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1. Full Text Higher susceptibility of spontaneous and NNK‐induced lung neoplasms in connexin 43 deficient CD1 × AJ F1 mice: Paradoxical expression of connexin 43 during lung carcinogenesis
by Fukumasu, Heidge and Avanzo, Jose L and Sanches, Daniel S and Mennecier, Gregory and Mori, Claudia M.C and Dagli, Maria L. Z
Molecular Carcinogenesis, ISSN 0899-1987, 07/2013, Volume 52, Issue 7, pp. 497 - 506
Connexins (Cxs) are proteins that form the communicating gap junctions, and reportedly have a role in carcinogenesis. Here, we evaluated the importance of... 
lung cancer | tobacco‐specific nitrosamine | connexin 43 | beta‐catenin | Tobacco-specific nitrosamine | Connexin 43 | Beta-catenin | Lung cancer | Disease Susceptibility | Lung Neoplasms - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Lung Neoplasms - pathology | Male | Nitrosamines - toxicity | Reverse Transcriptase Polymerase Chain Reaction | beta Catenin - metabolism | Blotting, Western | Immunoenzyme Techniques | beta Catenin - genetics | Carcinogens - toxicity | Mice, Knockout | Animals | Connexin 43 - physiology | Lung Neoplasms - etiology | Female | Mice | Cell Transformation, Neoplastic - pathology | Real-Time Polymerase Chain Reaction | Proteins | Genotype & phenotype | Oncology | Gene expression | Index Medicus
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2. Full Text Connexin 43 Hemichannels Are Permeable to ATP
by Kang, Jian and Kang, Ning and Lovatt, Ditte and Torres, Arnulfo and Zhao, Zhuo and Lin, Jane and Nedergaard, Maiken
Journal of Neuroscience, ISSN 0270-6474, 04/2008, Volume 28, Issue 18, pp. 4702 - 4711
Astrocytes are electrically nonexcitable cells that communicate by means of Ca2+ signaling. Long-distance intercellular Ca2+ waves are initiated by release of... 
Cx43 hemichannel | Bioluminescence | Astrocyte | ATP | Gap junction | P2X | CELLS | RAT | GAP-JUNCTION HEMICHANNELS | RELEASE | ASTROCYTES | NEUROSCIENCES | INHIBITION | AMPLIFICATION | CONDUCTANCES | bioluminescence | gap junction | CHANNELS | NEOCORTICAL PYRAMIDAL NEURONS | astrocyte | Animals, Newborn | Green Fluorescent Proteins - metabolism | Tetraethylammonium - pharmacology | Connexin 43 - metabolism | Nitrobenzoates - pharmacology | Rats | Green Fluorescent Proteins - genetics | Rats, Sprague-Dawley | Adenosine Triphosphate - pharmacology | Carbenoxolone - pharmacology | Ion Channel Gating - physiology | Patch-Clamp Techniques | Animals | Transfection - methods | Analysis of Variance | Adenosine Triphosphate - metabolism | Mutation | In Vitro Techniques | Dose-Response Relationship, Radiation | Electric Stimulation - methods | Cell Line, Transformed | Potassium Channel Blockers - pharmacology | Connexin 43 - genetics | Ion Channel Gating - drug effects | Index Medicus
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3. Full Text Gating and Regulation of Connexin 43 (Cx43) Hemichannels
by Jorge E. Contreras and Juan C. Sáez and Feliksas F. Bukauskas and Michael V. L. Bennett
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2003, Volume 100, Issue 20, pp. 11388 - 11393
Connexin 43 (Cx43) nonjunctional or "unapposed" hemichannels can open under physiological or pathological conditions. We characterize hemichannels comprised of... 
