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PloS one, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, p. e0132977
Cancer stem cells are cancer cells characterized by stem cell properties and represent a small population of tumor cells that drives tumor development,... 
TESTIS ANTIGEN BORIS | HEPATOCELLULAR-CARCINOMA | SIDE POPULATION | IN-VITRO | INDUCED APOPTOSIS | TELOMERASE | MULTIDISCIPLINARY SCIENCES | IMPRINTED SITES | CTCF | IDENTIFICATION | TUMOR-INITIATING CELLS | ATP Binding Cassette Transporter, Sub-Family G, Member 2 | RNA, Small Interfering - genetics | Cell Proliferation | Epithelial Cells - metabolism | Polycomb Repressive Complex 1 - metabolism | Homeodomain Proteins - metabolism | Humans | Retinal Dehydrogenase - metabolism | Gene Expression Regulation, Neoplastic | Spheroids, Cellular - pathology | Epithelial-Mesenchymal Transition - genetics | Neoplasm Proteins - metabolism | SOXB1 Transcription Factors - metabolism | DNA-Binding Proteins - metabolism | Octamer Transcription Factor-3 - genetics | Polycomb Repressive Complex 1 - genetics | Telomerase - genetics | ATP-Binding Cassette Transporters - genetics | Neoplastic Stem Cells - metabolism | SOXB1 Transcription Factors - genetics | Isoenzymes - metabolism | Hyaluronan Receptors - metabolism | ATP-Binding Cassette Transporters - metabolism | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Telomerase - metabolism | Neoplasm Proteins - genetics | DNA-Binding Proteins - antagonists & inhibitors | Nanog Homeobox Protein | Signal Transduction | Isoenzymes - genetics | Spheroids, Cellular - metabolism | Epithelial Cells - pathology | Retinal Dehydrogenase - genetics | DNA-Binding Proteins - genetics | Organ Specificity | Hyaluronan Receptors - genetics | Homeodomain Proteins - genetics | Phenotype | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Biomarkers, Tumor - genetics | Cell Movement | RNA, Small Interfering - metabolism | RNA | Genes | Colorectal cancer | Stem cells | Genetic aspects | Gene expression | Cancer | Protein binding | Neurosciences | Germ cells | Telomerase reverse transcriptase | Populations | Dyes | Mesenchyme | Oct-4 protein | Gene regulation | Oncology | Spheres | Drug resistance | Tissues | Metastases | DNA-binding protein | CD44 antigen | Cell cycle | Life sciences | Colon | Telomerase | Growth factors | Deoxyribonucleic acid--DNA | Medical research | Antigens | Cell survival | Tumor cells | Invasiveness | Cervix | Survival | Brain research | Molecular modelling | Medical prognosis | Epigenetics | Breast | Tumors | Deoxyribonucleic acid | DNA
Journal Article
Applied and environmental microbiology, ISSN 1098-5336, 2012, Volume 78, Issue 7, pp. 2179 - 2189
Trimeric autotransporter proteins (TAAs) are important virulence factors of many Gram-negative bacterial pathogens. A common feature of most TAAs is the... 
