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Gut, ISSN 0017-5749, 03/2016, Volume 65, Issue 3, pp. 415 - 425
Background Crohn's disease (CD)-associated dysbiosis is characterised by a loss of Faecalibacterium prausnitzii, whose culture supernatant exerts an... 
EPITHELIAL BARRIER | INFLAMMATORY-BOWEL-DISEASE | IMMUNE-SYSTEM | COLITIS | CAMPYLOBACTER-CONCISUS | FECAL MICROBIOTA | GUT | REGULATORY T-CELLS | GASTROENTEROLOGY & HEPATOLOGY | TIGHT JUNCTION | SEGMENTED FILAMENTOUS BACTERIA | Intestinal Mucosa - metabolism | Humans | Bacterial Proteins - chemistry | Crohn Disease - metabolism | Molecular Sequence Data | Male | NF-kappa B - metabolism | Dysbiosis - microbiology | Bacterial Proteins - therapeutic use | Colitis - chemically induced | Anti-Inflammatory Agents - therapeutic use | Crohn Disease - microbiology | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization | Biomarkers - metabolism | Amino Acid Sequence | Cell Line | Intestinal Mucosa - microbiology | Clostridiales - metabolism | Dysbiosis - metabolism | Animals | Crohn Disease - pathology | Colitis - metabolism | Bacterial Proteins - metabolism | Mice | Mice, Inbred BALB C | Dysbiosis - pathology | Bacterial Proteins - isolation & purification | Colitis - prevention & control | Intestinal Mucosa - pathology | Proteins | Inflammatory bowel disease | Probiotics | Immunology | Metabolites | E coli | Work stations | Homeostasis | Bacteria | Scientific imaging | Mass spectrometry | Crohns disease | Index Medicus | Abridged Index Medicus | Chemical Sciences | microbiota | Faecalibacterium prausnitzii | dysbiosis | Crohn’s disease | anti-inflammatory protein
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 146, Issue 2, pp. 508 - 519
Journal Article
Gastroenterology, ISSN 0016-5085, 2015, Volume 149, Issue 6, pp. 1564 - 1574.e3
Background & Aims Many patients with inflammatory bowel disease (IBD) fail to respond to anti–tumor necrosis factor (TNF) agents such as infliximab and... 
Gastroenterology and Hepatology | Therapy | Inflammation | EFFECTOR FUNCTIONS | CROHNS-DISEASE | VIVO | ULCERATIVE-COLITIS | MATRIX METALLOPROTEINASES | IN-VITRO | HINGE | MONOCLONAL-ANTIBODIES | INFLIXIMAB | ETANERCEPT | GASTROENTEROLOGY & HEPATOLOGY | Tumor Necrosis Factor-alpha - metabolism | Intestinal Mucosa - metabolism | Epitopes - metabolism | Humans | Inflammatory Bowel Diseases - immunology | Middle Aged | Crohn Disease - metabolism | Etanercept - metabolism | Colon - immunology | Male | Colitis, Ulcerative - immunology | Inflammatory Bowel Diseases - metabolism | Case-Control Studies | Intestinal Mucosa - drug effects | Immunoblotting - methods | Infliximab - metabolism | Proteolysis - drug effects | Intestinal Mucosa - immunology | Female | Colitis, Ulcerative - drug therapy | Biological Factors - metabolism | Inflammatory Bowel Diseases - drug therapy | Colon - pathology | Colitis, Ulcerative - metabolism | Antibodies, Monoclonal, Humanized - metabolism | Matrix Metalloproteinase 3 - metabolism | Matrix Metalloproteinase 12 - metabolism | Adalimumab - metabolism | Crohn Disease - immunology | Colon - metabolism | Biopsy | Crohn Disease - drug therapy | Biological Factors - pharmacology | Immunoglobulin G - metabolism | Medical colleges | Tumor necrosis factor | Gastrointestinal diseases | Tumors | Necrosis | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, pp. e49138 - e49138
Crohn's disease (CD) is an inflammatory bowel disease of complex etiology, although dysbiosis of the gut microbiota has been implicated in chronic... 
INFLAMMATORY-BOWEL-DISEASE | TWINS | DATABASE | SHOTGUN PROTEOMICS | PORPHYROMONAS-GINGIVALIS | MULTIDISCIPLINARY SCIENCES | PROTEIN FAMILIES | ULCERATIVE-COLITIS | RAG LOCUS | GUT MICROBIOTA | MASS-SPECTROMETRY | Metagenomics | Bacteria - metabolism | Ileum - pathology | Intestinal Mucosa - metabolism | Humans | Crohn Disease - metabolism | Gastrointestinal Tract - microbiology | Ileum - metabolism | Male | Proteome | Intestinal Mucosa - microbiology | Twins, Monozygotic | Bacteria - genetics | Gastrointestinal Tract - metabolism | Metabolic Networks and Pathways | Proteomics | Crohn Disease - microbiology | Female | Ileum - microbiology | Metagenome | Cluster Analysis | Intestinal Mucosa - pathology | Proteins | Microbiota (Symbiotic organisms) | Gastrointestinal diseases | Development and progression | Carbohydrate metabolism | Inflammation | Shotguns | Bioengineering | Dysbacteriosis | Inflammatory bowel diseases | Target recognition | Laboratories | Genomes | Biology | Data bases | Microbiota | Alterations | Acquired immune deficiency syndrome--AIDS | Etiology | Intestine | Gastroenterology | Bacteria | Bioindicators | Colon | Signatures | Bioinformatics | Carbohydrates | Internal medicine | Earth science | Metabolism | Ileum | Crohns disease | Medicine | Studies | Inflammatory bowel disease | Crohn's Disease | Acids | Biomarkers | Scientific imaging | Diagnostic systems | Mass spectrometry | Index Medicus | Medicin | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Acquired immune deficiency syndrome | AIDS
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2012, Volume 7, Issue 12, pp. e52132 - e52132
Crohn's disease (CD) is associated with intestinal dysbiosis evidenced by an altered microbiome forming thick biofilms on the epithelium. Additionally,... 
