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1. Full Text The Transcription Factors T-bet and Runx Are Required for the Ontogeny of Pathogenic Interferon-γ-Producing T Helper 17 Cells
by Wang, Yan and Godec, Jernej and Ben-Aissa, Khadija and Cui, Kairong and Zhao, Keji and Pucsek, Alexandra B and Lee, Yun Kyung and Weaver, Casey T and Yagi, Ryoji and Lazarevic, Vanja
Immunity, ISSN 1074-7613, 03/2014, Volume 40, Issue 3, pp. 355 - 366
T helper 17 (Th17) cells can give rise to interleukin-17A (IL-17A)- and interferon (IFN)-γ-double-producing cells that are implicated in development... 
ENCEPHALOMYELITIS | T(H)17 CELLS | IFNG GENE-EXPRESSION | ROR-GAMMA | HUMAN TH17 CELLS | CYTOKINE | DIFFERENTIATION | IMMUNOLOGY | LINEAGE | FOXP3 | PLASTICITY | Core Binding Factor Alpha 3 Subunit - metabolism | Encephalomyelitis, Autoimmune, Experimental - metabolism | Encephalomyelitis, Autoimmune, Experimental - immunology | Genetic Loci | Th1 Cells - immunology | Th1 Cells - metabolism | Th17 Cells - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Core Binding Factor alpha Subunits - metabolism | Th17 Cells - cytology | Cell Differentiation | Interferon-gamma - genetics | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Disease Models, Animal | Gene Expression | Core Binding Factor Alpha 2 Subunit - metabolism | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Mice, Knockout | Core Binding Factor alpha Subunits - genetics | Animals | Core Binding Factor Alpha 3 Subunit - genetics | Protein Binding | Th17 Cells - immunology | Mice | Th1 Cells - cytology | Core Binding Factor Alpha 2 Subunit - genetics | Interferon-gamma - biosynthesis | Index Medicus
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2. Full Text Sequence analysis of mutations and translocations across breast cancer subtypes
by Banerji, Shantanu and Cibulskis, Kristian and Rangel-Escareno, Claudia and Brown, Kristin K and Carter, Scott L and Frederick, Abbie M and Lawrence, Michael S and Sivachenko, Andrey Y and Sougnez, Carrie and Zou, Lihua and Cortes, Maria L and Fernandez-Lopez, Juan C and Peng, Shouyong and Ardlie, Kristin G and Auclair, Daniel and Bautista-Piña, Veronica and Duke, Fujiko and Francis, Joshua and Jung, Joonil and Maffuz-Aziz, Antonio and Onofrio, Robert C and Parkin, Melissa and Pho, Nam H and Quintanar-Jurado, Valeria and Ramos, Alex H and Rebollar-Vega, Rosa and Rodriguez-Cuevas, Sergio and Romero-Cordoba, Sandra L and Schumacher, Steven E and Stransky, Nicolas and Thompson, Kristin M and Uribe-Figueroa, Laura and Baselga, Jose and Beroukhim, Rameen and Polyak, Kornelia and Sgroi, Dennis C and Richardson, Andrea L and Jimenez-Sanchez, Gerardo and Lander, Eric S and Gabriel, Stacey B and Garraway, Levi A and Golub, Todd R and Melendez-Zajgla, Jorge and Toker, Alex and Getz, Gad and Hidalgo-Miranda, Alfredo and Meyerson, Matthew
Nature, ISSN 0028-0836, 06/2012, Volume 486, Issue 7403, pp. 405 - 409
Breast carcinoma is the leading cause of cancer-related mortality in women worldwide, with an estimated 1.38 million new cases and 458,000 deaths in 2008... 
