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Journal of Neurochemistry, ISSN 0022-3042, 09/2011, Volume 118, Issue 5, pp. 826 - 840
Inflammation contributes to neurodegeneration in post-ischemic brain, diabetes, and Alzheimer's disease. Participants in this inflammatory response include... 
gap junctions | Alzheimer’s disease | amyloid β‐peptide | cytokines | diabetes mellitus | pannexin | stroke | connexin | amyloid β-peptide | Alzheimer's disease | BRAIN-INJURY | GAP JUNCTIONAL COMMUNICATION | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMYLOID PLAQUES | ASTROCYTES | NEUROTOXICITY | NEUROSCIENCES | BETA | IN-VIVO | CHANNELS | RECEPTORS | amyloid beta-peptide | Tumor Necrosis Factor-alpha - metabolism | Oxygen - pharmacology | Cell Hypoxia - physiology | Amyloid beta-Peptides - pharmacology | Microtubule-Associated Proteins - pharmacology | Culture Media, Conditioned - pharmacology | Lanthanum - pharmacology | Peptide Fragments - pharmacology | Cerebral Cortex - cytology | Adenosine Triphosphate - pharmacology | Time Factors | Interleukin-1beta - metabolism | Adenosine Triphosphate - metabolism | Neuropeptides - pharmacology | Nerve Tissue Proteins - pharmacology | Neurons - physiology | Astrocytes - chemistry | Female | Cell Death - drug effects | Connexins - pharmacology | Neurons - drug effects | Animals, Newborn | Astrocytes - drug effects | Cell Hypoxia - drug effects | Connexin 43 - metabolism | Glutamic Acid - pharmacology | Cells, Cultured | Fluoresceins | Coculture Techniques - methods | Connexin 43 - deficiency | Connexins - metabolism | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Biotinylation - methods | Glutamic Acid - metabolism | Mice | Astrocytes - metabolism | Methyl aspartate | Neurosciences | Peptides | Neurons | Diabetes | Glucose | Glutamate | Dextrose | Index Medicus
Journal Article
Journal Article
Nature Neuroscience, ISSN 1097-6256, 01/2005, Volume 8, Issue 1, pp. 79 - 84
A central unresolved problem in research on Alzheimer disease is the nature of the molecular entity causing dementia. Here we provide the first direct... 
Cricetinae | Cricetulus | Amyloid beta-Peptides - pharmacology | Cognition Disorders - chemically induced | Humans | Injections, Intraventricular | Rats | Culture Media, Conditioned - pharmacology | Male | Rats, Sprague-Dawley | Animals | Conditioning, Operant - drug effects | Female | Amyloid beta-Peptides - chemistry | CHO Cells | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 01/2009, Volume 457, Issue 7225, pp. 102 - 106
Metastatic progression depends on genetic alterations intrinsic to cancer cells as well as the inflammatory microenvironment of advanced tumours. To understand... 
LEWIS-LUNG-CARCINOMA | ANGIOGENESIS | CANCER METASTASIS | INFLAMMATION | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | HYALURONAN | PROTEOGLYCAN | TUMOR-GROWTH | INNATE IMMUNITY | Tumor Necrosis Factor-alpha - metabolism | Neoplasm Transplantation | Carcinoma, Lewis Lung - secretion | Toll-Like Receptor 2 - agonists | Lung Neoplasms - metabolism | Culture Media, Conditioned - pharmacology | Lung Neoplasms - pathology | Culture Media, Serum-Free - metabolism | Lipopolysaccharide Receptors - metabolism | Macrophages - secretion | Lung Neoplasms - secondary | Liver Neoplasms - secondary | Interleukin-6 - metabolism | Toll-Like Receptor 6 - metabolism | Mice, Inbred C57BL | Tumor Necrosis Factor-alpha - secretion | Toll-Like Receptor 2 - metabolism | Versicans - pharmacology | Carcinoma, Lewis Lung - metabolism | Interleukin-6 - secretion | Macrophage Activation | Macrophages - metabolism | Animals | Neoplasm Metastasis - pathology | Carcinoma, Lewis Lung - pathology | Mice | Versicans - metabolism | Culture Media, Conditioned - metabolism | Care and treatment | Cell receptors | Cancer cells | Physiological aspects | Genetic aspects | Metastasis | Research | Health aspects | Risk factors | Proteins | Immunization | Adrenal glands | Rodents | Mortality | Gram-positive bacteria | Cancer | Apoptosis | Cell adhesion & migration | Index Medicus
Journal Article
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 02/2010, Volume 30, Issue 7, pp. 2636 - 2649
Amyloid beta (A beta)-containing plaques are surrounded by dystrophic neurites in the Alzheimer's disease (AD) brain, but whether and how plaques induce these... 
HIPPOCAMPAL SYNAPTIC PLASTICITY | COGNITIVE DECLINE | ALZHEIMERS-DISEASE |