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Blood, ISSN 0006-4971, 05/2014, Volume 123, Issue 19, pp. 2968 - 2977
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2011, Volume 108, Issue 24, pp. 9863 - 9868
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2010, Volume 107, Issue 46, pp. 19879 - 19884
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2007, Volume 282, Issue 19, pp. 14515 - 14524
Journal Article
Circulation Research, ISSN 0009-7330, 03/2009, Volume 104, Issue 6, pp. 733 - 741
Vascular calcification is a major risk factor for cardiovascular morbidity and mortality. To develop appropriate prevention and/or therapeutic strategies for... 
Vascular calcification | Smooth muscle cells | Genetic fate mapping | Lineage reprogramming | Runx2/Cbfa1 | WALL | CARDIAC & CARDIOVASCULAR SYSTEMS | vascular calcification | ATHEROSCLEROSIS | lineage reprogramming | genetic fate mapping | CARTILAGE | smooth muscle cells | IN-VITRO | DEFICIENT | CALCIFICATION | PERIPHERAL VASCULAR DISEASE | MICE | MUTATIONS | MATRIX-GLA-PROTEIN | HEMATOLOGY | EXPRESSION | Calcinosis - genetics | Vascular Diseases - pathology | Homeodomain Proteins - metabolism | Humans | Myocytes, Smooth Muscle - pathology | Extracellular Signal-Regulated MAP Kinases - metabolism | RNA, Messenger - metabolism | Stem Cells - metabolism | Wnt Proteins - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Arteries - metabolism | MAP Kinase Signaling System - genetics | Wnt Proteins - genetics | Mice, Mutant Strains | Bone Morphogenetic Protein 2 - metabolism | Calcinosis - metabolism | Myocytes, Smooth Muscle - metabolism | Chondrocytes - metabolism | SOX9 Transcription Factor - metabolism | Bone Morphogenetic Protein 2 - genetics | Chondrocytes - pathology | RNA, Messenger - genetics | Vascular Diseases - genetics | Arteries - pathology | Transcription Factors - genetics | Down-Regulation - genetics | Homeodomain Proteins - genetics | Mice, Knockout | Proteins - genetics | Transcription Factors - metabolism | Animals | Proteins - metabolism | Sp7 Transcription Factor | Stem Cells - pathology | Wnt3 Protein | Mice | Calcinosis - pathology | Vascular Diseases - metabolism | Wnt3A Protein | Cell Dedifferentiation - genetics | SOX9 Transcription Factor - genetics | Core Binding Factor Alpha 1 Subunit - genetics | Cbfa1 | Runx2
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2015, Volume 195, Issue 1, pp. 217 - 226
Proper regulation of microbial-induced cytokines is critical to intestinal immune homeostasis. Acute stimulation of nucleotide-binding oligomerization domain 2... 
CELLS | PROTEIN | CROHNS-DISEASE | MURAMYL DIPEPTIDE | SAETHRE-CHOTZEN SYNDROME | CROSS-TOLERANCE | IMMUNOLOGY | NF-KAPPA-B | C-MAF | HUMAN MONOCYTES | IL-10 GENE-EXPRESSION | RNA, Small Interfering - genetics | Humans | Polycomb Repressive Complex 1 - immunology | Nod2 Signaling Adaptor Protein - genetics | Core Binding Factor Alpha 1 Subunit - antagonists & inhibitors | Activating Transcription Factor 4 - immunology | Polycomb Repressive Complex 1 - genetics | RNA, Small Interfering - immunology | CCAAT-Enhancer-Binding Proteins - antagonists & inhibitors | Twist-Related Protein 1 - immunology | Transforming Growth Factor beta - antagonists & inhibitors | Nod2 Signaling Adaptor Protein - agonists | Transcription, Genetic | CCAAT-Enhancer-Binding Proteins - genetics | Nuclear Proteins - genetics | Macrophages - immunology | Proto-Oncogene Proteins c-maf - immunology | Transforming Growth Factor beta - immunology | Promoter Regions, Genetic | Activating Transcription Factor 4 - antagonists & inhibitors | Signal Transduction | Core Binding Factor Alpha 2 Subunit - antagonists & inhibitors | Proto-Oncogene Proteins c-maf - genetics | Gene Expression Regulation | Activating Transcription Factor 4 - genetics | Core Binding Factor Alpha 1 Subunit - immunology | Nod2 Signaling Adaptor Protein - immunology | Repressor Proteins - genetics | Antibodies, Neutralizing - pharmacology | Macrophages - cytology | Nuclear Proteins - immunology | Acetylmuramyl-Alanyl-Isoglutamine - pharmacology | Core Binding Factor Alpha 2 Subunit - immunology | Transforming Growth Factor beta - genetics | CCAAT-Enhancer-Binding Proteins - immunology | Interleukin-10 - antagonists & inhibitors | Interleukin-10 - genetics | Repressor Proteins - immunology | Twist-Related Protein 1 - genetics | Protein Binding | Macrophages - drug effects | Primary Cell Culture | Macrophage Activation - drug effects | Core Binding Factor Alpha 2 Subunit - genetics | Interleukin-10 - immunology | Core Binding Factor Alpha 1 Subunit - genetics
Journal Article
Molecular Endocrinology, ISSN 0888-8809, 07/2016, Volume 30, Issue 7, pp. 733 - 747
Journal Article
Experimental Cell Research, ISSN 0014-4827, 12/2017, Volume 361, Issue 2, pp. 257 - 264
Deregulation of the bone morphogenetic protein (BMP) pathway has been documented in colorectal cancer (CRC). Previously, we investigated possible associations... 
