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Cancer research (Chicago, Ill.), ISSN 1538-7445, 2018, Volume 78, Issue 1, pp. 143 - 156
.... However, complement activation is also peculiarly associated with tumor progression. Here we report mechanistic insights into this association in multiple immunocompetent orthotopic models of lung cancer... 
SYSTEM | HOMEOSTASIS | THERAPY | METASTASIS | CELL RESPONSES | MICROENVIRONMENT | ONCOLOGY | TUMOR | ANTITUMOR IMMUNITY | C5A | MODULATION | Adenocarcinoma - pathology | Adenocarcinoma of Lung | Complement Activation | Adenocarcinoma - immunology | Humans | Mice, Inbred C57BL | Lung Neoplasms - pathology | Male | Mice, Transgenic | CD4-Positive T-Lymphocytes - pathology | Receptors, Complement - metabolism | CD4-Positive T-Lymphocytes - immunology | Complement C3d - metabolism | Immunoglobulin M - metabolism | Xenograft Model Antitumor Assays | Lung Neoplasms - immunology | Animals | Receptors, Complement - immunology | Oncogene Proteins, Fusion - genetics | Cell Line, Tumor | Female | Complement C3 - genetics | Complement receptors | Animal models | Complement component C3a | Complement component C5a | CD8 antigen | Lung cancer | Activation | Complement | Lymphocytes T | Metastases | Receptors | Cell activation | Clonal deletion | Lymphocytes | Interleukin 1 | Inhibition | Autocrine signalling | Activation analysis | Immune system | Tumor cell lines | Tumor necrosis factor-α | CD4 antigen | Complement activation | Complement component C3 | γ-Interferon | Interleukin 10 | Mice | Tumors | Cancer | C3a | orthotopic mouse model | CD4+ T Lymphocytes | complement | C3aR Autocrine Loop
Journal Article
Seminars in immunopathology, ISSN 1863-2300, 2017, Volume 40, Issue 1, pp. 15 - 35
Journal Article