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1. Full Text Characterization of a Novel Coronavirus Associated with Severe Acute Respiratory Syndrome
by Paul A. Rota and M. Steven Oberste and Stephan S. Monroe and W. Allan Nix and Ray Campagnoli and Joseph P. Icenogle and Silvia Peñaranda and Bettina Bankamp and Kaija Maher and Min-hsin Chen and Suxiong Tong and Azaibi Tamin and Luis Lowe and Michael Frace and Joseph L. DeRisi and Qi Chen and David Wang and Dean D. Erdman and Teresa C. T. Peret and Cara Burns and Thomas G. Ksiazek and Pierre E. Rollin and Anthony Sanchez and Stephanie Liffick and Brian Holloway and Josef Limor and Karen McCaustland and Melissa Olsen-Rasmussen and Ron Fouchier and Stephan Günther and Albert D. M. E. Osterhaus and Christian Drosten and Mark A. Pallansch and Larry J. Anderson and William J. Bellini
Science (American Association for the Advancement of Science), ISSN 0036-8075, 5/2003, Volume 300, Issue 5624, pp. 1394 - 1399
Proteins | Coronavirus | Messenger RNA | RNA | Genomics | Viruses | Amino acids | Genomes | Research Articles | Open reading frames | SARS virus | SARS Virus/chemistry/classification/genetics/isolation & purification | Genome, Viral | Amino Acid Sequence | Nucleocapsid Proteins/chemistry/genetics | Humans | Membrane Glycoproteins/chemistry/genetics | Open Reading Frames | RNA, Viral/genetics | Regulatory Sequences, Nucleic Acid | Molecular Sequence Data | RNA, Messenger/genetics/metabolism | Viral Matrix Proteins/chemistry/genetics | Coronavirus/classification/genetics | Viral Envelope Proteins/chemistry/genetics | RNA Replicase/chemistry/genetics | Severe Acute Respiratory Syndrome/virology | Endopeptidases/chemistry/genetics | Phylogeny | Sequence Analysis, DNA | Conserved Sequence | Polyproteins/chemistry/genetics | Transcription, Genetic | Viral Proteins/chemistry/genetics | DNA, Complementary | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Genetics | Microbiology | Virology | Coronavirus - classification | Viral Matrix Proteins - genetics | Severe Acute Respiratory Syndrome - virology | Membrane Glycoproteins - chemistry | RNA, Messenger - metabolism | SARS Virus - classification | Endopeptidases - chemistry | RNA, Viral - genetics | SARS Virus - genetics | SARS Virus - isolation & purification | Viral Envelope Proteins - genetics | RNA Replicase - genetics | Viral Proteins - chemistry | RNA, Messenger - genetics | Polyproteins - chemistry | Viral Proteins - genetics | Nucleocapsid Proteins - chemistry | Polyproteins - genetics | SARS Virus - chemistry | Membrane Glycoproteins - genetics | Spike Glycoprotein, Coronavirus | Endopeptidases - genetics | Nucleocapsid Proteins - genetics | Viral Matrix Proteins - chemistry | RNA Replicase - chemistry | Viral Envelope Proteins - chemistry | Coronavirus - genetics | Epidemics | Control | Hong Kong | DNA | Severe acute respiratory syndrome | China | Causes of | Development and progression | Coronaviruses | Research | Observations | Index Medicus
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2. Full Text Epigenetic dysregulation of ACE2 and interferon-regulated genes might suggest increased COVID-19 susceptibility and severity in lupus patients
by Sawalha, Amr H and Zhao, Ming and Coit, Patrick and Lu, Qianjin
Clinical immunology (Orlando, Fla.), ISSN 1521-6616, 06/2020, Volume 215, pp. 108410 - 108410
