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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2013, Volume 110, Issue 30, pp. 12325 - 12330
Pancreatic ductal adenocarcinoma (PDA) is characterized by abundant desmoplasia and poor tissue perfusion. These features are proposed to limit the access of... 
Connective tissues | Animal models | Cell growth | Patent ductus arteriosus | Medical treatment | Mice | Pancreatic neoplasms | Tumors | Cancer | Apoptosis | Genetically engineered mouse models | Pancreatic tumor stroma | Chemoresistance | X-LINKED INHIBITOR | MULTIDISCIPLINARY SCIENCES | GEMCITABINE | NEOPLASTIC-CELLS | chemoresistance | pancreatic tumor stroma | TISSUE GROWTH-FACTOR | MOUSE MODEL | GENE-EXPRESSION | RESISTANCE | genetically engineered mouse models | SMALL-MOLECULE ANTAGONISTS | CARCINOMA | TUMOR MICROENVIRONMENT | Pancreatic Neoplasms - metabolism | Connective Tissue Growth Factor - antagonists & inhibitors | Connective Tissue Growth Factor - immunology | Deoxycytidine - administration & dosage | Pancreatic Neoplasms - pathology | Carcinoma, Pancreatic Ductal - metabolism | Antineoplastic Agents - therapeutic use | Antineoplastic Agents - administration & dosage | Carcinoma, Pancreatic Ductal - pathology | Pancreatic Neoplasms - drug therapy | Carcinoma, Pancreatic Ductal - drug therapy | Deoxycytidine - therapeutic use | Animals | Survival Analysis | X-Linked Inhibitor of Apoptosis Protein - metabolism | Liver Neoplasms - secondary | Antibodies, Monoclonal - immunology | Connective Tissue Growth Factor - metabolism | Deoxycytidine - analogs & derivatives | Antagonists (Biochemistry) | Drugs | Chemotherapy | Drug delivery systems | Pancreatic cancer | Physiological aspects | Research | Drug therapy | Health aspects | Vehicles | Biological Sciences
Journal Article
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 08/2017, Volume 109, Issue 8, p. djw339
Journal Article
Hypertension, ISSN 0194-911X, 2014, Volume 63, Issue 6, pp. 1235 - 1240
Connective tissue growth factor (CTGF) is involved in the pathogenesis of various fibrotic disorders. However, its role in the heart is not clear. To... 
Connective tissue growth factor | Ventricular remodeling | Hypertrophy, left ventricular | Ventricular dysfunction, left | Fibrosis | TGF-BETA | MYOCYTES | hypertrophy, left ventricular | ventricular remodeling | MATRICELLULAR PROTEINS | ventricular dysfunction, left | MECHANISMS | ANGIOTENSIN | connective tissue growth factor | FIBROBLAST ACTIVATION | MYOCARDIAL FIBROSIS | CARDIAC-HYPERTROPHY | PERIPHERAL VASCULAR DISEASE | fibrosis | ENDOTHELIN-1 | EXPRESSION | Gene Expression - drug effects | Connective Tissue Growth Factor - immunology | Heart Failure - physiopathology | Male | Constriction, Pathologic - physiopathology | Collagen Type I - genetics | Myocardium - metabolism | Aorta, Thoracic - drug effects | Plasminogen Activator Inhibitor 1 - genetics | Antibodies, Monoclonal - immunology | Aorta, Thoracic - pathology | Fibroblasts - metabolism | Heart - physiopathology | Heart Failure - complications | Angiotensin II - pharmacology | Matrix Metalloproteinase 2 - metabolism | Antibodies, Monoclonal - pharmacology | Connective Tissue Growth Factor - antagonists & inhibitors | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Ventricular Dysfunction, Left - etiology | Myocardium - pathology | Reverse Transcriptase Polymerase Chain Reaction | Pressure | Ventricular Dysfunction, Left - prevention & control | Weight-Bearing - physiology | Matrix Metalloproteinase 2 - genetics | Animals | Myocytes, Cardiac - drug effects | Fibroblasts - drug effects | Heart - drug effects | Myocytes, Cardiac - metabolism | Connective Tissue Growth Factor - genetics | Mice | Ventricular Remodeling - drug effects | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 5392 - 13
Peritoneal fibrosis (PF) is a serious complication in various clinical settings, but the mechanisms driving it remain to be fully determined. Connective tissue... 
