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1. Full Text Epigallocatechin 3‐gallate attenuates arthritis by regulating Nrf2, HO‐1, and cytokine levels in an experimental arthritis model
by Karatas, Ahmet and Dagli, Adile Ferda and Orhan, Cemal and Gencoglu, Hasan and Ozgen, Metin and Sahin, Nurhan and Sahin, Kazim and Koca, Suleyman Serdar
Biotechnology and applied biochemistry, ISSN 0885-4513, 05/2020, Volume 67, Issue 3, pp. 317 - 322
epigallocatechin 3‐gallate | rheumatoid arthritis | collagen‐induced arthritis | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Biotechnology & Applied Microbiology | Science & Technology | Glutathione peroxidase | Animal models | Cytokines | Destruction | Epigallocatechin-3-gallate | Superoxide dismutase | Inflammation | Arthritis | Malondialdehyde | Antioxidants | Oxygenase | Cartilage | Catalase | Tumor necrosis factor | Rheumatoid arthritis | Collagen | Heme | Peroxidase | Glutathione | Index Medicus
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2. Full Text Taraxasterol suppresses inflammation in IL-1β-induced rheumatoid arthritis fibroblast-like synoviocytes and rheumatoid arthritis progression in mice
by Chen, Jianfeng and Wu, Weibo and Zhang, Miaomiao and Chen, Caiming
International immunopharmacology, ISSN 1567-5769, 05/2019, Volume 70, pp. 274 - 283
NF-κB | NLRP3 inflammasomes | Taraxasterol | Rheumatoid arthritis | Fibroblast-like synoviocytes rheumatoid arthritis | Collagen-induced arthritis | Pharmacology & Pharmacy | Immunology | Life Sciences & Biomedicine | Science & Technology | Triterpenes | Animal models | Matrix metalloproteinases | Transforming growth factor | Interleukin | Arthritis | Matrix metalloproteinase | Kinases | IKK protein | Caspase-1 | Interleukin 6 | Proteins | Animal tissues | Fibroblasts | Tumor necrosis factor-TNF | Growth factors | Interleukin 8 | Interstitial collagenase | NF-κB protein | Stromelysin 1 | Progressions | Cytokines | TAK1 protein | Caspase | Pharmacology | Inflammation | Tumor necrosis factor-α | IL-1β | Chemical compounds | Synoviocytes | Herbal medicine | Collagen | Mice | In vivo methods and tests | Apoptosis
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3. Full Text Mouse Models of Rheumatoid Arthritis
by Caplazi, P and Baca, M and Barck, K and Carano, R. A. D and DeVoss, J and Lee, W. P and Bolon, B and Diehl, L
Veterinary pathology, ISSN 0300-9858, 9/2015, Volume 52, Issue 5, pp. 819 - 826
models | TNFΔ ARE mouse | collagen antibody–induced arthritis | arthritis | collagen-induced arthritis | animal | experimental | k/bxn antibody transfer arthritis | mice | rheumatoid | Pathology | Veterinary Sciences | Life Sciences & Biomedicine | Science & Technology | Arthritis, Rheumatoid - veterinary | Inflammation - veterinary | Animals | Arthritis - veterinary | Arthritis, Rheumatoid - etiology | Collagen - pharmacology | Mice | Arthritis, Rheumatoid - pathology | Disease Models, Animal | Index Medicus
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4. Full Text IL-1β and TNFα-initiated IL-6-STAT3 pathway is critical in mediating inflammatory cytokines and RANKL expression in inflammatory arthritis
by Mori, Tomoaki and Miyamoto, Takeshi and Yoshida, Hideyuki and Asakawa, Mayoko and Kawasumi, Miyuri and Kobayashi, Takashi and Morioka, Hideo and Chiba, Kazuhiro and Toyama, Yoshiaki and Yoshimura, Akihiko
International immunology, ISSN 0953-8178, 11/2011, Volume 23, Issue 11, pp. 701 - 712
Joint destruction | Chronic inflammation | Collagen-induced arthritis | Rheumatoid arthritis | IL-6 | Life Sciences & Biomedicine | Immunology | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Arthritis, Experimental - drug therapy | RANK Ligand - metabolism | Humans | Arthritis, Rheumatoid - complications | Interleukin-1beta - adverse effects | Arthritis, Experimental - pathology | RANK Ligand - immunology | Inflammation - complications | Signal Transduction - immunology | Osteoclasts - immunology | Arthritis, Rheumatoid - drug therapy | Inflammation - drug therapy | Interleukin-1beta - metabolism | Piperidines | Gene Expression - immunology | STAT3 Transcription Factor - deficiency | Tumor Necrosis Factor-alpha - immunology | Mice, Inbred DBA | Pyrroles - therapeutic use | Interleukin-6 - metabolism | Joints - metabolism | STAT3 Transcription Factor - genetics | Joints - drug effects | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Interleukin-1beta - immunology | Joints - pathology | Arthritis, Experimental - immunology | Inflammation - immunology | Pyrimidines - pharmacology | Arthritis, Rheumatoid - pathology | Pyrroles - pharmacology | Animals | Tumor Necrosis Factor-alpha - adverse effects | Interleukin-6 - pharmacology | RANK Ligand - antagonists & inhibitors | Pyrimidines - therapeutic use | Fibroblasts - drug effects | Interleukin-6 - immunology | Fibroblasts - immunology | Mice | STAT3 Transcription Factor - immunology | Arthritis, Rheumatoid - immunology | STAT3 Transcription Factor - antagonists & inhibitors | Chronic Disease | Osteoclasts - drug effects | Index Medicus | Animal models | Transcription | Leukemia inhibitory factor | Stat3 protein | Data processing | Inflammation | Joint diseases | Osteoblasts | Inflammatory diseases | Interleukin 6 | Osteoclastogenesis | Collagen | Interleukin 1 | TRANCE protein | Tumor necrosis factor- alpha
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5. Full Text Arthritis susceptibility and the gut microbiome
by Taneja, Veena
FEBS letters, ISSN 0014-5793, 11/2014, Volume 588, Issue 22, pp. 4244 - 4249
Humanized mice | Gut microbiome | Rheumatoid arthritis | Collagen-induced arthritis | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Microbiota | Animals | Disease Susceptibility | Arthritis, Rheumatoid - etiology | Humans | Gastrointestinal Tract - microbiology | Arthritis, Rheumatoid - immunology | Arthritis, Rheumatoid - microbiology | Hygiene Hypothesis | Rheumatoid factor | Development and progression | Arthritis | Microbiota (Symbiotic organisms) | Analysis | Index Medicus
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6. Full Text Arthritis models: usefulness and interpretation
by Bessis, Natacha and Decker, Patrice and Assier, Eric and Semerano, Luca and Boissier, Marie-Christophe
Seminars in immunopathology, ISSN 1863-2297, 06/2017, Volume 39, Issue 4, pp. 469 - 486
Inflammation | Arthritis models | Collagen-induced arthritis | Rheumatoid arthritis | Pathology | Immunology | Life Sciences & Biomedicine | Science & Technology | Adaptive Immunity | Arthritis, Rheumatoid - metabolism | Animals | Arthritis, Rheumatoid - etiology | Humans | Environment | Immunity, Innate | Arthritis, Experimental | Arthritis, Rheumatoid - pathology | Disease Models, Animal | Animal experimentation | Rheumatoid factor | Usage | Microbiota (Symbiotic organisms) | Collagen | Analysis | Models | Arthritis | Genetic engineering | B cells | Translation | Animal models | Transgenic mice | Data processing | Innate immunity | Interleukin 6 | Ethics | Pain | Lymphocytes B | Tumor necrosis factor | Rodents | Replication | Design of experiments | Index Medicus
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7. Full Text Anti-arthritis effect of a novel quinazoline derivative through inhibiting production of TNF-α mediated by TNF-α converting enzyme in murine collagen-induced arthritis model
by Pu, Yuzhi and Cao, Dong and Xie, Caifeng and Pei, Heying and Li, Dan and Tang, Minghai and Chen, Lijuan
Biochemical and biophysical research communications, ISSN 0006-291X, 07/2015, Volume 462, Issue 4, pp. 288 - 293
TNF-α converting enzyme | Quinazoline | TNF-α | Rheumatoid arthritis | Collagen-induced arthritis | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | ADAM17 Protein | Cell Line | Arthritis, Experimental - drug therapy | Enzyme-Linked Immunosorbent Assay | RNA, Messenger - genetics | Tumor Necrosis Factor-alpha - genetics | Male | Collagen - administration & dosage | ADAM Proteins - metabolism | Animals | Quinazolines - therapeutic use | Mice, Inbred DBA | Mice | Quinazolines - pharmacology | Tumor Necrosis Factor-alpha - biosynthesis | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Disease Models, Animal | Index Medicus
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