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Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 3/2011, Volume 108, Issue 12, pp. 4974 - 4979
C-reactivé protein (CRP) is an important biomarker for inflammatory diseases. However, its role in inflammation beyond complementmediated pathogen clearance... 
Receptors | Cytokines | Neutrophils | Cell lines | Antibodies | Ligands | Mast cells | Macrophages | Phagocytosis | Binding sites | Serum amyloid P component | CD89 | Acute phase protein | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Antigens, CD - immunology | Humans | Tumor Necrosis Factor-alpha - genetics | Immunity, Innate - genetics | Antigens, CD - genetics | Complement C1q - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Extracellular Signal-Regulated MAP Kinases - immunology | Phosphorylation - genetics | Receptors, IgG - genetics | Tumor Necrosis Factor-alpha - immunology | Antigens, CD - chemistry | Phosphorylation - immunology | Binding Sites | Complement C1q - chemistry | Macrophages - immunology | Receptors, Fc - chemistry | Neutrophils - immunology | Phagocytosis - immunology | C-Reactive Protein - genetics | Inflammation | Complement C1q - genetics | Receptors, IgG - immunology | C-Reactive Protein - chemistry | Immunity, Innate - immunology | Receptors, Fc - immunology | Tumor Necrosis Factor-alpha - chemistry | Protein Binding | Receptors, Fc - genetics | Mutation | Biomarkers - chemistry | C-Reactive Protein - immunology | Genetic aspects | C-reactive protein | Biological markers | Immunoglobulin A | Health aspects | Biological Sciences | serum amyloid P component | acute phase protein
Journal Article
Journal Article
Neurobiology of disease, ISSN 0969-9961, 2007, Volume 26, Issue 3, pp. 497 - 511
...), and in the chronic epileptic phase in both experimental as well as human TLE. Previous rat gene expression analysis using microarrays indicated prominent activation of the classical complement pathway which peaked at 1... 
Neurology | Human | Epileptogenesis | Rat | Astrocytes | Neurons | Immunocytochemistry | Microarray | Hippocampus | PCR | Microglia | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Microglia - metabolism | Complement C3d - genetics | Complement C5b - immunology | Humans | Middle Aged | Status Epilepticus - immunology | Male | Status Epilepticus - genetics | RNA, Messenger - metabolism | Complement C1q - metabolism | Complement System Proteins - metabolism | Complement C3d - metabolism | Encephalitis - physiopathology | Epilepsy, Temporal Lobe - genetics | Gliosis - immunology | Disease Models, Animal | Gliosis - genetics | Complement C5b - genetics | Rats | Encephalitis - immunology | Complement C3c - genetics | Hippocampus - pathology | Complement C1q - genetics | Rats, Sprague-Dawley | Up-Regulation - immunology | Adolescent | Complement C3c - immunology | Hippocampus - physiopathology | Astrocytes - metabolism | Complement C3d - immunology | Complement C1q - immunology | Complement System Proteins - immunology | Epilepsy, Temporal Lobe - physiopathology | Epilepsy, Temporal Lobe - immunology | Gliosis - physiopathology | Complement C5b - metabolism | Microglia - immunology | Astrocytes - immunology | Complement C3c - metabolism | Adult | Complement System Proteins - genetics | Female | Hippocampus - immunology | Encephalitis - genetics | RNA, Messenger - genetics | Up-Regulation - genetics | Animals | Status Epilepticus - physiopathology | Aged | Epilepsy
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 08/2017, Volume 292, Issue 32, pp. 13345 - 13360
