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Immunologic research, ISSN 1559-0755, 2011, Volume 51, Issue 1, pp. 45 - 60
Complement system activation plays an important role in both innate and acquired immunity... 
Allergology | Signal transduction | Immunology | Medicine & Public Health | Cell cycle | Internal Medicine | Membrane attack complex | C5b-9 terminal complement complex | Transcriptional regulation | Medicine/Public Health, general | Apoptosis | ACTIVATED PROTEIN-KINASE | SYSTEMIC LUPUS-ERYTHEMATOSUS | GUILLAIN-BARRE-SYNDROME | ULCERATIVE-COLITIS | GLOMERULAR EPITHELIAL-CELLS | C5B-9 COMPLEX | IMMUNOLOGY | S-PROTEIN | MANNAN-BINDING LECTIN | ACUTE MYOCARDIAL-INFARCTION | SMOOTH-MUSCLE-CELLS | Forkhead Transcription Factors - immunology | CDC2 Protein Kinase | Complement Membrane Attack Complex - metabolism | Cyclin-Dependent Kinase 4 - genetics | Humans | Caspase 8 - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Complement Membrane Attack Complex - immunology | MAP Kinase Signaling System - immunology | Phosphatidylinositol 3-Kinases - immunology | Cell Cycle Proteins - immunology | Cyclin B - immunology | Forkhead Transcription Factors - metabolism | Muscle Proteins - metabolism | bcl-Associated Death Protein - metabolism | Cell Membrane - metabolism | Phosphorylation - immunology | Cyclin-Dependent Kinases | Proto-Oncogene Proteins c-akt - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | Cyclin-Dependent Kinase 2 - metabolism | Mitogen-Activated Protein Kinase 3 - immunology | Muscle Proteins - immunology | G1 Phase - immunology | S Phase - immunology | Nerve Tissue Proteins - immunology | Cell Cycle Proteins - metabolism | bcl-Associated Death Protein - immunology | Cyclin-Dependent Kinase 4 - metabolism | Nerve Tissue Proteins - metabolism | Caspase 8 - immunology | Animals | Apoptosis - immunology | Proto-Oncogene Proteins c-akt - immunology | Mitogen-Activated Protein Kinase 3 - metabolism | BH3 Interacting Domain Death Agonist Protein - immunology | Cyclin B - metabolism | Cell Membrane - immunology | Forkhead Box Protein O1 | Cyclin-Dependent Kinase 2 - immunology | Medical colleges | Membranes | Immune system | apoptosis | membrane attack complex | cell cycle | signal transduction | transcriptional regulation
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2018, Volume 200, Issue 10, pp. 3495 - 3505
.... This gram-positive bacterium has developed several strategies to evade the human immune system, enabling... 
C4B-BINDING PROTEIN | VACCINE DEVELOPMENT | M1 PROTEIN | PROTEIN FACTOR-H | GROUP-A STREPTOCOCCUS | SURFACE-PROTEINS | ALBUMIN | IMMUNOLOGY | BINDING PROTEIN | CYSTEINE PROTEINASE | IMMUNOGLOBULIN-G | Bacterial Outer Membrane Proteins - immunology | Streptococcus pyogenes - immunology | Humans | Antigens, Bacterial - immunology | Phagocytosis - immunology | Complement System Proteins - immunology | Male | Virulence Factors - immunology | Protein Binding - immunology | Animals | Immunoglobulin G - immunology | Virulence - immunology | Female | Complement C4b-Binding Protein - immunology | Complement Inactivating Agents - immunology | Mice | Mice, Inbred BALB C | Streptococcal Infections - immunology | Carrier Proteins - immunology | Complement inhibitors | Virulence | Immunoglobulin G | Complement | Infections | Blood plasma | Proteins | H protein | Plasma proteins | Virulence factors | Rodents | Necrotizing fasciitis | Strains (organisms) | Immune system | Pathogens | Septicemia | Opsonization | C4b-binding protein | Transgenic mice | Electron microscopy | Pharynx | Fasciitis | Fc receptors | Streptococcus | Skin | Complement component C4b-binding protein | M protein | Basic Medicine | Immunologi inom det medicinska området | Medical and Health Sciences | Medicin och hälsovetenskap | Immunology in the medical area | Medicinska och farmaceutiska grundvetenskaper
Journal Article
Trends in immunology, ISSN 1471-4906, 2009, Volume 30, Issue 2, pp. 83 - 90
.... This process of opsonisation must be tightly regulated to prevent immunopathology. Endogenous ligands such as dying cells, extracellular matrix proteins, pentraxins... 
