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Glia, ISSN 0894-1491, 01/2015, Volume 63, Issue 1, pp. 104 - 117
Journal Article
Journal Article
Nature Neuroscience, ISSN 1097-6256, 08/2011, Volume 14, Issue 8, pp. 1009 - 1016
Permanent damage to white matter tracts, comprising axons and myelinating oligodendrocytes, is an important component of brain injuries of the newborn that... 
FUNCTIONAL INTERACTION | MULTIPLE-SCLEROSIS | CNS REMYELINATION | MYELINATION | SIGNALING PATHWAY | TRANSCRIPTION | DIFFERENTIATION | BETA-CATENIN | NEUROSCIENCES | WHITE-MATTER INJURY | NEGATIVE REGULATOR | Humans | Ki-67 Antigen - metabolism | Male | Brain Injuries - metabolism | Wnt Proteins - metabolism | Oligodendroglia - drug effects | Hypoxia-Ischemia, Brain - therapy | Infant, Newborn | Organ Culture Techniques | Disease Models, Animal | Animals, Newborn | Basic Helix-Loop-Helix Transcription Factors - genetics | Mice, Transgenic | Oligodendrocyte Transcription Factor 2 | Brain Injuries - therapy | Multiple Sclerosis - therapy | Myelin Proteins - genetics | beta Catenin - metabolism | Heterocyclic Compounds, 3-Ring - therapeutic use | Spinal Cord - physiology | Axin Protein | Mice | Myelin Sheath - ultrastructure | beta-Galactosidase - genetics | Cerebellum - ultrastructure | Myelin Proteins - metabolism | Corpus Callosum - metabolism | Spinal Cord - drug effects | Heterocyclic Compounds, 3-Ring - pharmacology | Myelin Proteins - therapeutic use | Cerebellum - drug effects | Myelin Sheath - drug effects | Hypoxia-Ischemia, Brain - pathology | Cerebral Cortex - cytology | Cytoskeletal Proteins - deficiency | Dose-Response Relationship, Drug | Oligodendroglia - physiology | Wnt Proteins - genetics | beta-Galactosidase - metabolism | Adult | Cytoskeletal Proteins - metabolism | Female | Demyelinating Diseases - chemically induced | Demyelinating Diseases - pathology | Neurons - drug effects | Cell Differentiation - physiology | Hypoxia-Ischemia, Brain - metabolism | Microscopy, Electron, Transmission | Myelin Sheath - pathology | Gene Expression Regulation - genetics | Cerebellum - metabolism | Cells, Cultured | Gene Expression Regulation - physiology | Corpus Callosum - drug effects | Nerve Tissue Proteins - genetics | beta Catenin - genetics | Multiple Sclerosis - complications | Nerve Tissue Proteins - metabolism | Animals | Cell Differentiation - drug effects | Multiple Sclerosis - pathology | Stem Cells - drug effects | Brain Injuries - etiology | Lysophosphatidylcholines - toxicity | Postmortem Changes | Infants (Newborn) | Brain | Care and treatment | Physiological aspects | Research | Binding proteins | Health aspects | Injuries
Journal Article
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2008, Volume 31, Issue 3, pp. 316 - 326
Abstract We examined the potential protective effect of BDNF against β-amyloid-induced neurotoxicity in vitro and in vivo in rats. In neuronal cultures, BDNF... 
Neurology | Neuroprotection | TrkB | BDNF | Somatostatin | Push-pull perfusion | Indusium griseum | Dentate gyrus hilus | β-Amyloid peptides | Corpus callosum | ALZHEIMERS-DISEASE | NEUROTROPHIC FACTOR | CORPUS-CALLOSUM ATROPHY | RECEPTOR EXPRESSION | NEUROSCIENCES | CULTURED CORTICAL-NEURONS | indusium griseum | MESSENGER-RNA | neuroprotection | somatostatin | push-pull perfusion | dentate gyrus hilus | HIPPOCAMPAL CONTINUATION | CENTRAL-NERVOUS-SYSTEM | corpus callosum | beta-amyloid peptides | DENTATE GYRUS | Neuroprotective Agents - therapeutic use | Corpus Callosum - metabolism | Peptide Fragments - toxicity | Male | Wallerian Degeneration - prevention & control | Receptor, trkB - genetics | Hippocampus - drug effects | Nerve Degeneration - chemically induced | Brain - metabolism | Dose-Response Relationship, Drug | Receptors, Somatostatin - genetics | Wallerian Degeneration - drug therapy | Brain-Derived Neurotrophic Factor - pharmacology | Neuroprotective Agents - pharmacology | Nerve Degeneration - prevention & control | Amyloid beta-Peptides - metabolism | Female | Receptors, Somatostatin - metabolism | Brain-Derived Neurotrophic Factor - therapeutic use | Disease Models, Animal | Wallerian Degeneration - chemically induced | Alzheimer Disease - physiopathology | Peptide Fragments - metabolism | Amyloid beta-Peptides - toxicity | Cells, Cultured | Alzheimer Disease - drug therapy | Rats | Corpus Callosum - drug effects | Treatment Outcome | Rats, Sprague-Dawley | Brain - drug effects | Hippocampus - metabolism | Amyloid beta-Peptides - antagonists & inhibitors | Animals | Peptide Fragments - antagonists & inhibitors | Corpus Callosum - pathology | Alzheimer Disease - metabolism | Receptors, Somatostatin - drug effects | Brain - pathology | Receptor, trkB - metabolism | Nerve Degeneration - drug therapy | Growth factors | Index Medicus
Journal Article
Neurochemical Research, ISSN 0364-3190, 12/2017, Volume 42, Issue 12, pp. 3525 - 3536
Multiple sclerosis (MS) is an inflammatory demyelinating disease of the central nervous system (CNS). The release of inflammatory cytokines and pro-oxidant... 
Neurochemistry | Biochemistry, general | Neurology | BV2 cells | Neurosciences | Multiple sclerosis | Biomedicine | Yokukansan | Cuprizone | Demyelination | Cell Biology | Microglia | CONTROLLED-TRIAL | MULTIPLE-SCLEROSIS LESIONS | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | UNCARIA HOOK | PSYCHOLOGICAL SYMPTOMS | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | GEISSOSCHIZINE METHYL-ETHER | CENTRAL-NERVOUS-SYSTEM | INFLAMMATORY CYTOKINES | ACTIVATED MICROGLIA | Astrocytes - drug effects | Oligodendroglia - metabolism | Cytokines - metabolism | Microglia - drug effects | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Drugs, Chinese Herbal - pharmacology | Corpus Callosum - drug effects | Inflammation - metabolism | Animals | Cuprizone - pharmacology | Inflammation - drug therapy | Female | Demyelinating Diseases - chemically induced | Astrocytes - metabolism | Demyelinating Diseases - drug therapy | Disease Models, Animal | Multiple Sclerosis - metabolism | Interleukins | Anti-inflammatory drugs | RNA | Analysis | Medicine, Botanic | Medicine, Herbal | Mice | Myelin proteins | Cytokines | Myelin | Interleukin | Central nervous system | mRNA | Gene expression | Corpus callosum | Nitric-oxide synthase | Myelin basic protein | Lipopolysaccharides | Herbal medicine | Cell activation | Cyclohexanone | Rodents | Microglial cells
Journal Article
Journal Article