CELL LUNG-CANCER | FUSION | COLORECTAL CANCERS | PIK3CA GENE | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | KINASE | HIGH-FREQUENCY | PATTERNS | LANDSCAPES | GENOME | Breast Neoplasms - classification | Membrane Proteins - genetics | Humans | Core Binding Factor beta Subunit - genetics | Mutation - genetics | Proto-Oncogene Proteins c-akt - genetics | Gene Fusion - genetics | Exome - genetics | Vietnam | Algorithms | Breast Neoplasms - genetics | Breast Neoplasms - pathology | DNA Mutational Analysis | Female | Mexico | Proto-Oncogene Proteins c-akt - metabolism | Core Binding Factor Alpha 2 Subunit - genetics | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Translocation, Genetic - genetics | Translocation (Genetics) | Gene mutations | Exome sequencing | Breast cancer | Genetic aspects | Research | Cancer | Genetics | Mutation | Kinases | Genes | Index Medicus
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3. Full Text Direct Recruitment of Polycomb Repressive Complex 1 to Chromatin by Core Binding Transcription Factors
by Yu, Ming and Mazor, Tali and Huang, Hui and Huang, Hsuan-Ting and Kathrein, Katie L and Woo, Andrew J and Chouinard, Candace R and Labadorf, Adam and Akie, Thomas E and Moran, Tyler B and Xie, Huafeng and Zacharek, Sima and Taniuchi, Ichiro and Roeder, Robert G and Kim, Carla F and Zon, Leonard I and Fraenkel, Ernest and Cantor, Alan B
Molecular Cell, ISSN 1097-2765, 02/2012, Volume 45, Issue 3, pp. 330 - 343
Polycomb repressive complexes (PRCs) play key roles in developmental epigenetic regulation. Yet the mechanisms that target PRCs to specific loci in mammalian... 
TRANSFORMATION | MAINTENANCE | ADULT HEMATOPOIESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEMATOPOIETIC STEM-CELLS | CBFA2 | MEGAKARYOCYTE | MICE | DIFFERENTIATION | RUNX1 | HAPLOINSUFFICIENCY | CELL BIOLOGY | Chromatin - metabolism | Thymocytes - metabolism | Protein Multimerization | Gene Expression Profiling | Core Binding Factor beta Subunit - genetics | Zebrafish - embryology | Gene Knockdown Techniques | T-Lymphocytes - metabolism | Hematopoietic Stem Cells - physiology | Repressor Proteins - isolation & purification | Megakaryocytes - metabolism | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Core Binding Factor beta Subunit - metabolism | Proto-Oncogene Proteins - metabolism | Cell Line | Core Binding Factor Alpha 2 Subunit - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Ubiquitin-Protein Ligases - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Polycomb-Group Proteins | Zebrafish - genetics | Mice, Knockout | Animals | Chromatography, Affinity | Polycomb Repressive Complex 1 | Protein Binding | Mice | Ubiquitin-Protein Ligases - genetics | Core Binding Factor Alpha 2 Subunit - genetics | Cluster Analysis | Computer science | Employee recruitment | Chromatin | Genomics | Genes | Stem cells | Chemical properties | DNA binding proteins | Artificial intelligence | Genetic research | Index Medicus
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4. Full Text Foxp3 controls regulatory T-cell function by interacting with AML1/Runx1
by Ono, Masahiro and Ohkura, Naganari and Kitabayashi, Issay and Miyachi, Yoshiki and Nagamura, Yuko and Sakaguchi, Shimon and Yaguchi, Hiroko and Tsukada, Toshihiko and Nomura, Takashi
Nature, ISSN 0028-0836, 04/2007, Volume 446, Issue 7136, pp. 685 - 689
Naturally arising CD25+CD4+ regulatory T cells (TR cells) are engaged in the maintenance of immunological self-tolerance and immune homeostasis by suppressing... 
RUNX1 BINDING-SITE | ACTIVATION | HEMATOPOIESIS | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION FACTOR FOXP3 | SUSCEPTIBILITY | IMMUNOLOGICAL SELF-TOLERANCE | LEUKEMIA | DIFFERENTIATION | COOPERATION | INTERLEUKIN-2 GENE | Interleukin-2 - metabolism | T-Lymphocytes, Regulatory - metabolism | Core Binding Factor Alpha 2 Subunit - metabolism | Core Binding Factor Alpha 2 Subunit - antagonists & inhibitors | Lymphocyte Activation | Humans | Gene Expression Regulation | Forkhead Transcription Factors - genetics | Promoter Regions, Genetic - genetics | T-Lymphocytes, Regulatory - immunology | Interleukin-2 - genetics | Phenotype | Animals | Forkhead Transcription Factors - metabolism | Protein Binding | Mice | Core Binding Factor Alpha 2 Subunit - genetics | Proteins | Immunology | Cytokines | Gene expression | Molecular biology | Binding sites | Index Medicus
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5. Full Text Blood stem cells emerge from aortic endothelium by a novel type of cell transition
by Herbomel, Philippe and Kissa, Karima
Nature, ISSN 0028-0836, 03/2010, Volume 464, Issue 7285, pp. 112 - 115
The ontogeny of haematopoietic stem cells (HSCs) during embryonic development is still highly debated, especially their possible lineage relationship to... 
