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Journal of Experimental Medicine, ISSN 0022-1007, 07/2017, Volume 214, Issue 7, pp. 2103 - 2119
Neutrophils play a crucial role in host defense. However, neutrophil activation is also linked to autoimmune diseases such as systemic lupus erythematosus (SLE... 
PLASMACYTOID DENDRITIC CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | NADPH OXIDASE | SYSTEMIC-LUPUS-ERYTHEMATOSUS | PROPROTEIN CONVERTASES | C1Q DEFICIENCY | INFLAMMATION | FC-GAMMA-RIIIB | TOLL-LIKE RECEPTORS | ALPHA | IMMUNOLOGY | EXTRACELLULAR TRAPS | Extracellular Traps - metabolism | Dendritic Cells - immunology | Humans | Complement C5a - metabolism | Lupus Erythematosus, Systemic - metabolism | Monocytes - metabolism | Monocytes - immunology | Complement Activation - immunology | Receptors, IgG - metabolism | Toll-Like Receptor 8 - immunology | Extracellular Traps - immunology | Flow Cytometry | Toll-Like Receptor 7 - immunology | Lupus Erythematosus, Systemic - immunology | Neutrophils - metabolism | Dendritic Cells - metabolism | Toll-Like Receptor 7 - metabolism | Cells, Cultured | Neutrophils - immunology | Phagocytosis - immunology | Receptors, IgG - immunology | Blotting, Western | Complement C5a - immunology | Antigen-Antibody Complex - immunology | Toll-Like Receptor 8 - metabolism | Shedding | Complement component C5a | Immune clearance | Activation | Proteins | Cell activation | Systemic lupus erythematosus | Proteolysis | Toll-like receptors | Furin | Cleavage | Antigen-antibody complexes | Immune system | TLR7 protein | Dendritic cells | Neutrophils | Leukocytes (neutrophilic) | Inflammation | Nucleic acids | Signaling | Monocytes | Chronic conditions | Complement activation | Information processing | Autoimmune diseases | Phagocytosis | 311 | 300 | Extracellular Traps/immunology | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Phagocytosis/immunology | Immunologi inom det medicinska området | Lupus Erythematosus, Systemic/immunology | Toll-Like Receptor 7/immunology | Neutrophils/immunology | Complement Activation/immunology | Monocytes/immunology | Antigen-Antibody Complex/immunology | Receptors, IgG/immunology | Medicinska och farmaceutiska grundvetenskaper | Dendritic Cells/immunology | Complement C5a/immunology | Toll-Like Receptor 8/immunology | Immunology in the medical area
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2014, Volume 192, Issue 6, pp. 2837 - 2845
...) have long been recognized as a hallmark of atherosclerotic lesions. CC appear early in the atheroma development and trigger inflammation by NLRP3 inflammasome activation... 
ATHEROSCLEROTIC LESIONS | TUMOR-NECROSIS-FACTOR | C5A RECEPTOR | MEMBRANE ATTACK | STERILE INFLAMMATION | EPITHELIAL-CELLS | CARDIOVASCULAR EVENTS | INTERLEUKIN-1-BETA | OXIDATIVE BURST | ANAPHYLATOXINS | IMMUNOLOGY | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Complement C1q - immunology | Humans | Complement Pathway, Classical - drug effects | Complement C5a - metabolism | Complement System Proteins - immunology | Caspase 1 - metabolism | Monocytes - metabolism | Caspase 1 - immunology | Macrophage-1 Antigen - immunology | Monocytes - immunology | Interleukin-1beta - genetics | Complement Activation - immunology | Complement C1q - metabolism | Complement System Proteins - metabolism | Inflammasomes - drug effects | Reactive Oxygen Species - immunology | Complement C5 - immunology | Tumor Necrosis Factor-alpha - immunology | Macrophage-1 Antigen - metabolism | Complement Activation - drug effects | Cytokines - immunology | Interleukin-1beta - secretion | Complement C5 - metabolism | Tumor Necrosis Factor-alpha - secretion | Cells, Cultured | Cytokines - secretion | Phagocytosis - immunology | Interleukin-1beta - immunology | Cholesterol - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Monocytes - drug effects | Complement Pathway, Classical - immunology | Microscopy, Confocal | Complement Pathway, Alternative - drug effects | Complement Pathway, Alternative - immunology | Inflammasomes - immunology | Cholesterol - pharmacology | Complement C5a - immunology
Journal Article
Trends in Immunology, ISSN 1471-4906, 2009, Volume 30, Issue 2, pp. 83 - 90
... inhibitors C4b-binding protein and factor H. This contrasts to the interaction between C1q and immune complexes, in which case no inhibitors bind, resulting in full complement activation... 