TRANSLOCATOR DOMAIN | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | NEISSERIA-MENINGITIDIS | URINARY-TRACT | CHROMOSOMAL GENES | MICROBIOLOGY | H-NS | INFLUENZAE HIA AUTOTRANSPORTER | YERSINIA | EXPRESSION | ADHESIN YADA | BIOFILM FORMATION | Colon - cytology | Epithelial Cells - metabolism | Virulence Factors - genetics | Fimbriae Proteins - metabolism | Humans | Biofilms - growth & development | Extracellular Matrix - metabolism | Molecular Sequence Data | Adhesins, Escherichia coli - genetics | Escherichia coli O157 - pathogenicity | Uropathogenic Escherichia coli - metabolism | Uropathogenic Escherichia coli - pathogenicity | Urinary Bladder - cytology | Urinary Bladder - microbiology | Caco-2 Cells | Rectum - microbiology | Amino Acid Sequence | Adhesins, Escherichia coli - metabolism | Escherichia coli O157 - genetics | Escherichia coli O157 - metabolism | Escherichia coli Infections - microbiology | Escherichia coli Proteins - metabolism | Uropathogenic Escherichia coli - genetics | Sequence Analysis, DNA | Bacterial Adhesion | Fimbriae Proteins - genetics | Rectum - cytology | Epithelial Cells - microbiology | Cell Line, Tumor | Escherichia coli Proteins - genetics | Virulence Factors - metabolism | Colon - microbiology | Gene Expression Regulation, Bacterial | Carrier proteins | Bacterial proteins | Escherichia coli | Physiological aspects | Microbiological synthesis | Chemical properties | Research | Protein binding | Extracellular Matrix/metabolism | Bacterial | Adhesins | Uropathogenic Escherichia coli/genetics/metabolism/pathogenicity | Fimbriae Proteins/genetics/metabolism | Escherichia coli O157/genetics/metabolism/pathogenicity | Escherichia coli/genetics/metabolism | Biofilms/growth & development | Epithelial Cells/metabolism/microbiology | Life Sciences | Tumor | Cell Line | Gene Expression Regulation | Escherichia coli Proteins/genetics/metabolism | Colon/cytology/microbiology | Bacteriology | Rectum/cytology/microbiology | Microbiology and Parasitology | DNA | Escherichia coli Infections/microbiology | Sequence Analysis | Urinary Bladder/cytology/microbiology | Virulence Factors/genetics/metabolism | Genetics and Molecular Biology
Journal Article
Scientific reports, ISSN 2045-2322, 2017, Volume 7, Issue 1, p. 43412
This study was conducted to investigate impacts of dietary protein levels on gut bacterial community and gut barrier. The intestinal microbiota of finishing... 
POSTWEANING DIARRHEA | HOST METABOLISM | CELLS | FINISHING PIGS | HOMEOSTASIS | PIGLETS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MODULATING INTESTINAL PERMEABILITY | BUTYRATE | PROMOTES | Dietary Proteins - administration & dosage | Peptostreptococcus - drug effects | Tight Junction Proteins - genetics | Receptors, G-Protein-Coupled - metabolism | Colon - drug effects | Ileum - metabolism | Escherichia - metabolism | Escherichia - classification | Fatty Acids, Volatile - metabolism | Intestinal Mucosa - drug effects | Occludin - metabolism | Genetic Variation | Polycomb Repressive Complex 1 - genetics | Dietary Proteins - metabolism | Ileum - drug effects | Swine | Shigella - metabolism | Tight Junction Proteins - metabolism | Firmicutes - isolation & purification | Intestinal Mucosa - microbiology | Peptostreptococcus - isolation & purification | Colon - metabolism | Gastrointestinal Microbiome - drug effects | Claudin-1 - genetics | Escherichia - drug effects | Receptors, G-Protein-Coupled - genetics | Escherichia - isolation & purification | Firmicutes - drug effects | Animal Feed | Peptostreptococcus - classification | Intestinal Mucosa - metabolism | Polycomb Repressive Complex 1 - metabolism | Shigella - classification | Gastrointestinal Microbiome - physiology | Clostridium - isolation & purification | Shigella - drug effects | Clostridium - classification | Occludin - genetics | Claudin-1 - metabolism | Biogenic Amines - metabolism | Diet, Protein-Restricted - methods | Firmicutes - metabolism | Digestion - drug effects | Firmicutes - classification | Shigella - isolation & purification | Peptostreptococcus - metabolism | Digestion - physiology | Gene Expression Regulation - drug effects | Animals | Clostridium - metabolism | Colon - microbiology | Ileum - microbiology | Clostridium - drug effects | Amines | Digestive system | Mucosa | Ileum | Studies | Proteins | Protein composition | Metabolites | Intestine | Fish | Intestinal microflora | Colon | Digestive tract | Biogenic amines | Colonization
Journal Article
Nature communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, p. 7629
...) and GLP-1 secretion by inhibiting glycolysis. In vivo, FXR deficiency increases GLP-1 gene expression and secretion in response to glucose hence improving glucose metabolism... 