Electric potential | Biological Sciences | Dyes | Gap junctions | Cell lines | HeLa cells | Fluorescence | Electrophysiology | Cultured cells | Connexins | Membrane potential | Dye uptake | Connexon | Gap junction | Permeation | BIOPHYSICAL PROPERTIES | GAP-JUNCTION CHANNELS | connexon | MULTIDISCIPLINARY SCIENCES | METABOLIC INHIBITION | PLASMA-MEMBRANE | GREEN FLUORESCENT PROTEIN | HELA-CELLS | permeation | CALCIUM | XENOPUS-OOCYTES | gap junction | ATP RELEASE | dye uptake | LIVING CELLS | Connexin 43 - chemistry | Green Fluorescent Proteins | Connexin 43 - physiology | Humans | Luminescent Proteins - chemistry | HeLa Cells | Recombinant Fusion Proteins - chemistry | Ion Channel Gating | Cells | Research | Proteins | Biophysics | Permeability | Mammals | Experiments | Index Medicus
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4. Full Text Possible role of gap junction intercellular channels and connexin 43 in satellite glial cells (SGCs) for preservation of human spiral ganglion neurons
: A comparative study with clinical implications
by Liu, Wei and Glueckert, Rudolf and Linthicum, Fred H and Rieger, Gunde and Blumer, Michael and Bitsche, Mario and Pechriggl, Elisabeth and Rask-Andersen, Helge and Schrott-Fischer, Annelies and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Öron-, näs- och halssjukdomar and Uppsala universitet and Institutionen för kirurgiska vetenskaper
Cell and Tissue Research, ISSN 0302-766X, 2/2014, Volume 355, Issue 2, pp. 267 - 278
Human spiral ganglion (SG) neurons show remarkable survival properties and maintain electric excitability for a long time after complete deafness and even... 
Human Genetics | Human | Spiral ganglion | Biomedicine | Guinea pig | Proteomics | Cochlear implant | Molecular Medicine | Connexin 43 | Gap junction | Satellite glial cells | TRIGEMINAL GANGLION | INNER-EAR | PROTEIN CONNEXIN43 | DEGENERATION | CELL BIOLOGY | CORTI LOSS | ADULT-RAT | COCHLEAR NERVE | SENSORY GANGLIA | EXPRESSION | RAT COCHLEA | Immunohistochemistry | Gap Junctions - metabolism | Spiral Ganglion - ultrastructure | Guinea Pigs | Connexin 43 - metabolism | Humans | Neurons - cytology | Spiral Ganglion - metabolism | Spiral Ganglion - cytology | Gap Junctions - ultrastructure | Extracellular Space - metabolism | Neuroglia - cytology | Animals | Neuroglia - metabolism | Deafness | Implants, Artificial | Prosthesis | Neurons | Comparative analysis | Electron microscopy | Ganglion | Myelin proteins | Cells | Index Medicus | Regular | Medical and Health Sciences | Medicin och hälsovetenskap
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5. Full Text ATP and glutamate released via astroglial connexin 43 hemichannels mediate neuronal death through activation of pannexin 1 hemichannels
by Orellana, Juan A and Froger, Nicolas and Ezan, Pascal and Jiang, Jean X and Bennett, Michael V. L and Naus, Christian C and Giaume, Christian and Sáez, Juan C
Journal of Neurochemistry, ISSN 0022-3042, 09/2011, Volume 118, Issue 5, pp. 826 - 840
Inflammation contributes to neurodegeneration in post-ischemic brain, diabetes, and Alzheimer's disease. Participants in this inflammatory response include... 
