X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
animals (189) 189
humans (179) 179
mice (150) 150
coxsackievirus infections - physiopathology (147) 147
male (139) 139
myocarditis - physiopathology (105) 105
female (93) 93
index medicus (90) 90
myocarditis (83) 83
myocarditis - virology (74) 74
coxsackievirus infections (58) 58
coxsackievirus infections - complications (51) 51
myocarditis - pathology (51) 51
coxsackievirus infections - pathology (49) 49
myocardium - pathology (49) 49
coxsackievirus infections - virology (47) 47
cardiac & cardiovascular systems (46) 46
infection (45) 45
enterovirus b, human (43) 43
mice, inbred balb c (42) 42
viruses (40) 40
dilated cardiomyopathy (38) 38
disease models, animal (38) 38
enterovirus b, human - pathogenicity (38) 38
virology (38) 38
adult (37) 37
coxsackievirus infections - immunology (35) 35
inflammation (33) 33
pathogenesis (33) 33
myocarditis - immunology (32) 32
virus diseases (31) 31
health aspects (30) 30
coxsackievirus (29) 29
adolescent (28) 28
enterovirus (28) 28
electrocardiography (27) 27
expression (27) 27
virus replication (27) 27
enterovirus b, human - isolation & purification (26) 26
immunology (26) 26
viral myocarditis (26) 26
acute disease (25) 25
child, preschool (25) 25
middle aged (25) 25
child (24) 24
enterovirus b, human - physiology (24) 24
infant (24) 24
cardiomyopathy (23) 23
mice, inbred c57bl (23) 23
coxsackievirus b3 (22) 22
enterovirus b, human - genetics (22) 22
analysis (20) 20
apoptosis (20) 20
disease (20) 20
myocardium - metabolism (20) 20
peripheral vascular disease (20) 20
virus (20) 20
article (19) 19
coxsackievirus infections - diagnosis (19) 19
enterovirus infections - physiopathology (19) 19
myocarditis - etiology (19) 19
cytokines (18) 18
enterovirus - isolation & purification (18) 18
heart (18) 18
time factors (18) 18
infant, newborn (17) 17
medicine, research & experimental (17) 17
mice, inbred strains (17) 17
cell biology (16) 16
cells (16) 16
coxsackievirus b3-induced myocarditis (16) 16
enterovirus b, human - immunology (16) 16
heart - physiopathology (16) 16
heart - virology (16) 16
mice, knockout (16) 16
research article (16) 16
cells, cultured (15) 15
children (15) 15
coxsackievirus infections - metabolism (15) 15
diagnosis (15) 15
heart diseases (15) 15
infections (15) 15
infectious diseases (15) 15
myocarditis - drug therapy (15) 15
autoimmunity (14) 14
cardiomyopathy, dilated - physiopathology (14) 14
myocarditis - complications (14) 14
pediatrics (14) 14
protein (14) 14
proteins (14) 14
biochemistry & molecular biology (13) 13
cardiomyopathies - physiopathology (13) 13
cell line (13) 13
coxsackievirus infections - drug therapy (13) 13
enterovirus - physiology (13) 13
enterovirus infections - virology (13) 13
heart failure (13) 13
hemodynamics (13) 13
myocarditis - genetics (13) 13
pathology (13) 13
more...
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (268) 268
Chinese (9) 9
Russian (7) 7
Japanese (6) 6
German (5) 5
Italian (2) 2
Spanish (2) 2
Bulgarian (1) 1
French (1) 1
Portuguese (1) 1
Ukrainian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Circulation research, ISSN 0009-7330, 2012, Volume 111, Issue 4, pp. 415 - U155
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2012, Volume 7, Issue 6, p. e39394
Background: Elevated heart rate is associated with increased cardiovascular morbidity. The selective I-f current inhibitor ivabradine reduces heart rate... 
