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European Journal of Cancer, ISSN 0959-8049, 2016, Volume 68, pp. 156 - 162
Abstract Purpose To assess preliminary safety and efficacy results of the anti-programmed cell death ligand-1 (anti-PD-L1) durvalumab in combination with... 
Hematology, Oncology and Palliative Medicine | Immunoradiotherapy | Phase 2 | Phase 1 | Radiotherapy | Anti-PD-L1 | ABSCOPAL | ONCOLOGY | MECHANISMS | PATIENT | RADIATION-THERAPY | Lymphatic Metastasis - radiotherapy | Bone Neoplasms - therapy | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Carcinoma, Transitional Cell - secondary | Radiosurgery | Bone Neoplasms - secondary | Antibodies, Monoclonal - therapeutic use | Lung Neoplasms - pathology | Male | Antineoplastic Agents - therapeutic use | Carcinoma, Transitional Cell - therapy | Liver Neoplasms - therapy | Eye Neoplasms - therapy | Chemoradiotherapy | Small Cell Lung Carcinoma - secondary | Breast Neoplasms - therapy | Brain Neoplasms - secondary | Neoplasms - therapy | Squamous Cell Carcinoma of Head and Neck | Urinary Bladder Neoplasms - pathology | Aged, 80 and over | Adult | Female | Liver Neoplasms - secondary | Colonic Neoplasms - therapy | Eye Neoplasms - pathology | Head and Neck Neoplasms - therapy | Kaplan-Meier Estimate | Carcinoma, Squamous Cell - therapy | Survival Rate | Lung Neoplasms - therapy | Adenocarcinoma - secondary | Melanoma - secondary | Head and Neck Neoplasms - pathology | Disease-Free Survival | B7-H1 Antigen - antagonists & inhibitors | Adenocarcinoma - therapy | Breast Neoplasms - pathology | Colonic Neoplasms - pathology | Urinary Bladder Neoplasms - therapy | Brain Neoplasms - therapy | Radiotherapy, Conformal | Aged | Small Cell Lung Carcinoma - therapy | Melanoma - therapy | Biochemistry | Apoptosis
Journal Article
Gastroenterology, ISSN 0016-5085, 2010, Volume 138, Issue 6, pp. 2101 - 2114.e5
The connection between inflammation and tumorigenesis is well-established and in the last decade has received a great deal of supporting evidence from genetic,... 
Gastroenterology and Hepatology | Inflammation | Cytokines | Immunity | Colon Cancer | CHRONIC INTESTINAL INFLAMMATION | COLITIS-ASSOCIATED CANCER | SEVERE CROHNS-DISEASE | EPITHELIAL-MESENCHYMAL TRANSITION | TUMOR-NECROSIS-FACTOR | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | KINASE-I-EPSILON | TOLL-LIKE RECEPTORS | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | GENOME-WIDE ASSOCIATION | Colonic Neoplasms - genetics | Colonic Neoplasms - prevention & control | Colonic Neoplasms - drug therapy | Colorectal Neoplasms - genetics | Humans | Inflammatory Bowel Diseases - immunology | Gene Expression Regulation, Neoplastic | Colon - immunology | Cell Transformation, Neoplastic - genetics | Colonic Neoplasms - immunology | Colorectal Neoplasms - drug therapy | Inflammation Mediators - metabolism | Inflammatory Bowel Diseases - genetics | Anti-Inflammatory Agents - therapeutic use | Inflammatory Bowel Diseases - complications | Anticarcinogenic Agents - therapeutic use | Inflammatory Bowel Diseases - drug therapy | Genetic Predisposition to Disease | Cytokines - metabolism | Risk Assessment | Signal Transduction | Neoplasm Invasiveness | Risk Factors | Colorectal Neoplasms - prevention & control | Colonic Neoplasms - secondary | Cell Transformation, Neoplastic - immunology | Animals | Colorectal Neoplasms - immunology | Colorectal Neoplasms - secondary | Colon - microbiology | Dendritic cells | Measles-mumps-rubella vaccine | Biological response modifiers | Sulfates | Risk factors | Dextran | Prostaglandins | Colon cancer | Gastrointestinal diseases | Vascular endothelial growth factor | Growth factors | Tumors
Journal Article
Nature, ISSN 0028-0836, 02/2018, Volume 554, Issue 7693, pp. 538 - 543
Most patients with colorectal cancer die as a result of the disease spreading to other organs. However, no prevalent mutations have been associated with... 
STEM-CELLS | GENE | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | MOUSE | LEADS | RECEPTOR | MICE | GROWTH-FACTOR | TUMORIGENESIS | EXPRESSION | Intestines - drug effects | Colonic Neoplasms - genetics | Intestinal Mucosa - metabolism | Colonic Neoplasms - drug therapy | Humans | Male | Programmed Cell Death 1 Receptor - antagonists & inhibitors | Stem Cells - metabolism | Th1 Cells - immunology | T-Lymphocytes, Cytotoxic - drug effects | Neoplasm Metastasis - drug therapy | Neoplasm Metastasis - immunology | Colonic Neoplasms - immunology | Immunotherapy | Transforming Growth Factor beta - antagonists & inhibitors | Female | Liver Neoplasms - secondary | Disease Models, Animal | T-Lymphocytes, Cytotoxic - immunology | Transforming Growth Factor beta - immunology | Tumor Microenvironment - drug effects | Th1 Cells - drug effects | Intestines - pathology | Liver Neoplasms - drug therapy | Liver Neoplasms - immunology | Drug Synergism | Tumor Microenvironment - immunology | Neoplasm Metastasis - genetics | Animals | Cell Differentiation - drug effects | Neoplasm Metastasis - pathology | Colonic Neoplasms - pathology | Alleles | Immune Evasion - drug effects | Stem Cells - drug effects | Stem Cells - pathology | Mice | Mutation | T-Lymphocytes, Cytotoxic - cytology | PD-1 protein | Liver | Colorectal carcinoma | Colorectal cancer | Stem cell transplantation | Cytotoxicity | Lymphocytes T | Metastasis | Metastases | Colon cancer | Intestine | Lymphocytes | Genetic analysis | Fibroblasts | Colon | Inhibition | Growth factors | Phenotypes | Data analysis | Liver diseases | Microsatellites | Organs | Gene expression | Patients | Signaling | Immune checkpoint | PD-L1 protein | Stem cells | Tumors | Cancer
Journal Article
International Journal of Clinical Oncology, ISSN 1341-9625, 4/2015, Volume 20, Issue 2, pp. 207 - 239
Journal Article
Ugeskrift for laeger, 01/2015, Volume 177, Issue 2A, p. 114
Secondary neoplasms of the larynx are rare and account for 0.09-0,4% of all laryngeal tumours. Cutaneous melanomas are the preponderant primaries metastasizing... 