Mouse model | Runx | Smad | Bone morphogenetic proteins (BMP) | Dextran sodium sulfate (DSS) | Colorectal cancer (CRC) | MORPHOGENETIC PROTEIN PATHWAY | TGF-BETA | TUMOR | PLASMINOGEN-ACTIVATOR SYSTEM | MOLECULAR JEKYLL | CELL BIOLOGY | CLAUDIN-1 | ONCOLOGY | COLORECTAL-CANCER | GROWTH | GASTRIC EPITHELIAL-CELLS | CARCINOMA | Dextran Sulfate | Core Binding Factor Alpha 3 Subunit - metabolism | Colitis - genetics | Colorectal Neoplasms - genetics | Humans | Gene Expression Regulation, Neoplastic | Colitis - pathology | Core Binding Factor Alpha 1 Subunit - metabolism | Bone Morphogenetic Proteins - metabolism | Snail Family Transcription Factors - genetics | Claudin-1 - metabolism | Colitis - chemically induced | Transcription, Genetic | Bone Morphogenetic Proteins - genetics | Colorectal Neoplasms - metabolism | Disease Models, Animal | Snail Family Transcription Factors - metabolism | Core Binding Factor Alpha 2 Subunit - metabolism | Signal Transduction | Smad Proteins - genetics | Bcl-2-Like Protein 11 - genetics | Urokinase-Type Plasminogen Activator - genetics | Colorectal Neoplasms - chemically induced | Mice, Knockout | Animals | Bcl-2-Like Protein 11 - metabolism | Claudin-1 - genetics | Colitis - metabolism | Core Binding Factor Alpha 3 Subunit - genetics | Mice | Colorectal Neoplasms - pathology | Core Binding Factor Alpha 2 Subunit - genetics | Smad Proteins - metabolism | Core Binding Factor Alpha 1 Subunit - genetics | Urokinase-Type Plasminogen Activator - deficiency | Dextran | Dysplasia | Analysis | Colorectal cancer | Bone morphogenetic proteins | Genetic aspects | Cellular signal transduction | Inflammation | DNA binding proteins | Genetic transcription | Transforming growth factors | Sulfates | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e46483
Background: Bone fracture initiates a series of cellular and molecular events including the expression of hypoxia-inducible factor (HIF)-1. HIF-1 is known to... 
OXYGEN | CAPACITY | STEM-CELLS | REPAIR | THERAPY | HIF | HUMAN BONE-MARROW | MULTIDISCIPLINARY SCIENCES | BIOLOGY | DIFFERENTIATION | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Humans | Transcriptional Activation | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Vascular Endothelial Growth Factor A - metabolism | PPAR gamma - metabolism | Vascular Endothelial Growth Factor A - genetics | Core Binding Factor Alpha 1 Subunit - metabolism | Antigens, CD - metabolism | Gene Knockdown Techniques | Cell Hypoxia | Phenotype | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - physiology | Core Binding Factor Alpha 1 Subunit - genetics | Mesenchymal Stromal Cells - physiology | Osteogenesis | Adipogenesis | PPAR gamma - genetics | Growth | Stem cells | Physiological aspects | Hypoxia (Aquatic ecology) | Bones | Research | Gene expression | Vascular endothelial growth factor | Hypoxia-inducible factor 1 | Transcription factors | Mesenchyme | Arthritis | Adipocytes | Impact analysis | Recruitment | Angiogenesis | Immunology | Bone marrow | Biocompatibility | Conditioning | Hypoxia-inducible factors | Cbfa-1 protein | Rheumatology | Bone healing | Stromal cells | Hypoxia | Peroxisome proliferator-activated receptors | Bone | Differentiation | Surface markers
Journal Article
Journal Article
FEBS Letters, ISSN 0014-5793, 04/2015, Volume 589, Issue 9, pp. 1040 - 1047
Cartilage generation and degradation are regulated by miRNAs. Our previous study has shown altered expression of miR-193b in chondrogenic human adipose-derived... 
MiR-193b | TGF-beta | Chondrogenesis | TNF-alpha | Osteoarthritis | BIOCHEMISTRY & MOLECULAR BIOLOGY | OSTEOGENIC DIFFERENTIATION | MICRORNAS | MESENCHYMAL STEM-CELLS | CELL BIOLOGY | GROWTH-FACTOR-BETA | BIOPHYSICS | REPRESSES CELL-PROLIFERATION | ARTICULAR-CARTILAGE | CHONDROCYTE PROLIFERATION | SOX9 | EXPRESSION | Tumor Necrosis Factor-alpha - metabolism | Chondrocytes - cytology | Interleukin-1beta - pharmacology | Phosphorylation | Receptors, Transforming Growth Factor beta - genetics | Chondrogenesis - genetics | Transforming Growth Factor beta2 - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | 3' Untranslated Regions - genetics | Adipose Tissue - cytology | Smad3 Protein - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Cell Differentiation - genetics | Smad3 Protein - genetics | Mesenchymal Stromal Cells - cytology | RNA Interference | Chondrocytes - metabolism | SOX9 Transcription Factor - metabolism | Mesenchymal Stromal Cells - drug effects | Gene Expression | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Proteoglycans - metabolism | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Animals | Receptors, Transforming Growth Factor beta - metabolism | Cell Differentiation - drug effects | Mice | MicroRNAs - genetics | Transforming Growth Factor beta2 - genetics | SOX9 Transcription Factor - genetics | Core Binding Factor Alpha 1 Subunit - genetics | Proteoglycans - genetics | Transforming growth factors
Journal Article