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1. Full Text Contaminated Heparin Associated with Adverse Clinical Events and Activation of the Contact System
by Kishimoto, Takashi Kei and Viswanathan, Karthik and Ganguly, Tanmoy and Elankumaran, Subbiah and Smith, Sean and Pelzer, Kevin and Lansing, Jonathan C and Sriranganathan, Nammalwar and Zhao, Ganlin and Galcheva-Gargova, Zoya and Al-Hakim, Ali and Bailey, Gregory Scott and Fraser, Blair and Roy, Sucharita and Rogers-Cotrone, Thomas and Buhse, Lucinda and Whary, Mark and Fox, James and Nasr, Moheb and Dal Pan, Gerald J and Shriver, Zachary and Langer, Robert S and Venkataraman, Ganesh and Austen, K. Frank and Woodcock, Janet and Sasisekharan, Ram
The New England journal of medicine, ISSN 0028-4793, 06/2008, Volume 358, Issue 23, pp. 2457 - 2467
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Biological and medical sciences | General aspects | Medical sciences | Complement C3a - drug effects | United States | Humans | Middle Aged | Complement C5a - biosynthesis | Heparin - adverse effects | Complement C5a - drug effects | Chondroitin Sulfates - adverse effects | Anaphylaxis - chemically induced | China | Chondroitin Sulfates - analysis | Female | Complement Activation - drug effects | United States Food and Drug Administration | Complement C3a - biosynthesis | Drug Industry | Chondroitin Sulfates - pharmacology | Hypotension - chemically induced | Animals | Kallikreins - drug effects | Sus scrofa | Drug Contamination | Germany | Heparin - chemistry | Kallikreins - metabolism | Composition | Heparin | Health aspects | Chondroitin | Drug therapy | Hemodialysis | Family medical history | Index Medicus | Abridged Index Medicus
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2. Full Text Cholesterol crystals induce complement-dependent inflammasome activation and cytokine release
by Samstad, Eivind O and Niyonzima, Nathalie and Nymo, Stig and Aune, Marie H and Ryan, Liv and Bakke, Siril S and Lappegård, Knut T and Brekke, Ole-Lars and Lambris, John D and Damås, Jan K and Latz, Eicke and Mollnes, Tom E and Espevik, Terje
The Journal of immunology (1950), ISSN 0022-1767, 03/2014, Volume 192, Issue 6, pp. 2837 - 2845
Life Sciences & Biomedicine | Immunology | Science & Technology | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Complement C1q - immunology | Humans | Complement Pathway, Classical - drug effects | Complement C5a - metabolism | Complement System Proteins - immunology | Caspase 1 - metabolism | Monocytes - metabolism | Caspase 1 - immunology | Macrophage-1 Antigen - immunology | Monocytes - immunology | Interleukin-1beta - genetics | Complement Activation - immunology | Complement C1q - metabolism | Complement System Proteins - metabolism | Inflammasomes - drug effects | Reactive Oxygen Species - immunology | Complement C5 - immunology | Tumor Necrosis Factor-alpha - immunology | Macrophage-1 Antigen - metabolism | Complement Activation - drug effects | Cytokines - immunology | Interleukin-1beta - secretion | Complement C5 - metabolism | Tumor Necrosis Factor-alpha - secretion | Cells, Cultured | Cytokines - secretion | Phagocytosis - immunology | Interleukin-1beta - immunology | Cholesterol - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Monocytes - drug effects | Complement Pathway, Classical - immunology | Microscopy, Confocal | Complement Pathway, Alternative - drug effects | Complement Pathway, Alternative - immunology | Inflammasomes - immunology | Cholesterol - pharmacology | Complement C5a - immunology | Index Medicus | Abridged Index Medicus
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3. Full Text The complement membrane attack complex triggers intracellular Ca2+ fluxes leading to NLRP3 inflammasome activation
