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1. Associations between adipokines and obesity-related cancer
by Paz-Filho, Gilberto and Lim, Ee Lyn and Wong, Ma-Li and Licinio, Julio
Frontiers in Bioscience, ISSN 1093-9946, 01/2011, Volume 16, Issue 5, pp. 1634 - 1650
There is increasing evidence that obesity may have pathophysiological effects that extend beyond its well-known co-morbidities; in particular its role in... 
Adipocytokine | Obesity | Leptin | Adipokine | Review | Adiponectin | Cancer | PLASMA ADIPONECTIN LEVELS | HUMAN ENDOMETRIAL CANCER | MESSENGER-RNA EXPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | LEPTIN-DEFICIENT ADULTS | JUN NH2-TERMINAL KINASE | RENAL-CELL CARCINOMA | CELL BIOLOGY | ESOPHAGEAL ADENOCARCINOMA CELLS | BODY-MASS-INDEX | PAPILLARY THYROID-CARCINOMA | COLONIC EPITHELIAL-CELLS | Body Mass Index | Esophageal Neoplasms - etiology | Neoplasms - etiology | Obesity - complications | Pancreatic Neoplasms - etiology | Humans | Thyroid Neoplasms - etiology | Breast Neoplasms - etiology | Endometrial Neoplasms - etiology | Prostatic Neoplasms - etiology | Male | Prostatic Neoplasms - physiopathology | Adiponectin - physiology | Endometrial Neoplasms - physiopathology | Breast Neoplasms - physiopathology | Colorectal Neoplasms - physiopathology | Leptin - physiology | Thyroid Neoplasms - physiopathology | Colorectal Neoplasms - etiology | Kidney Neoplasms - physiopathology | Kidney Neoplasms - etiology | Esophageal Neoplasms - physiopathology | Female | Pancreatic Neoplasms - physiopathology
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2. Full Text A Subpopulation of CD26 + Cancer Stem Cells with Metastatic Capacity in Human Colorectal Cancer
by Pang, Roberta and Law, Wai Lun and Chu, Andrew C.Y and Poon, Jensen T and Poon, Ronnie T and Lam, Colin S.C and Chow, Ariel K.M and Ng, Lui and Cheung, Leonard W.H and Lan, Hui Y and Lan, Xiao R and Tan, Victoria P.Y and Yau, Thomas C and Wong, Benjamin C.Y
Cell Stem Cell, ISSN 1934-5909, 2010, Volume 6, Issue 6, pp. 603 - 615
Recent evidence suggests that a subpopulation of cancer cells, cancer stem cells (CSCs), is responsible for tumor growth in colorectal cancer. However,... 
STEMCELL | CELLCYCLE | BREAST-CANCER | INITIATING CELLS | DIPEPTIDYL-PEPTIDASE-IV | CARCINOMA CELLS | EXTRACELLULAR-MATRIX | ACUTE MYELOID-LEUKEMIA | E-CADHERIN | IDENTIFICATION | TUMOR-GROWTH | HUMAN PANCREATIC-CANCER | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Neoplasm Transplantation | RNA, Small Interfering - genetics | Prognosis | Follow-Up Studies | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Colorectal Neoplasms - genetics | Humans | Apoptosis - genetics | Gene Expression Profiling | Colorectal Neoplasms - diagnosis | Organoplatinum Compounds - pharmacology | Neoplastic Stem Cells - metabolism | Cell Transformation, Neoplastic - genetics | Colorectal Neoplasms - drug therapy | Liver Neoplasms - physiopathology | Neoplastic Stem Cells - pathology | Tumor Burden - genetics | Tumor Cells, Cultured | Liver Neoplasms - secondary | Colorectal Neoplasms - metabolism | Carcinoma - secondary | Carcinoma - drug therapy | Liver Neoplasms - genetics | Dipeptidyl Peptidase 4 - biosynthesis | Liver Neoplasms - drug therapy | Carcinoma - diagnosis | Dipeptidyl Peptidase 4 - genetics | Mice, SCID | Carcinoma - physiopathology | Disease Progression | Colorectal Neoplasms - physiopathology | Drug Resistance, Neoplasm - genetics | Animals | Liver Neoplasms - diagnosis | Tumor Burden - drug effects | Liver Neoplasms - metabolism | Carcinoma - genetics | Biomarkers, Tumor - genetics | Fluorouracil - pharmacology | Mice | Carcinoma - metabolism | Colorectal Neoplasms - pathology | Neoplasm Invasiveness - genetics | Cell Migration Assays | Biomarkers, Tumor - biosynthesis
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3. Tight junctions in cancer metastasis
by Martin, Tracey A and Mason, Malcolm D and Jiang, Wen G
Frontiers in Bioscience, ISSN 1093-9946, 01/2011, Volume 16, Issue 3, pp. 898 - 936
Tight Junctions (TJ) are well known to function as a control for the paracellular diffusion of ions and certain molecules, it has however, become evident that... 
