc - metabolism | beta Catenin - metabolism | Blotting, Western | Culture Media, Conditioned | Mitogen-Activated Protein Kinase 3 - metabolism | Breast Neoplasms - pathology | Adaptor Proteins, Signal Transducing - genetics | Tamoxifen - pharmacology | Tamoxifen - analogs & derivatives | Enzyme Activation | Adaptor Proteins, Signal Transducing - metabolism | Apoptosis | Mitogen-Activated Protein Kinase 1 - metabolism | Physiological aspects | Development and progression | Breast cancer | Growth factor receptors | Metastasis | Growth factors | Health aspects | Index Medicus
Journal Article