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Nature, ISSN 0028-0836, 02/2011, Volume 470, Issue 7334, pp. 404 - 408
Activating AMPK or inactivating calcineurin slows ageing in Caenorhabditis elegans(1,2) and both have been implicated as therapeutic targets for age-related... 
COACTIVATOR CRTC2 | TORC2 | UNFOLDED PROTEIN RESPONSE | CAENORHABDITIS-ELEGANS | PATHWAY | INSULIN-RESISTANCE | ER STRESS | MULTIDISCIPLINARY SCIENCES | KINASE | GENE-REGULATION | C-ELEGANS | AMP-Activated Protein Kinases - metabolism | Phosphorylation | Caenorhabditis elegans Proteins - chemistry | Humans | Caenorhabditis elegans Proteins - metabolism | Trans-Activators - chemistry | Gene Knockdown Techniques | Caenorhabditis elegans - physiology | Calcineurin Inhibitors | HEK293 Cells | Trans-Activators - genetics | Transcription, Genetic | Protein-Serine-Threonine Kinases - metabolism | Cyclic AMP Response Element-Binding Protein - biosynthesis | Caenorhabditis elegans - metabolism | Caenorhabditis elegans - genetics | Down-Regulation | Transcription Factors - biosynthesis | Longevity - genetics | Transcription Factors - metabolism | Animals | Trans-Activators - deficiency | Aging - physiology | Energy Metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Trans-Activators - metabolism | Enzyme Activation | Caenorhabditis elegans Proteins - genetics | Longevity - physiology | Caenorhabditis elegans - enzymology | Calcineurin - metabolism | Aging - metabolism | Caenorhabditis elegans Proteins - biosynthesis | Physiological aspects | Chemical properties | Protein kinases | Calcineurin | Proteins | Homeostasis | Mutation | Kinases | DNA repair | Metabolic disorders | Binding sites
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 06/2005, Volume 25, Issue 23, pp. 5553 - 5562
The transcription factor cAMP response element binding protein (CREB) is implicated in the actions of drugs of abuse in several brain areas, but little... 
Mesocortical | CREB | Mesolimbic | Morphine | Cocaine | Reward | Ventral tegmental area | reward | CREB PHOSPHORYLATION | cocaine | mesolimbic | CHRONIC MORPHINE | mesocortical | MESOLIMBIC DOPAMINE SYSTEM | NEUROSCIENCES | morphine | HERPES-SIMPLEX-VIRUS | CYCLIC-AMP | ventral tegmental area | TYROSINE-HYDROXYLASE GENE | MEDIATED TRANSCRIPTION | NUCLEUS-ACCUMBENS SHELL | PHOSPHOLIPASE-C-GAMMA | TRANSCRIPTIONAL REGULATION | Tyrosine 3-Monooxygenase - metabolism | Up-Regulation | Male | Green Fluorescent Proteins - genetics | Ventral Tegmental Area - physiology | Nucleus Accumbens - physiology | Neurons - physiology | Behavior, Animal - drug effects | Female | Transcription, Genetic | Ventral Tegmental Area - anatomy & histology | Receptors, AMPA - genetics | Receptors, AMPA - metabolism | Recombinant Fusion Proteins - biosynthesis | Cyclic AMP Response Element-Binding Protein - biosynthesis | Morphine - pharmacology | Conditioning (Psychology) - drug effects | Mice, Inbred C57BL | Mice, Transgenic | Cyclic AMP Response Element-Binding Protein - physiology | Cocaine - pharmacology | Cyclic AMP Response Element-Binding Protein - genetics | Tyrosine 3-Monooxygenase - genetics | Animals | Ventral Tegmental Area - metabolism | Recombinant Fusion Proteins - genetics | Mice | Narcotics - pharmacology | Life Sciences | Neurons and Cognition | Plasticity | Development | Repair
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2012, Volume 287, Issue 2, pp. 905 - 915
Journal Article
Journal Article
Journal Article
The EMBO Journal, ISSN 0261-4189, 05/2018, Volume 37, Issue 9, p. n/a
The formation of neurites is an important process affecting the cognitive abilities of an organism. Neurite growth requires the addition of new membranes, but... 