HEMATOPOIETIC-CELLS | TRANSGENIC ZEBRAFISH | HAEMOGENIC ENDOTHELIUM | MULTIDISCIPLINARY SCIENCES | EMBRYOS | ZEBRAFISH DEVELOPMENT | Endothelium, Vascular - cytology | Core Binding Factor Alpha 2 Subunit - metabolism | Animals, Genetically Modified | Zebrafish Proteins - metabolism | Aorta - embryology | Zebrafish - embryology | Cell Lineage | Zebrafish Proteins - deficiency | Animals | Endothelial Cells - cytology | Hematopoietic Stem Cells - cytology | Cell Death | Zebrafish - blood | Cell Differentiation | Aorta - cytology | Zebrafish Proteins - genetics | Core Binding Factor Alpha 2 Subunit - genetics | Cell Movement | Core Binding Factor Alpha 2 Subunit - deficiency | Endothelium, Vascular - embryology | Physiological aspects | Research | Mammals | Hematopoietic stem cells | Endothelium | Proteins | Motion pictures | Blood | Veins & arteries | Index Medicus
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6. Full Text Comprehensive mutational profiling of core binding factor acute myeloid leukemia
by Duployez, Nicolas and Marceau-Renaut, Alice and Boissel, Nicolas and Petit, Arnaud and Bucci, Maxime and Geffroy, Sandrine and Lapillonne, Hélène and Renneville, Aline and Ragu, Christine and Figeac, Martin and Celli-Lebras, Karine and Lacombe, Catherine and Micol, Jean-Baptiste and Abdel-Wahab, Omar and Cornillet, Pascale and Ifrah, Norbert and Dombret, Hervé and Leverger, Guy and Jourdan, Eric and Preudhomme, Claude
Blood, ISSN 0006-4971, 05/2016, Volume 127, Issue 20, pp. 2451 - 2459
Acute myeloid leukemia (AML) with t(8; 21) or inv(16) have been recognized as unique entities within AML and are usually reported together as core binding... 
GENE-MUTATIONS | INV | IMPACT | PROGNOSIS | FLT3 | C-KIT | PATTERNS | PATIENT | SPECTRUM | COHESIN COMPLEX | HEMATOLOGY | Chromosomes, Human, Pair 8 - genetics | Translocation, Genetic | Prognosis | Humans | Middle Aged | Child, Preschool | Infant | Male | Chromosome Inversion | Young Adult | Core Binding Factors - genetics | DNA Mutational Analysis | Cell Cycle Proteins - genetics | Adult | Female | Child | Chromosomes, Human, Pair 16 - genetics | Chromatin - ultrastructure | Genetic Association Studies | Chromosomes, Human, Pair 21 - genetics | Chromosomal Proteins, Non-Histone - genetics | RUNX1 Translocation Partner 1 Protein | Oncogene Proteins, Fusion - genetics | Adolescent | Alleles | High-Throughput Nucleotide Sequencing | DNA, Neoplasm - genetics | Mutation | Chromatin - genetics | Core Binding Factor Alpha 2 Subunit - genetics | Leukemia, Myeloid, Acute - genetics | Index Medicus | Abridged Index Medicus | Myeloid Neoplasia
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7. Full Text Genome-wide meta-analysis identifies multiple novel associations and ethnic heterogeneity of psoriasis susceptibility
by Yin, Xianyong and Low, Hui Qi and Wang, Ling and Li, Yonghong and Ellinghaus, Eva and Han, Jiali and Estivill, Xavier and Sun, Liangdan and Zuo, Xianbo and Shen, Changbing and Zhu, Caihong and Zhang, Anping and Sanchez, Fabio and Padyukov, Leonid and Catanese, Joseph J and Krueger, Gerald G and Duffin, Kristina Callis and Mucha, Sören and Weichenthal, Michael and Weidinger, Stephan and Lieb, Wolfgang and Foo, Jia Nee and Li, Yi and Sim, Karseng and Liany, Herty and Irwan, Ishak and Teo, Yikying and Theng, Colin T S and Gupta, Rashmi and Bowcock, Anne and De Jager, Philip L and Qureshi, Abrar A and de Bakker, Paul I W and Seielstad, Mark and Liao, Wilson and Ståhle, Mona and Franke, Ane and Zhang, Xuejun and Liu, Jianjun
Nature Communications [E], ISSN 2041-1723, 2015, Volume 6, Issue 1, pp. 6916 - 6916
Psoriasis is a common inflammatory skin disease with complex genetics and different degrees of prevalence across ethnic populations. Here we present the... 
