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1. Full Text C1q restrains autoimmunity and viral infection by regulating CD8+ T cell metabolism
by Ling, Guang Sheng and Crawford, Greg and Buang, Norzawani and Bartok, Istvan and Tian, Kunyuan and Thielens, Nicole M and Bally, Isabelle and Harker, James A and Ashton-Rickardt, Philip G and Rutschmann, Sophie and Strid, Jessica and Botto, Marina
Science (American Association for the Advancement of Science), ISSN 0036-8075, 05/2018, Volume 360, Issue 6388, pp. 558 - 563
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Immunoglobulins - immunology | Complement C1q - physiology | Autoimmunity - genetics | Complement C1q - genetics | Lymphocytic Choriomeningitis - immunology | Complement Pathway, Classical - immunology | Autoantibodies - immunology | Animals | Autoimmunity - immunology | Mice, Mutant Strains | Lupus Erythematosus, Systemic - genetics | Lupus Erythematosus, Systemic - immunology | CD8-Positive T-Lymphocytes - metabolism | Mice | Complement C3 - genetics | Complement Pathway, Classical - genetics | Immunologic Memory - immunology | Complement C3 - physiology | Disease Models, Animal | Lymphocytic Choriomeningitis - genetics | Graft-versus-host reaction | CD8 antigen | Chronic infection | Effector cells | Complement | Lymphocytes T | Infections | Cell surface | Protein turnover | Proteins | Mitochondria | Systemic lupus erythematosus | Lymphocytes | Lupus | Antigens | Abnormalities | Autoantigens | Metabolism | Complement component C1q | Chronic conditions | Cellular biology | Complement component C3 | Autoimmune diseases | Viral infections | Classical pathway | Apoptosis | Index Medicus | Life Sciences | Biomolecules | Biochemistry, Molecular Biology
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2. Full Text Anti‐C1q autoantibodies from patients with systemic lupus erythematosus induce C1q production by macrophages
by Thanei, Sophia and Trendelenburg, Marten
Journal of leukocyte biology, ISSN 0741-5400, 02/2017, Volume 101, Issue 2, pp. 481 - 491
lupus nephritis | phagocytes | autoimmunity | complement | inflammation | Autoimmunity | Phagocytes | Complement | Inflammation | Lupus nephritis | Immunology | Life Sciences & Biomedicine | Hematology | Science & Technology | Cell Biology | Complement C1q - biosynthesis | Macrophages - pathology | Complement C1q - immunology | Humans | Middle Aged | Male | Lipopolysaccharide Receptors - metabolism | Autoantibodies - immunology | Macrophages - metabolism | Flow Cytometry | Intracellular Space - metabolism | Immobilized Proteins - metabolism | Lupus Erythematosus, Systemic - immunology | Monocytes - pathology | Adult | Female | Aged | Kinetics | Immunoglobulin G - metabolism | Lupus | Flow cytometry | Correlation | Nephritis | Myeloid cells | Autoantibodies | Dendritic cells | Immunomodulation | Immunoglobulin G | Antibodies | Macrophages | Patients | Complement component C1q | Cytometry | Monocytes | Chronic conditions | Systemic lupus erythematosus | Intracellular | Autoimmune diseases | Classical pathway | Index Medicus
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3. Full Text Complement component C1q inhibits β‐amyloid‐ and serum amyloid P‐induced neurotoxicity via caspase‐ and calpain‐independent mechanisms
by Pisalyaput, Karntipa and Tenner, Andrea J
Journal of neurochemistry, ISSN 0022-3042, 02/2008, Volume 104, Issue 3, pp. 696 - 707
