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index medicus (64) 64
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phosphorylation (59) 59
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collapsin response mediator protein 2 (53) 53
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Journal of Neurochemistry, ISSN 0022-3042, 11/2007, Volume 103, Issue 3, pp. 1132 - 1144
\ Collapsin response mediator protein 2 (CRMP2) is an abundant brain-enriched protein that can regulate microtubule assembly in neurons. This function of CRMP2... 
Glycogen synthase kinase 3 | Phosphorylation | Alzheimer's disease | Cyclin-dependent kinase 5 | Collapsin response mediator protein 2 | TAU-PROTEIN | NEUROFIBRILLARY TANGLES | CALPAIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLYCOGEN-SYNTHASE KINASE-3 | TRIPLE-TRANSGENIC MODEL | CRMP-2 | NEUROSCIENCES | collapsin response mediator protein 2 | P25 | A-BETA | MICE | cyclin-dependent kinase 5 | phosphorylation | glycogen synthase kinase 3 | Humans | Brain - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Microtubules - metabolism | Neurofibrillary Tangles - metabolism | Time Factors | Amyloid beta-Peptides - metabolism | Amyloid beta-Protein Precursor - metabolism | Neurons - metabolism | Binding Sites | Disease Models, Animal | Alzheimer Disease - physiopathology | Brain - physiopathology | Gene Expression Regulation - physiology | Mice, Transgenic | Glycogen Synthase Kinase 3 - metabolism | Disease Progression | Cyclin-Dependent Kinase 5 - metabolism | Nerve Tissue Proteins - metabolism | Amyloid beta-Protein Precursor - genetics | Animals | Alzheimer Disease - metabolism | Plaque, Amyloid - metabolism | Aged | Mice | Alzheimer Disease - genetics | Post-translational modification | Neurosciences | Synthesis | Glycogen | Phosphoproteins | Development and progression | Research institutes | Proteins | Neurology | Biochemistry | Alzheimers disease | Transgenic animals | Rodents
Journal Article
Journal Article
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 11/2018, Volume 35, Issue 21, pp. 253 - 2539
Journal Article
Journal of Inflammation (United Kingdom), ISSN 1476-9255, 2015, Volume 12, Issue 1, pp. 11 - 11
Background: Delayed M1 toward M2 macrophage phenotype transition is considered one of the major causes for the impaired healing after myocardial infarction... 
ApoE- | Myocardial infarction | Phenotypes | Fibrosis | Inflammation | Macrophages | Collapsin response mediator protein-2 | RECRUITMENT | INFLAMMATORY RESPONSES | PHOSPHORYLATION | IMMUNOLOGY | DELIVERY | NANOPARTICLES | REPAIR | MONOCYTE SUBSETS | MICE | POLARIZATION | EXPRESSION | ApoE(-/-) | Immunohistochemistry | Genetic aspects | Heart attack
Journal Article
Journal Article