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The Journal of clinical investigation, ISSN 0021-9738, 2011, Volume 121, Issue 7, pp. 2693 - 2708
Journal Article
Circulation (New York, N.Y.), ISSN 0009-7322, 03/2016, Volume 133, Issue 9, pp. 849 - 858
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 12/2015, Volume 35, Issue 12, pp. 2605 - 2616
OBJECTIVE—Kawasaki disease (KD) is the most common cause of acute vasculitis and acquired cardiac disease among US children... 
dendritic cells | interleukin-1 | mucocutaneous lymph node syndrome | MyD88 | Endothelial cells | protein | endothelial cells | ELEMENT-BINDING PROTEIN-2 | ACTIVATION | RECEPTOR | CORONARY-ARTERY ANEURYSMS | NLRP3 INFLAMMASOME | POLYMORPHISM | ACUTE-PHASE | INTRAVENOUS IMMUNOGLOBULIN | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ASSOCIATION | Lactobacillus casei | Mucocutaneous Lymph Node Syndrome - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Stromal Cells - pathology | Caspase 1 - metabolism | Interleukin-1alpha - metabolism | Aortitis - chemically induced | Aorta - metabolism | Coronary Vessels - metabolism | Cell Wall | DNA-Binding Proteins - metabolism | Receptors, Interleukin-1 Type I - metabolism | Interleukin-1beta - metabolism | Bone Marrow Transplantation | Coronary Artery Disease - pathology | Aortitis - pathology | Dendritic Cells - metabolism | Disease Models, Animal | Mucocutaneous Lymph Node Syndrome - pathology | Coronary Vessels - pathology | Signal Transduction | Coronary Artery Disease - chemically induced | Endothelial Cells - metabolism | Mucocutaneous Lymph Node Syndrome - chemically induced | Coronary Artery Disease - metabolism | Mice, Inbred C57BL | Stromal Cells - metabolism | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Aortitis - genetics | DNA-Binding Proteins - genetics | Transplantation Chimera | Mice, Knockout | Aorta - pathology | Mucocutaneous Lymph Node Syndrome - genetics | Aortitis - metabolism | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Coronary Artery Disease - genetics | Receptors, Interleukin-1 Type I - genetics | Myeloid Differentiation Factor 88 - metabolism | Bone Marrow Cells - metabolism | coronary artery vasculitis | Kawasaki disease | IL-1
Journal Article
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 10/2019, Volume 74, Issue 14, p. 1823
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 12, p. e0168773
.... But their role in type 2 diabetes mellitus (T2DM) and coronary artery disease (CAD) is still in infancy... 
OBESITY | ACTIVATION | ADIPONECTIN | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | KINASE | PPAR-GAMMA | ADIPOKINE | EXPRESSION | CORS-26 | FAMILY | Tumor Necrosis Factors - genetics | Leukocytes, Mononuclear - metabolism | Cytokines - metabolism | Humans | Middle Aged | Gene Expression Regulation | Tumor Necrosis Factors - blood | Male | Adipokines - metabolism | Biomarkers - blood | Complement C1q - genetics | Complement C1q - metabolism | Coronary Artery Disease - blood | Case-Control Studies | Adipokines - blood | Coronary Artery Disease - complications | Coronary Artery Disease - genetics | Female | Aged | Diabetes Mellitus, Type 2 - complications | Cytokines - blood | Type 2 diabetes | Physiological aspects | Development and progression | Research | Cytokines | Coronary heart disease | Pathogenesis | Leukocytes (mononuclear) | Cardiovascular disease | Biochemistry | Family medical history | Kinases | Metabolic syndrome | Adiponectin | Proteins | Glucose metabolism | Rodents | Atherosclerosis | Peripheral blood mononuclear cells | Tumor necrosis factor-TNF | Blood pressure | Lipid metabolism | Lipoproteins (high density) | Diabetes mellitus (non-insulin dependent) | Heart diseases | Obesity | Medical imaging | Diabetes mellitus | Coronary artery | Inflammation | Tumor necrosis factor-α | Gene expression | Metabolism | Angina pectoris | Insulin | Coronary artery disease | Medicine | Serum levels | Body mass | Coronary vessels | Insulin resistance | Diabetes | Acute coronary syndromes | Endocrinology | Metabolic disorders | Smoking
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2014, Volume 63, Issue 17, pp. 1724 - 1734
...)-1 genotype status on the risk for coronary artery disease (CAD), defined as >50% diameter stenosis, and cardiovascular events mediated by oxidized phospholipids (OxPLs) and lipoprotein (Lp... 