Lupus | COVID-19 | ACE2 | SARS-CoV-2 | Epigenetics | Interferon | Methylation | Life Sciences & Biomedicine | Immunology | Science & Technology | Betacoronavirus - pathogenicity | Lupus Erythematosus, Systemic - complications | Pandemics | Epigenesis, Genetic | Humans | NF-kappa B - immunology | Oxidative Stress - immunology | Host-Pathogen Interactions - immunology | Betacoronavirus - immunology | Peptidyl-Dipeptidase A - genetics | DNA Methylation | Pneumonia, Viral - genetics | CD11a Antigen - genetics | Coronavirus Infections - epidemiology | Pneumonia, Viral - epidemiology | Pneumonia, Viral - complications | Lupus Erythematosus, Systemic - immunology | Coronavirus Infections - complications | Spike Glycoprotein, Coronavirus - immunology | Cytokines - genetics | Cytokines - immunology | Genetic Predisposition to Disease | Spike Glycoprotein, Coronavirus - genetics | Receptors, KIR - genetics | Signal Transduction | Lupus Erythematosus, Systemic - epidemiology | Oxidative Stress - genetics | Coronavirus Infections - genetics | Interferon Regulatory Factors - genetics | Viremia - genetics | Coronavirus Infections - immunology | Pneumonia, Viral - immunology | Disease Progression | Angiotensin-Converting Enzyme 2 | Viremia - complications | Interferon Regulatory Factors - immunology | Host-Pathogen Interactions - genetics | NF-kappa B - genetics | Lupus Erythematosus, Systemic - genetics | Protein Binding | Viremia - epidemiology | Viremia - immunology | CD11a Antigen - immunology | Peptidyl-Dipeptidase A - immunology | Receptors, KIR - immunology | Virus diseases | Epigenetic inheritance | Genes | Genetic research | Disease susceptibility | Biological response modifiers | T cells | Health aspects | Risk factors | Index Medicus
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3. Full Text 2′- O methylation of the viral mRNA cap evades host restriction by IFIT family members
by Gale Jr, Michael and Errett, John and Daffis, Stephane and Li, Jianqing and Thiel, Volker and Fensterl, Volker and Shi, Pei-Yong and Buller, R. Mark and Youn, Soonjeon and Pierson, Theodore C and Schneller, Stewart and Szretter, Kristy J and Dong, Hongping and Diamond, Michael S and Klimstra, William B and Zust, Roland and Schriewer, Jill and Lin, Tsai-Yu and Sen, Ganes C
Nature (London), ISSN 0028-0836, 11/2010, Volume 468, Issue 7322, pp. 452 - 456
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Fundamental and applied biological sciences. Psychology | Replicative cycle, interference, host-virus relations, pathogenicity, miscellaneous strains | Biological and medical sciences | Microbiology | Virology | Poxviridae - genetics | RNA Caps - immunology | Humans | Methyltransferases - metabolism | Immunity, Innate - genetics | Protein Biosynthesis - immunology | Interferons - immunology | Neoplasm Proteins - metabolism | RNA Caps - metabolism | RNA, Viral - genetics | West Nile virus - genetics | Fibroblasts | Poxviridae - physiology | RNA, Viral - metabolism | Receptor, Interferon alpha-beta - genetics | Interferons - genetics | Neoplasm Proteins - genetics | Coronavirus - physiology | RNA Caps - genetics | West Nile virus - physiology | Poxviridae - enzymology | West Nile virus - immunology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Receptor, Interferon alpha-beta - deficiency | Mice, Inbred C57BL | Cells, Cultured | Survival Rate | Poxviridae - immunology | Interferons - deficiency | Coronavirus - immunology | Proteins - genetics | Carrier Proteins - genetics | Immunity, Innate - immunology | Models, Immunological | Animals | Carrier Proteins - metabolism | Proteins - metabolism | Virus Replication | Coronavirus - genetics | RNA, Viral - immunology | West Nile virus - enzymology | Mice | Models, Genetic | Coronavirus - enzymology | Methylation | 3T3 Cells | West Nile fever | Interferon | Messenger RNA | Methyltransferases | Mortality | Viruses | Infections | Pattern recognition | Experiments | Viral infections | Cytoplasm | Evacuations & rescues | Index Medicus
