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Nature Communications, ISSN 2041-1723, 02/2016, Volume 7, Issue 1, p. 10572
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2017, Volume 12, Issue 3, p. e0172965
Purpose Exertional rhabdomyolysis can occur in individuals performing various types of exercise but it is unclear why some individuals develop this condition... 
POLYMORPHISMS | PERFORMANCE | MULTIDISCIPLINARY SCIENCES | INJURY | ACTN3 GENOTYPE | EXERCISE | INDUCED MUSCLE DAMAGE | Myosin-Light-Chain Kinase - genetics | Prognosis | Humans | Middle Aged | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | Male | Gene Expression Profiling | Peptidyl-Dipeptidase A - genetics | Rhabdomyolysis - diagnosis | Insulin-Like Growth Factor II - genetics | Peptidyl-Dipeptidase A - blood | Interleukin-6 - blood | Actinin - genetics | Rhabdomyolysis - pathology | Adult | Female | Creatine Kinase, MM Form - blood | Physical Exertion | Creatine Kinase, MM Form - genetics | Interleukin-6 - genetics | Gene Expression Regulation | Insulin-Like Growth Factor II - metabolism | Actinin - blood | Rhabdomyolysis - genetics | Adolescent | Running | Myosin-Light-Chain Kinase - blood | Aged | Polymorphism, Single Nucleotide | Rhabdomyolysis - blood | Myoglobin - blood | Creatine kinase | Runners (Sports) | Genetic aspects | Research | Rhabdomyolysis | Single nucleotide polymorphisms | Marathon running | Health aspects | Risk factors | Myoglobin | Physical training | Laboratories | Anthropometry | Single-nucleotide polymorphism | Insulin-like growth factors | Creatine | Kinases | Myosin-light-chain kinase | Training | Interleukin 6 | Proteins | Signal transduction | Vitamin E | Rodents | Race | Genetics | Tumor necrosis factor-TNF | Insulin-like growth factor II | Genotypes | Enzymes | Cytokines | Muscles | Tumor necrosis factor-α | Metabolism | Gene expression | Musculoskeletal system | Physical fitness | Exercise physiology | Habits | Endocrinology
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2018, Volume 13, Issue 4, p. e0195764
Tenofovir (TFV), a nucleotide reverse transcriptase inhibitor, requires two phosphorylation steps to form a competitive inhibitor of HIV reverse transcriptase.... 
BLOOD SPOT SAMPLES | PYRUVATE-KINASE | MULTIDISCIPLINARY SCIENCES | AMINO-ACID SUBSTITUTIONS | MUTATIONS | PREEXPOSURE PROPHYLAXIS | CREATINE-KINASE | Anti-HIV Agents - pharmacology | United States - epidemiology | Creatine Kinase, MM Form - genetics | HIV-1 - drug effects | HIV Infections - epidemiology | HIV Infections - genetics | HIV Infections - virology | Humans | South Africa - epidemiology | Tenofovir - pharmacology | Thailand - epidemiology | HIV-1 - genetics | Genetic Variation | HIV Infections - drug therapy | HIV-1 - enzymology | High-Throughput Nucleotide Sequencing | Adenylate Kinase - genetics | Pyruvate Kinase - genetics | Tenofovir | Antiviral agents | Complications and side effects | Usage | Care and treatment | Creatine kinase | Analysis | DNA polymerases | Dosage and administration | HIV (Viruses) | Phosphorylation | RNA-directed DNA polymerase | Laboratories | Liver | Homeostasis | Pyruvic acid | Genomes | Infections | Kinases | Creatine | Epidemiology | Blood | Red blood cells | Enzymatic activity | Acquired immune deficiency syndrome--AIDS | E coli | Human immunodeficiency virus--HIV | Bioinformatics | Public health | Deoxyribonucleic acid--DNA | Medical research | Pyruvate kinase | Muscles | Pharmacology | Metabolism | Disease control | Medicine | Disease prevention | Genetic variance | Inhibitors | Adenylate kinase | Hepatocytes | Womens health | Predictions | Mutation | Acquired immune deficiency syndrome | Deoxyribonucleic acid | AIDS | HIV | DNA | Human immunodeficiency virus
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 06/2015, Volume 308, Issue 11, pp. C919 - C931
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, p. e102492
Low temperatures may cause severe growth inhibition and mortality in fish. In order to understand the mechanism of cold tolerance, a transgenic zebrafish Tg... 