gap junctions | Alzheimer’s disease | amyloid β‐peptide | cytokines | diabetes mellitus | pannexin | stroke | connexin | amyloid β-peptide | Alzheimer's disease | BRAIN-INJURY | GAP JUNCTIONAL COMMUNICATION | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMYLOID PLAQUES | ASTROCYTES | NEUROTOXICITY | NEUROSCIENCES | BETA | IN-VIVO | CHANNELS | RECEPTORS | amyloid beta-peptide | Tumor Necrosis Factor-alpha - metabolism | Oxygen - pharmacology | Cell Hypoxia - physiology | Amyloid beta-Peptides - pharmacology | Microtubule-Associated Proteins - pharmacology | Culture Media, Conditioned - pharmacology | Lanthanum - pharmacology | Peptide Fragments - pharmacology | Cerebral Cortex - cytology | Adenosine Triphosphate - pharmacology | Time Factors | Interleukin-1beta - metabolism | Adenosine Triphosphate - metabolism | Neuropeptides - pharmacology | Nerve Tissue Proteins - pharmacology | Neurons - physiology | Astrocytes - chemistry | Female | Cell Death - drug effects | Connexins - pharmacology | Neurons - drug effects | Animals, Newborn | Astrocytes - drug effects | Cell Hypoxia - drug effects | Connexin 43 - metabolism | Glutamic Acid - pharmacology | Cells, Cultured | Fluoresceins | Coculture Techniques - methods | Connexin 43 - deficiency | Connexins - metabolism | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Biotinylation - methods | Glutamic Acid - metabolism | Mice | Astrocytes - metabolism | Methyl aspartate | Neurosciences | Peptides | Neurons | Diabetes | Glucose | Glutamate | Dextrose | Index Medicus
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6. Full Text Astroglial connexin 43 sustains glutamatergic synaptic efficacy
by Chever, Oana and Pannasch, Ulrike and Ezan, Pascal and Rouach, Nathalie
Philosophical Transactions of the Royal Society B: Biological Sciences, ISSN 0962-8436, 10/2014, Volume 369, Issue 1654, pp. 20130596 - 20130596
Astrocytes dynamic interactions with neurons play an active role in neurotransmission. The gap junction (GJ) subunits connexins 43 and 30 are strongly... 
Connexin | Astrocytes | Hippocampus | Synapses | synapses | astrocytes | hippocampus | connexin | ASTROCYTE-DIRECTED INACTIVATION | PASSIVE CONDUCTANCE | TRAFFICKING | NETWORKS | TRANSMISSION | GAP-JUNCTIONS | HEMICHANNELS | BIOLOGY | MICE | EXPRESSION | COMMUNICATION | Immunohistochemistry | Synaptic Transmission - physiology | Connexin 43 - metabolism | Immunoblotting | Hippocampus - cytology | Mice, Knockout | Patch-Clamp Techniques | Animals | Synapses - metabolism | Analysis of Variance | Pyramidal Cells - physiology | Glutamic Acid - metabolism | Mice | Hippocampus - physiology | Astrocytes - metabolism | Connexin 43 - genetics | Index Medicus | 133 | 202 | 1001
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7. Full Text Connexin 43 inhibition sensitizes chemoresistant glioblastoma cells to temozolomide
by Murphy, Susan F and Varghese, Robin T and Lamouille, Samy and Guo, Sujuan and Pridham, Kevin J and Kanabur, Pratik and Osimani, Alyssa M and Sharma, Shaan and Jourdan, Jane and Rodgers, Cara M and Simonds, Gary R and Gourdie, Robert G and Sheng, Zhi
Cancer Research, ISSN 0008-5472, 01/2016, Volume 76, Issue 1, pp. 139 - 149
Resistance of glioblastoma (GBM) to the front-line chemotherapeutic agent temozolomide (TMZ) continues to challenge GBM treatment efforts. The repair of... 
GLIOMA-CELLS | TRIAL | SURVIVAL | STEM-CELLS | METHYLTRANSFERASE | ONCOLOGY | RESISTANCE | MIMETIC PEPTIDE | RADIOTHERAPY | ADJUVANT TEMOZOLOMIDE | ZONULA OCCLUDENS-1 | Dacarbazine - administration & dosage | Connexin 43 - metabolism | Signal Transduction | Humans | Brain Neoplasms - drug therapy | Brain Neoplasms - metabolism | Peptides - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Peptides - pharmacology | Drug Synergism | Xenograft Model Antitumor Assays | Biomimetic Materials - pharmacology | Animals | Dacarbazine - pharmacology | Mice, Nude | Dacarbazine - analogs & derivatives | Cell Line, Tumor | Glioblastoma - metabolism | Connexin 43 - antagonists & inhibitors | Mice | Mice, Inbred BALB C | Glioblastoma - drug therapy | Index Medicus
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8. Full Text Nanoengineering the heart: Conductive scaffolds enhance connexin 43 expression
by You, Jin-Oh and Rafat, Marjan and Ye, George J. C and Auguste, Debra T
Nano Letters, ISSN 1530-6984, 09/2011, Volume 11, Issue 9, pp. 3643 - 3648
Scaffolds that couple electrical and elastic properties may be valuable for cardiac cell function. However, existing conductive materials do not mimic... 