OXIDATIVE STRESS | ENDOTHELIAL DYSFUNCTION | BRADYCARDIAC AGENT | MULTIDISCIPLINARY SCIENCES | IMPROVES | NECROSIS-FACTOR-ALPHA | HEART-RATE REDUCTION | BLOOD-FLOW | EXPRESSION | FAILURE | LEFT-VENTRICULAR FUNCTION | Apoptosis - drug effects | Myocarditis - drug therapy | Enterovirus B, Human | Myocardial Contraction - drug effects | Male | Adrenergic beta-Antagonists - pharmacology | Coxsackievirus Infections - virology | Heart Rate - drug effects | Propanolamines - therapeutic use | Myocardium - metabolism | Adrenergic beta-Antagonists - therapeutic use | Disease Models, Animal | Heart - physiopathology | Coxsackievirus Infections - physiopathology | Cytokines - metabolism | Benzazepines - pharmacology | Coxsackievirus Infections - complications | Myocarditis - virology | Animals | Benzazepines - therapeutic use | Heart - drug effects | Carbazoles - therapeutic use | Mice | Oxidative Stress - drug effects | Carbazoles - pharmacology | Propanolamines - pharmacology | Myocarditis - physiopathology | Heart failure | Myocarditis | Heart | Heart beat | Cytokines | Analysis | Collagen | Atherosclerosis | Superoxide | Coxsackievirus infections | Noradrenaline | Apoptosis | Oxidative stress | Animal models | Superoxide dismutase | Congestive heart failure | Beta blockers | Interleukin 6 | Angiogenesis | Reduction | Tumor necrosis factor-TNF | Blood pressure | Lesions | Cardiology | Vascular endothelial growth factor | Contractility | Cardiomyocytes | Inflammation | Tumor necrosis factor-α | Muscle contraction | Malondialdehyde | Morbidity | Heart rate | Arteriosclerosis | Adrenergic receptors | Norepinephrine | Ventricle | Laboratory animals | Monocyte chemoattractant protein 1 | Viral infections
Journal Article
EMERGING INFECTIOUS DISEASES, ISSN 1080-6040, 01/2003, Volume 9, Issue 1, pp. 78 - 85
Journal Article
Circulation Research, ISSN 0009-7330, 04/2009, Volume 104, Issue 7, pp. 851 - 859
The characteristics of dilated cardiomyopathy (DCM) resulting from chronic viral myocarditis are remodeling processes of the extracellular matrix. Based on our... 
Infection | Myocarditis | Osteopontin | Inflammation | Remodeling | CELLS | infection | remodeling | CARDIAC & CARDIOVASCULAR SYSTEMS | VIRUS | myocarditis | osteopontin | AUTOIMMUNE MYOCARDITIS | DILATED CARDIOMYOPATHY | VIRAL MYOCARDITIS | PLASMINOGEN-ACTIVATOR | inflammation | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | HUMAN HEART | EXPRESSION | PULMONARY-FIBROSIS | Osteopontin - genetics | Cardiomyopathy, Dilated - pathology | Phosphorylation | Osteopontin - deficiency | Humans | Transforming Growth Factor beta1 - metabolism | Osteopontin - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | RNA, Messenger - metabolism | Calcitriol - analogs & derivatives | Coxsackievirus Infections - virology | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Macrophages - virology | Time Factors | Myocardium - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Biomarkers - metabolism | Acute Disease | Collagen Type I - metabolism | Coxsackievirus Infections - physiopathology | Coxsackievirus Infections - metabolism | Mice, Inbred C57BL | Rats | Matrix Metalloproteinase 3 - metabolism | Myocardium - pathology | Cardiomyopathy, Dilated - metabolism | Cardiomyopathy, Dilated - virology | Mice, Knockout | Coxsackievirus Infections - pathology | Myocarditis - virology | Macrophages - metabolism | Animals | Cardiomyopathy, Dilated - physiopathology | Urokinase-Type Plasminogen Activator - metabolism | Calcitriol - pharmacology | Fibrosis | Myocarditis - metabolism | Mice | Enterovirus B, Human - pathogenicity | Myocarditis - pathology | Chronic Disease | Myocarditis - physiopathology | Ventricular Remodeling - drug effects
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 11/2004, Volume 110, Issue 18, pp. 2938 - 2945
Background - Myocardial virus persistence is frequently observed in patients with cardiomyopathy. Endothelial dysfunction in patients with cardiomyopathy is... 
Viruses | Inflammation | Cardiomyopathy | Vasodilation | Endothelium | endothelium | SEQUENCE-ANALYSIS | viruses | CARDIAC & CARDIOVASCULAR SYSTEMS | RISK-FACTORS | PROTEIN | vasodilation | DILATED CARDIOMYOPATHY | ADHESION MOLECULES | LEFT-VENTRICULAR DYSFUNCTION | inflammation | DISEASE | PERIPHERAL VASCULAR DISEASE | cardiomyopathy | CONGESTIVE-HEART-FAILURE | EXPRESSION | Humans | Roseolovirus Infections - physiopathology | DNA, Viral - isolation & purification | Enterovirus Infections - physiopathology | Hemorheology | Roseolovirus Infections - blood | Coxsackievirus Infections - virology | Herpesvirus 4, Human - isolation & purification | Cardiomyopathies - physiopathology | Enterovirus Infections - virology | Coxsackievirus Infections - blood | Vasodilation - physiology | C-Reactive Protein - analysis | Enterovirus Infections - blood | Herpesvirus 6, Human - isolation & purification | Parvovirus B19, Human - isolation & purification | Parvoviridae Infections - virology | Coxsackievirus Infections - physiopathology | Heart - virology | Parvoviridae Infections - physiopathology | Endothelium, Vascular - physiopathology | Myocardium - pathology | Enterovirus - isolation & purification | Parvoviridae Infections - blood | Myocarditis - blood | Adenovirus Infections, Human - physiopathology | Myocarditis - virology | Adenovirus Infections, Human - blood | Biopsy | Adenoviruses, Human - isolation & purification | Arteries - physiopathology | Roseolovirus Infections - virology | Hemodynamics | Myocarditis - physiopathology | Adenovirus Infections, Human - virology
Journal Article
Circulation, ISSN 0009-7322, 10/2013, Volume 128, Issue 14, pp. 1542 - 1554
BACKGROUND—Viral myocarditis follows a fatal course in ≈30% of patients. Interleukin-1 receptor–associated kinase 4 (IRAK4), a major nodal signal transducer in... 
