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Molecular Therapy, ISSN 1525-0016, 11/2017, Volume 25, Issue 11, pp. 2513 - 2525
A causal role of hypercholesterolemia in non-ischemic heart failure has never been demonstrated. Adeno-associated viral serotype 8 (AAV8) (AAV8 ) gene transfer... 
metabolic remodeling | heart failure | gene transfer | low-density lipoprotein receptor | cholesterol-lowering therapy | adeno-associated viral vectors | gene therapy | cardiac hypertrophy | non-ischemic cardiomyopathy | hypercholesterolemia | MAMMALIAN TARGET | MEDICINE, RESEARCH & EXPERIMENTAL | PRESSURE-OVERLOAD | OXIDATIVE STRESS | LIPID-METABOLISM | FATTY-ACID-METABOLISM | PROLIFERATOR-ACTIVATED RECEPTOR | CELL-GROWTH | COENZYME Q | HYPERTROPHIED RAT-HEART | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | CARDIAC-HYPERTROPHY | GENETICS & HEREDITY | Constriction, Pathologic - metabolism | Dependovirus - genetics | Constriction, Pathologic - pathology | Genetic Vectors - administration & dosage | TOR Serine-Threonine Kinases - metabolism | Cardiomegaly - etiology | Cardiomyopathies - etiology | TOR Serine-Threonine Kinases - genetics | Cardiomegaly - mortality | Cardiomyopathies - mortality | Myocardium - metabolism | Receptors, LDL - deficiency | Female | Heart Function Tests | Biomarkers - metabolism | Receptors, LDL - genetics | Gene Expression | Dependovirus - metabolism | Genetic Vectors - chemistry | Genetic Vectors - metabolism | Myocardium - pathology | Cholesterol - metabolism | Constriction, Pathologic - complications | Cardiomyopathies - therapy | Mice, Knockout | Acetyl-CoA C-Acetyltransferase - genetics | Animals | Aorta - surgery | Cardiomyopathies - metabolism | Survival Analysis | Mice | Hemodynamics | Acetyl-CoA C-Acetyltransferase - metabolism | Cardiomegaly - metabolism | Cardiomegaly - therapy | Genetic Therapy - methods | TOR protein | Oxidative stress | Cardiomyopathy | Cardiovascular disease | Ischemia | Conflicts of interest | Aorta | Coenzyme A | Lipoprotein (low density) receptors | Heart diseases | Heart failure | Mortality | Cardiomyocytes | Rapamycin | LDLR gene | Metabolism | Pressure | Cholesterol | Lipoproteins | Hypercholesterolemia | Magnetic resonance imaging | Coronary vessels | Cardiac function | Fibrosis | Receptor density | Software | Gene therapy | Hypertrophy | Apoptosis | Index Medicus | Original
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/2014, Volume 289, Issue 30, pp. 20939 - 20952
Journal Article
Inflammatory Bowel Diseases, ISSN 1078-0998, 04/2013, Volume 19, Issue 5, pp. 891 - 903
Journal Article
Journal of Comparative Neurology, ISSN 0021-9967, 10/2017, Volume 525, Issue 15, pp. 3266 - 3285
Aging‐associated ependymal‐cell pathologies can manifest as ventricular gliosis, ventricle enlargement, or ventricle stenosis. Ventricle stenosis and fusion of... 
RRID:AB_2315304 | RRID:AB_1555288 | lateral ventricle | ventricle stenosis | ependymal cell | RRID:SCR_002285 | RRID:AB_2620025 | RRID:AB_221568 | RRID:AB_221569 | parvalbumin | RRID:AB_2665495 | aging | RRID:AB_10000344 | RRID:SCR_002798 | RRID:AB_839504 | RRID:SCR_002526 | PROTEIN | RAT | NICHE | ADULT MAMMALIAN BRAIN | SUBVENTRICULAR ZONE | ZOOLOGY | SURFACE | NEURONAL MIGRATION | LOCALIZATION | ASTROCYTES | NEUROSCIENCES | NEUROGENESIS | Constriction, Pathologic - metabolism | Microglia - metabolism | Constriction, Pathologic - pathology | Ependyma - pathology | Lateral Ventricles - pathology | Parvalbumins - metabolism | Lateral Ventricles - metabolism | Male | Green Fluorescent Proteins - genetics | Glial Fibrillary Acidic Protein - metabolism | Gliosis - pathology | Forkhead Transcription Factors - metabolism | Microglia - pathology | Parvalbumins - genetics | Female | S100 Calcium Binding Protein beta Subunit - metabolism | Green Fluorescent Proteins - metabolism | Gliosis - metabolism | Mice, Inbred C57BL | Ependyma - metabolism | Mice, Transgenic | Forkhead Transcription Factors - genetics | Aging - pathology | Microscopy, Confocal | Animals | Cell Adhesion - physiology | Fluorescent Antibody Technique | Aging - metabolism | Stenosis | Adhesions | Stem cells | Solar cells | Glial fibrillary acidic protein | S100b protein | Ventricles (cerebral) | Activation | Parvalbumin | Subventricular zone | Ependymal cells | Gliosis | Photovoltaic cells | Rodents | Aging | Mice | Ventricle | Immunofluorescence | Enlargement | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, pp. e53133 - e53133
Background: Left ventricular hypertrophy is enhanced by an inflammatory state and stimulation of various cytokines. Pentraxin 3 (PTX3) is rapidly produced in... 
