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Hypertension, ISSN 0194-911X, 10/2009, Volume 54, Issue 4, pp. 877 - 884
Journal Article
Circulation research, ISSN 0009-7330, 2009, Volume 104, Issue 2, pp. 170 - U61
Journal Article
Advances in experimental medicine and biology, ISSN 0065-2598, 2019, Volume 1165, p. 365
CCN2, also known as connective tissue growth factor (CTGF), is one of important members of the CCN family. Generally, CTGF expresses at low levels in normal... 
Kidney Diseases - physiopathology | Connective Tissue Growth Factor - physiology | Kidney - pathology | Humans | Transforming Growth Factor beta | Fibrosis
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 08/2019, Volume 9, Issue 1, pp. 1 - 11
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 09/2008, Volume 295, Issue 3, pp. 740 - 751
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 180, Issue 6, pp. 2386 - 2403
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2013, Volume 288, Issue 42, pp. 30708 - 30719
Journal Article
The Journal of Pathology, ISSN 0022-3417, 02/2018, Volume 244, Issue 2, pp. 227 - 241
Connective tissue growth factor (CCN2/CTGF) is a matricellular protein that is overexpressed in progressive human renal diseases, mainly in fibrotic areas. In... 
fibrosis | NF‐κB | CCN2 | EMT | EGFR | renal cells | NF-κB | NF-B | ANGIOTENSIN-II | PATHOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | TERMINAL MODULE IV | FIBROBLAST ACTIVATION | HUMAN MESANGIAL CELLS | ONCOLOGY | OBSTRUCTIVE NEPHROPATHY | MATRIX PROTEIN-PRODUCTION | TUBULOINTERSTITIAL FIBROSIS | INTERSTITIAL FIBROSIS | NF-KAPPA-B | NIH 3T3 Cells | Fibroblasts - enzymology | Peptide Fragments | Kidney - pathology | Humans | Kidney - enzymology | Epithelial-Mesenchymal Transition - drug effects | Male | NF-kappa B - metabolism | Kidney Diseases - enzymology | Kidney Diseases - chemically induced | Kidney Tubules, Proximal - enzymology | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Folic Acid | Kidney Tubules, Proximal - pathology | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Kidney - drug effects | Signal Transduction | Kidney Diseases - pathology | Kidney Diseases - prevention & control | Mice, Inbred C57BL | Fibroblasts - pathology | G2 Phase Cell Cycle Checkpoints | Mice, Knockout | Connective Tissue Growth Factor - deficiency | Animals | Receptor, trkA - metabolism | Erlotinib Hydrochloride - pharmacology | Fibrosis | Connective Tissue Growth Factor - genetics | Mice | Protein Kinase Inhibitors - pharmacology | Connective Tissue Growth Factor - metabolism | Immunohistochemistry | Fibronectins | Epidermal growth factor | Analysis | Kidney diseases | Folic acid | Animal models | Epidermal growth factor receptors | Mesenchyme | Syngeneic grafts | Epithelial cells | Connective tissue growth factor | Activation | Damage prevention | Proteins | Connective tissues | Signal transduction | Signaling | Rodents | Fibroblasts | Extracellular matrix | Receptor mechanisms | Damage accumulation | Index Medicus
Journal Article
Investigative Ophthalmology and Visual Science, ISSN 0146-0404, 05/2011, Volume 52, Issue 6, pp. 3660 - 3666
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e54652 - e54652
Glioblastomas (GBM), the most common and aggressive type of malignant glioma, are characterized by increased invasion into the surrounding brain tissues.... 
MALIGNANT GLIOMA | SURVIVAL | MIR-145 | STEM-CELLS | INVASION | MULTIDISCIPLINARY SCIENCES | FACTOR CTGF | DIFFERENTIATION | TUMORIGENICITY | CANCER | EXPRESSION | Focal Adhesion Kinase 1 - genetics | RNA, Small Interfering - genetics | Tumor Suppressor Proteins - antagonists & inhibitors | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Neural Stem Cells - cytology | Brain Neoplasms - metabolism | Transfection | Glioblastoma - genetics | Neoplastic Stem Cells - metabolism | Luciferases | Tumor Suppressor Proteins - genetics | Osteonectin | Neoplastic Stem Cells - pathology | Glioblastoma - metabolism | 3' Untranslated Regions | Focal Adhesion Kinase 1 - metabolism | Genes, Reporter | Astrocytes - cytology | Tumor Suppressor Proteins - metabolism | Signal Transduction | Gene Silencing | Brain Neoplasms - genetics | Glioblastoma - pathology | Cell Line, Tumor | Connective Tissue Growth Factor - genetics | MicroRNAs - genetics | Neural Stem Cells - metabolism | Connective Tissue Growth Factor - metabolism | Astrocytes - metabolism | Cell Movement | MicroRNA | Gliomas | Physiological aspects | Development and progression | Genetic aspects | Research | Cell migration | Brain | Pediatrics | Prognosis | Laboratories | Brain tumors | Brain cancer | Oncology | Neurosurgery | Kinases | Tissues | Cancer therapies | Cell adhesion & migration | Connective tissues | Angiogenesis | Glioma cells | Life sciences | Tumorigenesis | Growth factors | Recombinant | Astrocytes | MiRNA | Connective tissue growth factor | Gene expression | Ribonucleic acid--RNA | Chemotherapy | Medical prognosis | Stem cells | Neural stem cells | Focal adhesion kinase | Prostate | Tumors | Index Medicus | RNA | Ribonucleic acid
Journal Article