Allergy and Immunology | C-REACTIVE PROTEIN | APOPTOTIC CELL RECOGNITION | C4B-BINDING PROTEIN | AMYLOID-P COMPONENT | SYSTEMIC-LUPUS-ERYTHEMATOSUS | NECROTIC CELLS | MACULAR DEGENERATION | EXTRACELLULAR-MATRIX | IMMUNOLOGY | FACTOR-H BINDS | GLOBULAR HEAD | Alzheimer Disease - etiology | Macular Degeneration - immunology | Humans | Complement System Proteins - immunology | Intracellular Signaling Peptides and Proteins - immunology | Intracellular Signaling Peptides and Proteins - metabolism | Complement Activation - immunology | Complement C4b-Binding Protein | Complement System Proteins - metabolism | Prions - immunology | Muscle Proteins - metabolism | Alzheimer Disease - immunology | Histocompatibility Antigens - metabolism | Macular Degeneration - etiology | Histocompatibility Antigens - immunology | Muscle Proteins - immunology | Prions - metabolism | LIM Domain Proteins | Prion Diseases - immunology | Arthritis, Rheumatoid - etiology | Immunity, Innate | Animals | Prion Diseases - etiology | Protein Binding | Amyloid beta-Peptides - immunology | Ligands | Arthritis, Rheumatoid - immunology | Macular degeneration | Immune complexes | Immunoproteins | Protein binding | Proteins | Lupus | Aging | Alzheimers disease | Autoimmune diseases | Basic Medicine | Other Basic Medicine | Andra medicinska och farmaceutiska grundvetenskaper | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2014, Volume 192, Issue 6, pp. 2837 - 2845
.... In this study we hypothesized whether CC employ the complement system to activate inflammasome/caspase-1, leading to release of mature IL-1 beta, and whether complement activation regulates CC... 
ATHEROSCLEROTIC LESIONS | TUMOR-NECROSIS-FACTOR | C5A RECEPTOR | MEMBRANE ATTACK | STERILE INFLAMMATION | EPITHELIAL-CELLS | CARDIOVASCULAR EVENTS | INTERLEUKIN-1-BETA | OXIDATIVE BURST | ANAPHYLATOXINS | IMMUNOLOGY | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Complement C1q - immunology | Humans | Complement Pathway, Classical - drug effects | Complement C5a - metabolism | Complement System Proteins - immunology | Caspase 1 - metabolism | Monocytes - metabolism | Caspase 1 - immunology | Macrophage-1 Antigen - immunology | Monocytes - immunology | Interleukin-1beta - genetics | Complement Activation - immunology | Complement C1q - metabolism | Complement System Proteins - metabolism | Inflammasomes - drug effects | Reactive Oxygen Species - immunology | Complement C5 - immunology | Tumor Necrosis Factor-alpha - immunology | Macrophage-1 Antigen - metabolism | Complement Activation - drug effects | Cytokines - immunology | Interleukin-1beta - secretion | Complement C5 - metabolism | Tumor Necrosis Factor-alpha - secretion | Cells, Cultured | Cytokines - secretion | Phagocytosis - immunology | Interleukin-1beta - immunology | Cholesterol - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Monocytes - drug effects | Complement Pathway, Classical - immunology | Microscopy, Confocal | Complement Pathway, Alternative - drug effects | Complement Pathway, Alternative - immunology | Inflammasomes - immunology | Cholesterol - pharmacology | Complement C5a - immunology
Journal Article
The Journal of experimental medicine, ISSN 0022-1007, 07/2017, Volume 214, Issue 7, pp. 2103 - 2119
Neutrophils play a crucial role in host defense. However, neutrophil activation is also linked to autoimmune diseases such as systemic lupus erythematosus... 
PLASMACYTOID DENDRITIC CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | NADPH OXIDASE | SYSTEMIC-LUPUS-ERYTHEMATOSUS | PROPROTEIN CONVERTASES | C1Q DEFICIENCY | INFLAMMATION | FC-GAMMA-RIIIB | TOLL-LIKE RECEPTORS | ALPHA | IMMUNOLOGY | EXTRACELLULAR TRAPS | Extracellular Traps - metabolism | Dendritic Cells - immunology | Humans | Complement C5a - metabolism | Lupus Erythematosus, Systemic - metabolism | Monocytes - metabolism | Monocytes - immunology | Complement Activation - immunology | Receptors, IgG - metabolism | Toll-Like Receptor 8 - immunology | Extracellular Traps - immunology | Flow Cytometry | Toll-Like Receptor 7 - immunology | Lupus Erythematosus, Systemic - immunology | Neutrophils - metabolism | Dendritic Cells - metabolism | Toll-Like Receptor 7 - metabolism | Cells, Cultured | Neutrophils - immunology | Phagocytosis - immunology | Receptors, IgG - immunology | Blotting, Western | Complement C5a - immunology | Antigen-Antibody Complex - immunology | Toll-Like Receptor 8 - metabolism | Shedding | Complement component C5a | Immune clearance | Activation | Proteins | Cell activation | Systemic lupus erythematosus | Proteolysis | Toll-like receptors | Furin | Cleavage | Antigen-antibody complexes | Immune system | TLR7 protein | Dendritic cells | Neutrophils | Leukocytes (neutrophilic) | Inflammation | Nucleic acids | Signaling | Monocytes | Chronic conditions | Complement activation | Information processing | Autoimmune diseases | Phagocytosis | 311 | 300 | Extracellular Traps/immunology | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Phagocytosis/immunology | Immunologi inom det medicinska området | Lupus Erythematosus, Systemic/immunology | Toll-Like Receptor 7/immunology | Neutrophils/immunology | Complement Activation/immunology | Monocytes/immunology | Antigen-Antibody Complex/immunology | Receptors, IgG/immunology | Medicinska och farmaceutiska grundvetenskaper | Dendritic Cells/immunology | Complement C5a/immunology | Toll-Like Receptor 8/immunology | Immunology in the medical area
Journal Article
Journal of innate immunity, ISSN 1662-8128, 2014, Volume 6, Issue 2, pp. 192 - 204
... proteins Abstract E-Mail karger@karger.com Research Article J Innate Immun 2014;6:192–204 DOI: 10.1159/000353734 Genome-Wide T ranscriptional Analysis of Drosophila... 