Spontaneous activation enables the complement system to respond very rapidly to diverse threats... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Complement C3d - genetics | Complement Factor I - chemistry | Complement Activation | Sheep, Domestic | Humans | Bacterial Proteins - chemistry | Erythrocytes - chemistry | Recombinant Fusion Proteins - metabolism | Complement C3d - metabolism | Streptococcus pneumoniae - metabolism | Surface Properties | Complement C3d - chemistry | Protein Interaction Domains and Motifs | Atypical Hemolytic Uremic Syndrome - genetics | Binding Sites | Peptide Fragments - genetics | Hemolysis | Peptide Fragments - metabolism | Complement C3 Convertase, Alternative Pathway - chemistry | Immobilized Proteins - chemistry | Complement C3 Convertase, Alternative Pathway - metabolism | Immobilized Proteins - genetics | Bacterial Proteins - genetics | Solubility | Complement Factor I - genetics | Recombinant Fusion Proteins - chemistry | Macular Degeneration - metabolism | Peptide Fragments - chemistry | Animals | Complement Factor H - metabolism | Macular Degeneration - genetics | Atypical Hemolytic Uremic Syndrome - metabolism | Immobilized Proteins - metabolism | Bacterial Proteins - metabolism | Mutation | Complement Factor H - genetics | Complement Factor I - metabolism | Complement C3 Convertase, Alternative Pathway - genetics | Amino Acid Substitution | Complement Factor H - chemistry | Index Medicus | kidney | Immunology | Streptococcus | innate immunity | complement system | recombinant protein expression | surface plasmon resonance (SPR) | multiple domain | mutagenesis | CCP module | erythrocyte
Journal Article
The Journal of Immunology, ISSN 0022-1767, 10/2008, Volume 181, Issue 8, pp. 5537 - 5544
The periodontal pathogen Porphyromonas gingivalis is highly resistant to the bactericidal activity of human complement, which is present in the gingival... 
Life Sciences & Biomedicine | Immunology | Science & Technology | Porphyromonas gingivalis - enzymology | Protein Binding - genetics | Virulence Factors - genetics | Porphyromonas gingivalis - pathogenicity | Adhesins, Bacterial - genetics | Humans | Bacteroidaceae Infections - genetics | Virulence Factors - immunology | Complement Activation - immunology | Blood Bactericidal Activity - genetics | Adhesins, Bacterial - metabolism | Complement C9 - immunology | Cysteine Endopeptidases - metabolism | Complement C4b-Binding Protein - genetics | Complement C4b-Binding Protein - immunology | Porphyromonas gingivalis - immunology | Bacteroidaceae Infections - enzymology | Complement C9 - metabolism | Complement C4b-Binding Protein - metabolism | Blood Bactericidal Activity - immunology | Protein Structure, Tertiary - genetics | Protein Binding - immunology | Protein Structure, Secondary - genetics | Complement Activation - genetics | Bacteroidaceae Infections - immunology | Cysteine Endopeptidases - genetics | Ligands | Virulence Factors - metabolism | Complement C9 - genetics | Mutation | Adhesins, Bacterial - immunology | Cysteine Endopeptidases - immunology | Protein Binding: genetics | Adhesins | Medical and Health Sciences | Protein Binding: immunology | Complement C9: immunology | Medicin och hälsovetenskap | Complement C4b-Binding Protein: metabolism | Immunologi inom det medicinska området | Complement C4b-Binding Protein: immunology | Bacteroidaceae Infections: genetics | Porphyromonas gingivalis: pathogenicity | Cysteine Endopeptidases: metabolism | Virulence Factors: genetics | Cysteine Endopeptidases: genetics | Complement Activation: genetics | Porphyromonas gingivalis: immunology | Virulence Factors: metabolism | Immunology in the medical area | Basic Medicine | Bacterial: genetics | Bacterial: immunology | Bacteroidaceae Infections: enzymology | Protein Structure | Complement Activation: immunology | Secondary: genetics | Blood Bactericidal Activity: immunology | Complement C9: metabolism | Complement C4b-Binding Protein: genetics | Porphyromonas gingivalis: enzymology | Complement C9: genetics | Bacterial: metabolism | Medicinska och farmaceutiska grundvetenskaper | Virulence Factors: immunology | Cysteine Endopeptidases: immunology | Tertiary: genetics | Blood Bactericidal Activity: genetics | Bacteroidaceae Infections: immunology
Journal Article
Immunology and Cell Biology, ISSN 0818-9641, 03/2009, Volume 87, Issue 3, pp. 203 - 208
Journal Article