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Journal Article
Analytical Chemistry, ISSN 0003-2700, 05/2017, Volume 89, Issue 10, pp. 5467 - 5475
Journal Article
Nature Communications, ISSN 2041-1723, 02/2016, Volume 7, Issue 1, pp. 10572 - 10572
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2018, Volume 13, Issue 4, pp. e0195764 - e0195764
Tenofovir (TFV), a nucleotide reverse transcriptase inhibitor, requires two phosphorylation steps to form a competitive inhibitor of HIV reverse transcriptase.... 
BLOOD SPOT SAMPLES | PYRUVATE-KINASE | MULTIDISCIPLINARY SCIENCES | AMINO-ACID SUBSTITUTIONS | MUTATIONS | PREEXPOSURE PROPHYLAXIS | CREATINE-KINASE | Anti-HIV Agents - pharmacology | United States - epidemiology | Creatine Kinase, MM Form - genetics | HIV-1 - drug effects | HIV Infections - epidemiology | HIV Infections - genetics | HIV Infections - virology | Humans | South Africa - epidemiology | Tenofovir - pharmacology | Thailand - epidemiology | HIV-1 - genetics | Genetic Variation | HIV Infections - drug therapy | HIV-1 - enzymology | High-Throughput Nucleotide Sequencing | Adenylate Kinase - genetics | Pyruvate Kinase - genetics | Tenofovir | Antiviral agents | Complications and side effects | Usage | Care and treatment | Creatine kinase | Analysis | DNA polymerases | Dosage and administration | HIV (Viruses) | Phosphorylation | RNA-directed DNA polymerase | Laboratories | Liver | Escherichia coli | Homeostasis | Pyruvic acid | Genomes | Infections | Kinases | Creatine | Epidemiology | Blood | Red blood cells | Enzymatic activity | Acquired immune deficiency syndrome--AIDS | Human immunodeficiency virus--HIV | Bioinformatics | Public health | Deoxyribonucleic acid--DNA | Medical research | Pyruvate kinase | Muscles | Pharmacology | Metabolism | Disease control | Medicine | Disease prevention | Genetic variance | Inhibitors | Adenylate kinase | Hepatocytes | Womens health | Predictions | Mutation | Index Medicus | Acquired immune deficiency syndrome | Deoxyribonucleic acid | AIDS | HIV | DNA | Human immunodeficiency virus
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2017, Volume 12, Issue 3, pp. e0172965 - e0172965
Purpose Exertional rhabdomyolysis can occur in individuals performing various types of exercise but it is unclear why some individuals develop this condition... 
POLYMORPHISMS | PERFORMANCE | MULTIDISCIPLINARY SCIENCES | INJURY | ACTN3 GENOTYPE | EXERCISE | INDUCED MUSCLE DAMAGE | Myosin-Light-Chain Kinase - genetics | Prognosis | Humans | Middle Aged | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | Male | Gene Expression Profiling | Peptidyl-Dipeptidase A - genetics | Rhabdomyolysis - diagnosis | Insulin-Like Growth Factor II - genetics | Peptidyl-Dipeptidase A - blood | Interleukin-6 - blood | Actinin - genetics | Rhabdomyolysis - pathology | Adult | Female | Creatine Kinase, MM Form - blood | Physical Exertion | Creatine Kinase, MM Form - genetics | Interleukin-6 - genetics | Gene Expression Regulation | Insulin-Like Growth Factor II - metabolism | Actinin - blood | Rhabdomyolysis - genetics | Adolescent | Running | Myosin-Light-Chain Kinase - blood | Aged | Polymorphism, Single Nucleotide | Rhabdomyolysis - blood | Myoglobin - blood | Creatine kinase | Runners (Sports) | Genetic aspects | Research | Rhabdomyolysis | Single nucleotide polymorphisms | Marathon running | Health aspects | Risk factors | Myoglobin | Physical training | Laboratories | Anthropometry | Single-nucleotide polymorphism | Insulin-like growth factors | Creatine | Kinases | Myosin-light-chain kinase | Training | Interleukin 6 | Proteins | Signal transduction | Vitamin E | Rodents | Race | Genetics | Tumor necrosis factor-TNF | Insulin-like growth factor II | Genotypes | Enzymes | Cytokines | Muscles | Tumor necrosis factor-α | Metabolism | Gene expression | Musculoskeletal system | Physical fitness | Exercise physiology | Habits | Endocrinology | Index Medicus
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 06/2015, Volume 308, Issue 11, pp. C919 - C931
Myoblast fusion is critical for proper muscle growth and regeneration. During myoblast fusion, the localization of some molecules is spatially restricted;... 
Muscle growth | Myotube | Actin | Adenosine triphosphate | Myocyte | PHYSIOLOGY | myocyte | BINDING DOMAIN | GTPASE-ACTIVATING PROTEIN | BETA-ACTIN | muscle growth | SKELETAL-MUSCLE DIFFERENTIATION | FAMILY | CELL BIOLOGY | actin | IN-VITRO | myotube | GENE | adenosine triphosphate | MAMMALIAN SEPTIN | ACTIN CYTOSKELETON | CELL-FUSION | RNA, Small Interfering - genetics | Alternative Splicing | Creatine Kinase, MM Form - metabolism | Actins - metabolism | Actins - genetics | Cell Fusion | Protein Isoforms - metabolism | Gene Expression Regulation, Developmental | Myoblasts - cytology | Creatine Kinase, MM Form - genetics | Signal Transduction | Mice, Inbred C57BL | Creatine Kinase, BB Form - antagonists & inhibitors | Polymerization | Creatine Kinase, BB Form - metabolism | Protein Transport | Creatine Kinase, BB Form - genetics | Two-Hybrid System Techniques | Animals | Muscle Development - genetics | Muscle Fibers, Skeletal - cytology | Mice | Mice, Inbred BALB C | Primary Cell Culture | Myoblasts - enzymology | Muscle Fibers, Skeletal - enzymology | Protein Isoforms - genetics | RNA, Small Interfering - metabolism | Physiological aspects | Creatine | Phosphotransferases | Identification and classification | Protein-protein interactions | Heart muscle | Molecules | Biochemistry | Muscular system | Kinases | Fusion | Index Medicus
Journal Article