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Molecular Nutrition and Food Research, ISSN 1613-4125, 05/2012, Volume 56, Issue 5, pp. 691 - 701
Journal Article
Molecular and Cellular Biochemistry, ISSN 0300-8177, 2/2007, Volume 296, Issue 1, pp. 85 - 95
Many studies have been performed with the aim of developing effective resistance modulators to overcome the multidrug resistance (MDR) of human cancers. Potent... 
Life Sciences | Biochemistry, general | P-glycoprotein | tetrahydrocurcumin | multidrug resistance | ATP hydrolysis | drug transport | Oncology | Medical Biochemistry | Cardiology | chemosensitivity | ABC transporter | Drug transport | Multidrug resistance | Chemosensitivity | Tetrahydrocurcumin | MECHANISM | INVOLVEMENT | CHEMOPREVENTIVE AGENT CURCUMIN | CATALYTIC TRANSITION-STATE | CELL BIOLOGY | MICROORGANISMS | BREAST-CANCER CELLS | IN-VITRO | INHIBITION | PROTECTIVE ROLE | ATP Binding Cassette Transporter, Sub-Family G, Member 2 | Vinblastine - metabolism | Curcumin - analogs & derivatives | Fluorides - metabolism | Curcumin - chemistry | Fluoresceins - metabolism | Humans | Chlorophyll - analogs & derivatives | Fluorescent Dyes - metabolism | Drug Resistance, Neoplasm | Curcumin - metabolism | Neoplasm Proteins - metabolism | ATP-Binding Cassette, Sub-Family B, Member 1 | Organic Anion Transporters - metabolism | Antineoplastic Agents - metabolism | Beryllium - metabolism | Adenosine Triphosphate - metabolism | ATP-Binding Cassette Transporters - metabolism | Molecular Structure | Drug Resistance, Multiple - physiology | Mitoxantrone - metabolism | Etoposide - metabolism | Chlorophyll - metabolism | Antineoplastic Agents - chemistry | Rhodamine 123 - metabolism | Cell Line, Tumor | ATP Binding Cassette Transporter, Sub-Family B | Multidrug Resistance-Associated Proteins - metabolism | Proteins | Medical research | Binding sites | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2008, Volume 14, Issue 11, pp. 1247 - 1255
Journal Article
Nature, ISSN 0028-0836, 04/2011, Volume 472, Issue 7342, pp. 226 - 230
Genetic studies indicate that protein homeostasis is a major contributor to metazoan longevity(1). Collapse of protein homeostasis results in protein... 
LONGEVITY | INHIBITION | HEAT-SHOCK FACTOR | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | DAF-16 | NEMATODE CAENORHABDITIS-ELEGANS | STRESS | SELECTION | C.-ELEGANS | PROTEOSTASIS | Thiazoles - metabolism | Molecular Chaperones - metabolism | Paralysis - drug therapy | Longevity - drug effects | Humans | Aging - drug effects | Caenorhabditis elegans Proteins - metabolism | Autophagy | DNA-Binding Proteins - metabolism | Dose-Response Relationship, Drug | Caenorhabditis elegans - physiology | Amyloid - metabolism | Amyloid beta-Peptides - genetics | Protein Binding - drug effects | Amyloid beta-Peptides - metabolism | Homeostasis - drug effects | Caenorhabditis elegans - metabolism | Amyloid beta-Peptides - toxicity | Curcumin - pharmacology | Aging - pathology | Transcription Factors - metabolism | Amyloid beta-Peptides - antagonists & inhibitors | Phenotype | Animals | Caenorhabditis elegans - drug effects | Proteins - metabolism | Survival Analysis | Thiazoles - pharmacology | Proteasome Endopeptidase Complex - metabolism | Forkhead Transcription Factors | Longevity - physiology | Aging - metabolism | Caenorhabditis elegans | Amyloid beta-protein | Aging | Physiological aspects | Homeostasis | Genetic aspects | Research | Proteins | Aggregates | Mortality | Nematodes | Influence | Mutation | Stress response | Worms | Index Medicus
Journal Article
Molecular Nutrition and Food Research, ISSN 1613-4125, 12/2011, Volume 55, Issue 12, pp. 1829 - 1840
Scope: Atherosclerosis is a major cause of cardiovascular disease caused by high cholesterol. Stains are widely prescribed to lower cholesterol levels, but... 