heart failure | macrophages | interferons | cell movement | myocarditis | CARDIAC & CARDIOVASCULAR SYSTEMS | RECOGNITION | IFN-ALPHA/BETA | GENE INDUCTION | IRAK-4 | IRF-7 | RESPONSES | TOLL-LIKE RECEPTORS | PERIPHERAL VASCULAR DISEASE | INFECTION | EXPRESSION | VIRUSES | Interferons - biosynthesis | Interleukin-1 Receptor-Associated Kinases - genetics | Interferon Regulatory Factors - metabolism | Interleukin-1 Receptor-Associated Kinases - deficiency | Adoptive Transfer | Chemokine CCL5 - physiology | Enterovirus B, Human - physiology | Cell Movement - physiology | Interferon-Induced Helicase, IFIH1 | Chemokine CCL5 - deficiency | Coxsackievirus Infections - virology | STAT5 Transcription Factor - metabolism | STAT1 Transcription Factor - metabolism | CD11b Antigen - analysis | DEAD-box RNA Helicases - metabolism | Monocytes - physiology | Receptors, CCR5 - physiology | Dimerization | Myocarditis - immunology | Receptors, CCR5 - analysis | Disease Resistance | Coxsackievirus Infections - physiopathology | Mice, Inbred C57BL | Receptors, CCR5 - deficiency | Radiation Chimera | Mice, Knockout | Myocarditis - virology | Animals | Interleukin-1 Receptor-Associated Kinases - physiology | Virus Replication | Immunologic Deficiency Syndromes - physiopathology | Immunologic Deficiency Syndromes - genetics | Mice | Protein Processing, Post-Translational | Interferon Regulatory Factors - chemistry | Myocarditis - physiopathology | Coxsackievirus Infections - immunology | Myocarditis | Physiological aspects | Interleukin-1 | Monocytes | Interferon | Research
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2017, Volume 12, Issue 6, p. e0180029
Introduction Coxsackievirus B3 (CVB3) is known to induce acute and chronic myocarditis. Most infections are clinically unapparent but some patients suffer from... 
OVEREXPRESSION | ENTEROVIRUS-INDUCED MYOCARDITIS | POSTREPOLARIZATION REFRACTORINESS | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | MOUSE | INFECTION | DILATED CARDIOMYOPATHY | INDUCED PANCREATITIS | TRANSGENIC MICE | REPOLARIZATION | Coxsackievirus Infections - physiopathology | Tissue Culture Techniques | Mice, Inbred C57BL | Cells, Cultured | Viral Proteins - genetics | Male | Mice, Transgenic | Viral Proteins - metabolism | Arrhythmias, Cardiac - physiopathology | Arrhythmias, Cardiac - virology | Myocarditis - virology | Myocytes, Cardiac - virology | Patch-Clamp Techniques | Animals | Membrane Potentials | Myocytes, Cardiac - physiology | Heart Rate - physiology | Electrophysiological Phenomena | Chronic Disease | Myocarditis - physiopathology | Disease Models, Animal | Myocarditis | Complications and side effects | Care and treatment | Electrophysiology | Research | Diagnosis | Coxsackievirus infections | Risk factors | Cardiac arrhythmia | Animal models | Cardiomyopathy | Action potential | Infections | Genomes | Myocytes | Alterations | Thermal resistance | Transgenic animals | Rodents | Gangrene | Ion channels | Membrane potential | Cardiology | Heart diseases | Genotypes | Heart failure | Cardiomyocytes | Inflammation | Interdisciplinary aspects | Patients | Low level | Heart rate | Medicine | Hypotheses | Refractory period | Fibrosis | Death | Mice | Ventricle
Journal Article
Pediatric Diabetes, ISSN 1399-543X, 05/2013, Volume 14, Issue 3, pp. 149 - 158
Epidemiological data suggesting an infectious origin of diabetes pre-date the discovery of insulin; indeed it was the variation in mortality rates from... 