C-REACTIVE PROTEIN | CHRONIC HEART-FAILURE | TUMOR-NECROSIS-FACTOR | MULTIDISCIPLINARY SCIENCES | ACUTE MYOCARDIAL-INFARCTION | ANGIOTENSIN-II | INDUCED CARDIAC-HYPERTROPHY | ADVERSE CLINICAL-OUTCOMES | CAMP EARLY REPRESSOR | NF-KAPPA B | INNATE IMMUNITY | Constriction, Pathologic - metabolism | Constriction, Pathologic - pathology | Phosphorylation | Humans | NF-kappa B - metabolism | Aorta - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Myofibroblasts - metabolism | C-Reactive Protein - metabolism | Hypertrophy, Left Ventricular - genetics | Ventricular Dysfunction, Left - genetics | Hypertrophy, Left Ventricular - pathology | Myocardium - metabolism | Ultrasonography | Ventricular Dysfunction, Left - pathology | Interleukin-6 - metabolism | Myofibroblasts - pathology | Aorta - diagnostic imaging | Interleukin-6 - genetics | Hypertrophy, Left Ventricular - metabolism | Hydrogen Peroxide - pharmacology | Constriction, Pathologic - genetics | Mice, Transgenic | Myocardium - pathology | C-Reactive Protein - genetics | Myofibroblasts - drug effects | Nerve Tissue Proteins - genetics | Ventricular Dysfunction, Left - diagnostic imaging | Aorta - pathology | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Ventricular Dysfunction, Left - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | NF-kappa B - genetics | Signal Transduction - drug effects | Fibrosis | Myocytes, Cardiac - metabolism | Connective Tissue Growth Factor - genetics | Mice | Connective Tissue Growth Factor - metabolism | Hypertrophy, Left Ventricular - diagnostic imaging | Heart | Heart failure | Medical research | Interleukins | Heart enlargement | Medicine, Experimental | Genetic engineering | Nephrology | Hydrogen peroxide | Interleukin | Smooth muscle | Myocytes | Kinases | Eutrophication | Interleukin 6 | Proteins | Connective tissues | Rodents | Fibroblasts | Aorta | Cardiology | Heart diseases | Pentraxins | Recombinant | Echocardiography | Cytokines | Hematology | Transgenic mice | Extracellular signal-regulated kinase | Cardiomyocytes | Connective tissue growth factor | Inflammation | Gene expression | Pressure | Medicine | Coronary vessels | Ventricle | Laboratory animals | Hypertrophy | Index Medicus
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2008, Volume 50, Issue 1, pp. 69 - 79
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, pp. e104040 - e104040
The gene ankyrin repeat domain 1 (Ankrd1) is an enigmatic gene and may exert pleiotropic function dependent on its expression level, subcellular localization... 
ANKRD1 MUTATIONS | SKELETAL-MUSCLE | ANKYRIN REPEAT PROTEIN | CALPAIN | PATHWAY | MULTIDISCIPLINARY SCIENCES | CARDIAC-HYPERTROPHY | HEART-FAILURE | DISEASE | GENE-EXPRESSION | DILATED CARDIOMYOPATHY | Constriction, Pathologic - metabolism | RNA, Small Interfering - genetics | Constriction, Pathologic - pathology | Calpain - metabolism | Tetrazoles - pharmacology | Cyclosporine - pharmacology | Myosin Heavy Chains - genetics | Cardiomegaly - pathology | Aorta - metabolism | Calpain - genetics | Glycoproteins - pharmacology | Myosin Heavy Chains - metabolism | Repressor Proteins - antagonists & inhibitors | Calcineurin - genetics | Atrial Natriuretic Factor - genetics | Muscle Proteins - metabolism | Adenoviridae - genetics | Muscle Proteins - antagonists & inhibitors | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Animals, Newborn | Angiotensin II - pharmacology | Atrial Natriuretic Factor - metabolism | Signal Transduction | Aorta - drug effects | Gene Expression Regulation | Constriction, Pathologic - genetics | Rats | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Imidazoles - pharmacology | Protein Transport | Aorta - pathology | Muscle Proteins - genetics | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Nuclear Proteins - antagonists & inhibitors | Myocytes, Cardiac - metabolism | Mice | Primary Cell Culture | Cardiomegaly - genetics | Genetic Vectors | Calcineurin - metabolism | Cardiomegaly - metabolism | RNA, Small Interfering - metabolism | Infection | RNA | Adenoviruses | Angiotensin | Myosin | Calpain | Muscle proteins | Health aspects | Hypertrophy | Natriuretic peptides | Heart | Neonates | Transcription factors | Cardiomyopathy | Calcineurin | Body weight | Atrial natriuretic peptide | Cyclosporin A | NF-AT protein | Proteins | Signal transduction | Lymphocytes | Rodents | Aorta | Angiotensin II | Localization | Cardiology | Heart diseases | Recombinant | Heart failure | Translocation | Carp | Cardiomyocytes | Gene expression | Ribonucleic acid--RNA | Pressure | Nuclear transport | Musculoskeletal system | Hospitals | Ankyrin | Ventricle | Mutation | Laboratory animals | Index Medicus | Ribonucleic acid
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, pp. e110950 - e110950
Journal Article