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Journal of Immunology, ISSN 0022-1767, 03/2014, Volume 192, Issue 6, pp. 2837 - 2845
Inflammation is associated with development of atherosclerosis, and cholesterol crystals (CC) have long been recognized as a hallmark of atherosclerotic... 
ATHEROSCLEROTIC LESIONS | TUMOR-NECROSIS-FACTOR | C5A RECEPTOR | MEMBRANE ATTACK | STERILE INFLAMMATION | EPITHELIAL-CELLS | CARDIOVASCULAR EVENTS | INTERLEUKIN-1-BETA | OXIDATIVE BURST | ANAPHYLATOXINS | IMMUNOLOGY | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Complement C1q - immunology | Humans | Complement Pathway, Classical - drug effects | Complement C5a - metabolism | Complement System Proteins - immunology | Caspase 1 - metabolism | Monocytes - metabolism | Caspase 1 - immunology | Macrophage-1 Antigen - immunology | Monocytes - immunology | Interleukin-1beta - genetics | Complement Activation - immunology | Complement C1q - metabolism | Complement System Proteins - metabolism | Inflammasomes - drug effects | Reactive Oxygen Species - immunology | Complement C5 - immunology | Tumor Necrosis Factor-alpha - immunology | Macrophage-1 Antigen - metabolism | Complement Activation - drug effects | Cytokines - immunology | Interleukin-1beta - secretion | Complement C5 - metabolism | Tumor Necrosis Factor-alpha - secretion | Cells, Cultured | Cytokines - secretion | Phagocytosis - immunology | Interleukin-1beta - immunology | Cholesterol - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Monocytes - drug effects | Complement Pathway, Classical - immunology | Microscopy, Confocal | Complement Pathway, Alternative - drug effects | Complement Pathway, Alternative - immunology | Inflammasomes - immunology | Cholesterol - pharmacology | Complement C5a - immunology | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 05/2018, Volume 200, Issue 10, pp. 3495 - 3505
Streptococcus pyogenes is an exclusively human pathogen that can provoke mild skin and throat infections but can also cause fatal septicemia. This... 
C4B-BINDING PROTEIN | VACCINE DEVELOPMENT | M1 PROTEIN | PROTEIN FACTOR-H | GROUP-A STREPTOCOCCUS | SURFACE-PROTEINS | ALBUMIN | IMMUNOLOGY | BINDING PROTEIN | CYSTEINE PROTEINASE | IMMUNOGLOBULIN-G | Bacterial Outer Membrane Proteins - immunology | Streptococcus pyogenes - immunology | Humans | Antigens, Bacterial - immunology | Phagocytosis - immunology | Complement System Proteins - immunology | Male | Virulence Factors - immunology | Protein Binding - immunology | Animals | Immunoglobulin G - immunology | Virulence - immunology | Female | Complement C4b-Binding Protein - immunology | Complement Inactivating Agents - immunology | Mice | Mice, Inbred BALB C | Streptococcal Infections - immunology | Carrier Proteins - immunology | Complement inhibitors | Virulence | Immunoglobulin G | Complement | Infections | Blood plasma | Proteins | H protein | Plasma proteins | Virulence factors | Rodents | Necrotizing fasciitis | Strains (organisms) | Immune system | Pathogens | Septicemia | Opsonization | C4b-binding protein | Transgenic mice | Electron microscopy | Pharynx | Fasciitis | Fc receptors | Streptococcus | Skin | Complement component C4b-binding protein | M protein | Index Medicus | Abridged Index Medicus | Basic Medicine | Immunologi inom det medicinska området | Medical and Health Sciences | Medicin och hälsovetenskap | Immunology in the medical area | Medicinska och farmaceutiska grundvetenskaper
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2009, Volume 124, Issue 6, pp. 1161 - 1178
Journal Article
Nature, ISSN 0028-0836, 10/2011, Volume 478, Issue 7367, pp. 76 - 81
Oxidative stress and enhanced lipid peroxidation are linked to many chronic inflammatory diseases, including age-related macular degeneration (AMD). AMD is the... 
