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Journal of Immunology, ISSN 0022-1767, 03/2014, Volume 192, Issue 6, pp. 2837 - 2845
Inflammation is associated with development of atherosclerosis, and cholesterol crystals (CC) have long been recognized as a hallmark of atherosclerotic... 
ATHEROSCLEROTIC LESIONS | TUMOR-NECROSIS-FACTOR | C5A RECEPTOR | MEMBRANE ATTACK | STERILE INFLAMMATION | EPITHELIAL-CELLS | CARDIOVASCULAR EVENTS | INTERLEUKIN-1-BETA | OXIDATIVE BURST | ANAPHYLATOXINS | IMMUNOLOGY | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Complement C1q - immunology | Humans | Complement Pathway, Classical - drug effects | Complement C5a - metabolism | Complement System Proteins - immunology | Caspase 1 - metabolism | Monocytes - metabolism | Caspase 1 - immunology | Macrophage-1 Antigen - immunology | Monocytes - immunology | Interleukin-1beta - genetics | Complement Activation - immunology | Complement C1q - metabolism | Complement System Proteins - metabolism | Inflammasomes - drug effects | Reactive Oxygen Species - immunology | Complement C5 - immunology | Tumor Necrosis Factor-alpha - immunology | Macrophage-1 Antigen - metabolism | Complement Activation - drug effects | Cytokines - immunology | Interleukin-1beta - secretion | Complement C5 - metabolism | Tumor Necrosis Factor-alpha - secretion | Cells, Cultured | Cytokines - secretion | Phagocytosis - immunology | Interleukin-1beta - immunology | Cholesterol - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Monocytes - drug effects | Complement Pathway, Classical - immunology | Microscopy, Confocal | Complement Pathway, Alternative - drug effects | Complement Pathway, Alternative - immunology | Inflammasomes - immunology | Cholesterol - pharmacology | Complement C5a - immunology | Index Medicus | Abridged Index Medicus
Journal Article
Molecular Immunology, ISSN 0161-5890, 09/2017, Volume 89, pp. 111 - 114
The complement system has obtained renewed clinical focus due to increasing number of patients treated with eculizumab, a monoclonal antibody inhibiting... 
Eculizumab | Neoepitope | Complement | C5a | ACTIVATION | OXYGEN RADICALS | BIOCHEMISTRY & MOLECULAR BIOLOGY | 5TH | MONOCLONAL-ANTIBODY | C5B-LIKE C5 | IMMUNOLOGY | CLEAVAGE | INHIBITOR ECULIZUMAB | GENERATION | Epitopes - metabolism | Immunoglobulin G - blood | Outcome Assessment (Health Care) | Complement C5b - immunology | Humans | Complement C5a - metabolism | Atypical Hemolytic Uremic Syndrome - immunology | Complement C5b - metabolism | Complement Activation - immunology | Epitopes - immunology | Hemoglobinuria, Paroxysmal - immunology | Immunoglobulin G - immunology | Complement C5 - immunology | Atypical Hemolytic Uremic Syndrome - drug therapy | Hemoglobinuria, Paroxysmal - blood | Complement Activation - drug effects | Complement Inactivating Agents - metabolism | Antibodies, Monoclonal, Humanized - therapeutic use | Complement C5 - metabolism | Enzyme-Linked Immunosorbent Assay | Antibodies, Monoclonal, Humanized - metabolism | Protein Binding - immunology | Complement Inactivating Agents - therapeutic use | Hemoglobinuria, Paroxysmal - drug therapy | Complement C5a - immunology | Chromatography, Gel | Atypical Hemolytic Uremic Syndrome - blood | Hydrogen-Ion Concentration | Proteins | Monoclonal antibodies | Biological products | Drug approval | Analysis | Index Medicus | VDP | Medisinske Fag: 700 | Klinisk medisinske fag: 750 | Hematologi: 775 | Biological Sciences | Immunology | Naturvetenskap | Immunologi | Biologiska vetenskaper | Natural Sciences
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2018, Volume 13, Issue 12, pp. e0209509 - e0209509
Modulating the complement system is a promising strategy in drug discovery for disorders with uncontrolled complement activation. Although some of these... 