GLP-1 SECRETION | FXR | BILE-ACID RECEPTORS | PROTEIN | GLUCOSE | MULTIDISCIPLINARY SCIENCES | INTESTINE | METABOLIC-RATE | EXPRESSION | Colon - cytology | Intestinal Mucosa - metabolism | Sequestering Agents - pharmacology | Humans | Ileum - metabolism | RNA, Messenger - metabolism | Colesevelam Hydrochloride - pharmacology | Obesity - genetics | Glucagon-Like Peptide 1 - genetics | Jejunum - metabolism | Insulin-Secreting Cells - metabolism | Proglucagon - drug effects | Diet, High-Fat | Jejunum - cytology | Enteroendocrine Cells - metabolism | Ileum - cytology | Proglucagon - metabolism | Insulin Secretion | Glucagon-Like Peptide 1 - metabolism | Signal Transduction | Bile Acids and Salts - metabolism | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Colon - metabolism | Mice, Knockout | Obesity - metabolism | Transcription Factors - metabolism | Insulin - metabolism | Animals | Glycolysis | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice, Obese | Mice | Proglucagon - genetics | Receptors, G-Protein-Coupled - genetics | Blood Glucose - metabolism | Anticholesteremic Agents - pharmacology | Intestines - cytology | Carbohydrates | Glucose | Gene expression | Insulin | Cell and Molecular Biology | RAT SMALL-INTESTINE | GLUCOSE-HOMEOSTASIS | OBESITY | Endokrinologi och diabetes | MICE | Multidisciplinary Sciences | Cell- och molekylärbiologi | TYPE-2 DIABETES-MELLITUS | Endocrinology and Diabetes
Journal Article
PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 11, p. e14062
Background: The cancer stem cell theory hypothesizes that cancers are perpetuated by cancer stem cells (CSC) or tumor initiating cells (TIC) possessing... 
COLON-CANCER | INITIATING CELLS | POPULATION | MARKER | MULTIDISCIPLINARY SCIENCES | TUMOR | HYALURONAN | CHEMORESISTANCE | FLOW-CYTOMETRY | IDENTIFICATION | LINES | Immunohistochemistry | Humans | Lung Neoplasms - metabolism | Middle Aged | Glycoproteins - metabolism | Peptides - genetics | Immunoblotting | Lung Neoplasms - pathology | Male | Transplantation, Heterologous | Gene Expression Profiling | Antigens, CD - genetics | Antigens, CD - metabolism | Neoplasms, Experimental - pathology | Octamer Transcription Factor-3 - genetics | Flow Cytometry | Peptides - metabolism | Neoplastic Stem Cells - metabolism | Hyaluronan Receptors - metabolism | Neoplasms, Experimental - genetics | Neoplastic Stem Cells - pathology | Female | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Glycoproteins - genetics | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | AC133 Antigen | Hyaluronan Receptors - genetics | Animals | Polycomb Repressive Complex 1 | Mice, Nude | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Aged | Mice | Neoplasms, Experimental - metabolism | Squamous cell carcinoma | Lung cancer, Non-small cell | Analysis | Stem cells | Flow cytometry | Biotechnology | Laboratories | Heart surgery | Oct-4 protein | Lung cancer | Colorectal cancer | Stem cell transplantation | Proteins | Allografts | Epidermal growth factor | CD44 antigen | Xenografts | Cell cycle | CD34 antigen | Subpopulations | Cell survival | Tumor cells | Non-small cell lung carcinoma | Tumor cell lines | Cisplatin | Studies | Polymerase chain reaction | Pathology | Cytometry | Properties (attributes) | Medical prognosis | Biomarkers | In vivo methods and tests | Pluripotency | Tumors | Apoptosis | Cancer
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2011, Volume 141, Issue 2, pp. 621 - 632
Background & Aims Epithelial cells that cover the intestinal mucosal surface maintain immune homeostasis and tolerance in the gastrointestinal tract. However,... 
