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Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 12/2018, Volume 45, Issue 12, pp. 1341 - 1350
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2014, Volume 9, Issue 4, p. e94550
Background: Liver dysfunction and cirrhosis affect vasculature in several organ systems and cause impairment of organ functions, thereby increasing morbidity... 
PATHOGENESIS | ARTERIOVENOUS-MALFORMATIONS | ENDOTHELIN-B RECEPTOR | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | HEPATOPULMONARY-SYNDROME | MATRIX-METALLOPROTEINASE-9 | POTENTIAL ROLE | EXPRESSION | RAT MODEL | PORTAL-HYPERTENSION | Immunohistochemistry | Lung - pathology | Oligonucleotide Array Sequence Analysis | Hepatopulmonary Syndrome - physiopathology | Humans | Liver Diseases - pathology | Neovascularization, Pathologic | Inflammation | Lung - physiopathology | Lung Injury - etiology | Animals | Ligation | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Common Bile Duct - surgery | Mice | Mice, Inbred BALB C | Neutrophils - metabolism | Disease Models, Animal | Care and treatment | DNA microarrays | Diagnosis | Liver cirrhosis | Analysis | Cell proliferation | Pediatrics | Animal models | Thoracic surgery | Pathogenesis | Liver | Inflammatory response | Biochemistry | Proteins | Angiogenesis | Cell growth | Rodents | Animal tissues | Surgery | Gastroenterology | Tumor necrosis factor-TNF | Vascular endothelial growth factor | Injuries | University graduates | Medical research | Liver diseases | Mortality | Neutrophils | Pancreatitis | Committees | Tumor necrosis factor-α | Morbidity | Endothelial cells | Bile duct | Gelatinase B | Abdomen | Medicine | Studies | Polymerase chain reaction | Cirrhosis | Signaling | Lungs | Nitric oxide | Sepsis
Journal Article
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 2015, Volume 308, Issue 8, pp. G691 - G701
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 03/2018, Volume 314, Issue 3, pp. G319 - G333
Cholestatic liver injury results from impaired bile flow or metabolism and promotes hepatic inflammation and fibrogenesis. Toxic bile acids that accumulate in... 
CFLIP | Reactive oxygen species | Mitochondrial dysfunction | Hepatocytes | ASK1 | Bile duct ligation | JNK | Neutrophils | Cholestasis | KAPPA-B ACTIVATION | PHYSIOLOGY | cFLIP | RAT HEPATOCYTES | mitochondrial dysfunction | ACID-INDUCED APOPTOSIS | hepatocytes | MEDIATED-APOPTOSIS | JNK ACTIVATION | bile duct ligation | PRIMARY HUMAN HEPATOCYTES | cholestasis | OBSTRUCTIVE-CHOLESTASIS | reactive oxygen species | ANTI-APOPTOTIC MECHANISM | TNF-ALPHA | C-FLIPL | GASTROENTEROLOGY & HEPATOLOGY | neutrophils | Choledocholithiasis - metabolism | Liver - pathology | Oxidative Stress | Bile Acids and Salts - toxicity | Hepatic Stellate Cells - metabolism | Hepatocytes - pathology | Tumor Necrosis Factor alpha-Induced Protein 3 - metabolism | Hepatocytes - metabolism | Neutrophil Infiltration | Necrosis | Ligation | Liver - drug effects | Time Factors | Choledocholithiasis - etiology | Hepatitis - genetics | Hepatitis - metabolism | Inflammation Mediators - metabolism | Liver Cirrhosis - metabolism | Common Bile Duct - surgery | p38 Mitogen-Activated Protein Kinases - metabolism | Liver Cirrhosis - genetics | Hepatic Stellate Cells - pathology | Hepatocytes - drug effects | Hepatic Stellate Cells - drug effects | Liver Cirrhosis - etiology | Choledocholithiasis - genetics | Genetic Predisposition to Disease | Hepatitis - etiology | Cytokines - metabolism | Signal Transduction | CASP8 and FADD-Like Apoptosis Regulating Protein - genetics | Liver - metabolism | CASP8 and FADD-Like Apoptosis Regulating Protein - deficiency | Cells, Cultured | Hepatitis - pathology | Bile Acids and Salts - metabolism | Mice, Knockout | Phenotype | Animals | Transcription Factor RelA - metabolism | Liver Cirrhosis - pathology | Apoptosis | Choledocholithiasis - pathology | Physiological aspects | Common bile duct | Liver diseases | Health aspects | Bile ducts | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2009, Volume 137, Issue 1, pp. 297 - 308.e4
Background & Aims The hepatocyte growth factor (HGF)/mesenchymal-epithelial transition factor (c-Met) system is an essential inducer of hepatocyte growth and... 
