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American journal of transplantation, ISSN 1600-6135, 03/2013, Volume 13, Issue 3, pp. 663 - 675
.... We retrospectively studied 57 aHUS patients who had received 71 renal transplants. A mutation in complement gene was identified in 39 (68%), in factor H (CFH), factor I (CFI), membrane cofactor‐protein (MCP), C3 and factor B (CFB). At 5 years, death... 
Acute rejection | endothelial cell damage | mutations and polymorphisms in complement alternative pathway | thrombotic microangiopathy | atypical hemolytic and uremic syndrome | renal transplantation | Transplantation | Life Sciences & Biomedicine | Surgery | Science & Technology | Recurrence | Genetic Testing | Prognosis | Humans | Middle Aged | Male | Young Adult | Hemolytic-Uremic Syndrome - genetics | Complement Factor B - genetics | Adult | Complement System Proteins - genetics | Female | Hemolytic-Uremic Syndrome - therapy | Retrospective Studies | Complement C3 - genetics | Biomarkers - metabolism | Risk Factors | Biomarkers - analysis | Kidney Transplantation | Mutation - genetics | Fibrinogen - genetics | Graft Survival - genetics | Adolescent | Membrane Cofactor Protein - genetics | Aged | Complement Factor H - genetics | Graft Rejection - genetics | Atypical Hemolytic Uremic Syndrome | Medical research | Kidneys | Genes | Organ transplant recipients | Medicine, Experimental | Genetic research | Clinical trials | Mutation | Transplants & implants | Drug therapy | Index Medicus | Biological Markers | Complement C3 | Life Sciences | Immunology | Fibrinogen | Hemolytic-Uremic Syndrome | Antigens, CD46 | Graft Survival | Complement Factor H | Complement System Proteins | Complement Factor B | Graft Rejection
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 06/2004, Volume 24, Issue 6, pp. 1062 - 1067
OBJECTIVE—To investigate the effect of complement deficiency on atherogenesis and lipidemia, we used mice deficient in the third complement component (C3−/−) or factor B (FB... 
Complement | Factor B | Hyperlipidemia | Atherosclerosis | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Apolipoproteins E - deficiency | Aortic Diseases - blood | Male | Arteriosclerosis - genetics | Complement C3 - deficiency | Complement Factor B - genetics | Receptors, LDL - deficiency | Cholesterol, LDL - blood | Female | Complement Pathway, Alternative - genetics | Arteriosclerosis - blood | Complement C3 - genetics | Complement Factor B - deficiency | Aortic Diseases - pathology | Receptors, LDL - genetics | Complement Factor B - physiology | Genetic Predisposition to Disease | Hyperlipoproteinemia Type II - blood | Hyperlipoproteinemia Type IV - genetics | Mice, Knockout | Hyperlipoproteinemia Type IV - blood | Aortic Diseases - genetics | Animals | Lipoproteins, VLDL - blood | Apolipoproteins E - genetics | Triglycerides - blood | Mice | Arteriosclerosis - pathology | Complement Pathway, Classical - genetics | Complement C3 - physiology | Crosses, Genetic | Hyperlipoproteinemia Type II - genetics | Index Medicus | Research Support | Comparative Study | Genetic | Apolipoproteins E/deficiency/genetics | LDL Cholesterol/blood | Alternative/genetics | MEDICIN OCH HÄLSOVETENSKAP | Arteriosclerosis/blood/ genetics/pathology | Crosses | Receptors | Hyperlipoproteinemia Type IV/blood/genetics | VLDL/blood | MEDICAL AND HEALTH SCIENCES | Aortic Diseases/blood/ genetics/pathology | Complement Pathway | Complement C3/ deficiency/genetics/physiology | Knockout | Classical/genetics | Lipoproteins | Hypercholesterolemia | LDL/deficiency/genetics | Complement Factor B/ deficiency/genetics/physiology | Familial/blood/genetics | Triglycerides/blood | Non-U.S. Gov't
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 12/2006, Volume 104, Issue 1, pp. 240 - 245
Journal Article
The American journal of clinical nutrition, ISSN 1938-3207, 04/2016, Volume 103, Issue 4, pp. 1135 - 1144
...) is available, and prevention could be of great value. Objective: We investigated associations between dietary folate, B vitamins, and progression to GA and whether these associations might be modified by genetic susceptibility. Design... 