Gastroenterology and Hepatology | IgA | Inflammatory Bowel Disease | T Cell | Immune Response | CELLS | COMPLEX | ACTIVATION | POLYMERIC IMMUNOGLOBULIN RECEPTOR | INTESTINAL EPITHELIUM | TRANSPORT | INFLAMMATORY-BOWEL-DISEASE | ADAPTER | HOST-DEFENSE | INFECTION | GASTROENTEROLOGY & HEPATOLOGY | Tumor Necrosis Factor-alpha - metabolism | Intestinal Mucosa - metabolism | Adaptor Protein Complex mu Subunits - metabolism | Epithelial Cells - metabolism | Adaptor Protein Complex beta Subunits - deficiency | Humans | Cell Membrane Permeability | Crohn Disease - metabolism | Cell Membrane - physiology | Ribonucleases - metabolism | Adaptor Protein Complex 1 - metabolism | Th17 Cells - metabolism | Intestinal Mucosa - immunology | alpha-Defensins - metabolism | Adaptor Protein Complex beta Subunits - immunology | Cell Membrane - metabolism | beta-Defensins - metabolism | Colon | Receptors, Cytokine - metabolism | Colitis - immunology | Adaptor Protein Complex 1 - deficiency | Lipocalins - metabolism | Adaptor Protein Complex 1 - immunology | Immunoglobulin A - metabolism | Signal Transduction | Acute-Phase Proteins - metabolism | Down-Regulation | Oncogene Proteins - metabolism | Homeostasis - immunology | Epithelial Cells - pathology | S100 Proteins - metabolism | Muramidase - metabolism | Intestinal Mucosa - microbiology | Mice, Knockout | Interleukin-17 - metabolism | Adaptor Protein Complex beta Subunits - metabolism | Animals | Colitis - microbiology | Proteins - metabolism | Ribonuclease, Pancreatic - metabolism | Lipocalin-2 | Cathelicidins - metabolism | Epithelial Cells - immunology | Receptors, Cytokine - immunology | Mice | Intestinal Mucosa - pathology | Medical colleges | Fc receptors | Gastrointestinal diseases | Homeostasis | Biological response modifiers | Immunoglobulin A | T cells
Journal Article
Molecular cell, ISSN 1097-2765, 2010, Volume 39, Issue 4, pp. 493 - 506
A transient inflammatory signal can initiate an epigenetic switch from nontransformed to cancer cells via a positive feedback loop involving NF-κB, Lin28,... 
RNA | HUMDISEASE | SIGNALING | Signaling | Humdisease | BREAST-CANCER | MICRORNA MIR-21 | TARGET | TRANSFORMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | EMBRYONIC STEM-CELLS | LET-7 | IDENTIFICATION | NF-KAPPA-B | EXPRESSION | BINDING | CELL BIOLOGY | Colonic Neoplasms - genetics | Cell Proliferation | Epigenesis, Genetic | Humans | Transcriptional Activation | MicroRNAs - metabolism | NF-kappa B - metabolism | Colonic Neoplasms - metabolism | Inflammation - metabolism | Adenocarcinoma - metabolism | RNA Interference | Cell Transformation, Neoplastic - genetics | Proto-Oncogene Proteins c-akt - metabolism | Binding Sites | Colonic Neoplasms - therapy | Tumor Suppressor Proteins - metabolism | Signal Transduction | HCT116 Cells | Computational Biology | Mammary Glands, Human - pathology | Tumor Burden | Breast Neoplasms - genetics | Adenocarcinoma - therapy | Mice, Nude | Mice | Proto-Oncogene Proteins c-myc - genetics | Kinetics | Cell Movement | Deubiquitinating Enzyme CYLD | Gene Expression Regulation, Neoplastic | Mammary Glands, Human - metabolism | Breast Neoplasms - metabolism | Transfection | Tumor Suppressor Proteins - genetics | Inflammation Mediators - metabolism | Female | Adenocarcinoma - genetics | STAT3 Transcription Factor - metabolism | Promoter Regions, Genetic | Receptors, Estrogen - genetics | Neoplasm Invasiveness | PTEN Phosphohydrolase - metabolism | Cell Transformation, Neoplastic - metabolism | Proto-Oncogene Proteins c-myc - metabolism | HT29 Cells | Xenograft Model Antitumor Assays | Algorithms | Animals | Breast Neoplasms - pathology | Inflammation - genetics | Cell Transformation, Neoplastic - pathology | Genes, src | Medical colleges | Inflammation | Colon cancer | Cancer | Adenocarcinoma | microRNAs | cancer | transformation | transcription factors | inflammation | STAT3
Journal Article
Cell metabolism, ISSN 1550-4131, 2016, Volume 23, Issue 6, pp. 1140 - 1153
Journal Article
Nature cell biology, ISSN 1476-4679, 2017, Volume 19, Issue 9, pp. 1027 - 1036
...ART ICLES Control of intestinal stem cell function and proliferation by mitochondrial pyruvate metabolism John C. Schell1, Dona R. Wisidagama2, Claire Bensard1... 