by Triantafilou, Kathy and Hughes, Timothy R and Triantafilou, Martha and Morgan, B. Paul
Journal of cell science, ISSN 0021-9533, 07/2013, Volume 126, Issue 13, pp. 2903 - 2913
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Inflammasomes - metabolism | Respiratory Mucosa - drug effects | Calcium Channels - metabolism | Calcium - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Epithelial Cells - drug effects | Humans | Endoplasmic Reticulum - metabolism | Ryanodine Receptor Calcium Release Channel - metabolism | Membrane Potential, Mitochondrial - drug effects | Inflammasomes - drug effects | Inositol 1,4,5-Trisphosphate Receptors - metabolism | Respiratory Mucosa - immunology | Epithelial Cells - cytology | Calcium Channels - genetics | Carrier Proteins - immunology | Cell Membrane - drug effects | Complement Membrane Attack Complex - pharmacology | Interleukin-1beta - secretion | Calcium - agonists | Respiratory Mucosa - cytology | Interleukin-1beta - immunology | Mitochondria - metabolism | Mitochondria - drug effects | Carrier Proteins - genetics | Membrane Potential, Mitochondrial - immunology | Ryanodine Receptor Calcium Release Channel - genetics | Epithelial Cells - immunology | Cell Membrane - immunology | Primary Cell Culture | Inositol 1,4,5-Trisphosphate Receptors - genetics | Index Medicus
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4. Full Text Complement activation contributes to perioperative neurocognitive disorders in mice
by Xiong, Chao and Liu, Jinhu and Lin, Dandan and Zhang, Juxia and Terrando, Niccolò and Wu, Anshi
Journal of neuroinflammation, ISSN 1742-2094, 09/2018, Volume 15, Issue 1, pp. 254 - 254
Choroid plexus | Neuroinflammation | Complement | Hippocampus | Perioperative neurocognitive disorders | Neurosciences | Immunology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Microglia - metabolism | Neurocognitive Disorders - pathology | Blood-Brain Barrier - physiopathology | Choroid Plexus - metabolism | Male | Synapses - pathology | Complement System Proteins - metabolism | Tibial Fractures - surgery | Neurocognitive Disorders - drug therapy | Up-Regulation - physiology | Fear - psychology | Complement Factor I - pharmacology | Postoperative Complications - drug therapy | Neurocognitive Disorders - metabolism | Complement Activation - drug effects | Complement Activation - physiology | Disease Models, Animal | Astrocytes - drug effects | Neurocognitive Disorders - etiology | Synapses - drug effects | Postoperative Complications - physiopathology | Conditioning, Classical - drug effects | Microglia - drug effects | Mice, Inbred C57BL | Hippocampus - pathology | Conditioning, Classical - physiology | Cytophagocytosis - drug effects | Up-Regulation - drug effects | Hippocampus - metabolism | Animals | Signal Transduction - drug effects | Signal Transduction - physiology | Mice | Astrocytes - metabolism | Cellular signal transduction | Models | Rats as laboratory animals | Research | Cognition disorders | Complement receptors | Complement component C3a | Pathogenesis | Memory | Cognitive ability | Cognition | Cerebrospinal fluid | Bone surgery | Neuromodulation | Neurodegeneration | Rodents | Surgery | Bone marrow | Anesthesia | Pain perception | Astrocytes | Inflammation | Microglia | Neurological diseases | Analgesia | Complement activation | Fractures | Complement component C3 | Orthopedics | Regulation | Anesthesiology | Alzheimers disease | Index Medicus
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5. Full Text Uncoupling complement C1s activation from C1q binding in apoptotic cell phagocytosis and immunosuppressive capacity
