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Gut, ISSN 0017-5749, 2019, p. gutjnl-2018-317540
The crypt population in the human intestine is dynamic: crypts can divide to produce two new daughter crypts through a process termed crypt fission, but... 
crypt fusion | crypt fission | mathematical modelling | lineage tracing | colon crypt | evolutionary dynamics
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e56020 - e56020
Background: Colon carcinogenesis is a multistep process and it emanates from a series of molecular and histopathological alterations. Glycyrrhizic acid (GA) is... 
K-RAS MUTATIONS | OXIDATIVE STRESS | MULTIDISCIPLINARY SCIENCES | CHRONIC HEPATITIS-C | MAST-CELLS | PROLIFERATIVE ACTIVITY | PRENEOPLASTIC LESIONS | NF-KAPPA-B | ABERRANT CRYPT FOCI | MUCIN-DEPLETED FOCI | PROMYELOTIC LEUKEMIA | Rats, Wistar | Apoptosis - drug effects | Colon - drug effects | Colon - immunology | Male | Aberrant Crypt Foci - immunology | Mucins - analysis | Tumor Necrosis Factor-alpha - immunology | Anti-Inflammatory Agents - therapeutic use | Sialomucins - analysis | Aberrant Crypt Foci - prevention & control | Anticarcinogenic Agents - therapeutic use | Connexin 43 - analysis | Aberrant Crypt Foci - chemically induced | Mast Cells - immunology | Colon - pathology | Colorectal Neoplasms - prevention & control | Rats | Aberrant Crypt Foci - pathology | Connexin 43 - immunology | Colorectal Neoplasms - chemically induced | Mast Cells - drug effects | Tumor Necrosis Factor-alpha - analysis | Dimethylhydrazines | Glycyrrhizic Acid - therapeutic use | Animals | Colorectal Neoplasms - immunology | Mast Cells - pathology | Cell Proliferation - drug effects | Colorectal Neoplasms - pathology | Prevention | Antiviral agents | Colon cancer | Oncology, Experimental | Inflammation | Research | Tumor proteins | Vascular endothelial growth factor | Cancer | Apoptosis | Biological properties | p53 Protein | Colorectal cancer | Connexin 43 | Caspase-3 | Carcinogenesis | Angiogenesis | Carcinogens | Toxicology | Rodents | Tumor necrosis factor-TNF | Colon | Supplementation | Lesions | NF-κB protein | Cytokines | Cyclin-dependent kinases | Caspase | Pharmacology | Tumor necrosis factor-α | Nitric-oxide synthase | Mucin | Acids | Natural products | Caspase-9 | Mast cells | Infiltration | Mutation | Cyclooxygenase-2 | Aberration | Tumors | Index Medicus
Journal Article
European Journal of Cancer Prevention, ISSN 0959-8278, 2018, Volume 27, Issue 2, pp. 110 - 117
Journal Article
AMERICAN JOURNAL OF SURGICAL PATHOLOGY, ISSN 0147-5185, 01/2008, Volume 32, Issue 1, pp. 21 - 29
The morphologic distinction between various serrated polyps of the colorectum may be challenging. The distinction between sessile serrated adenoma (SSA) and... 
traditional serrated adenoma | SURGERY | HYPERPLASTIC POLYPS | MICROSATELLITE-UNSTABLE ADENOCARCINOMAS | LARGE-INTESTINE | SELF-RENEWAL | PATHOLOGY | BETA-CATENIN | ectopic crypt formation | IMMUNOHISTOCHEMICAL ANALYSIS | sessile serrated adenoma | COLORECTAL-CANCER | colon | CRYPT | STEM-CELL | JUVENILE POLYPOSIS | crypt compartmentalization aberration
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 03/2017, Volume 56, Issue 3, pp. 913 - 922
Journal Article
Carcinogenesis, ISSN 0143-3334, 12/2013, Volume 34, Issue 12, pp. 2814 - 2822
The antitumorigenic activities of polyphenols such as ellagitannins and anthocyanins in pomegranate (Punica granatum L.) have been previously studied where... 