TISSUE-GROWTH-FACTOR | DIALYSIS PATIENTS | MEMBRANE | MULTIDISCIPLINARY SCIENCES | LIVER FIBROSIS | RECEPTOR | MONOCLONAL-ANTIBODY | IDIOPATHIC PULMONARY-FIBROSIS | MORPHOLOGIC CHANGES | MOUSE EXPERIMENTAL-MODEL | MESOTHELIAL CELLS | NIH 3T3 Cells | Humans | Peritoneal Fibrosis - genetics | Transforming Growth Factor beta1 - metabolism | Green Fluorescent Proteins - genetics | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Neovascularization, Pathologic - pathology | Myofibroblasts - metabolism | Peritoneal Fibrosis - drug therapy | Collagen Type I - genetics | Neovascularization, Pathologic - prevention & control | Genes, Reporter | Disease Models, Animal | Fibroblasts - metabolism | Green Fluorescent Proteins - metabolism | Myofibroblasts - pathology | Collagen Type I - metabolism | Signal Transduction | Antibodies, Monoclonal - pharmacology | Connective Tissue Growth Factor - antagonists & inhibitors | Gene Expression Regulation | Angiogenesis Inhibitors - pharmacology | Mice, Transgenic | Peritoneal Fibrosis - pathology | Peritoneal Fibrosis - chemically induced | Transforming Growth Factor beta1 - genetics | Neovascularization, Pathologic - chemically induced | Fibroblasts - pathology | Myofibroblasts - drug effects | Animals | Chlorhexidine - analogs & derivatives | Cell Differentiation - drug effects | Fibroblasts - drug effects | Neovascularization, Pathologic - genetics | Connective Tissue Growth Factor - genetics | Mice | Connective Tissue Growth Factor - metabolism | Cell proliferation | Collagen (type I) | Mesenchyme | Connective tissue growth factor | Angiogenesis | Collagen | Rodents | Green fluorescent protein | Fibrosis | Fibroblasts | Monoclonal antibodies | Chlorhexidine | Vascular endothelial growth factor | Peritoneum
Journal Article
The Journal of Dermatology, ISSN 0385-2407, 01/2010, Volume 37, Issue 1, pp. 54 - 70
Systemic sclerosis (SSc) is an autoimmune disorder with clinical manifestations resulting from immune activation, fibrosis development and damage of small... 
immune dysfunction | fibrosis | vasculopathy | new treatments | scleroderma | Vasculopathy | Scleroderma | Immune dysfunction | Fibrosis | New treatments | PULMONARY ARTERIAL-HYPERTENSION | ACTIVATES LATENT TGF-BETA-1 | ANTIENDOTHELIAL CELL ANTIBODIES | BETA TYPE-I | TRANSCRIPTION FACTOR FLI1 | DERMATOLOGY | INTERSTITIAL LUNG-DISEASE | SCLERODERMA RENAL CRISIS | TRANSFORMING-GROWTH-FACTOR | PROTEIN-ASSOCIATED FACTOR | CONNECTIVE-TISSUE DISEASES | Endothelin-1 - antagonists & inhibitors | Autoimmune Diseases - physiopathology | Platelet-Derived Growth Factor - immunology | Receptors, Transforming Growth Factor beta - immunology | Connective Tissue Growth Factor - immunology | Humans | Phosphodiesterase 5 Inhibitors | Smad3 Protein - immunology | Scleroderma, Systemic - physiopathology | Transforming Growth Factor beta - antagonists & inhibitors | Autoimmune Diseases - drug therapy | Endothelin Receptor Antagonists | Transforming Growth Factor beta - immunology | Connective Tissue Growth Factor - antagonists & inhibitors | Autoimmune Diseases - immunology | Serotonin Uptake Inhibitors - therapeutic use | Piperazines - therapeutic use | Scleroderma, Systemic - immunology | Imatinib Mesylate | Smad3 Protein - antagonists & inhibitors | Randomized Controlled Trials as Topic | Endothelin-1 - immunology | Animals | B-Lymphocytes - immunology | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Pyrimidines - therapeutic use | Mice | Benzamides | Immunoglobulins - therapeutic use | Platelet-Derived Growth Factor - antagonists & inhibitors | Scleroderma, Systemic - drug therapy | Hypertension | Immunoglobulins | Platelet-derived growth factor | Peptides | Stem cells | Transplantation | Transforming growth factors
Journal Article