COMMON VARIANTS | RISK LOCI | INTERPLAY | EXPLAIN | HAPLOTYPE MAP | MULTIDISCIPLINARY SCIENCES | LUNG ADENOCARCINOMA | DIFFERENTIATION | PREVALENCE | HLA | RUNX1 | Index Medicus | Psoriasi
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8. Full Text Runx1 and Cbfβ regulate the development of Flt3 + dendritic cell progenitors and restrict myeloproliferative disorder
by Satpathy, Ansuman T and Briseño, Carlos G and Cai, Xiongwei and Michael, Drew G and Chou, Chun and Hsiung, Sunnie and Bhattacharya, Deepta and Speck, Nancy A and Egawa, Takeshi
Blood, ISSN 0006-4971, 05/2014, Volume 123, Issue 19, pp. 2968 - 2977
Runx1 and Cbf beta are critical for the establishment of definitive hematopoiesis and are implicated in leukemic transformation. Despite the absolute... 
LINEAGE COMMITMENT | STEM-CELLS | HUMAN BONE-MARROW | IN-VIVO | ACUTE MYELOID-LEUKEMIA | HEMATOLOGY | TRANSCRIPTION FACTOR | COMMITTED PROGENITORS | EXPRESSION | FETAL LIVER HEMATOPOIESIS | BINDING-FACTOR | Core Binding Factor Alpha 3 Subunit - metabolism | Interferon Regulatory Factors - metabolism | Anemia - pathology | Myeloproliferative Disorders - pathology | Core Binding Factor beta Subunit - genetics | Stem Cells - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Myeloproliferative Disorders - genetics | Cell Differentiation - genetics | Anemia - genetics | Flow Cytometry | Myeloproliferative Disorders - metabolism | Granulocytes - metabolism | Dendritic Cells - metabolism | Core Binding Factor beta Subunit - metabolism | Gene Expression | Core Binding Factor Alpha 2 Subunit - metabolism | Mice, Inbred C57BL | Cells, Cultured | Interferon Regulatory Factors - genetics | Hematopoiesis - genetics | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | fms-Like Tyrosine Kinase 3 - metabolism | Mice, Knockout | Anemia - metabolism | Animals | Core Binding Factor Alpha 3 Subunit - genetics | Mice | Core Binding Factor Alpha 2 Subunit - genetics | Bone Marrow Cells - metabolism | Core Binding Factor Alpha 1 Subunit - genetics | Index Medicus | Abridged Index Medicus | Hematopoiesis and Stem Cells
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9. Full Text In vivo imaging of haematopoietic cells emerging from the mouse aortic endothelium
by Galjart, Niels and van Cappellen, Wiggert and Robin, Catherine and Boisset, Jean-Charles and Dzierzak, Elaine and Andrieu-Soler, Charlotte
Nature, ISSN 0028-0836, 03/2010, Volume 464, Issue 7285, pp. 116 - 120
Haematopoietic stem cells (HSCs), responsible for blood production in the adult mouse, are first detected in the dorsal aorta starting at embryonic day 10.5... 
AGM REGION | MIGRATION | STEM-CELLS | DEFINITIVE HEMATOPOIESIS | EMBRYO | MULTIDISCIPLINARY SCIENCES | GENERATION | COLONIZATION | HAEMOGENIC ENDOTHELIUM | EXPRESSION | MARKS | Endothelium, Vascular - cytology | Core Binding Factor Alpha 2 Subunit - metabolism | Aorta - embryology | Male | Pregnancy | Cell Lineage | Microscopy, Confocal | Phenotype | Animals | Embryo, Mammalian - cytology | Endothelial Cells - cytology | Aorta - surgery | Hematopoietic Stem Cells - cytology | Female | Cell Differentiation | Mice | Aorta - cytology | Core Binding Factor Alpha 2 Subunit - genetics | Dissection | Core Binding Factor Alpha 2 Subunit - deficiency | Endothelium, Vascular - embryology | Physiological aspects | Usage | Diagnostic imaging | Research | Hematopoietic stem cells | Endothelium | Protocol | Motion pictures | Embryos | Index Medicus
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10. Full Text Depletion of RUNX1/ETO in t(8;21) AML cells leads to genome-wide changes in chromatin structure and transcription factor binding
by Ptasinska, A and Assi, S.A and Mannari, D and James, S.R and Williamson, D and Dunne, J and Hoogenkamp, M and Wu, M and Care, M and McNeill, H and Cauchy, P and Cullen, M and Tooze, R.M and Tenen, D.G and Young, B.D and Cockerill, P.N and Westhead, D.R and Heidenreich, O and Bonifer, C
Leukemia, ISSN 0887-6924, 2012, Volume 26, Issue 8, pp. 1829 - 1841
The t(8;21) translocation fuses the DNA-binding domain of the hematopoietic master regulator RUNX1 to the ETO protein. The resultant RUNX1/ETO fusion protein... 