COLON-CANCER CELLS | IN-VITRO | CARCINOGENESIS | ONCOLOGY | HIGH-FAT DIET | CHEMOPREVENTIVE ACTIVITIES | PROSTATE-CANCER | GROWTH | FACTOR-KAPPA-B | JUICE | EXTRACT | Inflammation - chemically induced | TOR Serine-Threonine Kinases - metabolism | Colorectal Neoplasms - genetics | Humans | Polyphenols - pharmacology | Male | MicroRNAs - metabolism | Azoxymethane - adverse effects | Proto-Oncogene Proteins c-akt - genetics | Aberrant Crypt Foci - genetics | Inflammation - metabolism | TOR Serine-Threonine Kinases - genetics | Cyclooxygenase 2 - genetics | Inflammation - drug therapy | Vascular Cell Adhesion Molecule-1 - genetics | Punicaceae | Beverages | Proto-Oncogene Proteins c-akt - metabolism | Anticarcinogenic Agents - pharmacology | Phosphatidylinositol 3-Kinase - metabolism | Colorectal Neoplasms - metabolism | Aberrant Crypt Foci - drug therapy | Aberrant Crypt Foci - chemically induced | Rats | Colorectal Neoplasms - chemically induced | Rats, Sprague-Dawley | Aberrant Crypt Foci - metabolism | HT29 Cells | Animals | Nitric Oxide Synthase Type II - genetics | Phosphatidylinositol 3-Kinase - genetics | Cyclooxygenase 2 - metabolism | Cell Line, Tumor | Inflammation - genetics | Cell Proliferation - drug effects | Colorectal Neoplasms - diet therapy | MicroRNAs - genetics | Vascular Cell Adhesion Molecule-1 - metabolism | Nitric Oxide Synthase Type II - metabolism | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e91246 - e91246
Background: Based on the potential of Schiff base compounds to act as sources for the development of cancer chemotherapeutic agents, this in vivo study was... 
COLON-CANCER | APOPTOSIS | CELLS | OXIDATIVE STRESS | CARCINOGENESIS | MULTIDISCIPLINARY SCIENCES | COMPOUND | PROLIFERATION | INDUCTION | PROTEASOME INHIBITORS | ABERRANT CRYPT FOCI | Blood Chemical Analysis | Body Weight - drug effects | Male | Toxicity Tests, Acute | Azoxymethane - adverse effects | Schiff Bases - pharmacology | Carcinogens | Kidney - metabolism | Copper - chemistry | Liver - drug effects | Colorectal Neoplasms - drug therapy | Female | Colorectal Neoplasms - metabolism | Disease Models, Animal | Aberrant Crypt Foci - drug therapy | Aberrant Crypt Foci - chemically induced | Kidney - drug effects | Oxidation-Reduction | Chemoprevention | Liver - metabolism | Rats | Aberrant Crypt Foci - pathology | Colorectal Neoplasms - chemically induced | Aberrant Crypt Foci - metabolism | Animals | Schiff Bases - chemistry | Colorectal Neoplasms - pathology | Proliferating Cell Nuclear Antigen - metabolism | Prevention | Immunohistochemistry | Schiff bases | Chemotherapy | Colorectal cancer | Copper compounds | Cancer | Bax protein | 5-Fluorouracil | Colorectal carcinoma | Superoxide dismutase | PCNA protein | Colon cancer | Azoxymethane | Catalase | Imines | Proliferating cell nuclear antigen | Peroxidase | Colon | Copper | Coordination compounds | Glutathione | Glutathione peroxidase | Dysplasia | Superoxide | Malondialdehyde | In vivo methods and tests | Cyclooxygenase-2 | Aberration | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, pp. e60939 - e60939
Obesity is an important risk factor for colon cancer in humans, and numerous studies have shown that a high fat diet enhances colon cancer development. As both... 