HMG-CoA | Lovastatin | Curcumin | Gene expression | Atherosclerosis | C-REACTIVE PROTEIN | COMPLEMENT ACTIVATION | HIGH-FAT DIET | FOOD SCIENCE & TECHNOLOGY | LOW-DENSITY-LIPOPROTEIN | DEFICIENT MICE | KNOCKOUT MICE | HYPERCHOLESTEROLEMIC RATS | APOLIPOPROTEIN-E | COA REDUCTASE | LIVER | Up-Regulation | Vascular Cell Adhesion Molecule-1 - drug effects | Male | Apolipoproteins B - blood | Apolipoproteins B - drug effects | Curcumin - administration & dosage | Orphan Nuclear Receptors - metabolism | Apolipoprotein A-I - genetics | Hydroxymethylglutaryl-CoA Reductase Inhibitors - metabolism | Hydroxymethylglutaryl CoA Reductases - metabolism | Liver - drug effects | Liver X Receptors | Lipoproteins - metabolism | Intercellular Adhesion Molecule-1 - drug effects | Cholesterol, LDL - blood | Lovastatin - administration & dosage | Cholesterol, HDL - drug effects | Liver - metabolism | Mice, Inbred C57BL | Cholesterol, LDL - drug effects | PPAR alpha - genetics | Apolipoprotein A-I - metabolism | Cholesterol - metabolism | Orphan Nuclear Receptors - genetics | Mice, Knockout | Acetyl-CoA C-Acetyltransferase - genetics | Intercellular Adhesion Molecule-1 - metabolism | Animals | Lipid Metabolism - drug effects | Triglycerides - blood | Cholesterol, HDL - blood | Mice | PPAR alpha - metabolism | Acetyl-CoA C-Acetyltransferase - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Cholesterol, Dietary | Atherosclerosis - prevention & control | Index Medicus
Journal Article
Experimental Gerontology, ISSN 0531-5565, 02/2013, Volume 48, Issue 2, pp. 269 - 276
We tested the hypothesis that curcumin supplementation would reverse arterial dysfunction and vascular oxidative stress with aging. Young (Y, 4–6 months) and... 
AGEs | Superoxide | Endothelial function | Arterial stiffness | Collagen | CARDIOVASCULAR EVENTS | PORCINE CORONARY-ARTERIES | AORTIC STIFFNESS | GERIATRICS & GERONTOLOGY | INDUCED HYPERTENSION | DEPENDENT DILATION | ADVANCED GLYCATION | NITRIC-OXIDE | CHEMOPREVENTIVE AGENT | VASCULAR ENDOTHELIAL DYSFUNCTION | AEROBIC EXERCISE | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Age Factors | Pulse Wave Analysis | Endothelium, Vascular - drug effects | Male | Aorta - metabolism | Phosphoproteins - metabolism | Tyrosine - analogs & derivatives | Dose-Response Relationship, Drug | Superoxides - metabolism | Aorta - physiopathology | Nitric Oxide Synthase Type III - metabolism | Superoxide Dismutase - metabolism | Elastin - metabolism | Collagen Type I - metabolism | Vasodilator Agents - pharmacology | Aorta - drug effects | Mice, Inbred C57BL | Curcumin - pharmacology | Endothelium, Vascular - physiopathology | Antioxidants - pharmacology | Tyrosine - metabolism | Animals | Carotid Arteries - physiopathology | Endothelium, Vascular - metabolism | Glycation End Products, Advanced - metabolism | Vascular Stiffness - drug effects | Mice | Oxidative Stress - drug effects | Vasodilation - drug effects | Nitric Oxide - metabolism | Aging - metabolism | Index Medicus | arterial stiffness | endothelial function | collagen
Journal Article
Journal Article
International Journal of Pharmaceutics, ISSN 0378-5173, 2007, Volume 330, Issue 1, pp. 155 - 163
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, pp. e0124000 - e0124000
Inhibition of carcinogenesis may be a consequence of attenuation of oxidative stress via activation of antioxidant defence system, restoration and... 