Cardiovascular | Internal Medicine | atherosclerosis | genetic risk stratification | haplotype | IL-1 | inflammation | oxidation | oxidized phospholipids | polymorphism | lipoprotein(a) | lipoproteins | C-REACTIVE PROTEIN | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | LOW-DENSITY-LIPOPROTEIN | MONONUCLEAR-CELLS | OXIDATION-SPECIFIC BIOMARKERS | HUMAN ENDOTHELIAL-CELLS | RECEPTOR ANTAGONIST GENE | E-DEFICIENT MICE | ISCHEMIC-STROKE | SINGLE NUCLEOTIDE POLYMORPHISMS | Interleukin-1 - genetics | Interleukin-1 - metabolism | United States - epidemiology | Prospective Studies | Survival Rate - trends | Follow-Up Studies | Oxidation-Reduction | Coronary Artery Disease - metabolism | Humans | Middle Aged | Risk Factors | Genotype | Male | Incidence | DNA - genetics | Disease-Free Survival | Inflammation - metabolism | Coronary Artery Disease - genetics | Inflammation - genetics | Female | Coronary Artery Disease - epidemiology | Polymorphism, Single Nucleotide | Phospholipids - metabolism | Lipoprotein(a) - metabolism | Sects | Interleukins | Lipids | Genetic aspects | Coronary heart disease | Risk factors | Lipoprotein A | Proteins | Haplotypes | Confidence intervals | Genotype & phenotype | Heart attacks | Coronary vessels | Cardiovascular disease | Trends | Cardiology | Acute coronary syndromes | Thrombosis | Age | lipoprotein (a)
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 10, p. e0186426
...) utilizing a Tie2-cre recombinase (WHC-eTNAP mice). WHC-eTNAP developed significant coronary artery calcification at baseline compared WHC controls (4312 vs 0 mu m(2... 
ENU MUTAGENESIS | HEART-DISEASE | OSTEOBLAST DIFFERENTIATION | RISK PREDICTION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MUSCLE | ATHEROSCLEROSIS | INHIBITS VASCULAR CALCIFICATION | CALCIUM SCORE | MICE | Immunohistochemistry | Blood Chemical Analysis | Humans | Alkaline Phosphatase - metabolism | Body Weight - drug effects | Endothelium, Vascular - enzymology | Core Binding Factor Alpha 1 Subunit - metabolism | Coronary Vessels - metabolism | Myocardium - metabolism | Hyperlipoproteinemia Type II - pathology | Cytokines - blood | Disease Models, Animal | Receptors, LDL - genetics | Coronary Vessels - pathology | Alkaline Phosphatase - genetics | Coronary Artery Disease - etiology | Echocardiography | Endothelial Cells - metabolism | Placebo Effect | Mice, Inbred C57BL | Ventricular Function, Left - drug effects | Cells, Cultured | Enzyme Inhibitors - pharmacology | Mice, Transgenic | Myocardium - pathology | Hyperlipoproteinemia Type II - metabolism | Diet, Atherogenic | Myocardium - enzymology | Alkaline Phosphatase - antagonists & inhibitors | Animals | Calcification, Physiologic | Endothelial Cells - cytology | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Mice | Core Binding Factor Alpha 1 Subunit - genetics | Alkaline phosphatase | Care and treatment | Atherosclerosis | Genetic aspects | Research | Diagnosis | Coronary heart disease | Endothelium | Calcification (ectopic) | Heart | Animal models | Calcium | Body weight | Lipids | Smooth muscle | Cardiovascular disease | Recombinase | Phosphatase | Alizarin | Arteries | Ischemia | Mineralization | Animal tissues | Lipoprotein (low density) receptors | Lesions | Osteopathic medicine | Heart diseases | Deposition | Thickening | Heart failure | Medical research | Coronary artery | Pharmacology | Coronary artery disease | Thrombosis | Cre recombinase | Endothelial cells | Medicine | Cores | Hypercholesterolemia | Foam | Arteriosclerosis | Coronary vessels | Weight reduction | Calcification | Myocardium | Receptor density | Ventricle | Mutation | Laboratory animals
Journal Article
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 10, p. e0140958
Small non-coding microRNAs (miRNAs) are implicated in gene regulation, including those involved in coronary artery disease (CAD... 
