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1. Full Text NLR members NLRC4 and NLRP3 mediate sterile inflammasome activation in microglia and astrocytes
by Freeman, Leslie and Guo, Haitao and David, Clément N and Brickey, W. June and Jha, Sushmita and Ting, Jenny P.-Y
The Journal of experimental medicine, ISSN 0022-1007, 05/2017, Volume 214, Issue 5, pp. 1351 - 1370
Immunology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Astrocytes - drug effects | Inflammation - pathology | Microglia - drug effects | Mice, Inbred C57BL | Astrocytes - pathology | Lysophosphatidylcholines - metabolism | Male | Mice, Knockout | Inflammasomes - physiology | Astrocytes - physiology | Animals | Microglia - physiology | Cuprizone - pharmacology | Calcium-Binding Proteins - physiology | Microglia - pathology | Apoptosis Regulatory Proteins - physiology | Mice | NLR Family, Pyrin Domain-Containing 3 Protein - physiology | Disease Models, Animal | Inflammation - physiopathology | Efflux | Brain | Multiple sclerosis | Stroke | Calcium mobilization | Calcium | Caspase-11 | Astrocytes | Caspase | Activation | Inflammation | Lysophosphatidylcholine | Caspase-1 | Microglia | Damage patterns | Neurological diseases | Microorganisms | Gliosis | Cuprizone | Neurodegeneration | Demyelination | Calcium efflux | Potassium | Apoptosis | Index Medicus | 306
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2. Full Text Cuprizone-Induced Demyelination as a Tool to Study Remyelination and Axonal Protection
by Zendedel, Adib and Beyer, Cordian and Kipp, Markus
Journal of molecular neuroscience, ISSN 0895-8696, 10/2013, Volume 51, Issue 2, pp. 567 - 572
Neurochemistry | Neurology | Neurosciences | Biomedicine | Cuprizone | Demyelination | Proteomics | Axon | Remyelination | Cell Biology | Biochemistry & Molecular Biology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Cuprizone - toxicity | Animals | Chelating Agents - toxicity | Cuprizone - pharmacology | Axons - pathology | Humans | Myelin Sheath - drug effects | Chelating Agents - pharmacology | Myelin Sheath - metabolism | Multiple sclerosis | Neurons | Physiological aspects | Research | Health aspects | Risk factors | Neurotoxic agents | Index Medicus
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3. Full Text Acute axonal damage in three different murine models of multiple sclerosis: A comparative approach
by Höflich, Katharina Marie and Beyer, Cordian and Clarner, Tim and Schmitz, Christoph and Nyamoya, Stella and Kipp, Markus and Hochstrasser, Tanja
Brain research, ISSN 0006-8993, 2016, Volume 1650, pp. 125 - 133
Neurology | Lysolecithin | Experimental autoimmune encephalomyelitis | APP | Axonal damage | Cuprizone | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Immunohistochemistry | Demyelinating Diseases - physiopathology | Microglia - metabolism | Encephalomyelitis, Autoimmune, Experimental - pathology | Spinal Cord - metabolism | Myelin Sheath - pathology | Mice, Inbred C57BL | Nerve Degeneration - physiopathology | Axons - metabolism | Brain - metabolism | Cuprizone - metabolism | Multiple Sclerosis - physiopathology | Inflammation - metabolism | Animals | Cuprizone - pharmacology | Corpus Callosum - pathology | Amyloid beta-Protein Precursor - metabolism | Models, Animal | Mice | Multiple sclerosis | Amyloid beta-protein | Models | T cells | Comparative analysis | Glutamate | Neurophysiology | Index Medicus
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4. Full Text Cuprizone-Containing Pellets Are Less Potent to Induce Consistent Demyelination in the Corpus Callosum of C57BL/6 Mice
by Hochstrasser, Tanja and Exner, Gianna Lisa and Nyamoya, Stella and Schmitz, Christoph and Kipp, Markus
Journal of molecular neuroscience, ISSN 0895-8696, 4/2017, Volume 61, Issue 4, pp. 617 - 624
Neurochemistry | Neurology | Pellet | Neurosciences | Biomedicine | Cuprizone | Demyelination | Proteomics | Corpus callosum | Cell Biology | Biochemistry & Molecular Biology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Cuprizone - toxicity | Administration, Oral | Mice, Inbred C57BL | Corpus Callosum - drug effects | Male | Drug Implants - adverse effects | Cuprizone - administration & dosage | Animals | Corpus Callosum - pathology | Demyelinating Diseases - etiology | Mice | Demyelinating Diseases - pathology | Disease Models, Animal | Index Medicus
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5. Full Text A regenerative approach to the treatment of multiple sclerosis