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4. Full Text Irisin modulates genes associated with severe coronavirus disease (COVID-19) outcome in human subcutaneous adipocytes cell culture
by de Oliveira, Miriane and De Sibio, Maria Teresa and Mathias, Lucas Solla and Rodrigues, Bruna Moretto and Sakalem, Marna Eliana and Nogueira, Célia Regina
Molecular and cellular endocrinology, ISSN 0303-7207, 09/2020, Volume 515, pp. 110917 - 110917
Irisin | ADAM10 | SARS-CoV-2 | TRIB3 | FURIN | Adipose tissue | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Amyloid Precursor Protein Secretases - genetics | Humans | ADAM10 Protein - metabolism | Adipocytes - drug effects | COVID-19 | Histone Acetyltransferases - metabolism | Peptidyl-Dipeptidase A - metabolism | Gene Ontology | Protein-Serine-Threonine Kinases - metabolism | Repressor Proteins - metabolism | Histone Deacetylase 2 - genetics | Fibronectins - pharmacology | Signal Transduction | rab1 GTP-Binding Proteins - metabolism | Membrane Proteins - genetics | Cell Cycle Proteins - metabolism | Repressor Proteins - genetics | Betacoronavirus - metabolism | Toll-Like Receptor 3 - metabolism | Angiotensin-Converting Enzyme 2 | Amyloid Precursor Protein Secretases - metabolism | Coronavirus Infections - virology | Models, Biological | Betacoronavirus - genetics | Jumonji Domain-Containing Histone Demethylases - metabolism | Sirtuin 1 - metabolism | Pandemics | ADAM10 Protein - genetics | Histone Acetyltransferases - genetics | Adipocytes - cytology | Sirtuin 1 - genetics | Peptidyl-Dipeptidase A - genetics | Furin - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cell Cycle Proteins - genetics | Membrane Proteins - metabolism | Toll-Like Receptor 3 - genetics | Nuclear Proteins - genetics | Furin - metabolism | Obesity - virology | Molecular Sequence Annotation | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Jumonji Domain-Containing Histone Demethylases - genetics | Fibronectins - metabolism | rab1 GTP-Binding Proteins - genetics | Gene Expression Regulation - drug effects | Adipocytes - metabolism | Fibronectins - genetics | Histone Deacetylase 2 - metabolism | Pneumonia, Viral - virology | Virus diseases | Enzymes | Anopheles | Genes | Genetic aspects | Coronaviruses | Health aspects | Cells | Risk factors | Index Medicus
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5. Full Text Describing variability in pig genes involved in coronavirus infections for a One Health perspective in conservation of animal genetic resources
by Bovo, Samuele and Schiavo, Giuseppina and Ribani, Anisa and Utzeri, Valerio J and Taurisano, Valeria and Ballan, Mohamad and Muñoz, Maria and Alves, Estefania and Araujo, Jose P and Bozzi, Riccardo and Charneca, Rui and Di Palma, Federica and Djurkin Kušec, Ivona and Etherington, Graham and Fernandez, Ana I and García, Fabián and García-Casco, Juan and Karolyi, Danijel and Gallo, Maurizio and Martins, José Manuel and Mercat, Marie-José and Núñez, Yolanda and Quintanilla, Raquel and Radović, Čedomir and Razmaite, Violeta and Riquet, Juliette and Savić, Radomir and Škrlep, Martin and Usai, Graziano and Zimmer, Christoph and Ovilo, Cristina and Fontanesi, Luca
Scientific reports, ISSN 2045-2322, 02/2021, Volume 11, Issue 1, pp. 3359 - 3359
Genetics, Population | Breeding | Gene Frequency | Humans | Coronavirus Infections - genetics | Dipeptidyl Peptidase 4 - genetics | Receptors, Virus - genetics | INDEL Mutation | Angiotensin-Converting Enzyme 2 - genetics | Whole Genome Sequencing | Genetic Variation | Sus scrofa - genetics | Animals | CD13 Antigens - genetics | Swine | Serine Endopeptidases - genetics | High-Throughput Nucleotide Sequencing | Polymorphism, Single Nucleotide | One Health | Risk assessment | Proteinase | Genetic resources | Conservation | Amino acid sequence | Infections | Livestock | Coronaviruses | Viral infections | Index Medicus | Animal genetics | Coronavirus Infections | Dipeptidyl Peptidase 4 | Life Sciences | Angiotensin-Converting Enzyme 2 | CD13 Antigens | Genetics | Serine Endopeptidases | Sus scrofa | Receptors, Virus
