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Lancet Neurology, The, ISSN 1474-4422, 2014, Volume 13, Issue 2, pp. 150 - 158
Journal Article
Journal of Toxicology and Environmental Health, Part A, ISSN 1528-7394, 11/2011, Volume 74, Issue 22-24, pp. 1460 - 1468
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, pp. e0170266 - e0170266
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 10/2017, Volume 37, Issue 43, pp. 10278 - 10289
Creutzfeldt-Jakob disease (CJD) is a neurodegenerative disorder caused by prion protein (PrP) misfolding, clinically recognized by cognitive and motor... 
Prion | IL-1 receptor antagonist | Creutzfeldt-Jakob disease | Synaptic plasticity | Transgenic mice | Seizures | HIPPOCAMPAL SLICES | TEMPORAL-LOBE EPILEPSY | STATUS EPILEPTICUS | NEUROSCIENCES | CELLULAR PRION PROTEIN | transgenic mice | prion | seizures | RAT HIPPOCAMPUS | synaptic plasticity | LONG-TERM POTENTIATION | KAINATE-INDUCED SEIZURES | INFLAMMATORY CYTOKINES | NEURONAL EXCITABILITY | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Synaptic Transmission - physiology | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Seizures - drug therapy | Neuronal Plasticity - drug effects | Male | Seizures - metabolism | Hippocampus - drug effects | N-Methylaspartate - antagonists & inhibitors | Neuronal Plasticity - physiology | Interleukin-1beta - metabolism | Female | Synaptic Transmission - drug effects | Interleukin 1 Receptor Antagonist Protein - pharmacology | Disease Models, Animal | Interleukin-1beta - antagonists & inhibitors | Disease Susceptibility | N-Methylaspartate - metabolism | Mice, Inbred C57BL | Mice, Transgenic | Random Allocation | Mice, Knockout | Creutzfeldt-Jakob Syndrome - metabolism | Hippocampus - metabolism | Creutzfeldt-Jakob Syndrome - drug therapy | Animals | Interleukin 1 Receptor Antagonist Protein - therapeutic use | Signal Transduction - drug effects | Signal Transduction - physiology | Mice | Potentiation | Phosphorylation | Glutamic acid receptors (ionotropic) | Cognitive ability | Glutamic acid receptors | Interleukin 1 receptors | Proteins | Protein folding | BSE | Rodents | Interleukin 1 | Prion protein | Seizing | Neurodegenerative diseases | Astrocytes | Abnormalities | EEG | Long-term potentiation | N-Methyl-D-aspartic acid receptors | Plasticity (hippocampal) | Signaling | Plasticity (synaptic) | Glutamatergic transmission | Kainic acid | Neurotransmission | Hippocampus | Synapses | Index Medicus | Creutzfeldt–Jakob disease
Journal Article
Journal Article
Journal Article
Neurology, ISSN 0028-3878, 03/2004, Volume 62, Issue 5, pp. 714 - 718
Background: In cell culture experiments, flupirtine maleate ( FLU), a triaminopyridine compound, was able to protect neuronal cells from apoptotic cell death... 
APOPTOSIS | IN-VITRO | THERAPY | PROTEIN | ALZHEIMERS-DISEASE | CREUTZFELDT-JAKOB-DISEASE | CLINICAL NEUROLOGY | Neuropsychological Tests | Creutzfeldt-Jakob Syndrome - drug therapy | Aminopyridines - therapeutic use | Double-Blind Method | Humans | Middle Aged | Adult | Female | Male | Aged | Cognition - drug effects | Index Medicus | Abridged Index Medicus
Journal Article
Lancet Neurology, The, ISSN 1474-4422, 2014, Volume 13, Issue 2, pp. 130 - 132
  Additionally, drugs reported to be effective against one prion strain or in one animal genetic background are often ineffective against other strains or in... 
Neurology | THERAPEUTICS | TRIAL | EFFICACY | TETRACYCLINES | HUMAN PRION DISEASE | QUINACRINE | INFUSION | CLINICAL NEUROLOGY | INTRAVENTRICULAR PENTOSAN POLYSULFATE | Doxycycline - pharmacology | Female | Male | Creutzfeldt-Jakob Syndrome - drug therapy | Humans | Studies | Animals | Prions | Creutzfeldt-Jakob disease | Television programs | Spongiform encephalopathies | Methods | Index Medicus
Journal Article