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Cancer Research, ISSN 0008-5472, 10/2009, Volume 69, Issue 20, pp. 8208 - 8215
Journal Article
Journal Article
Journal of Steroid Biochemistry and Molecular Biology, ISSN 0960-0760, 01/2016, Volume 155, Issue Pt B, pp. 207 - 216
•1βHSD isozymes can be modulated by several EDs and therapeutics.•They may possess adverse or beneficial effects.•Inhibition of 11βHSD in testes and placenta... 
Testis | Brain | Adipose tissue | Placenta | 11β-hydroxysteroid dehydrogenase | Inhibitor | Endocrine disruptor | Colon | TYPE-1 INHIBITOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | BETA-HYDROXYSTEROID DEHYDROGENASE | GLUCOCORTICOID-RECEPTOR ACTIVATION | INDUCED OBESE MICE | HEXOSE-6-PHOSPHATE DEHYDROGENASE | METABOLIC-SYNDROME | REDUCTASE-ACTIVITY | 11 beta-hydroxysteroid dehydrogenase | ENDOCRINOLOGY & METABOLISM | RAT LEYDIG-CELLS | BISPHENOL-A | ADIPOSE-TISSUE | Diabetes Mellitus - enzymology | Diabetes Mellitus - pathology | Colon - enzymology | Humans | Colon - drug effects | Brain - enzymology | Male | Metabolic Syndrome - chemically induced | Testis - drug effects | Endocrine Disruptors - pharmacology | Neoplasms - chemically induced | Glucocorticoids - metabolism | Testis - enzymology | Female | 11-beta-Hydroxysteroid Dehydrogenase Type 2 - antagonists & inhibitors | Metabolic Syndrome - enzymology | Obesity - chemically induced | Enzyme Inhibitors - pharmacology | Neoplasms - enzymology | Placenta - enzymology | Organ Specificity | 11-beta-Hydroxysteroid Dehydrogenase Type 1 - antagonists & inhibitors | Obesity - pathology | Brain - drug effects | Placenta - drug effects | Pregnancy | 11-beta-Hydroxysteroid Dehydrogenase Type 1 - metabolism | 11-beta-Hydroxysteroid Dehydrogenase Type 2 - metabolism | Diabetes Mellitus - chemically induced | Adipose Tissue - enzymology | Obesity - enzymology | Metabolic Syndrome - pathology | Neoplasms - pathology | Adipose Tissue - drug effects
Journal Article
Science Translational Medicine, ISSN 1946-6234, 05/2010, Volume 2, Issue 32, pp. 32ra36 - ra36
Journal Article
Pharmaceutical Research, ISSN 0724-8741, 1/2015, Volume 32, Issue 1, pp. 74 - 90
Journal Article
Journal Article
NATURE, ISSN 0028-0836, 09/2011, Volume 477, Issue 7364, pp. 330 - U102
Intestinal immune homeostasis depends on a tightly regulated cross talk between commensal bacteria, mucosal immune cells and intestinal epithelial cells... 
APOPTOSIS | REGULATOR | DEATH PATHWAY | NECROPTOSIS | B-CELL | MULTIDISCIPLINARY SCIENCES | NETWORKS | TNF-ALPHA | DEFICIENT MICE | BOWEL-DISEASE | NEMO/IKK-GAMMA | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Colon - enzymology | Epithelial Cells - metabolism | Paneth Cells - pathology | Colitis - pathology | Intracellular Signaling Peptides and Proteins - metabolism | Inflammatory Bowel Diseases - metabolism | Metagenome - physiology | Necrosis | Intracellular Signaling Peptides and Proteins - deficiency | Cysteine Endopeptidases - metabolism | Inflammatory Bowel Diseases - enzymology | Inflammatory Bowel Diseases - pathology | Enteritis - metabolism | Signal Transduction | Colon - pathology | Fas-Associated Death Domain Protein - metabolism | Fas-Associated Death Domain Protein - deficiency | Epithelial Cells - pathology | Enteritis - pathology | Tumor Necrosis Factors - deficiency | Colon - metabolism | Animals | Colitis - enzymology | Myeloid Differentiation Factor 88 - deficiency | Enteritis - enzymology | Colitis - metabolism | Epithelial Cells - enzymology | Mice | Receptor-Interacting Protein Serine-Threonine Kinases - antagonists & inhibitors | Chronic Disease | Myeloid Differentiation Factor 88 - metabolism | Apoptosis | Proteins | Inflammatory bowel disease | Pathogenesis | Colleges & universities | Homeostasis | Colon
Journal Article
Gastroenterology, ISSN 0016-5085, 2010, Volume 139, Issue 3, pp. 869 - 881.e9
Background & Aims Mechanisms responsible for crypt architectural distortion in chronic ulcerative colitis (CUC) are not well understood. Data indicate that... 
Gastroenterology and Hepatology | Intestinal Progenitor Cells | PI3K | Pik3r1 | Intestinal Stem Cells | ULCERATIVE-COLITIS | SELF-RENEWAL | COLORECTAL-CARCINOMA | IDENTIFICATION | INTERLEUKIN-10-DEFICIENT MICE | CANCER | INFLAMMATORY-BOWEL-DISEASE | PATHWAY | GENE-EXPRESSION | WNT | GASTROENTEROLOGY & HEPATOLOGY | Stem Cells - immunology | Colon - enzymology | Phosphorylation | Cell Proliferation | T-Lymphocytes - enzymology | Colitis - genetics | Humans | Colitis - complications | Colon - drug effects | Colitis - pathology | Colon - immunology | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Wnt Proteins - metabolism | Intestinal Mucosa - drug effects | Phosphatidylinositol 3-Kinases - deficiency | Stem Cells - enzymology | Time Factors | Intestinal Mucosa - immunology | Colonoscopy | Intestinal Mucosa - enzymology | Colitis - immunology | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Interleukin-10 - deficiency | Colon - pathology | Mice, Inbred C57BL | beta Catenin - metabolism | Mice, Knockout | Phosphatidylinositol 3-Kinases - genetics | Animals | Colitis - enzymology | Signal Transduction - drug effects | Biopsy | Colonic Neoplasms - pathology | Interleukin-10 - genetics | Stem Cells - drug effects | Stem Cells - pathology | T-Lymphocytes - immunology | Colonic Neoplasms - enzymology | Mice | Protein Kinase Inhibitors - pharmacology | Intestinal Mucosa - pathology
Journal Article