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Transplantation, ISSN 0041-1337, 2014, Volume 98, Issue 4, pp. 387 - 393
Journal Article
Lupus, ISSN 0961-2033, 11/2012, Volume 21, Issue 13, pp. 1423 - 1432
Objective: Patients with systemic lupus erythematosus (SLE) have an increased risk of developing vascular diseases (VD) such as myocardial infarction, stroke... 
cardiovascular disease | platelet | Systemic lupus erythematosus | complement | venous thrombosis | COMPLEMENT ACTIVATION | COMPLEXES | ANTIPHOSPHOLIPID ANTIBODIES | RECEPTOR | RHEUMATOLOGY | DAMAGE | DISEASE | INDEX | BINDING | ASSOCIATION | Lupus Coagulation Inhibitor - blood | Up-Regulation | Lupus Erythematosus, Systemic - complications | Blood Platelets - immunology | Humans | Middle Aged | Venous Thrombosis - blood | Male | Lupus Erythematosus, Systemic - blood | Case-Control Studies | Complement C4b - analysis | Complement C1q - analysis | Young Adult | Flow Cytometry | Venous Thrombosis - immunology | Lupus Erythematosus, Systemic - immunology | Aged, 80 and over | Adult | Female | Complement Pathway, Classical | Complement C3d - analysis | Risk Factors | Biomarkers - blood | Venous Thrombosis - etiology | Peptide Fragments - analysis | Platelet Activation | Aged | Index Medicus | Myocardial infarction | complement component C4 | Anticoagulants | Flow cytometry | Stroke | Centrifugation | Data processing | Thrombosis | Blood | Risk factors | antiphospholipid antibodies | Complement component C1q | Complement activation | Vascular diseases | Complement component C3 | Platelets | Sexually-transmitted diseases | Rheumatology and Autoimmunity | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Reumatologi och inflammation
Journal Article
Journal Article
Human Immunology, ISSN 0198-8859, 2011, Volume 72, Issue 10, pp. 849 - 858
Journal Article
Methods in Molecular Biology, ISSN 1064-3745, 2019, Volume 1901, pp. 183 - 189
Anti-C1q autoantibodies may be found in many conditions, most commonly in systemic lupus erythematosus (SLE) and hypocomplementemic urticarial vasculitis... 
Western blot | Hypocomplementemic urticarial vasculitis syndrome | Systemic lupus erythematosus | Anti-C1q autoantibodies | C1q protein chains B and C | Protein Binding | Autoantibodies - analysis | Blotting, Western - methods | Complement C1q - isolation & purification | Complement C1q - immunology | Humans
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2017, Volume 12, Issue 2, pp. e0172271 - e0172271
Objectives The C1q complement/TNF-related protein (CTRP) superfamily, which includes the adipokine adiponectin, has been shown in animal models to have... 
WOMEN | OBESITY | CHOLESTEROL | ADIPONECTIN | MULTIDISCIPLINARY SCIENCES | ADIPOKINE ADIPOLIN/CTRP12 | EX-VIVO | INSULIN SENSITIVITY | ASSOCIATION | METFORMIN INCREASES | ADIPOSE-TISSUE | Glucose Tolerance Test | Prognosis | Cross-Sectional Studies | Sweetening Agents - pharmacology | Humans | Middle Aged | Insulin Resistance | Male | Glucose - pharmacology | Glycoproteins - blood | Proteins - analysis | Biomarkers - blood | Diabetes Mellitus, Type 2 - metabolism | Case-Control Studies | Adipokines - blood | Adiponectin - blood | Complement C1q - analysis | Diabetes Mellitus, Type 2 - blood | Animals | Adult | Female | Diabetes Mellitus, Type 2 - drug therapy | Type 2 diabetes | Obesity | Complications and side effects | Fat cells | Research | Diagnosis | Biological markers | Risk factors | Lipoproteins (low density) | Animal models | C-reactive protein | High density | Glucose | Lipase | Blood | Adiponectin | Residential density | Rodents | Blood pressure | Bioindicators | Mathematical models | Diabetes mellitus (non-insulin dependent) | Lipoproteins (high density) | Enzyme-linked immunosorbent assay | Diabetes mellitus | Triglycerides | Pharmacology | Insulin | Cholesterol | Glucose tolerance | Complement component C1q | Body mass | Tumor necrosis factor | Leptin | Predictions | Biomarkers | Insulin resistance | In vivo methods and tests | Index Medicus
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