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Clinical and experimental immunology, ISSN 0009-9104, 2014, Volume 176, Issue 2, pp. 266 - 274
Summary Inflammatory bowel diseases are characterized by a deregulated immune response targeting the gut bacterial flora. Mucosal... 
immune system | MHC class I‐like | innate T cells | inflammatory bowel diseases | Inflammatory bowel diseases | Innate T cells | MHC class I-like | Immune system | NATURAL-KILLER-CELLS | CYTOKINES | CROHNS-DISEASE | ULCERATIVE-COLITIS | IMMUNOLOGY | SUBSET | NKG2D | INFECTION | DEFINES | EXPRESSION | IMPROVES CYTOTOXICITY | T-Lymphocyte Subsets - immunology | Humans | Inflammatory Bowel Diseases - immunology | Interleukin-17 - immunology | NK Cell Lectin-Like Receptor Subfamily K - immunology | Ki-67 Antigen - metabolism | Male | Colitis, Ulcerative - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Ki-67 Antigen - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Intestinal Mucosa - immunology | Natural Killer T-Cells - metabolism | Inflammatory Bowel Diseases - pathology | Interleukins - immunology | Natural Killer T-Cells - pathology | Adult | Female | Receptors, Immunologic - immunology | Interleukin-17 - blood | Interleukins - blood | Colitis, Ulcerative - pathology | Inflammatory Bowel Diseases - blood | Colitis, Ulcerative - blood | Crohn Disease - immunology | Immunity, Innate - immunology | Microscopy, Confocal | T-Lymphocyte Subsets - pathology | Crohn Disease - pathology | T-Lymphocyte Subsets - metabolism | Crohn Disease - blood | Interferon-gamma - immunology | Interferon-gamma - blood | Receptors, Immunologic - metabolism | Intestinal Mucosa - pathology | Natural Killer T-Cells - immunology | Biological response modifiers | T cells | Analysis | Ulcerative colitis | Medical research | Bowel disease | Original
Journal Article
Trends in immunology, ISSN 1471-4906, 2011, Volume 32, Issue 9, pp. 395 - 401
T helper (Th)17 cells have been proposed to represent a new CD4+ T cell lineage that is important for host defense against fungi and extracellular bacteria, and the development of autoimmune diseases... 
Allergy and Immunology | HYPER-IGE SYNDROME | GROWTH-FACTOR-BETA | TGF-BETA | T(H)17 CELLS | ROR-GAMMA-T | IN-VIVO | HUMAN TH17 CELLS | CENTRAL-NERVOUS-SYSTEM | IMMUNOLOGY | T-H-17 CELLS | GENOME-WIDE ASSOCIATION | Autoimmune Diseases - physiopathology | Species Specificity | Humans | Interleukin-17 - immunology | STAT5 Transcription Factor - immunology | Mycoses - microbiology | Cell Differentiation - genetics | STAT5 Transcription Factor - metabolism | Signal Transduction - immunology | Interleukin-23 Subunit p19 - biosynthesis | Th17 Cells - metabolism | Bacterial Infections - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Th17 Cells - cytology | Genetic Variation - immunology | Autoimmune Diseases - pathology | Bacterial Infections - microbiology | STAT3 Transcription Factor - metabolism | Transforming Growth Factor beta - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Immunity, Cellular - genetics | Interleukin-17 - biosynthesis | Cell Differentiation - immunology | Animals | Th17 Cells - immunology | Mice | Interleukin-23 Subunit p19 - immunology | STAT3 Transcription Factor - immunology | Mycoses - immunology | Transforming Growth Factor beta - metabolism | Cell Lineage - immunology | T cells | Pathology | Transcription factors | Lymphocytes | Genes | Rodents | Rheumatoid arthritis | Autoimmune diseases | Kinases | Immune system | Crohns disease
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, p. e24092
...) infection on the pathology of celiac disease was evaluated. We found that basal production of Interferon-(IFN... 
