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Journal of Forensic Sciences, ISSN 0022-1198, 05/2008, Volume 53, Issue 3, pp. 720 - 723
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2011, Volume 6, Issue 12, pp. e29017 - e29017
Background: TGF-beta is a pleiotropic cytokine that can have pro- or anti-inflammatory effects depending on the context. Elevated levels of bioactive TGF-beta... 
BARRIER DISRUPTION | GROWTH-FACTOR-BETA | DESICCATING STRESS | TEAR FLUID | INTERFERON-GAMMA | EXPERIMENTAL DRY EYE | BIOLOGY | REGULATORY T-CELLS | SJOGRENS-SYNDROME | CORNEAL EPITHELIUM | SALIVARY-GLANDS | Eye - pathology | Inflammation - pathology | Homeodomain Proteins - metabolism | Humans | Aging - drug effects | Adoptive Transfer | CD4-Positive T-Lymphocytes - pathology | Conjunctiva - metabolism | Dry Eye Syndromes - pathology | Conjunctiva - pathology | Inflammation - metabolism | Transforming Growth Factor beta - drug effects | Keratoconjunctivitis Sicca - complications | Eye - drug effects | Mucous Membrane - drug effects | Autoimmune Diseases - metabolism | Autoimmune Diseases - pathology | Protein-Serine-Threonine Kinases - metabolism | Eye - metabolism | Epithelium - drug effects | Epithelium - pathology | Epithelium - metabolism | Receptors, Chemokine - metabolism | Autoimmune Diseases - complications | Keratoconjunctivitis Sicca - metabolism | Aging - pathology | Mice, Knockout | Mucous Membrane - metabolism | Cell Movement - drug effects | Transforming Growth Factor beta - pharmacology | Mucous Membrane - pathology | Animals | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Conjunctiva - drug effects | Dry Eye Syndromes - metabolism | Keratoconjunctivitis Sicca - pathology | Cell Proliferation - drug effects | Mice | Genes, Dominant - genetics | Transforming Growth Factor beta - metabolism | CD4-Positive T-Lymphocytes - drug effects | Cornea | Sjogren's syndrome | Physicians | Glands | Helper cells | Interleukin 23 | Lymphocytes T | Kinases | Cell density | Eye | Keratoconjunctivitis | Lymphocytes | Rodents | Age | Scopolamine | Cytokines | Desiccants | Salivary glands | Inflammation | Conjunctiva | Disease control | Smoothness | Gelatinase B | CD4 antigen | Medicine | Signaling | RAG1 protein | γ-Interferon | Eye diseases | Infiltration | Interferon | Disruption | Tears | Index Medicus
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, pp. e78508 - e78508
CD4(+) T cells are essential to pathogenesis of ocular surface disease in dry eye. Two subtypes of CD4(+) T cells, Th1 and Th17 cells, function concurrently in... 
PATHOGENESIS | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | DESICCATING STRESS | INTERFERON-GAMMA | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | DISTINCT | PREVALENCE | LACRIMAL KERATOCONJUNCTIVITIS | EXPRESSION | TH17 CELLS | Receptors, CCR6 - immunology | Th1 Cells - pathology | Matrix Metalloproteinases - genetics | Lymph Nodes - pathology | Cornea - immunology | Interleukin-17 - immunology | Adoptive Transfer | Th1 Cells - immunology | Xerophthalmia - genetics | Goblet Cells - immunology | Conjunctiva - pathology | Matrix Metalloproteinases - immunology | Scopolamine Hydrobromide | Female | Cornea - pathology | Interferon-gamma - genetics | Receptors, CXCR3 - deficiency | Disease Models, Animal | Th17 Cells - pathology | Xerophthalmia - pathology | Conjunctiva - immunology | Interleukin-17 - genetics | Epithelial Cells - pathology | Receptors, CCR6 - deficiency | Lymph Nodes - immunology | Mice, Knockout | Animals | Epithelial Cells - immunology | Interferon-gamma - immunology | Receptors, CXCR3 - genetics | Receptors, CCR6 - genetics | Receptors, CXCR3 - immunology | Th17 Cells - immunology | Mice | Goblet Cells - pathology | Xerophthalmia - chemically induced | Xerophthalmia - immunology | Cell Movement | Animal models | Cornea | Pathogenesis | Epithelial cells | Trafficking | Helper cells | Lymphocytes T | Matrix metalloproteinase | Cell surface | Lymph nodes | Eye | Receptors | Lymphocytes | Rodents | Animal tissues | Metalloproteinase | Localization | Lymphatic system | Immune response | Psoriasis | Cytokines | Desiccants | Therapeutic applications | Inflammation | T cell receptors | CXCR3 protein | Conjunctiva | Chemokine receptors | CCR6 protein | Crohns disease | CD4 antigen | Immune systems | Medicine | γ-Interferon | Ligands | Eye diseases | Infiltration | Cell migration | Chemokines | Index Medicus
Journal Article
Development (Cambridge), ISSN 0950-1991, 02/2013, Volume 140, Issue 3, pp. 594 - 605
Conjunctival goblet cells primarily synthesize mucins to lubricate the ocular surface, which is essential for normal vision. Notch signaling has been known to... 
Goblet cell | Cell fate | Mouse | Notch signaling | Klf4/5 | Differentiation | Ocular surface | STEM-CELLS | ALLERGIC CONJUNCTIVITIS | DOWN-REGULATION | HUMAN CORNEAL | DEVELOPMENTAL BIOLOGY | OCULAR SURFACE EPITHELIUM | GOBLET CELLS | MUCIN GENE | IN-VIVO | GENE-EXPRESSION | MUCOUS METAPLASIA | Inflammation - pathology | Cell Proliferation | Cornea - embryology | Metaplasia - metabolism | Transcriptional Activation | Epithelium, Corneal - pathology | Conjunctiva - metabolism | Conjunctiva - pathology | Inflammation - metabolism | Hyperplasia - genetics | Gene Expression Regulation, Developmental | Kruppel-Like Transcription Factors - metabolism | Gene Deletion | Cell Differentiation | Cornea - pathology | Lacrimal Apparatus - pathology | Nuclear Proteins - genetics | Epithelium, Corneal - embryology | Promoter Regions, Genetic | Conjunctiva - embryology | Signal Transduction | Mice, Transgenic | Nuclear Proteins - metabolism | Receptor, Notch1 - metabolism | Transcription Factors - genetics | Lacrimal Apparatus - metabolism | Goblet Cells - metabolism | Metaplasia - pathology | Hyperplasia - pathology | Transcription Factors - metabolism | Cornea - metabolism | Phenotype | Animals | Mucin 5AC - genetics | Epithelium, Corneal - metabolism | Inflammation - genetics | Mucin 5AC - metabolism | Mice | Goblet Cells - pathology | Receptor, Notch1 - genetics | Kruppel-Like Transcription Factors - genetics | Index Medicus | s | 5 | Klf4
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