NRF2-MEDIATED ANTIOXIDANT | QUINONE OXIDOREDUCTASE-1 | GROWTH-FACTOR-BETA | LYMPHOMA-BEARING MICE | NAD(P)H-QUINONE OXIDOREDUCTASE-1 | TGF-BETA | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | SMOOTH-MUSCLE-CELLS | GLUTATHIONE-S-TRANSFERASE | Consensus Sequence | Glutathione Reductase - metabolism | Antioxidants - metabolism | Male | NAD(P)H Dehydrogenase (Quinone) - genetics | RNA, Messenger - metabolism | Lymphoma, T-Cell - prevention & control | Glutathione Reductase - genetics | Lymphoma, T-Cell - metabolism | Glutathione Transferase - genetics | Liver - drug effects | Cyclooxygenase 2 - genetics | Protein Binding - drug effects | Inflammation Mediators - metabolism | NF-E2-Related Factor 2 - genetics | Anticarcinogenic Agents - pharmacology | Tumor Microenvironment - drug effects | Liver - metabolism | RNA, Messenger - genetics | Curcumin - pharmacology | Tumor Suppressor Protein p53 - metabolism | Glutathione Transferase - metabolism | Transforming Growth Factor beta1 - genetics | Lymphoma, T-Cell - drug therapy | Animals | Nitric Oxide Synthase Type II - genetics | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | Cyclooxygenase 2 - metabolism | NAD(P)H Dehydrogenase (Quinone) - metabolism | Mice | Mice, Inbred AKR | Nitric Oxide Synthase Type II - metabolism | Prevention | Antioxidants | Patient outcomes | Development and progression | Genetic aspects | Research | Non-Hodgkin's lymphomas | Transforming growth factors | Tumor proteins | Risk factors | Oxidative stress | Regulations | Laboratories | Zoology | Liver | p53 Protein | Smooth muscle | Biochemistry | Lymphocytes T | Activation | Metastasis | Carcinogenesis | Bearing | Proteins | Signal transduction | Angiogenesis | Carcinogens | Metabolites | Restoration | Rodents | Modulation | Cell cycle | Attenuation | Curcumin | Inhibition | Growth factors | Deoxyribonucleic acid--DNA | Enzymes | Inflammation | Gene expression | Lymphoma | Nitric-oxide synthase | Signaling | Phytochemicals | Hepatocytes | Nitric oxide | Glycolysis | Lymphomas | Cyclooxygenase-2 | Molecular biology | T-cell lymphoma | Cancer | Apoptosis | Tumors | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Biomedicine & Pharmacotherapy, ISSN 0753-3322, 2017, Volume 92, pp. 1103 - 1110
Abstract Background Schwann cells (SCs) play an indispensable role in the repair and regeneration of injured peripheral nerve. Curcumin can reduce SCs... 
Internal Medicine | Medical Education | Erk1/2 | Curcumin | Akt | Autophagy | Schwann cells | Myelinization | SURVIVAL | MEDICINE, RESEARCH & EXPERIMENTAL | FUNCTIONAL RECOVERY | REGENERATION | SPINAL-CORD | MODEL | CRUSH | INHIBITION | SIGNALING PATHWAY | DISEASE | PHARMACOLOGY & PHARMACY | Phosphorylation | Sciatic Nerve - injuries | Apoptosis - drug effects | Microtubule-Associated Proteins - metabolism | Myelin Sheath - drug effects | Male | Autophagy - drug effects | Myelin Sheath - metabolism | Sciatic Nerve - metabolism | Neuroprotective Agents - pharmacology | Sciatic Nerve - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Sciatic Neuropathy - drug therapy | Sequestosome-1 Protein - metabolism | Disease Models, Animal | Schwann Cells - drug effects | Cell Line | Myelin Sheath - pathology | Sciatic Nerve - pathology | Curcumin - pharmacology | Sciatic Neuropathy - pathology | S100 Proteins - metabolism | Schwann Cells - pathology | Schwann Cells - metabolism | Rats, Sprague-Dawley | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Nerve Regeneration - drug effects | Sciatic Neuropathy - physiopathology | Beclin-1 - metabolism | Fibrin - metabolism | Myelin Proteins - metabolism | Sciatic Neuropathy - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Apoptosis | Immunohistochemistry | Fibrin | Index Medicus
Journal Article