PHAGOCYTOSIS | MULTIDISCIPLINARY SCIENCES | APOPTOTIC CELLS | MACULAR DEGENERATION | RISK | PHOSPHOLIPIDS | PROTEINS | END-PRODUCTS | RETINAL-PIGMENT EPITHELIUM | INNATE IMMUNITY | AGE | Inflammation - pathology | Protein Binding - genetics | Retina - metabolism | Epitopes - metabolism | Oxidative Stress | Humans | Malondialdehyde - chemistry | Complement System Proteins - immunology | Male | Complement System Proteins - metabolism | Necrosis | Inflammation - metabolism | Female | Malondialdehyde - metabolism | Protein Structure, Tertiary | Complement Factor H - immunology | Complement Inactivator Proteins - metabolism | Enzyme-Linked Immunosorbent Assay | Mice, Inbred C57BL | Complement Inactivator Proteins - immunology | Inflammation - immunology | Binding Sites - genetics | Mutation - genetics | Macular Degeneration - metabolism | Animals | Complement Factor H - metabolism | Malondialdehyde - immunology | Epitopes - chemistry | Mice | Complement Factor H - genetics | Lipid Peroxidation | Macrophages, Peritoneal - metabolism | Malondialdehyde - antagonists & inhibitors | Apoptosis | Complement Inactivator Proteins - genetics | Macular Degeneration - pathology | Macular degeneration | Oxidative stress | Complement factor H | Care and treatment | Physiological aspects | Research | Health aspects | Risk factors | Proteins | Flow cytometry | Plasma | Pathogenesis | Low density lipoprotein | Mass spectrometry | Molecular weight | Binding sites | Index Medicus
Journal Article
Journal Article
Nature, ISSN 0028-0836, 06/2016, Volume 534, Issue 7608, pp. 538 - 543
Journal Article
Nature, ISSN 0028-0836, 09/2007, Volume 449, Issue 7158, pp. 101 - 104
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e59372 - e59372
Background: Vascular endothelial cells (ECs) express and release protein components of the complement pathways, as well as secreting and anchoring ultra-large... 
ADAMTS-13 | HEMOLYTIC-UREMIC SYNDROME | ATYPICAL HUS | FACTOR-B | PATHWAY | MULTIDISCIPLINARY SCIENCES | FACTOR MULTIMERS | FACTOR-H | MUTATIONS | PROTEINS | BINDING | ADAM Proteins - immunology | Gene Expression | Hemostasis - immunology | ADAM Proteins - antagonists & inhibitors | Human Umbilical Vein Endothelial Cells - metabolism | von Willebrand Factor - immunology | Humans | Protein Multimerization | Cells, Cultured | Thrombosis - immunology | Complement System Proteins - immunology | Human Umbilical Vein Endothelial Cells - immunology | von Willebrand Factor - genetics | Hot Temperature | von Willebrand Factor - secretion | ADAMTS13 Protein | Thrombosis - pathology | Human Umbilical Vein Endothelial Cells - cytology | Protein Binding | Complement System Proteins - genetics | ADAM Proteins - genetics | Complement Pathway, Alternative | Complement Pathway, Mannose-Binding Lectin | Complement Pathway, Classical | Lectins | Gene expression | Thrombosis | Blood clot | Von Willebrand factor | Endothelium | Bioengineering | Antibodies | Fluorescence | Activation | Complement | Proteins | Thrombotic thrombocytopenic purpura | Pathways | Hemostasis | Hemostatics | Thromboembolism | Assembly | Anchoring | Strings | Endothelial cells | Thrombocytopenic purpura | Complement activation | Complement component C5 | Microscopy | Complement component C4 | Purpura | Complement component C3 | Mutation | Umbilical vein | Veins & arteries | Index Medicus
Journal Article
Annals of Neurology, ISSN 0364-5134, 2008, Volume 63, Issue 1, pp. 16 - 25
Journal Article