HEMA | Au nanoparticles | connexin 43 | Sxcaffold | cardiomyocyte | conductive | GOLD NANOPARTICLES | PHYSICS, CONDENSED MATTER | PHYSICS, APPLIED | POLYPYRROLE | MATERIALS SCIENCE, MULTIDISCIPLINARY | CARDIAC MYOCYTES | TISSUE | CHITOSAN SCAFFOLDS | CHEMISTRY, PHYSICAL | NANOSCIENCE & NANOTECHNOLOGY | Scaffold | MECHANICAL-PROPERTIES | CHEMISTRY, MULTIDISCIPLINARY | IN-VITRO | SURFACE-DENSITY | STIFFNESS | ELECTRICAL-STIMULATION | Animals, Newborn | Gold - chemistry | Myocytes, Cardiac - cytology | Nanotechnology - methods | Heart - physiology | Metal Nanoparticles - chemistry | Gene Expression Regulation | Connexin 43 - biosynthesis | Rats | Electric Conductivity | Methacrylates - chemistry | Animals | Anisotropy | Myocardium - metabolism | Polymers - chemistry | Nanoparticles | Hydrogels | Stimulation | Modulus of elasticity | Joining | Polypyrroles | Nanostructure | Scaffolds | Nanoengineering | Index Medicus
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9. Full Text Selectivity of Connexin 43 Channels Is Regulated Through Protein Kinase C–Dependent Phosphorylation
by Ek-Vitorin, Jose F and King, Timothy J and Heyman, Nathanael S and Lampe, Paul D and Burt, Janis M
Circulation Research, ISSN 0009-7330, 06/2006, Volume 98, Issue 12, pp. 1498 - 1505
Coordinated contractile activation of the heart and resistance to ischemic injury depend, in part, on the intercellular communication mediated by Cx43-composed... 
Phosphorylation | Selectivity | Connexin 43 | Ischemia | Gap junction | selectivity | CONDUCTANCE | GAP-JUNCTION CHANNELS | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PERMEABILITY | ADENOSINE | ischemia | HEART | INTERCELLULAR COMMUNICATION | PERIPHERAL VASCULAR DISEASE | gap junction | connexin 43 | HEMATOLOGY | phosphorylation | EXPRESSION | Gap Junctions - metabolism | Myocardial Ischemia - metabolism | Cricetinae | Connexin 43 - metabolism | Cricetulus | Cells, Cultured | Intercellular Junctions - metabolism | Rats | Permeability | Mice, Inbred Strains | Animals | Ion Channels - metabolism | Protein Kinase C - metabolism | Coloring Agents - pharmacokinetics | Mice | CHO Cells | Index Medicus
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10. Full Text HIV-Associated Cardiovascular Disease: Role of Connexin 43
by Prevedel, Lisa and Morocho, Camilla and Bennett, Michael V.L and Eugenin, Eliseo A
American Journal of Pathology, ISSN 0002-9440, 09/2017, Volume 187, Issue 9, pp. 1960 - 1970
Chronic HIV infection due to effective antiretroviral treatment has resulted in a broad range of clinical complications, including accelerated heart disease.... 
INDIVIDUALS | HUMAN ASTROCYTES | GAP-JUNCTION CHANNELS | CONNEXIN-43 | PROTEIN | CA2 | ANTIRETROVIRAL THERAPY | RAT-HEART | DYSFUNCTION | PATHOLOGY | HUMAN-IMMUNODEFICIENCY-VIRUS | Cardiovascular Diseases - etiology | Cardiovascular Diseases - metabolism | Connexin 43 - metabolism | Humans | Middle Aged | Male | CD4 Lymphocyte Count | Young Adult | Inflammation - metabolism | Myocardium - metabolism | HIV Infections - complications | Aged, 80 and over | Adult | Female | Aged | HIV Infections - metabolism | Index Medicus | Abridged Index Medicus
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11. Full Text Connexin 43 is critical to maintain the homeostasis of the blood–testis barrier via its effects on tight junction reassembly
by Michelle W. M. Li and Dolores D. Mruk and Will M. Lee and C. Yan Cheng and Ryuzo Yanagimachi
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2010, Volume 107, Issue 42, pp. 17998 - 18003
In mammalian testes, the blood–testis barrier (BTB) or Sertoli cell barrier created by specialized junctions between Sertoli cells near the basement membrane... 