Metastasis | Review | Barrier function | Cancer | Tight Junction | BIOCHEMISTRY & MOLECULAR BIOLOGY | COXSACKIE-ADENOVIRUS RECEPTOR | CLOSTRIDIUM-PERFRINGENS ENTEROTOXIN | TRANSENDOTHELIAL RESISTANCE TER | HUMAN BREAST-CANCER | EPITHELIAL OVARIAN-CANCER | CELL BIOLOGY | GROWTH-FACTOR RECEPTOR | PARACELLULAR BARRIER FUNCTION | HUMAN ENDOMETRIAL CARCINOMA | SQUAMOUS-CELL CARCINOMA | VASCULAR ENDOTHELIAL-CELLS | Receptors, Virus - biosynthesis | Humans | Brain Neoplasms - physiopathology | Claudins - biosynthesis | Male | Receptors, Cell Surface | Prostatic Neoplasms - physiopathology | Coxsackie and Adenovirus Receptor-Like Membrane Protein | Lung Neoplasms - physiopathology | Stomach Neoplasms - physiopathology | Breast Neoplasms - physiopathology | Nectins | Occludin | Tight Junctions - physiology | Urinary Bladder Neoplasms - physiopathology | Kidney Neoplasms - physiopathology | Neoplasm Invasiveness - pathology | Liver Neoplasms - physiopathology | Female | Immunoglobulins - biosynthesis | Genital Neoplasms, Female - physiopathology | Melanoma - physiopathology | Epithelium - pathology | Cell Adhesion Molecules - biosynthesis | Colorectal Neoplasms - physiopathology | Thyroid Neoplasms - physiopathology | Membrane Proteins - biosynthesis | Animals | Neoplasm Metastasis - pathology | Sarcoma, Synovial - physiopathology | Esophageal Neoplasms - physiopathology | Pancreatic Neoplasms - physiopathology | Neoplasms - pathology
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4. Full Text Tumor metastasis: mechanistic insights and clinical challenges
by Steeg, Patricia S
Nature Medicine, ISSN 1078-8956, 08/2006, Volume 12, Issue 8, pp. 895 - 904
Metastatic disease is the primary cause of death for most cancer patients. Complex and redundant pathways involving the tumor cell and the microenvironment... 
MEDICINE, RESEARCH & EXPERIMENTAL | NUDE-MICE | MAMMARY-CARCINOMA CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | LIVER METASTASIS | HUMAN PANCREATIC-CANCER | CELL BIOLOGY | BREAST-CANCER CELLS | MATRIX-METALLOPROTEINASE INHIBITORS | FOCAL-ADHESION KINASE | COLORECTAL-CANCER | IN-VIVO | ENDOTHELIAL GROWTH-FACTOR | Neoplasms - metabolism | Capillary Permeability | Neoplastic Cells, Circulating - pathology | Humans | Gene Expression Regulation, Neoplastic | Neovascularization, Pathologic | Neoplasm Invasiveness - physiopathology | Neoplasm Metastasis - genetics | Animals | Models, Biological | Neoplasm Invasiveness - pathology | Neoplasm Metastasis - pathology | Neoplasm Metastasis - physiopathology | Neoplasms - physiopathology | Neoplasms, Experimental - metabolism | Neoplasm Invasiveness - genetics | Cell growth | Biomedical research | Molecular biology | Cancer | Tumors
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5. Full Text Lymphangiogenesis and lymphatic vessel remodelling in cancer
by Stacker, Steven A and Williams, Steven P and Karnezis, Tara and Shayan, Ramin and Fox, Stephen B and Achen, Marc G
Nature Reviews Cancer, ISSN 1474-175X, 03/2014, Volume 14, Issue 3, pp. 159 - 172
The generation of new lymphatic vessels through lymphangiogenesis and the remodelling of existing lymphatics are thought to be important steps in cancer... 