SERUM HALF-LIFE | IMMUNOGENICITY | PHARMACOKINETICS | COMPLEMENT INHIBITOR ECULIZUMAB | MULTIDISCIPLINARY SCIENCES | IN-VIVO | HEMOPHILIA-A | MONOCLONAL-ANTIBODY | EMICIZUMAB PROPHYLAXIS | DEPENDENT ANTIGEN-BINDING | THERAPEUTIC ANTIBODY | Endosomes - immunology | Protein Engineering - methods | Humans | Macaca fascicularis | Antibodies, Monoclonal - therapeutic use | Drug Discovery - methods | Complement Activation - immunology | Complement C5 - isolation & purification | Recombinant Proteins - isolation & purification | Time Factors | Complement C5 - immunology | Computer Simulation | Antibodies, Monoclonal - immunology | Complement Activation - drug effects | Histocompatibility Antigens Class I - immunology | Immune System Diseases - immunology | Antibody Affinity | Immune System Diseases - drug therapy | Mice, Transgenic | Histocompatibility Antigens Class I - genetics | Antibodies, Monoclonal - genetics | Animals | Receptors, Fc - immunology | Mutagenesis | Recombinant Proteins - immunology | Receptors, Fc - genetics | Mice | Complement C5 - antagonists & inhibitors | Hydrogen-Ion Concentration | Viral antibodies | Protein engineering | Usage | Physiological aspects | Antibodies | Genetic aspects | Research | Complement (Immunology) | Neonates | Plasma | Intravenous administration | Immune clearance | Disorders | Complement | Monkeys | Dosage | pH effects | Accumulation | Engineering | Salvage | Hemophilia | Recycling | Drug dosages | Binding | Antigens | Surface charge | Immunoglobulins | Fc receptors | Complement activation | Complement component C5 | Mutation | Endosomes | Neutralization | Pharmaceuticals | Polymorphism | Index Medicus | pH
Journal Article
Kidney International, ISSN 0085-2538, 06/2017, Volume 91, Issue 6, pp. 1386 - 1397
C3 glomerulopathy is a potentially life-threatening disease of the kidney caused by dysregulated alternative pathway complement activation. The specific... 
properdin | C5a receptor | C3 glomerulopathy | complement factor H | C5 | ECULIZUMAB | ACTIVATION | ABNORMALITIES | COMPLEMENT ALTERNATIVE PATHWAY | FOLLOW-UP | NEPHRITIC FACTOR | IN-VIVO | UROLOGY & NEPHROLOGY | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | DENSE-DEPOSIT DISEASE | Receptor, Anaphylatoxin C5a - genetics | Kidney - pathology | Glomerulonephritis - prevention & control | Properdin - deficiency | Properdin - genetics | Kidney - immunology | Complement Factor H - deficiency | Renal Insufficiency - prevention & control | Mice, 129 Strain | Glomerulonephritis - metabolism | Proteinuria - prevention & control | Kidney - metabolism | Time Factors | Complement C5 - immunology | Renal Insufficiency - immunology | Disease Models, Animal | Renal Insufficiency - genetics | Genetic Predisposition to Disease | Complement C5 - metabolism | Kidney - drug effects | Proteinuria - immunology | Complement C3 - metabolism | Antibodies, Monoclonal - pharmacology | Mice, Inbred C57BL | Glomerulonephritis - genetics | Proteinuria - metabolism | Glomerulonephritis - immunology | Renal Insufficiency - metabolism | Complement C3 - immunology | Mice, Knockout | Phenotype | Animals | Signal Transduction - drug effects | Complement Inactivating Agents - pharmacology | Complement Pathway, Alternative - drug effects | Receptor, Anaphylatoxin C5a - deficiency | Complement Factor H - genetics | Complement C5 - antagonists & inhibitors | Index Medicus | complement | factor H
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 04/2016, Volume 184, Issue 1, pp. 118 - 125
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2016, Volume 196, Issue 5, pp. 2293 - 2299
Endothelial cells (EC) play a central role in inflammation. E-selectin and ICAM-1 expression are essential for leukocyte recruitment and are good markers of EC... 
ADHESION MOLECULE EXPRESSION | COMBINED INHIBITION | COMPLEMENT ACTIVATION | NEISSERIA-MENINGITIDIS | OXIDATIVE BURST | E-SELECTIN | LIPOPOLYSACCHARIDE | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | INFLAMMATORY RESPONSE | GENETIC DEFICIENCIES | Human Umbilical Vein Endothelial Cells | Cytokines - metabolism | Endothelial Cells - metabolism | Lymphocyte Antigen 96 - metabolism | Antibodies, Monoclonal - pharmacology | Humans | Cells, Cultured | Tumor Necrosis Factors - metabolism | Escherichia coli - immunology | Complement Activation - immunology | Lipopolysaccharide Receptors - metabolism | Lymphocyte Antigen 96 - antagonists & inhibitors | Interleukin-1beta - metabolism | Complement C5 - immunology | Biomarkers | Staphylococcus aureus - immunology | Complement C5 - antagonists & inhibitors | Endothelial Cells - drug effects | Index Medicus | Abridged Index Medicus | Innate Immunity and Inflammation | necrosis-factor-alpha | Basic Medicine | Immunologi | Medical and Health Sciences | Medicin och hälsovetenskap | inflammatory response | Immunologi inom det medicinska området | e-selectin | complement activation | oxidative burst | combined inhibition | Immunology | Medicinska och farmaceutiska grundvetenskaper | lipopolysaccharide | adhesion molecule expression | genetic deficiencies | Immunology in the medical area | neisseria-meningitidis
Journal Article