by Colonna, Lucrezia and Parry, Graham C and Panicker, Sandip and Elkon, Keith B
Clinical immunology (Orlando, Fla.), ISSN 1521-6616, 2016, Volume 163, pp. 84 - 90
Allergy and Immunology | C1q | Complement | Systemic lupus erythematosus | C1s | Phagocytosis | Apoptotic cells | Life Sciences & Biomedicine | Immunology | Science & Technology | Interleukin-1beta - drug effects | Apoptosis - drug effects | Complement C1q - immunology | Humans | Complement C1s - metabolism | Complement Activation - immunology | Complement C1q - metabolism | Complement C3b - drug effects | Complement C3b - metabolism | Tumor Necrosis Factor-alpha - immunology | Complement C1s - antagonists & inhibitors | Complement Activation - drug effects | Macrophages - immunology | Cytokines - immunology | Immune Tolerance - drug effects | Phagocytosis - drug effects | Jurkat Cells | Antibodies, Monoclonal - pharmacology | Phagocytosis - immunology | Interleukin-1beta - immunology | Complement C1q - drug effects | Cytophagocytosis - drug effects | Apoptosis - immunology | Tumor Necrosis Factor-alpha - drug effects | Cytokines - drug effects | Interleukin-6 - immunology | Macrophages - drug effects | Cytophagocytosis - immunology | In Vitro Techniques | Complement C3b - immunology | Apoptosis | Index Medicus
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6. Full Text Complement activation-related pseudoallergy: A new class of drug-induced acute immune toxicity
by Szebeni, Janos
Toxicology (Amsterdam), ISSN 0300-483X, 2005, Volume 216, Issue 2, pp. 106 - 121
Allergy | Cancer chemotherapy | Cremophor EL | Anaphylactoid reaction | Taxol | Micelles | Radiocontrast agents | Anaphylatoxins | Pharmacology & Pharmacy | Toxicology | Life Sciences & Biomedicine | Science & Technology | Biological and medical sciences | Medical sciences | Complement Pathway, Classical - immunology | Animals | Anaphylaxis - chemically induced | Humans | Anaphylaxis - immunology | Nanostructures - adverse effects | Disease Models, Animal | Liposomes - adverse effects | Chemotherapy | Anthracyclines | Polyethylene glycol | Analysis | Immunoglobulin E | Lipids | Allergic reaction | Cancer | Index Medicus
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7. Full Text Association of COVID-19 inflammation with activation of the C5a–C5aR1 axis
by Carvelli, Julien and Demaria, Olivier and Vély, Frédéric and Batista, Luciana and Chouaki Benmansour, Nassima and Fares, Joanna and Carpentier, Sabrina and Thibult, Marie-Laure and Morel, Ariane and Remark, Romain and André, Pascale and Represa, Agnès and Piperoglou, Christelle and Assante  Miranda, Laura and Baron, William and Belaid, Nourhène and Caillet, Clarisse and Caraguel, Flavien and Carrette, Barbara and Carrette, Florent and Chanuc, Fabien and Courtois, Rachel and Fenis, Aurore and Giordano, Marilyn and Girard-Madoux, Mathilde and Giraudon-Paoli, Marc and Gourdin, Nicolas and Grondin, Gwendoline and Guillot, Franceline and Habif, Guillaume and Jaubert, Solène and Lopez, Julie and Le Van, Mélanie and Lovera, Naouel and Mansuy, Marine and Bonnet, Elodie and Sansaloni, Audrey and Reboul, Annick and Mitry, Emmanuel and Nekkar-Constant, Camille and Péri, Valentine and Ricaut, Paul and Simon, Léa and Vallier, Jean-Baptiste and Vétizou, Marie and Zerbib, Robert and Ugolini, Sophie and Etiennot, Marion and Galluso, Justine and Lyonnet, Luc and Forel, Jean-Marie and Papazian, Laurent and Velly, Lionel and André, Baptiste and Briantais, Antoine and Faucher, Benoit and Jean, Estelle and Seguier, Julie and Veit, Veronique and Harlé, Jean-Robert and Pastorino, Boris and Delteil, Clémence and Daniel, Laurent and Boudsocq, Jean-Paul and Clerc, Axelle and Delmond, Emmanuel and Vidal, Pierre-Olivier and Savini, Hélène and Coutard, Bruno and Cordier, Pierre Yves and Le Dault, Erwan and Guervilly, Christophe and Simeone, Pierre and Gainnier, Marc and Morel, Yannis and Ebbo, Mikael and Schleinitz, Nicolas and Vivier, Eric and Explore COVID-19 Marseille Immunop and Explore COVID-19 IPH Grp and Explore COVID-19 Marseille Immunopole group and Explore COVID-19 IPH group and the Explore COVID-19 IPH group and the Explore COVID-19 Marseille Immunopole group