Calcium | Bisphenols | Gap junctions | Small interfering RNA | Cadherins | Connexins | Testes | Epithelium | Sertoli cells | Metrorrhagia | Bisphenol A | Calcium switch | Seminiferous epithelial cycle | Spermatogenesis | Gap junction | ANTIBODIES | CELLS | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | GAP-JUNCTIONS | spermatogenesis | DYNAMICS | seminiferous epithelial cycle | bisphenol A | gap junction | KNOCKOUT | MICE | PROTEINS | EXPRESSION | calcium switch | Blood-Testis Barrier | Connexin 43 - physiology | Benzhydryl Compounds | RNA Interference | Homeostasis - physiology | Humans | Tight Junctions | Male | Sertoli Cells - drug effects | Phenols - pharmacology | Bisphenol-A | Homeostasis | Models | Research | Properties | Proteins | Membranes | Cellular biology | Cell division | Reproductive system | Gene expression | Chemical compounds | Blood | Index Medicus | Biological Sciences
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12. Full Text Osteocytic connexin 43 channels affect fracture healing
by Chen, Yunhe and Chen, Meng and Xue, Tong and Li, Guobin and Wang, Dongen and Shang, Peng and Jiang, Jean X and Xu, Huiyun
Journal of Cellular Physiology, ISSN 0021-9541, 11/2019, Volume 234, Issue 11, pp. 19824 - 19832
The cross‐talk between cells is very critical for moving forward fracture healing in an orderly manner. Connexin (Cx) 43‐formed gap junctions and hemichannels... 
gap junction | Cx43 | transgenic mouse model | hemichannel | fracture healing | OSTEOBLAST | PHYSIOLOGY | SCLEROSTIN ANTIBODY | RELEASE | CELL BIOLOGY | OSTEOCLASTOGENESIS | REPAIR | GAP-JUNCTIONS | HEMICHANNELS | MICE | BONE | Analysis | Genetic engineering | Fractures | Frogs | Animal models | Gap junctions | Transgenic mice | Osteocytes | Connexin 43 | Bone healing | Promotion | Cell interactions | Osteoblasts | Channels | Mutants | Osteoclastogenesis | Bone growth | Healing | Biocompatibility | Mice | Bone | Repair | Osteogenesis | Index Medicus
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13. Full Text Theaflavin 3,3′‐digallate reverses the downregulation of connexin 43 and autophagy induced by high glucose via AMPK activation in cardiomyocytes
by Shen, Zhida and Chen, Qi and Jin, Tingting and Wang, Meihui and Ying, Hangying and Lu, Jiangting and Wang, Ming and Zhang, Wenbin and Qiu, Fuyu and Jin, Chongying and Zhao, Yanbo and Fu, Guosheng
Journal of Cellular Physiology, ISSN 0021-9541, 10/2019, Volume 234, Issue 10, pp. 17999 - 18016
Theaflavin 3,3′‐digallate (TF3), is reported to protect cardiomyocytes from lipotoxicity and reperfusion injury. However, the role of TF3 in the protection of... 