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6. Full Text Ribose 2′- O -methylation provides a molecular signature for the distinction of self and non-self mRNA dependent on the RNA sensor Mda5
by Cervantes-Barragan, Luisa and Siddell, Stuart G and Habjan, Matthias and Thiel, Volker and Ziebuhr, John and Züst, Roland and Ludewig, Burkhard and Neuman, Benjamin W and Szretter, Kristy J and Baker, Susan C and Diamond, Michael S and Barchet, Winfried and Maier, Reinhard
Nature immunology, ISSN 1529-2908, 02/2011, Volume 12, Issue 2, pp. 137 - 143
Life Sciences & Biomedicine | Immunology | Science & Technology | Virus Replication - genetics | Humans | Methyltransferases - metabolism | Immunity, Innate - genetics | Methyltransferases - genetics | Viral Proteins - immunology | Methyltransferases - immunology | Interferon Type I - immunology | Viral Proteins - metabolism | Interferon-Induced Helicase, IFIH1 | Coronavirus Infections - metabolism | Virulence - genetics | Interferon Type I - metabolism | RNA, Viral - metabolism | Receptor, Interferon alpha-beta - genetics | DEAD-box RNA Helicases - metabolism | Coronavirus - physiology | Cell Line | Coronavirus - pathogenicity | Ribose - metabolism | Mice, Inbred C57BL | Coronavirus Infections - genetics | Viral Proteins - genetics | Coronavirus Infections - immunology | Mice, Knockout | DEAD-box RNA Helicases - genetics | Animals | DEAD-box RNA Helicases - immunology | Interferon Type I - genetics | Receptors, Pattern Recognition - genetics | Mice | Methylation | Messenger RNA | Physiological aspects | Viruses | Genetic aspects | Research | Health aspects | Index Medicus
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7. Full Text Genetic gateways to COVID‐19 infection: Implications for risk, severity, and outcomes
by Debnath, Monojit and Banerjee, Moinak and Berk, Michael
The FASEB journal, ISSN 0892-6638, 07/2020, Volume 34, Issue 7, pp. 8787 - 8795
ACE | cytokine storm | genetics | COVID‐19 | SARS‐CoV‐2 | immunity | HLA | COVID-19 | SARS-CoV-2 | Biochemistry & Molecular Biology | Biology | Life Sciences & Biomedicine - Other Topics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Metagenomics | Pandemics | Prognosis | Humans | Peptidyl-Dipeptidase A - physiology | Risk | Disease Resistance - immunology | HLA Antigens - genetics | Mutation, Missense | Receptors, Virus - genetics | Host-Pathogen Interactions - immunology | Receptors, Virus - physiology | Coronavirus Infections - transmission | Genetic Variation | Pneumonia, Viral - transmission | Peptidyl-Dipeptidase A - genetics | Pneumonia, Viral - genetics | Betacoronavirus - physiology | Pneumonia, Viral - complications | Coronavirus Infections - complications | Genetic Predisposition to Disease | Toll-Like Receptors - immunology | Cytokine Release Syndrome - genetics | Coronavirus Infections - genetics | HLA Antigens - immunology | Treatment Outcome | Inflammation | Continental Population Groups - genetics | Coronavirus Infections - immunology | Pneumonia, Viral - immunology | Angiotensin-Converting Enzyme 2 | Complement Activation - genetics | Cytokine Release Syndrome - etiology | Host-Pathogen Interactions - genetics | Toll-Like Receptors - genetics | Quantitative Trait Loci | Index Medicus
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8. Full Text Genomewide Association Study of Severe Covid-19 with Respiratory Failure