Macular degeneration | Geographic atrophy | Genetics | B vitamins | Folate | Life Sciences & Biomedicine | Nutrition & Dietetics | Science & Technology | Prospective Studies | Geographic Atrophy - genetics | Humans | Middle Aged | Complement C2 - genetics | Male | Folic Acid - administration & dosage | Vitamin B Complex - administration & dosage | Complement Factor B - genetics | Complement Factor B - metabolism | Serine Endopeptidases - genetics | Female | Complement C3 - genetics | Body Mass Index | Genetic Predisposition to Disease | Complement C3 - metabolism | High-Temperature Requirement A Serine Peptidase 1 | Complement C2 - metabolism | Disease Progression | Proteins - genetics | Complement Factor H - metabolism | Proteins - metabolism | Macular Degeneration - genetics | Collagen Type VIII - metabolism | Thiamine - administration & dosage | Aged | Serine Endopeptidases - metabolism | Complement Factor H - genetics | Collagen Type VIII - genetics | Riboflavin - administration & dosage | Collagen | Analysis | Physiological aspects | Development and progression | Genetic aspects | Disease susceptibility | Health aspects | Folic acid | Genotype & phenotype | Demographics | Nutrition | Peptides | Correlation analysis | Vitamin B | Index Medicus | Abridged Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 3, pp. e93459 - e93459
Journal Article
The Journal of neuroscience, ISSN 0270-6474, 08/2012, Volume 32, Issue 34, pp. 11511 - 11523
.... We have investigated the function of the proinflammatory acting inhibitor of kappa B kinase (IKK)/nuclear factor kappa B (NF-kappa B... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Glial Fibrillary Acidic Protein - genetics | Cell Adhesion Molecules - genetics | Lateral Ventricles - pathology | Age Factors | Humans | Brain - enzymology | Hydrocephalus - pathology | Astrocytes - enzymology | Brain - growth & development | I-kappa B Proteins - metabolism | Cerebral Cortex - cytology | Complement System Proteins - metabolism | Doxycycline - administration & dosage | I-kappa B Kinase - metabolism | Microarray Analysis | Statistics, Nonparametric | Glioma, Subependymal - etiology | Complement System Proteins - genetics | Hydrocephalus - enzymology | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Gene Expression Regulation, Developmental - physiology | Animals, Newborn | Astrocytes - drug effects | Glioma, Subependymal - pathology | Microscopy, Electron, Scanning | NF-KappaB Inhibitor alpha | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation, Developmental - drug effects | Protein-Serine-Threonine Kinases - genetics | Mice, Transgenic | Hydrocephalus - complications | Signal Transduction - genetics | I-kappa B Kinase - genetics | Lateral Ventricles - growth & development | Enzyme Activation - drug effects | Cell Adhesion Molecules - metabolism | Chemokines - genetics | Lateral Ventricles - ultrastructure | Animals | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Brain - pathology | Chemokines - metabolism | Mice | Enzyme Activation - genetics | Encephalitis - etiology | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, pp. e54339 - e54339
Background: Mitochondrial dysfunction is associated with the development and progression of age-related macular degeneration (AMD). Recent studies using... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Haplotypes | Retinal Pigment Epithelium - metabolism | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Humans | Adenosine Triphosphate - biosynthesis | DNA, Mitochondrial - genetics | Mitochondria - genetics | Myosins - metabolism | Epithelial Cells - cytology | Complement C3 - genetics | Extracellular Matrix Proteins - metabolism | Hybrid Cells - metabolism | Gene Expression | DNA, Mitochondrial - metabolism | Complement C3 - metabolism | Reactive Nitrogen Species - metabolism | Extracellular Matrix Proteins - genetics | Lactic Acid - metabolism | Cells, Cultured | Mitochondria - metabolism | Signal Transduction - genetics | Myosins - genetics | Macular Degeneration - metabolism | Complement Factor H - metabolism | Hybrid Cells - pathology | Macular Degeneration - genetics | Models, Biological | Complement Factor H - genetics | Retinal Pigment Epithelium - cytology | Macular Degeneration - pathology | Macular degeneration | Cellular proteins | Care and treatment | Genetic variation | Development and progression | Genetic aspects | Mitochondrial DNA | Health aspects | Glaucoma | Diabetic retinopathy | Cybrids | Disease | Physicians | Epithelial cells | Genes | Retina | Population studies | Arthritis | Proteins | Signal transduction | Mitochondria | Pathways | Energy | Aging | Age | Deoxyribonucleic acid--DNA | Oxygen | Cell division | Metabolism | Gene expression | Age related diseases | Signaling | Retinal degeneration | Lactic acid | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article