C1q | amyloid | neurotoxicity | SAP | neurons | neuroprotection | Neuroprotection | Neurotoxicity | Amyloid | Neurons | Biochemistry & Molecular Biology | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neurology | Organic mental disorders. Neuropsychology | Psychology. Psychoanalysis. Psychiatry | Adult and adolescent clinical studies | Degenerative and inherited degenerative diseases of the nervous system. Leukodystrophies. Prion diseases | Psychopathology. Psychiatry | Biological and medical sciences | Medical sciences | Cell Survival - drug effects | Brain - cytology | Calpain - metabolism | Amyloid beta-Peptides - toxicity | Embryo, Mammalian | Cells, Cultured | Etoposide - pharmacology | Rats | Membrane Potential, Mitochondrial - drug effects | Complement C1q - pharmacology | Rats, Sprague-Dawley | Serum Amyloid P-Component - toxicity | Dose-Response Relationship, Drug | Animals | Caspases - metabolism | Drug Interactions | Antineoplastic Agents, Phytogenic - pharmacology | Neurons - drug effects | Immunologic Factors - pharmacology | Index Medicus
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4. Full Text MafB is a critical regulator of complement component C1q
by Tran, Mai Thi Nhu and Hamada, Michito and Jeon, Hyojung and Shiraishi, Risako and Asano, Keigo and Hattori, Motochika and Nakamura, Megumi and Imamura, Yuki and Tsunakawa, Yuki and Fujii, Risa and Usui, Toshiaki and Kulathunga, Kaushalya and Andrea, Christina-Sylvia and Koshida, Ryusuke and Kamei, Risa and Matsunaga, Yurina and Kobayashi, Makoto and Oishi, Hisashi and Kudo, Takashi and Takahashi, Satoru
Nature communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 1700 - 14
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Complement component C1q | Monocytes | Complement activation | Rodents | Complement | Mice | Gene expression | Autoimmune diseases | Macrophages | Apoptosis | Index Medicus
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5. Full Text ApoE attenuates unresolvable inflammation by complex formation with activated C1q
by Yin, Changjun and Ackermann, Susanne and Ma, Zhe and Mohanta, Sarajo K and Zhang, Chuankai and Li, Yuanfang and Nietzsche, Sandor and Westermann, Martin and Peng, Li and Hu, Desheng and Bontha, Sai Vineela and Srikakulapu, Prasad and Beer, Michael and Megens, Remco T. A and Steffens, Sabine and Hildner, Markus and Halder, Luke D and Eckstein, Hans-Henning and Pelisek, Jaroslav and Herms, Jochen and Roeber, Sigrun and Arzberger, Thomas and Borodovsky, Anna and Habenicht, Livia and Binder, Christoph J and Weber, Christian and Zipfel, Peter F and Skerka, Christine and Habenicht, Aneas J. R
Nature medicine, ISSN 1078-8956, 03/2019, Volume 25, Issue 3, pp. 496 - 506
IMMUNE | ALZHEIMERS-DISEASE | A-BETA | NEUROFIBRILLARY PATHOLOGY | ATHEROSCLEROSIS | NEURODEGENERATION | MICE | FACTOR-H BINDS | LASER-CAPTURE MICRODISSECTION | APOLIPOPROTEIN-E | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Complement C5 | Complement C1q - immunology | Humans | Middle Aged | Choroid Plexus - immunology | Male | Leukocytes | Cognitive Dysfunction - immunology | Carotid Artery Diseases - pathology | Aged, 80 and over | Female | Plaque, Amyloid - immunology | Carotid Artery Diseases - immunology | Atherosclerosis - pathology | Atherosclerosis - immunology | Plaque, Amyloid - pathology | Choroid Plexus - pathology | Aorta - immunology | Cognitive Dysfunction - pathology | Mice, Knockout, ApoE | Aorta - pathology | Complement Pathway, Classical - immunology | Animals | Apolipoproteins E - immunology | Carotid Arteries - immunology | Brain - pathology | Amyloid beta-Peptides - immunology | Aged | Carotid Arteries - pathology | RNA, Small Interfering | Antigen-Antibody Complex - immunology | Protein Isoforms - immunology | Brain - immunology | Microscopy, Fluorescence | Cogeneration power plants | Analysis | Atherosclerosis | Lipids | Research | Apolipoproteins | Alzheimer's disease | Choroid plexus | Complex formation | Cognitive ability | Complement | Inflammation | siRNA | Ribonucleic acid--RNA | Arteries | Microglia | White blood cells | Proteins | Complement component C1q | Immune checkpoint | Apolipoprotein E | Arteriosclerosis | Isoforms | Attenuation | Infiltration | Plaques | Index Medicus
