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Neurobiology of Disease, ISSN 0969-9961, 2009, Volume 37, Issue 2, pp. 370 - 383
Abstract Transgenic mice expressing the human superoxide dismutase 1 (SOD-1) mutant at position 93 (G93A) develop a phenotype resembling amyotrophic lateral... 
Neurology | Choline acetyl transferase | Amyotrophic lateral sclerosis | Lithium | Synaptic boutons | Transgenic G93A mouse | Brainstem motor neurons | ADULT RATS | CELLS IN-VITRO | NITRIC-OXIDE SYNTHASE | CU/ZN SUPEROXIDE-DISMUTASE | SPINAL-CORD | AMYOTROPHIC-LATERAL-SCLEROSIS | NEUROSCIENCES | DIFFERENTIAL VULNERABILITY | HYPOGLOSSAL NERVE ANASTOMOSIS | NEURON DEGENERATION | DISEASE PROGRESSION | Neuroprotective Agents - therapeutic use | Amyotrophic Lateral Sclerosis - physiopathology | Humans | Nerve Degeneration - physiopathology | Vagus Nerve - drug effects | Facial Nerve - drug effects | Male | Cytoprotection - physiology | Amyotrophic Lateral Sclerosis - drug therapy | Brain Stem - drug effects | Choline O-Acetyltransferase - metabolism | Motor Neurons - pathology | Vagus Nerve - pathology | Hypoglossal Nerve - drug effects | Lithium - therapeutic use | Neuroprotective Agents - pharmacology | Cranial Nerves - pathology | Brain Stem - pathology | Nerve Degeneration - prevention & control | Cytoprotection - drug effects | Trigeminal Nerve - physiopathology | Disease Models, Animal | Biomarkers - metabolism | Drug Administration Schedule | Lithium - pharmacology | Biomarkers - analysis | Axons - drug effects | Trigeminal Nerve - pathology | Mice, Transgenic | Treatment Outcome | Brain Mapping - methods | Vagus Nerve - physiopathology | Hypoglossal Nerve - pathology | Motor Neurons - metabolism | Cranial Nerves - drug effects | Amyotrophic Lateral Sclerosis - pathology | Trigeminal Nerve - drug effects | Animals | Axons - pathology | Choline O-Acetyltransferase - analysis | Cranial Nerves - physiopathology | Brain Stem - physiopathology | Mice | Facial Nerve - pathology | Hypoglossal Nerve - physiopathology | Facial Nerve - physiopathology | Nerve Degeneration - drug therapy | Superoxide dismutase | Superoxide | Neurons | Analysis | Choline | Index Medicus
Journal Article
International Journal of Biological Macromolecules, ISSN 0141-8130, 03/2019, Volume 124, pp. 460 - 468
Facial nerve injury is a clinically common disease accompanied by demyelination of damaged nerves. The remyelination of damaged nerves and the unsatisfactory... 
PI3K-AKT-mTOR | Facial nerve injury | CXCL12 | Autophagy | Migration | ACTIVATION | POLYMER SCIENCE | REGENERATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CXCR4 | NLRP3 INFLAMMASOME | NGF | STEM | CRUSH INJURY | NEURITE OUTGROWTH | NEURONS | SECRETION | CHEMISTRY, APPLIED | Insulin-Like Growth Factor I - pharmacology | Microtubule-Associated Proteins - genetics | TOR Serine-Threonine Kinases - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Facial Nerve - drug effects | Male | Facial Nerve Injuries - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Autophagy - drug effects | Facial Nerve Injuries - drug therapy | Proto-Oncogene Proteins c-akt - genetics | Neuroprotective Agents - pharmacology | Protein Isoforms - metabolism | TOR Serine-Threonine Kinases - genetics | Cranial Nerves - pathology | Facial Nerve - metabolism | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Schwann Cells - drug effects | Adenine - analogs & derivatives | Signal Transduction | Facial Nerve Injuries - genetics | Cranial Nerves - metabolism | Gene Expression Regulation | Morpholines - pharmacology | Rats | Schwann Cells - pathology | Adenine - pharmacology | Schwann Cells - metabolism | Recombinant Proteins - pharmacology | Rats, Sprague-Dawley | Phosphatidylinositol 3-Kinases - genetics | Cell Movement - drug effects | Cranial Nerves - drug effects | Animals | Chemokine CXCL12 - pharmacology | Facial Nerve Injuries - pathology | Cell Proliferation - drug effects | Primary Cell Culture | Facial Nerve - pathology | Protein Isoforms - genetics | Index Medicus
Journal Article
American Journal of Therapeutics, ISSN 1075-2765, 2017, Volume 24, Issue 2, pp. e227 - e233
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, pp. e0122048 - e0122048
Dysregulation of the complement system is evident in many CNS diseases but mechanisms regulating complement activation in the CNS remain unclear. In a recent... 