acute myeloid leukemia | chromatin | epigenetic regulation | integrated analysis of high-throughput data | RUNX1/ETO | AML1-ETO | STEM-CELLS | ACUTE MYELOGENOUS LEUKEMIA | SELF-RENEWAL | ACUTE MYELOID-LEUKEMIA | HEMATOPOIESIS | ONCOLOGY | IN-VIVO | FUSION PROTEIN | GENE-EXPRESSION | DIFFERENTIATION | HEMATOLOGY | Chromatin - metabolism | Translocation, Genetic | Oncogene Proteins, Fusion - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Transcriptional Activation | CCAAT-Binding Factor - genetics | Gene Expression Profiling | RNA Polymerase II - metabolism | Cell Differentiation - genetics | CCAAT-Binding Factor - metabolism | Acetylation | Binding Sites | Chromosomes, Human, Pair 21 | Proto-Oncogene Proteins - metabolism | Core Binding Factor Alpha 2 Subunit - metabolism | Gene Silencing | Proto-Oncogene Proteins - genetics | Chromosomes, Human, Pair 8 | Transcription Factors - genetics | RUNX1 Translocation Partner 1 Protein | Transcription Factors - metabolism | Oncogene Proteins, Fusion - genetics | Cell Line, Tumor | Protein Binding | Histones - metabolism | Mutation | Chromatin - genetics | Core Binding Factor Alpha 2 Subunit - genetics | Cluster Analysis | Leukemia, Myeloid, Acute - genetics | Physiological aspects | Chromatin | Transcription factors | Research | Gene expression | Cell lines | Index Medicus | ETO | Original | RUNX1
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11. Full Text FTO-dependent demethylation of N6-methyladenosine regulates mRNA splicing and is required for adipogenesis
by Xu Zhao Ying Yang Bao-Fa Sun Yue Shi Xin Yang Wen Xiao Ya-Juan Hao Xiao-Li Ping Yu-Sheng Chen Wen-Jia Wang Kang-Xuan Jin Xing Wang Chun-Min Huang Yu Fu Xiao-Meng Ge Shu-Hui Song Hyun Seok Jeong Hiroyuki Yanagisawa Yamei Niu Gui-Fang Jia Wei Wu Wei-Min Tong Akimitsu Okamoto Chuan He Jannie M Rendtlew Danielsen Xiu-Jie Wang Yun-Gui Yang
细胞研究：英文版, ISSN 1001-0602, 2014, Volume 24, Issue 12, pp. 1403 - 1419
The role of Fat Mass and Obesity-associated protein （FTO） and its substrate N6-methyladenosine （m6A） in mRNA processing and adipogenesis remains largely... 
FTO | 去甲基化 | 剪接位点 | RNA剪接 | 调控因子 | 脂肪形成 | 基因表达 | 相关蛋白 | N6-methyladenosine (m | A | adipogenesis | mRNA splicing | METTL3 | MOUSE | MECHANISMS | CELL BIOLOGY | N-6-METHYLADENOSINE | TRANSCRIPTOMES | GENE | N6-methyladenosine (mA) | NUCLEAR-RNA | PROTEINS | EXPRESSION | REVEALS | Cell Line | Core Binding Factor Alpha 2 Subunit - metabolism | RNA, Messenger - genetics | Adipocytes - cytology | Alpha-Ketoglutarate-Dependent Dioxygenase FTO | Mixed Function Oxygenases - metabolism | RNA, Messenger - metabolism | RNA Splicing | Animals | Adenosine - analogs & derivatives | Adipocytes - metabolism | Adenosine - metabolism | Mice | Oxo-Acid-Lyases - metabolism | Methylation | Core Binding Factor Alpha 2 Subunit - genetics | Adipogenesis | Index Medicus | N6-methyladenosine (m6A) | Original
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