CELL LUNG-CANCER | VASCULAR NETWORK FORMATION | NODE METASTASIS | PROMOTES TUMOR-METASTASIS | ONCOLOGY | ENDOTHELIAL-GROWTH-FACTOR | VEGF-D PROMOTES | CHEMOKINE RECEPTOR CXCR4 | FACTOR-C EXPRESSION | I COLORECTAL-CARCINOMA | HUMAN BREAST-CANCER | Neoplasms - metabolism | Lymphatic Vessels - metabolism | Animals | Neoplasms - genetics | Humans | Lymphatic Vessels - pathology | Neovascularization, Pathologic | Neoplasms - physiopathology | Lymphangiogenesis | Lymphatic Vessels - physiopathology | Neoplasms - pathology | Metastasis | Research | Oncology, Experimental | Cancer
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6. Full Text Opinion - Migrating cancer stem cells - an integrated concept of malignant tumour progression
by Brabletz, T and Jung, A and Spaderna, S and Hlubek, F and Kirchner, T
NATURE REVIEWS CANCER, ISSN 1474-175X, 09/2005, Volume 5, Issue 9, pp. 744 - 749
The dissemination of tumour cells is the prerequisite of metastases and is correlated with a loss of epithelial differentiation and the acquisition of a... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | TRANSCRIPTION FACTORS | RECTAL-CANCER | ONCOLOGY | COLORECTAL-CANCER | E-CADHERIN EXPRESSION | DOWN-REGULATION | HAIR FOLLICLE MORPHOGENESIS | SELF-RENEWAL | NUCLEAR BETA-CATENIN | Colorectal Neoplasms - physiopathology | Animals | Models, Biological | Neoplasm Metastasis - pathology | Humans | Neoplasm Metastasis - physiopathology | Stem Cells - pathology | Neoplasms - physiopathology | Colorectal Neoplasms - pathology | Neoplasms - pathology | Stem Cells - metabolism | Cell Movement - physiology
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7. Full Text Bile acid–microbiota crosstalk in gastrointestinal inflammation and carcinogenesis
by Jia, Wei and Xie, Guoxiang and Jia, Weiping
Nature Reviews Gastroenterology and Hepatology, ISSN 1759-5045, 02/2018, Volume 15, Issue 2, pp. 111 - 128
Emerging evidence points to a strong association between the gut microbiota and the risk, development and progression of gastrointestinal cancers such as... 
FARNESOID-X | COLON-CANCER CELLS | BETA-MURICHOLIC ACID | CHAIN FATTY-ACIDS | NUCLEAR RECEPTORS | TUMOR-ASSOCIATED MACROPHAGES | GUT MICROBIOTA | CHOLESTATIC LIVER | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | PREGNANE-X-RECEPTOR | Carcinoma, Hepatocellular - physiopathology | Humans | Bile Acids and Salts - metabolism | Gastroenteritis - therapy | Gastrointestinal Microbiome - physiology | Liver Neoplasms - therapy | Carcinogenesis | Digestive System Neoplasms - etiology | Liver Neoplasms - etiology | Gastroenteritis - etiology | Hepatitis - physiopathology | Liver Neoplasms - physiopathology | Signal Transduction - physiology | Biological Transport - physiology | Carcinoma, Hepatocellular - etiology | Carcinoma, Hepatocellular - therapy | Digestive System Neoplasms - physiopathology | Bile Acids and Salts - physiology | Gastroenteritis - physiopathology | Digestive System Neoplasms - therapy | Liver cancer | Microbiota (Symbiotic organisms) | Analysis | Colorectal cancer | Carbohydrate metabolism | Development and progression | Bile acids | Digestive system | Liver | Colorectal carcinoma | Hepatocellular carcinoma | Innate immunity | Metabolism | Insulin | Microbiota | Acids | Intestine | Lipid metabolism | Digestive tract | Bile | Index Medicus
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8. Full Text Adrenocortical Carcinoma
by Else, Tobias and Kim, Alex C and Sabolch, Aaron and Raymond, Victoria M and Kandathil, Asha and Caoili, Elaine M and Jolly, Shruti and Miller, Barbra S and Giordano, Thomas J and Hammer, Gary D
Endocrine Reviews, ISSN 0163-769X, 04/2014, Volume 35, Issue 2, pp. 282 - 326
Adrenocortical carcinoma (ACC) is a rare endocrine malignancy, often with an unfavorable prognosis. Here we summarize the knowledge about diagnosis,... 