by Deshmukh, Vishal A and Tardif, Virginie and Lyssiotis, Costas A and Green, Chelsea C and Kerman, Bilal and Kim, Hyung Joon and Padmanabhan, Krishnan and Swoboda, Jonathan G and Ahmad, Insha and Kondo, Toru and Gage, Fred H and Theofilopoulos, Argyrios N and Lawson, Brian R and Schultz, Peter G and Lairson, Luke L
Nature (London), ISSN 0028-0836, 2013, Volume 502, Issue 7471, pp. 327 - 332
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Cuprizone - therapeutic use | Oligodendroglia - metabolism | Optic Nerve - cytology | Recurrence | Antiparkinson Agents - pharmacology | Coculture Techniques | Myelin Sheath - drug effects | Receptor, Muscarinic M1 - antagonists & inhibitors | Stem Cells - cytology | Myelin Proteolipid Protein - pharmacology | Myelin Sheath - metabolism | Antiparkinson Agents - therapeutic use | Oligodendroglia - drug effects | Cuprizone - pharmacology | Encephalomyelitis, Autoimmune, Experimental - chemically induced | Propylene Glycols - therapeutic use | Immune System - immunology | Female | Oligodendroglia - cytology | Benztropine - therapeutic use | Propylene Glycols - pharmacology | Encephalomyelitis, Autoimmune, Experimental - pathology | Myelin Sheath - pathology | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Mice, Inbred C57BL | Receptor, Muscarinic M3 - metabolism | Fingolimod Hydrochloride | Rats | Receptor, Muscarinic M3 - antagonists & inhibitors | Oligodendroglia - pathology | Sphingosine - pharmacology | Regeneration - drug effects | Sphingosine - analogs & derivatives | Animals | Cell Differentiation - drug effects | Models, Biological | Immune System - drug effects | Multiple Sclerosis - pathology | Stem Cells - drug effects | Sphingosine - therapeutic use | Mice | Benztropine - pharmacology | Receptor, Muscarinic M1 - metabolism | Multiple Sclerosis - drug therapy | Benztropine | Multiple sclerosis | Myelination | Physiological aspects | Oligodendroglia | Dosage and administration | Drug therapy | Cell differentiation | Cell culture | Dopamine | Drug dosages | Rodents | Cell cycle | Index Medicus
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6. Full Text TREM2 sustains microglial expansion during aging and response to demyelination
by Poliani, Pietro Luigi and Wang, Yaming and Fontana, Elena and Robinette, Michelle L and Yamanishi, Yoshinori and Gilfillan, Susan and Colonna, Marco
The Journal of clinical investigation, ISSN 0021-9738, 05/2015, Volume 125, Issue 5, pp. 2161 - 2170
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Receptors, Immunologic - physiology | Cell Count | Humans | Nerve Tissue Proteins - deficiency | Gene Expression Profiling | Phagocytosis - genetics | Myelin Sheath - metabolism | Membrane Glycoproteins - physiology | Microglia - immunology | Osteochondrodysplasias - genetics | Nerve Tissue Proteins - biosynthesis | Microglia - pathology | Transcription, Genetic | Demyelinating Diseases - chemically induced | Demyelinating Diseases - pathology | Genes, Reporter | Disease Models, Animal | Cuprizone - toxicity | Myelin Sheath - physiology | Nerve Tissue Proteins - physiology | Receptors, Immunologic - deficiency | Nerve Tissue Proteins - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Lipodystrophy - genetics | Regeneration | Animals | Axons - pathology | Subacute Sclerosing Panencephalitis - genetics | Brain - pathology | Mice | Receptors, Immunologic - genetics | Membrane Glycoproteins - deficiency | Aging - metabolism | Physiological aspects | Development and progression | Cell receptors | Research | Demyelinating diseases | Phosphorylation | Statistical analysis | Lipids | Data processing | Genomes | Kinases | Gene expression | Brain research | Blood-brain barrier | Rodents | Morphology | Aging | Alzheimers disease | Expansion | Apoptosis | Index Medicus | Abridged Index Medicus
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7. Full Text Glial response during cuprizone-induced de- and remyelination in the CNS: Lessons learned
by Gudi, Viktoria and Gingele, Stefan and Skripuletz, Thomas and Stangel, Martin
Frontiers in cellular neuroscience, ISSN 1662-5102, 03/2014, Volume 8, pp. 73 - 73
Astrocytes | Demyelination | Growth factors | Remyelination | Cuprizone mouse model | Microglia | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Animal models | Multiple sclerosis | Transcription factors | Myelin | Central nervous system | Nervous system | Insulin-like growth factors | Gene expression | Substantia grisea | Insulin | Pathology | Myelination | Gliosis | Molecular modelling | Diet | Cuprizone | Oligodendrocytes | Fibroblasts | Copper | Phagocytosis | Apoptosis | cuprizone mouse model | growth factors | demyelination | remyelination
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