cytokine | epidemiology | pathogenesis | type 1 diabetes | viral infection | cytomegalovirus | enterovirus | rotavirus | coxsackievirus | β‐cell | apoptosis | Coxsackievirus | Cytomegalovirus | Rotavirus | Pathogenesis | Type 1 diabetes | Cytokine | Enterovirus | β-cell | Epidemiology | Viral infection | Apoptosis | ENCEPHALOMYOCARDITIS VIRUS | ROTAVIRUS INFECTION | BETA-CELLS | SEASONAL-VARIATION | cell | ENTEROVIRUS INFECTION | PANCREATIC-ISLET CELLS | BLOOD MONONUCLEAR-CELLS | CAPSID PROTEIN VP1 | RISING INCIDENCE | ENDOCRINOLOGY & METABOLISM | PEDIATRICS | COXSACKIE-B VIRUSES | Humans | Diabetes Mellitus, Type 1 - metabolism | Virus Diseases - virology | Enterovirus Infections - physiopathology | Interferon-Induced Helicase, IFIH1 | DEAD-box RNA Helicases - biosynthesis | Enterovirus Infections - virology | Virus Diseases - physiopathology | Diabetes Mellitus, Type 1 - etiology | Enterovirus Infections - immunology | DEAD-box RNA Helicases - metabolism | Disease Models, Animal | Genetic Predisposition to Disease | Virus Diseases - immunology | Islets of Langerhans - pathology | Diabetes Mellitus, Type 1 - genetics | Islets of Langerhans - immunology | Immunity, Innate | DEAD-box RNA Helicases - genetics | Animals | Virus Diseases - pathology | Enterovirus - immunology | Islets of Langerhans - virology | Enterovirus Infections - pathology | Virus diseases | Coxsackievirus infections
Journal Article
Circulation: Heart Failure, ISSN 1941-3289, 05/2015, Volume 8, Issue 4, pp. 809 - 818
Background-This study tested the hypothesis whether endoplasmic reticulum (ER) stress/C/EBP homologous protein (CHOP) signaling is linked with coxsackievirus... 
apoptosis | C/EBP homologous protein | endoplasmic reticulum stress | CELLS | PRESSURE-OVERLOAD | CARDIAC & CARDIOVASCULAR SYSTEMS | ER STRESS | AORTIC-VALVE CALCIFICATION | REPLICATION | HEPATITIS-C VIRUS | PATHWAY | B3 INFECTION | MICE | Transcription Factor CHOP - genetics | Caspase 3 - metabolism | Endoplasmic Reticulum - metabolism | Male | Taurochenodeoxycholic Acid - pharmacology | Coxsackievirus Infections - virology | Proto-Oncogene Proteins c-bcl-2 - metabolism | Endoplasmic Reticulum - drug effects | Myocarditis - prevention & control | Disease Models, Animal | Myocarditis - genetics | Enterovirus B, Human - growth & development | Virus Replication - drug effects | Antiviral Agents - pharmacology | Coxsackievirus Infections - physiopathology | Endoplasmic Reticulum Stress - drug effects | Signal Transduction | Coxsackievirus Infections - metabolism | Mice, Inbred C57BL | bcl-2-Associated X Protein - metabolism | Endoplasmic Reticulum - virology | Transcription Factor CHOP - deficiency | Enterovirus B, Human - drug effects | Mice, Knockout | Coxsackievirus Infections - pathology | Coxsackievirus Infections - prevention & control | Myocarditis - virology | Myocytes, Cardiac - pathology | Coxsackievirus Infections - genetics | Myocytes, Cardiac - virology | Animals | Myocytes, Cardiac - drug effects | Myocarditis - metabolism | Myocytes, Cardiac - metabolism | Enterovirus B, Human - pathogenicity | Myocarditis - pathology | Transcription Factor CHOP - metabolism | Myocarditis - physiopathology | Apoptosis
Journal Article
Archives of Virology, ISSN 0304-8608, 9/2013, Volume 158, Issue 9, pp. 1953 - 1963
Viral myocarditis (VMC) is a common cardiovascular disease, and microRNAs (miRNAs) have been postulated to be involved in its pathology. Using microarrays, we... 
Medical Microbiology | Infectious Diseases | Biomedicine | Virology | RHEUMATOID-ARTHRITIS | TGF-BETA | VIROLOGY | SYNOVIAL TISSUE | PATHWAY | ROR-GAMMA