cardiomyocytes | AMPK | autophagy | Cx43 | theaflavin 3 | 3′‐digallate | PHYSIOLOGY | MECHANISMS | DIABETIC CARDIOMYOPATHY | CELL BIOLOGY | HEART | 3 '-digallate | PATHWAY | INFLAMMATION | CACO-2 CELL MONOLAYERS | DYSFUNCTION | STRESS | EXPRESSION | INHIBIT | Proteins | Adenosine | Glucose | Protein kinases | Dextrose | Heart cells | Neonates | Intercellular signalling | Adenosine monophosphate | AMP | Gap junctions | Cardiomyocytes | Activation | siRNA | Connexin 43 | Kinases | Autophagy | Time dependence | Reperfusion | Protein kinase | Adenosine kinase | Connexins | Inhibition | Phagocytosis
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14. Full Text Astroglial connexin 43 hemichannels modulate olfactory bulb slow oscillations
by Roux, Lisa and Madar, Antoine and Lacroix, Marie Masako and Yi, Chenju and Benchenane, Karim and Giaume, Christian
Journal of Neuroscience, ISSN 0270-6474, 11/2015, Volume 35, Issue 46, pp. 15339 - 15352
An emergent concept in neurosciences consists in considering brain functions as the product of dynamic interactions between neurons and glial cells,... 
Olfactory glomeruli | UP state | DOWN state | Astrocyte | Mitral cells | Hemichannel | GAP-JUNCTION CHANNELS | olfactory glomeruli | GLUTAMATE RELEASE | hemichannel | HIPPOCAMPAL ASTROCYTES | SYNAPTIC-TRANSMISSION | FEEDBACK INHIBITION | NEUROSCIENCES | LAMINAR DISTRIBUTION | EXTERNAL TUFTED CELLS | mitral cells | IN-VIVO | astrocyte | NEURON-GLIA INTERACTIONS | Membrane Potentials - genetics | Synaptic Transmission - genetics | Adenosine A1 Receptor Antagonists - pharmacology | Nerve Net - drug effects | Xanthines - pharmacology | Excitatory Postsynaptic Potentials - drug effects | Nerve Net - physiology | Biological Clocks - drug effects | Synaptic Transmission - drug effects | Tetrodotoxin - pharmacology | Excitatory Postsynaptic Potentials - genetics | Connexin 43 - genetics | Olfactory Bulb - cytology | Animals, Newborn | Membrane Potentials - drug effects | Connexins - deficiency | Connexin 43 - metabolism | Mice, Inbred C57BL | Connexins - genetics | Connexin 30 | Mice, Transgenic | Sodium Channel Blockers - pharmacology | Membrane Potentials - physiology | Peptides - pharmacology | Carbenoxolone - pharmacology | Animals | Biological Clocks - physiology | Biological Clocks - genetics | Olfactory Bulb - physiology | Glutamic Acid - metabolism | Mice | In Vitro Techniques | Astrocytes - metabolism | Index Medicus
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15. Full Text N ε -lysine acetylation determines dissociation from GAP junctions and lateralization of connexin 43 in normal and dystrophic heart
by Claudia Colussi and Jessica Rosati and Stefania Straino and Francesco Spallotta and Roberta Berni and Donatella Stilli and Stefano Rossi and Ezio Musso and Emilio Macchi and Antonello Mai and Gianluca Sbardella and Sabrina Castellano and Cristina Chimenti and Andrea Frustaci and Angela Nebbioso and Lucia Altucci and Maurizio C. Capogrossi and Carlo Gaetano and Eric N. Olson
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2011, Volume 108, Issue 7, pp. 2795 - 2800
Wanting to explore the epigenetic basis of Duchenne cardiomyopathy, we found that global histone acetylase activity was abnormally elevated and the acetylase... 
Heart | Intercellular junctions | 3T3 cells | Histone deacetylase inhibitors | Gap junctions | Histones | Epigenetics | Myocardium | Connexins | Acetylation | Protein acetylation | Muscular dystrophy | MYOCARDIUM | SMALL-MOLECULE MODULATORS | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | MDX MICE | muscular dystrophy | protein acetylation | NITRIC-OXIDE | DUCHENNE MUSCULAR-DYSTROPHY | HISTONE DEACETYLASE INHIBITORS | PROTEINS | EXPRESSION | ACETYLTRANSFERASES | Gap Junctions - metabolism | Immunoprecipitation | Connexin 43 - metabolism | Muscular Dystrophy, Duchenne - complications | Cardiomyopathies - etiology | Hydroxamic Acids | p300-CBP Transcription Factors - metabolism | Acetylation - drug effects