Allergy and Immunology | C-REACTIVE PROTEIN | APOPTOTIC CELL RECOGNITION | C4B-BINDING PROTEIN | AMYLOID-P COMPONENT | SYSTEMIC-LUPUS-ERYTHEMATOSUS | NECROTIC CELLS | MACULAR DEGENERATION | EXTRACELLULAR-MATRIX | IMMUNOLOGY | FACTOR-H BINDS | GLOBULAR HEAD | Alzheimer Disease - etiology | Macular Degeneration - immunology | Humans | Complement System Proteins - immunology | Intracellular Signaling Peptides and Proteins - immunology | Intracellular Signaling Peptides and Proteins - metabolism | Complement Activation - immunology | Complement C4b-Binding Protein | Complement System Proteins - metabolism | Prions - immunology | Muscle Proteins - metabolism | Alzheimer Disease - immunology | Histocompatibility Antigens - metabolism | Macular Degeneration - etiology | Histocompatibility Antigens - immunology | Muscle Proteins - immunology | Prions - metabolism | LIM Domain Proteins | Prion Diseases - immunology | Arthritis, Rheumatoid - etiology | Immunity, Innate | Animals | Prion Diseases - etiology | Protein Binding | Amyloid beta-Peptides - immunology | Ligands | Arthritis, Rheumatoid - immunology | Macular degeneration | Immune complexes | Immunoproteins | Protein binding | Proteins | Lupus | Aging | Alzheimers disease | Autoimmune diseases | Basic Medicine | Other Basic Medicine | Andra medicinska och farmaceutiska grundvetenskaper | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2017, Volume 114, Issue 45, pp. E9618 - E9625
Journal Article
Blood, ISSN 1528-0020, 2017, Volume 129, Issue 16, pp. 2291 - 2302
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2009, Volume 123, Issue 2, pp. 342 - 351
.... Resistance to digestion and dendritic cell activation by the major peanut allergen Ara h 1 are reported to contribute to its allergenicity... 
Allergy and Immunology | C3a | Peanut | macrophages | histamine | shock | anaphylaxis | platelet-activating factor | basophils | complement | mast cells | MAST-CELLS | IMMUNOLOGY | mart cells | CUTTING EDGE | EFFECTOR PHASE | TARGETED DISRUPTION | ALLERGY | ACQUIRED-IMMUNITY | CELL WALL COMPONENTS | IN-VIVO | ALLERGIC-ASTHMA | IGE-BINDING EPITOPES | BETA-BLOCKERS | Humans | Platelet Activating Factor - immunology | Histamine - immunology | Complement Activation - immunology | Complement C3a - genetics | Immunoglobulin E - blood | Receptors, IgG - metabolism | Adrenergic beta-Antagonists - pharmacology | Histamine - metabolism | Interleukin-4 - pharmacology | Propranolol - pharmacology | Receptors, IgG - genetics | Allergens - immunology | Basophils - immunology | Mast Cells - metabolism | Anaphylaxis - immunology | Toll-Like Receptors - metabolism | Complement C3a - metabolism | Macrophages - immunology | Mast Cells - immunology | Chymases - blood | Anaphylaxis - etiology | Toll-Like Receptors - immunology | Arachis - metabolism | Peanut Hypersensitivity - immunology | Receptors, IgG - immunology | Cell Degranulation - immunology | Platelet Activating Factor - metabolism | Macrophages - metabolism | Animals | Peanut Hypersensitivity - metabolism | Arachis - immunology | Mice | Mice, Inbred BALB C | Complement C3a - immunology | Basophils - metabolism | Allergens | Medical colleges | Food allergy | Anaphylaxis | Dendritic cells | Proteases | Emergency medicine | Medical law | Platelet activating factor | Human subjects | Food allergies | Plasma | Peanuts | Rodents | Allergies | Binding sites | PAF | peanut
Journal Article
American journal of respiratory and critical care medicine, ISSN 1535-4970, 2020, Volume 201, Issue 2, pp. 224 - 239
Journal Article
PLoS medicine, ISSN 1549-1676, 2018, Volume 15, Issue 5, p. e1002572
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, p. e99386
.... Complement activation on platelets is seen in SLE patients, especially in patients with aPL antibodies, and has been related to venous thrombosis and stroke... 
Complement C4b - immunology | Blood Platelets - immunology | Humans | Middle Aged | Platelet Activation - immunology | Risk Factors | Antibodies, Antiphospholipid - immunology | Thrombosis - immunology | Cardiolipins - immunology | Male | Lupus Erythematosus, Systemic - blood | Complement Activation - immunology | Young Adult | Peptide Fragments - immunology | Adolescent | Lupus Erythematosus, Systemic - immunology | Aged, 80 and over | Adult | Female | Aged | Thrombosis - blood | Stroke (Disease) | Lupus | Rheumatoid factor | Systemic lupus erythematosus | Phospholipids | Antiphospholipid antibodies | Thrombosis | Blood clot | Shipping industry | Heart attacks | Laboratories | Antibodies | Cardiovascular disease | Activation | Complement | Arthritis | Kinases | Risk factors | Inflammatory diseases | Immunology | Blood platelets | Thromboembolism | Deposition | Stroke | Immunoglobulins | Mortality | Rheumatology | Health risks | Rheumatic diseases | Risk analysis | Patients | Studies | Complement component C1q | Chronic conditions | Complement activation | Hospitals | Rheumatoid arthritis | Systemic sclerosis | Autoimmune diseases | Cardiovascular diseases | Platelets | Classical pathway | Rheumatology and Autoimmunity | Clinical Medicine | Basic Medicine | Medical and Health Sciences | Microbiology in the medical area | Medicin och hälsovetenskap | Reumatologi och inflammation | Medicinska och farmaceutiska grundvetenskaper | Immunologi inom det medicinska området | Klinisk medicin | Mikrobiologi inom det medicinska området | Immunology in the medical area
Journal Article