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JACC: Cardiovascular Interventions, ISSN 1936-8798, 08/2015, Volume 8, Issue 9, pp. 1166 - 1176
Objectives This study sought to evaluate the incidence of plaque rupture (PR), plaque erosion (PE), and calcified nodule (CN) using optical coherence... 
intravascular ultrasound | ST-segment elevation myocardial infarction | calcified nodule | plaque erosion | optical coherence tomography | MICROVASCULAR OBSTRUCTION | DIAGNOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | NO-REFLOW | THROMBOSIS | ACQUISITION | ARTERIES | DOCUMENT | THIN-CAP FIBROATHEROMA | ATHEROSCLEROTIC PLAQUE | CULPRIT LESION | Coronary Vessels - diagnostic imaging | Japan - epidemiology | Predictive Value of Tests | Myocardial Infarction - epidemiology | Vascular Remodeling | Humans | Middle Aged | Ultrasonography, Interventional | Vascular Calcification - diagnostic imaging | Male | Myocardial Infarction - diagnostic imaging | Incidence | Myocardial Infarction - therapy | Time Factors | Myocardial Infarction - pathology | Coronary Artery Disease - pathology | Female | Retrospective Studies | Vascular Calcification - diagnosis | Myocardial Infarction - diagnosis | Coronary Vessels - pathology | No-Reflow Phenomenon - epidemiology | Tomography, Optical Coherence | No-Reflow Phenomenon - diagnosis | Treatment Outcome | Plaque, Atherosclerotic | Vascular Calcification - pathology | Coronary Angiography | Coronary Artery Disease - diagnostic imaging | Thrombectomy | Coronary Artery Disease - therapy | Coronary Artery Disease - diagnosis | Fibrosis | Vascular Calcification - epidemiology | Aged | Coronary Artery Disease - epidemiology | Vascular Calcification - therapy | Percutaneous Coronary Intervention | Rupture, Spontaneous | Index Medicus
Journal Article
Arteriosclerosis Thrombosis and Vascular Biology, ISSN 1079-5642, 02/2015, Volume 35, Issue 2, pp. 399 - 408
OBJECTIVE—Vascular and valvular calcifications are pathological processes regulated by resident cells, and depending on a complex interplay between... 
aortic valve | multivesicular bodies | calcification of | gene expression | vascular calcification | AORTIC-STENOSIS | MATRIX VESICLES | VALVE | aortic valve, calcification of | CARTILAGE | DISEASE | VITAMIN-K | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | FETUIN-A | INSIGHTS | Calcinosis - genetics | Aortic Valve Stenosis - genetics | Calcium - metabolism | Humans | Middle Aged | Vascular Calcification - genetics | Actins - metabolism | Osteocalcin - genetics | Male | Aorta - metabolism | Case-Control Studies | Aortic Valve - pathology | Coronary Vessels - metabolism | Aortic Valve Stenosis - prevention & control | Vascular Calcification - metabolism | Coronary Artery Disease - pathology | Aged, 80 and over | Adult | Female | Calcinosis - metabolism | Extracellular Matrix Proteins - metabolism | Calcium-Binding Proteins - metabolism | Coronary Vessels - pathology | Osteocalcin - metabolism | Tissue Culture Techniques | Coronary Artery Disease - metabolism | Extracellular Matrix Proteins - genetics | Coronary Artery Disease - prevention & control | Gene Expression Regulation | Vascular Calcification - prevention & control | Vascular Calcification - pathology | Aortic Valve - metabolism | Aorta - pathology | Proteins - genetics | Aortic Valve Stenosis - pathology | Proteins - metabolism | Coronary Artery Disease - genetics | alpha-2-HS-Glycoprotein - metabolism | Aortic Valve Stenosis - metabolism | Aged | Calcinosis - prevention & control | Calcinosis - pathology | Calcium-Binding Proteins - genetics | Index Medicus
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 07/2016, Volume 7, Issue 1, pp. 12092 - 12092
Coronary artery disease (CAD) is the leading cause of mortality and morbidity, driven by both genetic and environmental risk factors. Meta-analyses of... 
MUSCLE-CELL DIFFERENTIATION | SMOOTH-MUSCLE | METAANALYSIS | OPEN CHROMATIN | MULTIDISCIPLINARY SCIENCES | SYSTEMS GENETICS | SUSCEPTIBILITY LOCI | RISK | WIDE ASSOCIATION | EXPRESSION | REVEALS | Chromatin - metabolism | Coronary Vessels - pathology | Genetic Predisposition to Disease | Genome-Wide Association Study | Basic Helix-Loop-Helix Transcription Factors - genetics | Transcription Factor AP-1 - genetics | Coronary Artery Disease - metabolism | Humans | Gene Expression Regulation | Myocytes, Smooth Muscle - pathology | Gene Expression Profiling | Transcription Factor AP-1 - metabolism | Coronary Vessels - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Base Sequence | Alleles | Coronary Artery Disease - genetics | Coronary Artery Disease - pathology | Primary Cell Culture | Genome, Human | Genomics - methods | Quantitative Trait Loci | Myocytes, Smooth Muscle - metabolism | Chromatin - chemistry | Myocardial infarction | Profiling | Smooth muscle | Cardiovascular disease | Smad3 protein | Genomes | Risk factors | Ecological risk assessment | Interleukin 6 | Interleukin 6 receptors | Genetic analysis | Heart diseases | Transforming growth factor-b1 | Bone morphogenetic protein 1 | Coronary artery | Muscles | Risk analysis | Coronary artery disease | Environmental risk | Loci | Morbidity | Quantitative trait loci | Coronary vessels | Regulatory mechanisms (biology) | Infarction | Gene mapping | Index Medicus | Kardiologi | Clinical Medicine | Cardiac and Cardiovascular Systems | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
Journal Article
Journal Article
Catheterization and Cardiovascular Interventions, ISSN 1522-1946, 09/2013, Volume 82, Issue 3, pp. E173 - E183
Journal Article
Journal Article