CYTOKINES | GLIADIN | NECATOR-AMERICANUS | EXPANSION | BIOLOGY | REGULATORY T-CELLS | PARASITE INFECTION | ETHIOPIA | INTERLEUKIN-10 | PEPTIDE | CHILDREN | Forkhead Transcription Factors - immunology | Immunohistochemistry | Leukocytes, Mononuclear - metabolism | Intestinal Mucosa - metabolism | Humans | Interleukin-17 - immunology | Interferon-gamma - metabolism | Interleukin-2 - immunology | Time Factors | Celiac Disease - immunology | Forkhead Transcription Factors - metabolism | Intestinal Mucosa - immunology | Leukocytes, Mononuclear - immunology | Duodenum - metabolism | Celiac Disease - parasitology | Necator americanus - immunology | Interleukin-2 Receptor alpha Subunit - immunology | Duodenum - immunology | Interleukin-2 - metabolism | CD4 Antigens - immunology | Necatoriasis - parasitology | Gliadin - immunology | Necatoriasis - pathology | Interleukin-5 - metabolism | Necatoriasis - immunology | Cells, Cultured | Necator americanus - physiology | Clinical Trials as Topic | Host-Parasite Interactions - immunology | Interleukin-5 - immunology | Celiac Disease - pathology | Interleukin-17 - metabolism | Animals | Interleukin-2 Receptor alpha Subunit - metabolism | Biopsy | Duodenum - pathology | Interferon-gamma - immunology | Leukocytes, Mononuclear - cytology | CD4 Antigens - metabolism | Infection | Celiac disease | Immune response | Interleukins | Interferon | Biological response modifiers | Comparative analysis | Health aspects | Gluten | Cell culture | Mucosa | Interleukin | Helper cells | Clinical trials | Inflammatory response | Lymphocytes T | Infections | Parasites | Small intestine | Interleukin 5 | Immunology | Interleukin 2 | Lymphocytes | Bowel disease | Gastroenterology | Gliadin | CD25 antigen | Foxp3 protein | Medical research | Cytokines | Inflammation | Crohns disease | CD4 antigen | Whooping cough | Studies | Pathology | Hospitals | Diet | γ-Interferon | Interleukin 10 | Autoimmune diseases
Journal Article
Annual review of immunology, ISSN 1545-3278, 2009, Volume 27, Issue 1, pp. 621 - 668
The autoinflammatory diseases are characterized by seemingly unprovoked episodes of inflammation, without high-titer autoantibodies or antigen-specific T cells... 
Pulmonary fibrosis | Atherosclerosis | Innate immunity | Crohn's disease | Inflammasome | Type 2 diabetes mellitus | IL-1β | Ankylosing spondylitis | atherosclerosis | HEMOLYTIC-UREMIC SYNDROME | INTERLEUKIN-1 RECEPTOR ANTAGONIST | ankylosing spondylitis | JUVENILE IDIOPATHIC ARTHRITIS | IL-1 beta | INNATE IMMUNE-SYSTEM | ENDOPLASMIC-RETICULUM STRESS | IMMUNOLOGY | ENCODING MEVALONATE KINASE | UNFOLDED PROTEIN RESPONSE | innate immunity | FAMILIAL MEDITERRANEAN FEVER | inflammasome | MACROPHAGE ACTIVATION SYNDROME | pulmonary fibrosis | type 2 diabetes mellitus | NF-KAPPA-B | Tumor Necrosis Factor-alpha - metabolism | Phosphotransferases (Alcohol Group Acceptor) - immunology | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Tumor Necrosis Factor-alpha - genetics | NF-kappa B - immunology | NF-kappa B - metabolism | Interleukin-1beta - genetics | Autoimmune Diseases - genetics | Interleukin-1beta - metabolism | Tumor Necrosis Factor-alpha - immunology | Cytokines - genetics | Carrier Proteins - immunology | Cytokines - immunology | Autoimmune Diseases - classification | Cytokines - metabolism | Autoimmune Diseases - immunology | Interleukin-1beta - immunology | Phosphotransferases (Alcohol Group Acceptor) - genetics | Inflammation - immunology | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | NF-kappa B - genetics | Inflammation - classification | Inflammation - genetics | Crohn’s disease
Journal Article