by Ellinghaus, David and Degenhardt, Frauke and Buti, Maria and Albillos, Agustin and Prati, Daniele and Asselta, Rosanna and Milani, Chiara and Kassens, Jan and May, Sandra and Wienbrandt, Lars and Uellendahl-Werth, Florian and Zheng, Tenghao and Yi, Xiaoli and Rodriguez-Frias, Francisco and Zanella, Alberto and Bandera, Alessandra and Protti, Alessandro and Aghemo, Alessio and Lleo, Ana and Biondi, Andrea and Caballero-Garralda, Andrea and Latiano, Anna and Fracanzani, Anna Ludovica and Peschuck, Anna and Pesenti, Antonio and Mateos, Beatriz and Jimenez, Beatriz Nafria and Quereda, Carmen and Paccapelo, Cinzia and Angelini, Claudio and Cea, Cristina and Solier, Aurora and Sandoval, Elena and Sanchez, Felix Garcia and Ceriotti, Ferruccio and Martinelli-Boneschi, Filippo and Blasi, Francesco and Tellez, Luis and Blanco-Grau, Albert and Hemmrich-Stanisak, Georg and Grasselli, Giacomo and Cardamone, Giulia and Aneli, Serena and Kurihara, Hayato and ElAbd, Hesham and Galvan-Femenia, Ivan and Erdmann, Jeanette and Ferrusquia-Acosta, Jose and Garcia-Etxebarria, Koldo and Santoro, Luigi and Roade, Luisa and Ruhlemann, Malte C and Carrabba, Maria and Riveiro-Barciela, Mar and Valsecchi, Maria G and Hernandez-Tejero, Maria and Acosta-Herrera, Marialbert and D'Angio, Mariella and Baldini, Marina and Cazzaniga, Marina and Schulzky, Martin and Cecconi, Maurizio and Rodriguez-Gandia, Miguel and Bocciolone, Monica and Miozzo, Monica and Montano, Nicola and Braun, Nicole and Sacchi, Nicoletta and Faverio, Paola and Preatoni, Paoletta and Bonfanti, Paolo and Omodei, Paolo and Tentorio, Paolo and Castro, Pedro and Rodrigues, Pedro M and Blandino, Aaron and de Cid, Rafael and Ferrer, Ricard and Nieto, Rosa and Goerg, Siegfried and Badalamenti, Salvatore and Marsal, Sara and Matullo, Giuseppe and Juzenas, Simonas and Aliberti, Stefano and Monzani, Valter and Moreno, Victor and Lenz, Tobias L and Pumarola, Tomas and Rimoldi, Valeria and Bosari, Silvano and Albrecht, Wolfgang and Peter, Wolfgang and D'Amato, Mauro and Duga, Stefano and Banales, Jesus M and Hov, Johannes R and Folseraas, Trine and Franke, Andre and Karlsen, Tom H and The Severe Covid-19 GWAS Group and Severe Covid-19 GWAS Grp and Severe Covid-19 GWAS Group
The New England journal of medicine, ISSN 0028-4793, 10/2020, Volume 383, Issue 16, pp. 1522 - 1534
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Multigene Family | Pandemics | Humans | Middle Aged | Respiratory Insufficiency - etiology | Male | Genetic Loci | Case-Control Studies | COVID-19 | Chromosomes, Human, Pair 9 - genetics | SARS-CoV-2 | Pneumonia, Viral - genetics | Pneumonia, Viral - complications | ABO Blood-Group System - genetics | Betacoronavirus | Coronavirus Infections - complications | Female | Genetic Predisposition to Disease | Genome-Wide Association Study | Coronavirus Infections - genetics | Spain | Respiratory Insufficiency - genetics | Italy | Aged | Polymorphism, Single Nucleotide | Chromosomes, Human, Pair 3 - genetics | Disease susceptibility | Genetic aspects | Respiratory insufficiency | Epidemics | Blood group A | Disease | Pathogenesis | Association analysis | Single-nucleotide polymorphism | Ethics | Genetic factors | Blood groups | Drug dosages | Age | Ventilators | Medical research | Blood & organ donations | Susceptibility | Mortality | Phlebotomy | ABO system | RNA polymerase | Coronaviruses | Chromosome 3 | Severe acute respiratory syndrome coronavirus 2 | Blood group O | Hospitals | Chromosome 9 | Quality control | Informed consent | CCR9 protein | Index Medicus | Abridged Index Medicus | Medicin och hälsovetenskap
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