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Journal of the American College of Cardiology, ISSN 0735-1097, 02/2016, Volume 67, Issue 6, pp. 618 - 626
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2016, Volume 11, Issue 6, p. e0158421
Journal Article
Journal Article
Journal of the Royal Society Interface, ISSN 1742-5689, 11/2017, Volume 14, Issue 136, pp. 20170327 - 20170327
The embryonic lineage of intramural cells, microstructural organization of the extracellular matrix, local luminal and wall geometry, and haemodynamic loads... 
Smooth muscle | Inflammatory cells | Angiotensin II | Stress | Aortic stiffness | stress | MULTIDISCIPLINARY SCIENCES | INVOLVEMENT | smooth muscle | ATHEROSCLEROSIS | ANGIOTENSIN-II | MODEL | II-INDUCED HYPERTENSION | aortic stiffness | angiotensin II | inflammatory cells | GROWTH | SMOOTH-MUSCLE-CELLS | EXTRACELLULAR-MATRIX | MICE | MECHANOTRANSDUCTION | Aneurysm, Dissecting - genetics | Aneurysm, Dissecting - metabolism | Aortic Aneurysm - metabolism | Muscle, Smooth, Vascular - metabolism | Models, Cardiovascular | Aortic Aneurysm - physiopathology | Extracellular Matrix - metabolism | Male | Aneurysm, Dissecting - pathology | Muscle, Smooth, Vascular - physiopathology | Neointima - metabolism | Mice, Knockout | Extracellular Matrix - genetics | Neointima - physiopathology | Muscle, Smooth, Vascular - pathology | Neointima - genetics | Animals | Aneurysm, Dissecting - physiopathology | Fibrosis | Neointima - pathology | Mice | Aortic Aneurysm - pathology | Aortic Aneurysm - genetics | Extracellular Matrix - pathology | Hypertension | Animal models | Cyclic loads | Aneurysm | Muscles | Mechanical properties | Aneurysms | Stiffening | Muscle contraction | Biomechanics | Arteriosclerosis | Coronary vessels | Collagen | Atherosclerosis | Aorta | Extracellular matrix | Stiffness | Blood pressure | Dissection | Stretching | 1004 | Life Sciences–Engineering interface
Journal Article
Journal of Cardiology, ISSN 0914-5087, 2011, Volume 59, Issue 2, pp. 139 - 146
Journal Article
PLOS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e89830
The pathogenesis of abdominal aortic aneurysm (AAA) is characterized to be inflammation-associated degeneration of vascular wall. Neovascularization is... 
BREAST-CANCER | PATHOGENESIS | CELLS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | PODOPLANIN | ATHEROSCLEROSIS | MICROVESSEL DENSITY | MACROPHAGE RECRUITMENT | UP-REGULATION | TUMORS | Inflammation - pathology | Microvessels - physiopathology | Humans | Microvessels - pathology | Aortic Aneurysm, Abdominal - diagnostic imaging | Male | Case-Control Studies | Neovascularization, Pathologic - complications | Neovascularization, Pathologic - diagnostic imaging | Neovascularization, Pathologic - pathology | Inflammation - complications | Lymphography | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Microvessels - diagnostic imaging | Female | Neovascularization, Pathologic - physiopathology | Tunica Intima - diagnostic imaging | Lymphangiogenesis | Aortic Aneurysm, Abdominal - pathology | Aortic Aneurysm, Abdominal - complications | Tunica Intima - pathology | Aortic Aneurysm, Abdominal - physiopathology | Lymphatic Vessels - pathology | Aged | Lymphatic Vessels - diagnostic imaging | Lymphatic Vessels - physiopathology | Cell Movement | Fluorescence | Neovascularization | Abdominal aneurysm | Hyaluronic acid | Vascular endothelial growth factor | Fluorescence microscopy | Neurosciences | Laboratories | Pathogenesis | Aneurysm | Aneurysms | Arthritis | Matrix metalloproteinase | Macrophages | Dermatitis | Vascularization | Photonics | Angiogenesis | Hyaluronan | Ischemia | Autopsy | Rodents | Surgery | Aorta | Degeneration | Metalloproteinase | Hypoxia-inducible factors | Hypertension | Vascular surgery | Autopsies | Dermatology | Inflammation | Gelatinase B | Abdomen | Medicine | Coronary vessels | Hypoxia | Infiltration | Diabetes | Lymph
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e67098
Abdominal aortic aneurysm (AAA) is a common chronic degenerative disease characterized by progressive aortic dilation and rupture. The mechanisms underlying... 