BIOMARKERS | CELLS | PLASMA | METABOLISM | MULTIDISCIPLINARY SCIENCES | ACUTE MYOCARDIAL-INFARCTION | CARDIOVASCULAR-DISEASE | ATHEROSCLEROSIS | MICRORNAS | EXPRESSION | HIGH-DENSITY-LIPOPROTEIN | Biomarkers - metabolism | Myocardial Infarction - diagnosis | Angina, Unstable - metabolism | Diagnosis, Differential | Lipoproteins, HDL - blood | Angina, Stable - diagnosis | Coronary Artery Disease - metabolism | Humans | Middle Aged | Angina, Stable - metabolism | Angina, Unstable - diagnosis | Male | MicroRNAs - metabolism | Myocardial Infarction - metabolism | Young Adult | Coronary Artery Disease - diagnosis | Adult | Female | Aged | MicroRNAs - genetics | Unstable angina | Development and progression | Disease susceptibility | Research | Health aspects | Heart attack | Risk factors | Myocardial infarction | Heart attacks | Gene regulation | Cardiovascular disease | Ultracentrifugation | Angina | Regression models | Atherosclerosis | miRNA | Bioindicators | Lipid metabolism | Cardiology | Heart diseases | Lipoproteins (high density) | Coronary artery | Regression analysis | Metabolism | Gene expression | Coronary artery disease | Patients | Low density lipoprotein | Cholesterol | Pathology | Screening | Lipoproteins | Hospitals | Cellular biology | MicroRNAs | Coronary vessels | Biomarkers | Infarction | Diabetes | Acute coronary syndromes | Cardiovascular diseases
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 12/2016, Volume 37, Issue 2, pp. 191 - 204
... appreciation of increased disease burden and higher levels of arterial calcification in these subjects. Plaques within the coronary arteries of patients with diabetes mellitus generally... 
atherosclerosis | coronary artery disease | carotid arteries | death, sudden | diabetes mellitus | 10-YEAR FOLLOW-UP | PLAQUE MORPHOLOGY | PROTEIN-KINASE-C | RISK-FACTORS | death | ALKALINE-PHOSPHATASE | BEAM COMPUTED-TOMOGRAPHY | sudden | ACUTE MYOCARDIAL-INFARCTION | GROWTH-FACTOR 23 | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | Carotid Arteries - metabolism | Diabetes Mellitus, Type 1 - epidemiology | Vascular Remodeling | Prognosis | Coronary Vessels - physiopathology | Humans | Carotid Artery Diseases - physiopathology | Diabetes Mellitus, Type 1 - metabolism | Diabetes Mellitus, Type 2 - metabolism | Coronary Vessels - metabolism | Diabetes Mellitus, Type 2 - epidemiology | Vascular Calcification - metabolism | Diabetic Angiopathies - physiopathology | Diabetic Angiopathies - pathology | Coronary Artery Disease - physiopathology | Carotid Artery Diseases - pathology | Coronary Artery Disease - pathology | Vascular Calcification - mortality | Vascular Calcification - physiopathology | Carotid Artery Diseases - metabolism | Diabetic Angiopathies - metabolism | Coronary Vessels - pathology | Coronary Artery Disease - metabolism | Risk Factors | Diabetic Angiopathies - mortality | Carotid Artery Diseases - mortality | Plaque, Atherosclerotic | Vascular Calcification - pathology | Disease Progression | Diabetes Mellitus, Type 1 - mortality | Animals | Carotid Arteries - physiopathology | Diabetes Mellitus, Type 2 - mortality | Death, Sudden, Cardiac - pathology | Carotid Arteries - pathology | Rupture, Spontaneous
Journal Article