SYSTEM | STRAIN LF82 | INFLAMMATORY-BOWEL-DISEASE | GUT MICROBIOME | MULTIDISCIPLINARY SCIENCES | ILEAL MUCOSA | MUCOSAL FLORA | GRADIENT GEL-ELECTROPHORESIS | TYPE-1 PILI | BIOFILM FORMATION | E. COLI | Intestinal Mucosa - metabolism | Epithelial Cells - metabolism | Diet - adverse effects | Humans | Crohn Disease - metabolism | Disease Susceptibility - metabolism | Ileum - metabolism | Escherichia coli - cytology | Antigens, CD - metabolism | Bacterial Adhesion - physiology | Escherichia coli - metabolism | Crohn Disease - microbiology | Polysaccharides - administration & dosage | Escherichia coli - physiology | Macrophages - microbiology | Caco-2 Cells | Food Additives - administration & dosage | Escherichia coli Infections - microbiology | Escherichia coli Infections - metabolism | Intestinal Mucosa - microbiology | Disease Susceptibility - microbiology | Cell Adhesion Molecules - metabolism | GPI-Linked Proteins - metabolism | HT29 Cells | Macrophages - metabolism | Animals | Biofilms | Cell Adhesion - physiology | Epithelial Cells - microbiology | Cell Line, Tumor | Mice | Ileum - microbiology | Fimbriae, Bacterial - metabolism | Gastrointestinal diseases | Food additives | Escherichia coli | Physiological aspects | Bacteria | Disease susceptibility | Dextrins | Additives | Dysbacteriosis | Pathogenesis | Epithelial cells | Mucosa | Epidemiology | Streptococcus infections | Stabilizers (agents) | Polysaccharides | Microbiota | E coli | Intestine | Rodents | Gastroenterology | Cell adhesion | Lesions | Strains (organisms) | Saccharides | Enzymes | Starch | Environmental factors | Exposure | Metabolism | Epithelium | Patients | Ileum | Adhesion | Crohns disease | Medicine | Studies | Inflammatory bowel disease | Nutrition research | Crohn's Disease | Pili | Diet | Maltodextrin | Emulsifiers | Index Medicus
Journal Article
Journal Article
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 06/2013, Volume 172, Issue 3, pp. 410 - 416
Interleukin ( IL )‐37 is a member of the IL ‐1 cytokine family. We investigated IL ‐37b expression in the inflamed mucosa of inflammatory bowel disease ( IBD )... 
innate immunity | TNF | anti‐inflammatory cytokine | TNF-α | Innate immunity | Anti-inflammatory cytokine | CROHNS-DISEASE | TNF-alpha | IL-1 FAMILY | KAPPA-B | IMMUNOLOGY | PROTEIN-KINASE KINASE | IDENTIFICATION | COLONIC SUBEPITHELIAL MYOFIBROBLASTS | PATHOGENESIS | OVEREXPRESSION | INHIBITOR | anti-inflammatory cytokine | CELL | Crohn Disease - genetics | Interleukin-1 - genetics | Intestinal Mucosa - metabolism | Gene Expression - drug effects | Myofibroblasts - immunology | Humans | Inflammatory Bowel Diseases - immunology | Colitis, Ulcerative - genetics | Crohn Disease - metabolism | NF-kappa B - metabolism | Phosphatidylinositol 3-Kinases - metabolism | RNA, Messenger - metabolism | Colitis, Ulcerative - immunology | Inflammatory Bowel Diseases - metabolism | Transcription Factor AP-1 - metabolism | Case-Control Studies | Intestinal Mucosa - drug effects | MAP Kinase Signaling System | Myofibroblasts - metabolism | Intestinal Mucosa - immunology | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Adaptive Immunity | Caco-2 Cells | Interleukin-1 - metabolism | Myofibroblasts - pathology | Colitis, Ulcerative - metabolism | RNA, Messenger - genetics | Cells, Cultured | Colitis, Ulcerative - pathology | Immunity, Innate | Myofibroblasts - drug effects | Crohn Disease - immunology | Tumor Necrosis Factor-alpha - pharmacology | Chemokine CXCL10 - genetics | Crohn Disease - pathology | Chemokine CXCL10 - metabolism | Intestinal Mucosa - pathology | Interleukins | Biological response modifiers | Gastrointestinal diseases | Inflammatory bowel disease | Proteins | Colon | Cytokines | Index Medicus | Original
Journal Article
Gut, ISSN 0017-5749, 07/2008, Volume 57, Issue 7, pp. 903 - 910
Background and aims: Reduced ileal Paneth cell alpha-defensin expression has been reported to be associated with Crohn's disease, especially in patients... 
RT-PCR | QUANTIFICATION | SUSCEPTIBILITY | HUMAN INTESTINAL DEFENSIN | PANETH CELLS | SECRETION | GASTROENTEROLOGY & HEPATOLOGY | HOUSEKEEPING GENES | DIPEPTIDE | BOWEL-DISEASE | TRANSGENIC MICE | Crohn Disease - genetics | Interleukin-8 - genetics | Humans | Middle Aged |