COLORECTAL CARCINOGENESIS | KK-A(Y) MICE | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | FAT | RATS | BODY-MASS INDEX | CANCER | TUMORIGENESIS | ABERRANT CRYPT FOCI | Obesity - immunology | Aberrant Crypt Foci - complications | Adenoma - immunology | Colon - immunology | Colonic Neoplasms - chemically induced | Male | Aberrant Crypt Foci - immunology | Adenoma - complications | Azoxymethane | Colonic Neoplasms - immunology | Diet, High-Fat | Tumor Necrosis Factor-alpha - immunology | Aberrant Crypt Foci - chemically induced | Colon - pathology | Obesity - complications | Mice, Inbred C57BL | Risk Factors | Interleukin-1beta - immunology | Aberrant Crypt Foci - pathology | Colonic Neoplasms - complications | Obesity - pathology | Animals | Colonic Neoplasms - pathology | Interferon-gamma - immunology | Interleukin-6 - immunology | Adenoma - pathology | Mice | Adenoma - chemically induced | Obesity | Development and progression | Colon cancer | Research | Risk factors | Tumors | Adipose tissue | Interleukin | High fat diet | Interleukin 6 | Carcinogens | Developmental stages | Rodents | Tumor necrosis factor-TNF | Tumorigenesis | Colon | Lesions | Cytokines | Health risks | Inflammation | Group dynamics | Tumor necrosis factor-α | Insulin resistance | Interferon | Aberration | Cancer | Apoptosis | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 9, pp. e13027 - e13027
Inflammatory bowel disease (IBD) is a disorder of chronic inflammation with increased susceptibility to colorectal cancer. The etiology of IBD is unclear but... 
INFLAMMATORY-BOWEL-DISEASE | TGF-BETA | DENDRITIC CELLS | EPITHELIAL-CELLS | NITRIC-OXIDE SYNTHASE | COLORECTAL-CANCER | IN-VIVO | BIOLOGY | REGULATORY T-CELLS | SPONTANEOUS INTESTINAL TUMORIGENESIS | CUTTING EDGE | Aberrant Crypt Foci - etiology | Toll-Like Receptor 2 - genetics | Colitis - genetics | Humans | Colitis - complications | Male | Aberrant Crypt Foci - immunology | Colorectal Neoplasms - etiology | Female | Colitis - immunology | Aberrant Crypt Foci - prevention & control | Disease Models, Animal | Signal Transduction | Mice, Inbred C57BL | Colorectal Neoplasms - prevention & control | Aberrant Crypt Foci - pathology | Immunity, Innate | Mice, Knockout | Neoplastic Processes | Animals | Colorectal Neoplasms - immunology | Interleukin-6 - immunology | Toll-Like Receptor 2 - immunology | Mice | Colorectal Neoplasms - pathology | Gastrointestinal diseases | Oncology, Experimental | Colorectal cancer | Colitis | Research | Risk factors | Cancer | Diseases | Immunohistochemistry | Haplotypes | Pediatrics | Inflammatory bowel diseases | Epithelial cells | Physicians | Colorectal carcinoma | Homeostasis | Innate immunity | Genomes | Immunity | Interleukin 6 | Proteins | Microorganisms | Histopathology | Immunology | Lymphocytes | Etiology | Intestine | Rodents | Toll-like receptors | Bacteria | Tumorigenesis | Medical research | Immune response | Cytokines | Stat3 protein | Inflammation | Epithelium | Crohns disease | Immune systems | Medicine | Inflammatory bowel disease | Signaling | Infectious diseases | TLR2 protein | Regulation | Aberration | Immunofluorescence | Tumors | Index Medicus
Journal Article
Journal Article
Journal Article
Gut, ISSN 0017-5749, 06/2013, Volume 62, Issue 6, pp. 882 - 889
Journal Article