CLINICAL-PRACTICE GUIDELINES | POSITRON-EMISSION-TOMOGRAPHY | BECKWITH-WIEDEMANN-SYNDROME | NONPOLYPOSIS COLORECTAL-CANCER | LONG-TERM SURVIVAL | ENDOCRINOLOGY & METABOLISM | FAMILIAL ADENOMATOUS POLYPOSIS | ADRENAL-CORTICAL CARCINOMA | OF-THE-LITERATURE | ENDOCRINE NEOPLASIA TYPE-1 | GROWTH-FACTOR-II | Adrenal Cortex Neoplasms - therapy | Adrenal Cortex Neoplasms - diagnosis | Adrenal Cortex Neoplasms - epidemiology | Adrenocortical Carcinoma - epidemiology | Prevalence | Humans | Adrenocortical Carcinoma - diagnosis | Adrenal Cortex Neoplasms - physiopathology | Genetic Predisposition to Disease - epidemiology | Adrenocortical Carcinoma - therapy | Adrenocortical Carcinoma - physiopathology | Reviews
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9. Full Text Association between physical activity and mortality among breast cancer and colorectal cancer survivors: A systematic review and meta-analysis
by Schmid, Daniela and Leitzmann, M.F
Annals of Oncology, ISSN 0923-7534, 2014, Volume 25, Issue 7, pp. 1293 - 1311
Physical activity improves physical function during and after cancer treatment, but whether physical activity imparts survival benefit remains uncertain. Using... 
Physical activity | Survival | Cancer | Meta-analysis | ACTIVITY GUIDELINES | DIAGNOSIS | survival | SIZE | meta-analysis | EXERCISE | physical activity | ONCOLOGY | INSULIN-RESISTANCE | BODY-MASS INDEX | cancer | RECURRENCE | HEALTH | POSTMENOPAUSAL WOMEN | LIFE | Colorectal Neoplasms - physiopathology | Humans | Survivors | Female | Male | Motor Activity | Breast Neoplasms - physiopathology
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10. Full Text Epithelial-mesenchymal plasticity in carcinoma metastasis
by Tsai, Jeff H and Yang, Jing
Genes and Development, ISSN 0890-9369, 10/2013, Volume 27, Issue 20, pp. 2192 - 2206
Tumor metastasis is a multistep process by which tumor cells disseminate from their primary site and form secondary tumors at a distant site. Metastasis occurs... 
Epithelial-mesenchymal transition (EMT) | Mesenchymal-epithelial transition (MET) | Extravasation | Carcinoma metastasis | Intravasation | Tumor dormancy | Invasion | STEM-CELLS | TRANSCRIPTION FACTOR SNAIL | carcinoma metastasis | DEVELOPMENTAL BIOLOGY | intravasation | mesenchymal-epithelial transition (MET) | MIR-200 FAMILY | NEGATIVE FEEDBACK LOOP | CELL BIOLOGY | epithelial-mesenchymal transition (EMT) | extravasation | BREAST-CANCER CELLS | INVADOPODIA FORMATION | invasion | CIRCULATING TUMOR-CELLS | tumor dormancy | COLORECTAL-CANCER | GENETICS & HEREDITY | E-CADHERIN GENE | NF-KAPPA-B | Neoplasms - therapy | Neoplasms - physiopathology | Epithelial-Mesenchymal Transition - physiology | Humans | Neoplasm Metastasis - physiopathology | epithelial–mesenchymal transition (EMT) | Review | mesenchymal–epithelial transition (MET)
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