ATHEROSCLEROTIC LESIONS | VITAMIN-E | ANTIOXIDANTS | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | MATRIX-METALLOPROTEINASE-9 | ALPHA-TOCOPHEROL | RECEPTOR | INHIBIT | alpha-Tocopherol - administration & dosage | Aortic Aneurysm, Abdominal - immunology | Apolipoproteins E - deficiency | Male | Aorta, Abdominal - pathology | Angiotensin II | Plaque, Atherosclerotic - pathology | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Lymphocytes - metabolism | Macrophages - pathology | Plaque, Atherosclerotic - diet therapy | Organ Size | beta Carotene - administration & dosage | Plaque, Atherosclerotic - immunology | Aortic Aneurysm, Abdominal - diet therapy | Mice, Knockout | Aorta, Abdominal - immunology | Lymphocytes - pathology | Macrophages - metabolism | Animals | Apolipoproteins E - genetics | Antioxidants - administration & dosage | Cholesterol, LDL - metabolism | Dietary Supplements | Lipoproteins (low density) | Oxidative stress | Vascular cell adhesion molecule 1 | Disease | Aneurysm | Lipid peroxidation | Aneurysms | Lipids | Kinases | Macrophages | Cell adhesion & migration | Recruitment | Antioxidants | Infusion | Apolipoprotein E | Vitamin E | Rodents | Atherosclerosis | Aorta | Supplementation | Diffusion | Plaques | Intercellular adhesion molecule 1 | Carotene | Oral administration | Dietary supplements | Macrophage colony-stimulating factor | Triglycerides | Inflammation | Metabolism | Gene expression | Cholesterol | Gelatinase B | β-Carotene | Abdomen | Gelatinase A | Diet | Medical prognosis | Coronary vessels | Angiotensin | Mice | Peroxisome proliferator-activated receptors | Tocopherol | Molecular biology | Monocyte chemoattractant protein 1
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6027, pp. 361 - 365
Angiotensin II (AngII) mediates progression of aortic aneurysm, but the relative contribution of its type 1 (AT1) and type 2 (AT2) receptors remains unknown.... 
Connective tissues | Receptors | Root growth | Medical treatment | REPORTS | Aneurysms | Placebos | Aorta | Mice | Aortic aneurysm | Marfan syndrome | PATHOGENESIS | ACTIVATION | MECHANISM | MULTIDISCIPLINARY SCIENCES | MARFAN-SYNDROME | MOUSE MODEL | GROWTH | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | EXPRESSION | Aortic Aneurysm - metabolism | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Aortic Rupture - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Enalapril - therapeutic use | MAP Kinase Signaling System | Marfan Syndrome - drug therapy | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Aortic Rupture - prevention & control | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Aortic Aneurysm - pathology | Aortic Aneurysm - prevention & control | Disease Models, Animal | Receptor, Angiotensin, Type 2 - genetics | Angiotensin II - metabolism | Signal Transduction | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Losartan - pharmacology | Aortic Rupture - pathology | Disease Progression | Mice, Knockout | Animals | Enalapril - pharmacology | Receptor, Angiotensin, Type 2 - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Aortic Aneurysm - drug therapy | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Aortic aneurysms | Development and progression | Genetic aspects | Health aspects | Angiotensin | Signal transduction | Peptides | Cellular biology | Coronary vessels | Rodents
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2016, Volume 36, Issue 5, pp. 886 - 897
OBJECTIVE—Kawasaki disease (KD) is the most common cause of acquired cardiac disease in US children. In addition to coronary artery abnormalities and... 
vasculitis | caspase-1 | abdominal aortic aneurysm | aortitis | dilatation | CORONARY ARTERITIS | IL-1 | INFLAMMASOME ACTIVATION | NLRP3 INFLAMMASOME | CHILDREN | IL-1-ALPHA | INTRAVENOUS IMMUNOGLOBULIN | PERIPHERAL VASCULAR DISEASE | MACROPHAGE ELASTASE | IL-1-BETA | HEMATOLOGY | INSIGHTS | Aortic Aneurysm, Abdominal - prevention & control | Cell Proliferation | Humans | Male | Gene Expression Profiling | Interleukin-1beta - genetics | Aorta, Abdominal - drug effects | Interleukin-1beta - deficiency | Aortic Aneurysm, Abdominal - metabolism | Time Factors | Interleukin-1beta - metabolism | Interleukin-1alpha - genetics | Interleukin 1 Receptor Antagonist Protein - pharmacology | Aortitis - pathology | Dilatation, Pathologic | Disease Models, Animal | Macrophages - pathology | Mucocutaneous Lymph Node Syndrome - chemically induced | Genotype | Mice, Knockout | Aortitis - metabolism | Macrophages - metabolism | Phenotype | Signal Transduction - drug effects | Caspase 1 - deficiency | Interleukin-1alpha - deficiency | Lactobacillus casei | Muscle, Smooth, Vascular - metabolism | Myocytes, Smooth Muscle - pathology | Interleukin-1alpha - metabolism | Cell Wall | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Receptors, Interleukin-1 Type I - metabolism | Mucocutaneous Lymph Node Syndrome - complications | Female | Aorta, Abdominal - pathology | Aorta, Abdominal - metabolism | Myocytes, Smooth Muscle - metabolism | Aortic Aneurysm, Abdominal - pathology | Receptors, Interleukin-1 Type I - deficiency | Elastin - metabolism | Mucocutaneous Lymph Node Syndrome - drug therapy | Mice, Inbred C57BL | Aortitis - genetics | Aortic Aneurysm, Abdominal - genetics | NLR Family, Pyrin Domain-Containing 3 Protein - deficiency | Muscle, Smooth, Vascular - pathology | Animals | Caspase 1 - genetics | Receptors, Interleukin-1 Type I - genetics
Journal Article