Research Article | Recognition molecule | Thioester-containing proteins | Hemocyte | Coagulation | Nematodes | Insect immunity | Basement membrane | IMMUNE-RESPONSE | ADULT FLIES | BACTERIA | IMMUNOLOGY | PGRP-LC | ARMIGERES-SUBALBATUS | GENE-EXPRESSION | HOST-DEFENSE | MELANOGASTER | WOUND REPAIR | RNAI SCREEN | Drosophila melanogaster - immunology | Membrane Glycoproteins - metabolism | Oligonucleotide Array Sequence Analysis | Hemocytes - immunology | Complement System Proteins - immunology | Transcriptome - immunology | Drosophila Proteins - immunology | Drosophila Proteins - metabolism | Drosophila melanogaster - genetics | Complement System Proteins - metabolism | Host-Pathogen Interactions - immunology | Photorhabdus - physiology | Larva - immunology | RNA Interference | Rhabditoidea - physiology | Complement System Proteins - genetics | Larva - genetics | Genome, Insect | Membrane Glycoproteins - immunology | Extracellular Matrix Proteins - metabolism | Gene Ontology | Rhabditoidea - microbiology | Animals, Genetically Modified | Extracellular Matrix Proteins - genetics | Host-Parasite Interactions - immunology | Reverse Transcriptase Polymerase Chain Reaction | Hemocytes - metabolism | Photorhabdus - immunology | Membrane Glycoproteins - genetics | Microscopy, Confocal | Animals | Extracellular Matrix Proteins - immunology | Larva - parasitology | Drosophila melanogaster - parasitology | Rhabditoidea - immunology | Luminescent Proteins - genetics | Drosophila Proteins - genetics | Cluster Analysis | Luminescent Proteins - metabolism
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 3, p. e59372
Background: Vascular endothelial cells (ECs) express and release protein components of the complement pathways, as well as secreting and anchoring ultra-large von Willebrand factor (ULVWF... 
ADAMTS-13 | HEMOLYTIC-UREMIC SYNDROME | ATYPICAL HUS | FACTOR-B | PATHWAY | MULTIDISCIPLINARY SCIENCES | FACTOR MULTIMERS | FACTOR-H | MUTATIONS | PROTEINS | BINDING | ADAM Proteins - immunology | Gene Expression | Hemostasis - immunology | ADAM Proteins - antagonists & inhibitors | Human Umbilical Vein Endothelial Cells - metabolism | von Willebrand Factor - immunology | Humans | Protein Multimerization | Cells, Cultured | Thrombosis - immunology | Complement System Proteins - immunology | Human Umbilical Vein Endothelial Cells - immunology | von Willebrand Factor - genetics | Hot Temperature | von Willebrand Factor - secretion | ADAMTS13 Protein | Thrombosis - pathology | Human Umbilical Vein Endothelial Cells - cytology | Protein Binding | Complement System Proteins - genetics | ADAM Proteins - genetics | Complement Pathway, Alternative | Complement Pathway, Mannose-Binding Lectin | Complement Pathway, Classical | Lectins | Gene expression | Thrombosis | Blood clot | Von Willebrand factor | Endothelium | Bioengineering | Antibodies | Fluorescence | Activation | Complement | Proteins | Thrombotic thrombocytopenic purpura | Pathways | Hemostasis | Hemostatics | Thromboembolism | Assembly | Anchoring | Strings | Endothelial cells | Thrombocytopenic purpura | Complement activation | Complement component C5 | Microscopy | Complement component C4 | Purpura | Complement component C3 | Mutation | Umbilical vein | Veins & arteries
Journal Article