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6. Full Text Complement component C1q is produced by isolated articular chondrocytes
by Lubbers, R and van Schaarenburg, R.A and Kwekkeboom, J.C and Levarht, E.W.N and Bakker, A.M and Mahdad, R and Monteagudo, S and Cherifi, C and Lories, R.J and Toes, R.E.M and Ioan-Facsinay, A and Trouw, L.A
Osteoarthritis and cartilage, ISSN 1063-4584, 05/2020, Volume 28, Issue 5, pp. 675 - 684
Cartilage | C1q | Complement | Osteoarthritis | Chondrocytes | Life Sciences & Biomedicine | Orthopedics | Rheumatology | Science & Technology | Index Medicus
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7. Full Text Complement component C1q: Historical perspective of a functionally versatile, and structurally unusual, serum protein
by Reid, Kenneth B.M
Frontiers in immunology, ISSN 1664-3224, 04/2018, Volume 9, pp. 764 - 764
Aging | Tumor | C1q | Collagen-like structure | Globular heads | C1 activation | Life Sciences & Biomedicine | Immunology | Science & Technology | Animals | History, 21st Century | History, 20th Century | Humans | Complement C1q - physiology | Complement C1q - history | Complement C1q - chemistry | Physiological aspects | Genetic aspects | Immune response | Research | Complement (Immunology) | Blood proteins | globular heads | aging | collagen-like structure | tumor
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8. Full Text Complement C1q subunit molecules from Xenopus laevis possess conserved function in C1q-immunoglobulin interaction
by Yan, Fangfang and Zhou, Enxu and Liu, Shuo and Gao, Along and Kong, Linghe and Li, Bingxi and Tu, Xiao and Guo, Zheng and Mo, Jinfeng and Chen, Meng and Ye, Jianmin
Developmental and comparative immunology, ISSN 0145-305X, 02/2020, Volume 103, pp. 103532 - 103532
Immunoglobulin (Ig) | Hemolysis | C1q-Ig interaction | Complement component 1q (C1q) | Xenopus laevis | Veterinary Sciences | Fisheries | Immunology | Life Sciences & Biomedicine | Zoology | Science & Technology | Immunoglobulin M | Immunoglobulins | Nucleotide sequence | Blood cells | Immunoglobulin G | Erythrocytes | Amino acids | Tilapia | Complement | Nucleotides | C-Terminus | Phylogeny | Blood levels | Complement component C1q | Sheep red blood cells | Red blood cells | Collagen | N-Terminus | Sheep | Open reading frames | Index Medicus
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9. Full Text C1q-induced LRP1B and GPR6 proteins expressed early in Alzheimer disease mouse models, are essential for the C1q-mediated protection against amyloid- β neurotoxicity
by Benoit, Marie E and Hernandez, Michael X and Dinh, Minhan L and Benavente, Francisca and Vasquez, Osvaldo and Tenner, Andrea J
The Journal of biological chemistry, ISSN 0021-9258, 01/2013, Volume 288, Issue 1, pp. 654 - 665
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Up-Regulation | Tumor Suppressor Proteins - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Gene Expression Regulation | Rats | Receptors, LDL - metabolism | Gene Expression Profiling | Neurodegenerative Diseases - metabolism | Complement C1q - metabolism | Tumor Suppressor Proteins - physiology | Animals | Models, Biological | Alzheimer Disease - metabolism | Amyloid beta-Peptides - metabolism | Mice | Receptors, LDL - physiology | Disease Models, Animal | Receptors, G-Protein-Coupled - physiology | Index Medicus | Neuroprotection | Gene Expression | C1q | Alzheimer disease | Neurodegeneration | Neurobiology | Amyloid | Complement | GPR6 | LRP1B
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