HIF‐1α | synaptic activity‐mediated transcription | neurite growth | glycolysis | Siah2 | HIF-1α | synaptic activity-mediated transcription | NEURONAL-ACTIVITY | DENDRITIC GROWTH | HIF-1 alpha | BIOCHEMISTRY & MOLECULAR BIOLOGY | ACTIVITY-DEPENDENT REGULATION | HYPOXIA | MICE LACKING | CELL BIOLOGY | BRAIN ENERGY-METABOLISM | AEROBIC GLYCOLYSIS | GENE-EXPRESSION | HIF-ALPHA | RECEPTOR-ACTIVITY | Synapses - genetics | Glucose Transporter Type 3 - metabolism | Synapses - pathology | Gene Knockdown Techniques | Glycolysis - genetics | Glucose Transporter Type 3 - genetics | Cell Membrane Structures - metabolism | Synapses - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - biosynthesis | Cell Membrane Structures - genetics | Cyclic AMP Response Element-Binding Protein - biosynthesis | Cell Membrane Structures - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Gene Expression Regulation | Rats | Neurites - metabolism | Rats, Sprague-Dawley | Ubiquitin-Protein Ligases - biosynthesis | Cyclic AMP Response Element-Binding Protein - genetics | Neurites - pathology | Animals | Cyclic AMP Response Element-Binding Protein - metabolism | Glucose Transporter Type 3 - biosynthesis | Mice | Ubiquitin-Protein Ligases - genetics | Glucose transporter | Brain | Membranes | Transcription | Stabilization | Axonogenesis | Genes | Cognitive ability | Lipids | Pharmacology | Biosynthesis | Glucose | Metabolism | Neurodevelopmental disorders | Cyclic AMP response element-binding protein | Gene expression | Ablation | Constraining | Glucose metabolism | Glycolysis | Lipid metabolism | Transporter | Inducers | Neuroscience
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2010, Volume 285, Issue 2, pp. 1529 - 1543
Journal Article
Life Sciences, ISSN 0024-3205, 03/2017, Volume 173, pp. 43 - 54
To investigate the effect of FTY720 on the valproic acid (VPA) rat model of autism. As an animal model of autism, we used intraperitoneal injection of VPA on... 
Oxidative stress | Hippocampal cell | Neuroinflammatory | Fingolimod (FTY720) | VPA | Autism spectrum disorder | HIPPOCAMPAL SYNAPTIC PLASTICITY | MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS | PRENATAL EXPOSURE | VALPROIC ACID | MOUSE MODEL | PHARMACOLOGY & PHARMACY | ANIMAL-MODEL | UP-REGULATION | EXPRESSION | COGNITIVE DEFICITS | Autistic Disorder - chemically induced | Inflammation - chemically induced | Fingolimod Hydrochloride - pharmacology | Inflammation - pathology | Pyramidal Cells - metabolism | Autistic Disorder - pathology | Autistic Disorder - metabolism | Rats, Wistar | Apoptosis - drug effects | Learning - drug effects | Memory Disorders - metabolism | Male | Inflammation - metabolism | Social Behavior | Female | Brain-Derived Neurotrophic Factor - biosynthesis | Interleukin-1beta - biosynthesis | Fingolimod Hydrochloride - adverse effects | Memory Disorders - pathology | Disease Models, Animal | Oxidoreductases - biosynthesis | Apoptosis Regulatory Proteins - biosynthesis | Malondialdehyde - metabolism | Cyclic AMP Response Element-Binding Protein - biosynthesis | Rats | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - biosynthesis | Gene Expression Regulation - drug effects | Animals | Interleukin-6 - biosynthesis | Pyramidal Cells - pathology | Oxidative Stress - drug effects | Memory Disorders - chemically induced | Autism | Divalproex | Valproic acid | Neurons | Memory | Interleukins | Superoxide | Social aspects | Calcium-binding proteins | Protein kinases | Protein binding | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2015, Volume 195, Issue 3, pp. 1218 - 1232
Journal Article