ACTIVATION | INHIBITION | ALZHEIMERS-DISEASE | BIOLOGICAL MARKERS | MULTIDISCIPLINARY SCIENCES | NICOTINIC ACETYLCHOLINE-RECEPTOR | REGULATOR FACTOR-H | CENTRAL-NERVOUS-SYSTEM | CEREBROSPINAL-FLUID | CHOLINERGIC SYSTEMS | MASS-SPECTROMETRY | Recurrence | Cranial Nerve Injuries - immunology | Humans | Acetylcholinesterase - cerebrospinal fluid | Cranial Nerve Injuries - pathology | Male | Case-Control Studies | Neurofilament Proteins - cerebrospinal fluid | Butyrylcholinesterase - cerebrospinal fluid | Cranial Nerves - pathology | GPI-Linked Proteins - cerebrospinal fluid | Adult | Female | Cranial Nerves - immunology | Disability Evaluation | Severity of Illness Index | Multiple Sclerosis - cerebrospinal fluid | Cranial Nerves - metabolism | Complement C3 - cerebrospinal fluid | Remission Induction | Cranial Nerves - drug effects | Magnetic Resonance Imaging | Cranial Nerve Injuries - drug therapy | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Biomarkers - cerebrospinal fluid | Cranial Nerve Injuries - cerebrospinal fluid | Immunologic Factors - therapeutic use | Multiple Sclerosis - drug therapy | Disability | Complications and side effects | Multiple sclerosis | Care and treatment | Influence | Acetylcholine | Inflammation | Research | Risk factors | Neurosciences | Correlation | Nuclear magnetic resonance--NMR | Disease | Neurobiology | Central nervous system | Nervous system | Activation | Complement | Genomes | Cerebrospinal fluid | Acetylcholinesterase | Sclerosis | Immunology | Neurodegeneration | Rodents | Lesions | Light levels | Linkage analysis | Enzymes | Cytokines | Immunoregulation | Gene expression | Patients | Neurology | Complement activation | Magnetic resonance imaging | Complement component C3 | Index Medicus | Basic Medicine | Biological Sciences | Naturvetenskap | Immunologi | Medical and Health Sciences | Medicin och hälsovetenskap | Biologiska vetenskaper | Medicinska och farmaceutiska grundvetenskaper | Natural Sciences | Neurovetenskaper | Nuclear magnetic resonance | NMR
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2008, Volume 28, Issue 45, pp. 11731 - 11740
Journal Article
Clinical Neurology and Neurosurgery, ISSN 0303-8467, 2016, Volume 152, pp. 12 - 15
Journal Article
Journal Article
Journal Article
Neuroscience Research, ISSN 0168-0102, 2007, Volume 59, Issue 4, pp. 446 - 456
Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative disease characterized by progressive loss of brainstem and spinal motoneurons. Although... 
c-Met | Caspases | X chromosome-linked inhibitor of apoptosis protein (XIAP) | Monocyte chemoattractant protein-1 (MCP-1) | Microglia | PROLONGS SURVIVAL | MINOCYCLINE | NEUROTROPHIC FACTOR | NEURON DEATH | SPINAL-CORD | AMYOTROPHIC-LATERAL-SCLEROSIS | NEUROSCIENCES | CHEMOATTRACTANT PROTEIN-1 | MICE | caspases | microglia | monocyte chemoattractant protein-1 (MCP-1) | EXPRESSION | DISEASE PROGRESSION | Hepatocyte Growth Factor - therapeutic use | Neuroprotective Agents - therapeutic use | Microglia - metabolism | Superoxide Dismutase - genetics | Amyotrophic Lateral Sclerosis - physiopathology | Humans | Astrocytes - pathology | Brain Stem - metabolism | Nerve Degeneration - physiopathology | Male | Gliosis - physiopathology | Hepatocyte Growth Factor - pharmacology | Apoptosis Regulatory Proteins - drug effects | Amyotrophic Lateral Sclerosis - drug therapy | Brain Stem - drug effects | Motor Neurons - pathology | Neuroprotective Agents - pharmacology | Cranial Nerves - pathology | Nerve Degeneration - prevention & control | Microglia - pathology | Female | Chemokine CCL2 - metabolism | Gliosis - prevention & control | Motor Neurons - drug effects | Disease Models, Animal | Astrocytes - drug effects | Microglia - drug effects | Cranial Nerves - metabolism | Mice, Inbred C57BL | Mice, Transgenic | Treatment Outcome | Apoptosis Regulatory Proteins - metabolism | Motor Neurons - metabolism | Cranial Nerves - drug effects | Animals | Amyotrophic Lateral Sclerosis - metabolism | Brain Stem - physiopathology | Gliosis - drug therapy | Mice | Superoxide Dismutase-1 | Chemokine CCL2 - drug effects | Nerve Degeneration - drug therapy | Astrocytes - metabolism | Physiological aspects | Amyotrophic lateral sclerosis | Genetic engineering | Growth factors | Index Medicus
Journal Article
Journal Article