PROTEIN | MEDIATED BAC TRANSGENESIS | ACCLIMATION | MULTIDISCIPLINARY SCIENCES | GROWTH | TEMPERATURE TOLERANCE | GENE-EXPRESSION | MUSCLE | STEAROYL-COA DESATURASE-1 | FOS | CREATINE-KINASE | Creatine Kinase, MM Form - metabolism | Muscle, Skeletal - metabolism | Gene Expression Profiling | Biological Transport | Adenosine Triphosphate - metabolism | Energy Metabolism - genetics | Transgenes | Carps - metabolism | Promoter Regions, Genetic | Cold Temperature | Histone-Lysine N-Methyltransferase - genetics | Creatine Kinase, MM Form - genetics | Animals, Genetically Modified | Zebrafish Proteins - metabolism | Molecular Sequence Annotation | Circadian Rhythm - genetics | Gene Expression Regulation | Lipid Metabolism | Zebrafish - genetics | Animals | Histone-Lysine N-Methyltransferase - metabolism | Zebrafish - metabolism | Acclimatization - genetics | Zebrafish Proteins - genetics | Carps - genetics | Enzymes | DNA microarrays | Analysis | Fishes | Muscles | Animal genetic engineering | Health aspects | Energy metabolism | Neurosciences | Laboratories | Creatine kinase | Transgenic | Homeostasis | Lipids | Biochemistry | Kinases | Creatine | Proteins | Immunology | Enzymatic activity | Energy | Cold tolerance | Rodents | Fish | Physiology | Swimming | Lipid metabolism | Low temperature | Circadian rhythms | Medical research | Carp | Cytokines | Zebrafish | Marine biology | Acclimatization | Metabolism | Gene expression | Muscle contraction | Fatty acids | Biological activity | Skeletal muscle | Acclimation | Energy balance | Brain research | Swimming behavior | Thermal analysis | ATP
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2012, Volume 303, Issue 7, pp. H844 - H852
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2012, Volume 109, Issue 50, pp. 20590 - 20595
There is no effective treatment for the cardiomyopathy of the most common autosomal recessive ataxia, Friedreich ataxia (FA). This disease is due to decreased... 
Heart | Protein metabolism | Energy metabolism | Diet | Liver | Cardiomyopathies | Mice | Down regulation | Ferritins | Skeletal muscle | Ferroportin 1 | Hemojuvelin | Transferrin receptor 1 | Heme oxygenase | heme oxygenase | MECHANISM | HEME-SYNTHESIS | MULTIDISCIPLINARY SCIENCES | CARDIOMYOPATHY | TRAFFICKING | hemojuvelin | RETICULOCYTES | ferroportin 1 | SUCCINYLACETONE | METABOLISM | transferrin receptor 1 | TISSUES | FERRITIN | PROTEINS | Mitochondria, Heart - metabolism | Creatine Kinase, MM Form - metabolism | Friedreich Ataxia - pathology | Humans | Cardiomegaly - pathology | Iron, Dietary - administration & dosage | Iron-Binding Proteins - metabolism | Friedreich Ataxia - genetics | Mice, Mutant Strains | Myocardium - metabolism | Disease Models, Animal | Iron - blood | Myocardium - ultrastructure | Microscopy, Electron, Transmission | Friedreich Ataxia - metabolism | Creatine Kinase, MM Form - genetics | Signal Transduction | Spectroscopy, Mossbauer | Liver - metabolism | Iron - metabolism | Mice, Knockout | Animals | Cardiomegaly - prevention & control | Iron-Binding Proteins - antagonists & inhibitors | Iron-Binding Proteins - genetics | Iron Regulatory Protein 2 - metabolism | Cardiomegaly - metabolism | Physiological aspects | Ataxia | Development and progression | Iron in the body | Metabolism | Health aspects | Phosphotransferases | Biological Sciences
Journal Article
Journal Article