Laryngeal Neoplasms - radiotherapy | Adenocarcinoma - pathology | Laryngeal Neoplasms - surgery | Colonic Neoplasms - pathology | Humans | Female | Laryngeal Neoplasms - diagnostic imaging | Tomography, X-Ray Computed | Aged | Laryngeal Neoplasms - secondary
Journal Article
Gastroenterology, ISSN 0016-5085, 07/2018, Volume 155, Issue 1, pp. 33 - 37.e6
We studied the effects of gut microbiome depletion by oral antibiotics on tumor growth in subcutaneous and liver metastases models of pancreatic cancer, colon... 
Immune Regulation | Tumor Promotion | Gut Bacteria | Metastases | GASTROENTEROLOGY & HEPATOLOGY | IL-17 | T-Lymphocyte Subsets - immunology | Neoplasm Transplantation | Interleukin-17 - immunology | Soft Tissue Neoplasms - immunology | Dysbiosis - immunology | Melanoma, Experimental - immunology | Neoplasm Metastasis - immunology | Melanoma, Experimental - secondary | Liver Neoplasms - secondary | Carcinoma - secondary | Disease Models, Animal | Skin Neoplasms - pathology | Skin Neoplasms - immunology | Soft Tissue Neoplasms - secondary | Pancreatic Neoplasms - pathology | Liver Neoplasms - immunology | Melanoma - secondary | Mice, Knockout | Tumor Microenvironment - immunology | Animals | Gastrointestinal Microbiome - drug effects | Melanoma - immunology | Neoplasms - immunology | Colonic Neoplasms - pathology | Interferon-gamma - immunology | Pancreatic Neoplasms - immunology | Cell Line, Tumor | T-Lymphocytes - immunology | Anti-Bacterial Agents - pharmacology | Mice | Gastrointestinal Microbiome - immunology | Interleukin-10 - immunology | Medical colleges | Microbiota (Symbiotic organisms) | Growth | Liver | Melanoma | Metastasis | B cells | T cells | Biological response modifiers | Liver cancer | Colon cancer | Interleukins | Immunotherapy | Pancreatic cancer | Bacteria | Interferon gamma | metastases | gut bacteria | immune regulation | tumor promotion
Journal Article
Journal Article
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 03/2011, Volume 17, Issue 5, pp. 1122 - 1130
Journal Article
Cancer Cell, ISSN 1535-6108, 2011, Volume 19, Issue 1, pp. 125 - 137
Metastasis is responsible for most cancer deaths. Here, we show that (or ) gene functions as an endogenous metastasis suppressor. Expression of Aes was... 
COMPLEX | GENE | REPRESSION | ONCOLOGY | PATHWAY | DISEASE | RECEPTOR | MICE | INDUCTION | TUMORIGENESIS | MOUSE MODELS | CELL BIOLOGY | Gene Expression - genetics | Receptors, Notch - metabolism | Humans | Neoplasm Invasiveness - prevention & control | Colonic Neoplasms - metabolism | Receptors, Notch - antagonists & inhibitors | Liver Neoplasms - pathology | Transendothelial and Transepithelial Migration - physiology | Liver Neoplasms - secondary | Immunoglobulin J Recombination Signal Sequence-Binding Protein - genetics | Repressor Proteins - metabolism | Nuclear Matrix - metabolism | HCT116 Cells | Repressor Proteins - genetics | Mice, Transgenic | Down-Regulation - genetics | Intestinal Polyposis - metabolism | Lung Neoplasms - prevention & control | Signal Transduction - drug effects | Mice, Nude | Models, Biological | Cell Line, Tumor | Ligands | Mice | Mice, Inbred BALB C | Colonic Neoplasms - drug therapy | Lung Neoplasms - pathology | Lung Neoplasms - secondary | Mice, Mutant Strains | Neoplasm Invasiveness - pathology | Neoplasm Metastasis - prevention & control | Immunoglobulin J Recombination Signal Sequence-Binding Protein - metabolism | Intestinal Polyposis - pathology | Benzodiazepinones - pharmacology | Mice, Inbred C57BL | Stromal Cells - metabolism | Intestinal Polyposis - drug therapy | Receptor, Notch1 - metabolism | Transcription Factors - genetics | Transcription Factors - metabolism | Neoplasm Metastasis - genetics | Animals | Neoplasm Metastasis - pathology | Colonic Neoplasms - pathology | Signal Transduction - physiology | Co-Repressor Proteins | Neoplasm Invasiveness - genetics | Benzodiazepinones - therapeutic use | Gene Silencing - physiology
Journal Article