COLON-CANCER | PROGENITORS | INHIBITION | TCA CYCLE | GROWTH | VALPROIC ACID | DIFFERENTIATION | IDENTIFICATION | ADULT DROSOPHILA MIDGUT | CARRIER | CELL BIOLOGY | Intestines - drug effects | Intestinal Mucosa - metabolism | Mitochondrial Membrane Transport Proteins - antagonists & inhibitors | Receptors, G-Protein-Coupled - metabolism | Humans | Drosophila Proteins - metabolism | Stem Cells - metabolism | Mitochondrial Membrane Transport Proteins - genetics | Drosophila melanogaster - metabolism | Monocarboxylic Acid Transporters | Transfection | Anion Transport Proteins - antagonists & inhibitors | RNA Interference | Time Factors | Mitochondrial Proteins - metabolism | Acrylates - pharmacology | Pyruvic Acid - metabolism | Cell Differentiation | Mitochondrial Membrane Transport Proteins - metabolism | Signal Transduction | Tissue Culture Techniques | Lactic Acid - metabolism | Cells, Cultured | Drosophila melanogaster - cytology | Genotype | Mitochondria - metabolism | Mitochondria - drug effects | Anion Transport Proteins - metabolism | Mice, Knockout | Phenotype | Animals | Glycolysis | Stem Cells - drug effects | Cell Proliferation - drug effects | Receptors, G-Protein-Coupled - genetics | Drosophila Proteins - genetics | Anion Transport Proteins - genetics | Intestines - cytology | Physiological aspects | Glucose metabolism | Cell differentiation | Stem cells | Cell proliferation | Pyruvic acid | Metabolism | Cytosol | Mitochondria | Clonal deletion | Organoids | Intestine | Fruit flies | Oxidation | Differentiation | Cancer
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2015, Volume 149, Issue 6, pp. 1564 - 1574.e3
Background & Aims Many patients with inflammatory bowel disease (IBD) fail to respond to anti–tumor necrosis factor (TNF) agents such as infliximab and... 
Gastroenterology and Hepatology | Therapy | Inflammation | EFFECTOR FUNCTIONS | CROHNS-DISEASE | VIVO | ULCERATIVE-COLITIS | MATRIX METALLOPROTEINASES | IN-VITRO | HINGE | MONOCLONAL-ANTIBODIES | INFLIXIMAB | ETANERCEPT | GASTROENTEROLOGY & HEPATOLOGY | Tumor Necrosis Factor-alpha - metabolism | Intestinal Mucosa - metabolism | Epitopes - metabolism | Humans | Inflammatory Bowel Diseases - immunology | Middle Aged | Crohn Disease - metabolism | Etanercept - metabolism | Colon - immunology | Male | Colitis, Ulcerative - immunology | Inflammatory Bowel Diseases - metabolism | Case-Control Studies | Intestinal Mucosa - drug effects | Immunoblotting - methods | Infliximab - metabolism | Proteolysis - drug effects | Intestinal Mucosa - immunology | Female | Colitis, Ulcerative - drug therapy | Biological Factors - metabolism | Inflammatory Bowel Diseases - drug therapy | Colon - pathology | Colitis, Ulcerative - metabolism | Antibodies, Monoclonal, Humanized - metabolism | Matrix Metalloproteinase 3 - metabolism | Matrix Metalloproteinase 12 - metabolism | Adalimumab - metabolism | Crohn Disease - immunology | Colon - metabolism | Biopsy | Crohn Disease - drug therapy | Biological Factors - pharmacology | Immunoglobulin G - metabolism | Medical colleges | Tumor necrosis factor | Gastrointestinal diseases | Tumors | Necrosis
Journal Article