Nature (London), ISSN 0028-0836, 12/2020, Volume 588, Issue 7836, pp. 146 - 150
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | COVID-19 - immunology | Inflammation - pathology | Humans | Bronchoalveolar Lavage Fluid - immunology | Respiratory Distress Syndrome - immunology | Complement C5a - biosynthesis | Inflammation - complications | Respiratory Distress Syndrome - prevention & control | Acute Lung Injury - prevention & control | COVID-19 - complications | Inflammation - drug therapy | Myeloid Cells - immunology | COVID-19 - blood | Receptor, Anaphylatoxin C5a - immunology | SARS-CoV-2 - drug effects | Female | Myeloid Cells - drug effects | Respiratory Distress Syndrome - drug therapy | SARS-CoV-2 - immunology | Acute Lung Injury - drug therapy | Cytokine Release Syndrome - drug therapy | Disease Models, Animal | Cytokine Release Syndrome - immunology | Lung - pathology | CD11b Antigen - immunology | Cytokine Release Syndrome - prevention & control | Mice, Inbred C57BL | Inflammation - immunology | Complement C5a - antagonists & inhibitors | Animals | SARS-CoV-2 - pathogenicity | Receptor, Anaphylatoxin C5a - blood | Lung - drug effects | COVID-19 - pathology | Acute Lung Injury - immunology | Mice | Bronchoalveolar Lavage Fluid - chemistry | CD11b Antigen - metabolism | Complement C5a - immunology | Myeloid Cells - pathology | Receptor, Anaphylatoxin C5a - antagonists & inhibitors | Lung - immunology | Myeloid cells | Immune response | Complement component C5a | Severe acute respiratory syndrome | Viral diseases | Organs | Bronchus | Leukocytes (neutrophilic) | Inflammation | Coronaviruses | Blood | Recruitment | Severe acute respiratory syndrome coronavirus 2 | COVID-19 | Smoke inhalation | Receptors | Cell activation | Respiratory distress syndrome | Lungs | Bronchoalveolar lavage | Respiratory diseases | Monoclonal antibodies | Alveoli | Immune system | Index Medicus
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8. Full Text Activation of complement by therapeutic liposomes and other lipid excipient-based therapeutic products: Prediction and prevention
by Szebeni, Janos and Muggia, Franco and Gabizon, Alberto and Barenholz, Yechezkel
Advanced drug delivery reviews, ISSN 0169-409X, 2011, Volume 63, Issue 12, pp. 1020 - 1030
Allergy | Cancer chemotherapy | Nanomedicines | Immune toxicity | Adverse drug effects | CARPA | Hypersensitivity reactions | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Drug Hypersensitivity - etiology | Antineoplastic Agents - immunology | Liposomes - administration & dosage | Humans | Complement System Proteins - immunology | Lipids - immunology | Lipids - administration & dosage | Antineoplastic Agents - administration & dosage | Complement Activation - immunology | Excipients - administration & dosage | Animals | Drug Hypersensitivity - immunology | Complement Activation - drug effects | Liposomes - immunology | Prevention | Drugs | Chemotherapy | Anthracyclines | Drug delivery systems | Sulfates | Allergic reaction | Health aspects | Cancer | Vehicles | Index Medicus | Animal models
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