Gastroenterology and Hepatology | mouse | expression | cells | receptor | cxc chemokines | regeneration | nemo/ikk-gamma | hepatocyte growth-factor | hepatic-injury | cre recombinase | CELLS | REGENERATION | HEPATOCYTE GROWTH-FACTOR | MOUSE | CRE RECOMBINASE | RECEPTOR | CXC CHEMOKINES | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | HEPATIC-INJURY | NEMO/IKK-GAMMA | Proto-Oncogene Proteins c-met - metabolism | Liver - pathology | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Hepatic Stellate Cells - metabolism | Apoptosis - genetics | Hepatocytes - pathology | Hepatocytes - metabolism | Cholestasis, Extrahepatic - complications | Neutrophil Infiltration | Necrosis | Ligation | Time Factors | Hepatitis - metabolism | Inflammation Mediators - metabolism | Liver Cirrhosis - metabolism | Common Bile Duct - surgery | Liver Cirrhosis - genetics | Cholestasis, Extrahepatic - pathology | Extracellular Matrix Proteins - metabolism | Hepatic Stellate Cells - pathology | Disease Models, Animal | Liver Cirrhosis - prevention & control | Cytokines - metabolism | Liver - metabolism | Hepatitis - pathology | Gene Expression Profiling - methods | Cholestasis, Extrahepatic - genetics | Disease Progression | Hepatocyte Growth Factor - metabolism | Mice, Knockout | Proto-Oncogene Proteins c-met - genetics | Animals | Proto-Oncogene Proteins c-met - deficiency | Liver Cirrhosis - pathology | Mice | Cholestasis, Extrahepatic - metabolism | Chronic Disease | Liver diseases | Liver | Fibrosis | Physiological aspects | Albumin | Growth factors | Tumors
Journal Article
Hepatobiliary & Pancreatic Diseases International, ISSN 1499-3872, 2017, Volume 16, Issue 1, pp. 88 - 95
Background Bile duct ligation (BDL) and subsequent cholestasis are correlated with oxidative stress, hepatocellular injury and fibrosis. Quercetin is a... 
Endocrinology & Metabolism | Gastroenterology and Hepatology | quercetin | liver fibrosis | Rac1 | NOX1 | oxidative stress | FIBROSIS | OXIDATIVE-STRESS | ACTIVATION | HEDGEHOG | HEPATIC STELLATE CELLS | CARBONYLATION | GLUTATHIONE | DISEASE | DRUGS | UPDATE | GASTROENTEROLOGY & HEPATOLOGY | Liver - pathology | Liver - enzymology | Cholestasis - complications | Rats, Wistar | Transforming Growth Factor beta1 - metabolism | Actins - metabolism | Liver Cirrhosis, Experimental - etiology | Male | Protein Carbonylation - drug effects | Actins - genetics | Liver Cirrhosis, Experimental - prevention & control | Liver Cirrhosis, Experimental - enzymology | Sulfhydryl Compounds - metabolism | Collagen Type I - genetics | Liver - drug effects | Liver Cirrhosis, Experimental - pathology | Cholestasis - drug therapy | NADH, NADPH Oxidoreductases - metabolism | Superoxide Dismutase - metabolism | Cytoprotection | NADH, NADPH Oxidoreductases - genetics | Collagen Type I - metabolism | Cholestasis - enzymology | Down-Regulation | Hydroxyproline - metabolism | Transforming Growth Factor beta1 - genetics | Antioxidants - pharmacology | NADPH Oxidase 1 | Catalase - metabolism | Cholestasis - pathology | Animals | Quercetin - pharmacology | Signal Transduction - drug effects | Oxidative Stress - drug effects | rac1 GTP-Binding Protein - metabolism | rac1 GTP-Binding Protein - genetics | Prevention | Common bile duct | Usage | Liver diseases | Bile ducts | NADP (Coenzyme) | Quercetin | Research
Journal Article
Liver International, ISSN 1478-3223, 04/2015, Volume 35, Issue 4, pp. 1373 - 1382
Journal Article