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Journal Article
Cancer Research, ISSN 0008-5472, 01/2018, Volume 78, Issue 1, pp. 143 - 156
The complement cascade is a part of the innate immune system that acts primarily to remove pathogens and injured cells. However, complement activation is also... 
SYSTEM | HOMEOSTASIS | THERAPY | METASTASIS | CELL RESPONSES | MICROENVIRONMENT | ONCOLOGY | TUMOR | ANTITUMOR IMMUNITY | C5A | MODULATION | Adenocarcinoma - pathology | Adenocarcinoma of Lung | Complement Activation | Adenocarcinoma - immunology | Humans | Mice, Inbred C57BL | Lung Neoplasms - pathology | Male | Mice, Transgenic | CD4-Positive T-Lymphocytes - pathology | Receptors, Complement - metabolism | CD4-Positive T-Lymphocytes - immunology | Complement C3d - metabolism | Immunoglobulin M - metabolism | Xenograft Model Antitumor Assays | Lung Neoplasms - immunology | Animals | Receptors, Complement - immunology | Oncogene Proteins, Fusion - genetics | Cell Line, Tumor | Female | Complement C3 - genetics | Complement receptors | Animal models | Complement component C3a | Complement component C5a | CD8 antigen | Lung cancer | Activation | Complement | Antagonists | Lymphocytes T | Metastases | Receptors | Cell activation | Clonal deletion | Lymphocytes | Interleukin 1 | Deletion | Inhibition | Autocrine signalling | Activation analysis | Immune system | Tumor cell lines | Tumor necrosis factor-α | CD4 antigen | Complement activation | Complement component C3 | γ-Interferon | Interleukin 10 | Mice | Tumors | Cancer | Index Medicus | C3a | orthotopic mouse model | CD4+ T Lymphocytes | complement | C3aR Autocrine Loop
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2011, Volume 108, Issue 12, pp. 4974 - 4979
C-reactivé protein (CRP) is an important biomarker for inflammatory diseases. However, its role in inflammation beyond complementmediated pathogen clearance... 
Receptors | Cytokines | Neutrophils | Cell lines | Antibodies | Ligands | Mast cells | Macrophages | Phagocytosis | Binding sites | Serum amyloid P component | CD89 | Acute phase protein | serum amyloid P component | AMYLOID-P COMPONENT | COMPLEX | PHAGOCYTOSIS | SERUM IGA | CRYSTAL-STRUCTURE | MULTIDISCIPLINARY SCIENCES | MAST-CELLS | RELEASE | GAMMA RECEPTORS | INFLAMMATION | acute phase protein | KINASES | Antigens, CD - immunology | Humans | Tumor Necrosis Factor-alpha - genetics | Immunity, Innate - genetics | Antigens, CD - genetics | Complement C1q - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Extracellular Signal-Regulated MAP Kinases - immunology | Phosphorylation - genetics | Receptors, IgG - genetics | Tumor Necrosis Factor-alpha - immunology | Antigens, CD - chemistry | Phosphorylation - immunology | Binding Sites | Complement C1q - chemistry | Macrophages - immunology | Receptors, Fc - chemistry | Neutrophils - immunology | Phagocytosis - immunology | C-Reactive Protein - genetics | Inflammation | Complement C1q - genetics | Receptors, IgG - immunology | C-Reactive Protein - chemistry | Immunity, Innate - immunology | Receptors, Fc - immunology | Tumor Necrosis Factor-alpha - chemistry | Protein Binding | Receptors, Fc - genetics | Mutation | Biomarkers - chemistry | C-Reactive Protein - immunology | Genetic aspects | C-reactive protein | Biological markers | Immunoglobulin A | Health aspects | Proteins | Immunoglobulins | Phosphorylation | Biomarkers | TNF inhibitors | Kinases | Inflammatory diseases | Index Medicus | Pathogens | biomarkers | Extracellular signal-regulated kinase | Complement | Leukocytes (neutrophilic) | pentraxins | Complement component C1q | Fc receptors | Degranulation | Tumor necrosis factor- alpha | Biological Sciences
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 10/2018, Volume 94, Issue 4, pp. 689 - 700
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2014, Volume 192, Issue 6, pp. 2837 - 2845
Inflammation is associated with development of atherosclerosis, and cholesterol crystals (CC) have long been recognized as a hallmark of atherosclerotic... 
ATHEROSCLEROTIC LESIONS | TUMOR-NECROSIS-FACTOR | C5A RECEPTOR | MEMBRANE ATTACK | STERILE INFLAMMATION | EPITHELIAL-CELLS | CARDIOVASCULAR EVENTS | INTERLEUKIN-1-BETA | OXIDATIVE BURST | ANAPHYLATOXINS | IMMUNOLOGY | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Complement C1q - immunology | Humans | Complement Pathway, Classical - drug effects | Complement C5a - metabolism | Complement System Proteins - immunology | Caspase 1 - metabolism | Monocytes - metabolism | Caspase 1 - immunology | Macrophage-1 Antigen - immunology | Monocytes - immunology | Interleukin-1beta - genetics | Complement Activation - immunology | Complement C1q - metabolism | Complement System Proteins - metabolism | Inflammasomes - drug effects | Reactive Oxygen Species - immunology | Complement C5 - immunology | Tumor Necrosis Factor-alpha - immunology | Macrophage-1 Antigen - metabolism | Complement Activation - drug effects | Cytokines - immunology | Interleukin-1beta - secretion | Complement C5 - metabolism | Tumor Necrosis Factor-alpha - secretion | Cells, Cultured | Cytokines - secretion | Phagocytosis - immunology | Interleukin-1beta - immunology | Cholesterol - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Monocytes - drug effects | Complement Pathway, Classical - immunology | Microscopy, Confocal | Complement Pathway, Alternative - drug effects | Complement Pathway, Alternative - immunology | Inflammasomes - immunology | Cholesterol - pharmacology | Complement C5a - immunology | Index Medicus | Abridged Index Medicus
Journal Article