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Journal Article
Cell, ISSN 0092-8674, 2007, Volume 128, Issue 2, pp. 281 - 294
The kinase inhibitor p27 regulates the G cell cycle phase. Here, we present data indicating that the oncogenic kinase Src regulates p27 stability through... 
GROWTH-FACTOR RECEPTOR | FACTOR-BETA | HUMAN-BREAST-CANCER | DEPENDENT-KINASE | TYROSINE KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL-CYCLE | CDK INHIBITOR | MEDIATED G ARREST | ESTROGEN-RECEPTOR | C-SRC | CELL BIOLOGY | Phosphorylation | Protein Binding - genetics | Humans | Cyclin E - genetics | Antineoplastic Agents, Hormonal - metabolism | Breast Neoplasms - metabolism | Breast Neoplasms - therapy | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Cell Nucleus - metabolism | Cell Transformation, Neoplastic - genetics | Cell Cycle Proteins - genetics | Female | Tamoxifen - metabolism | Cyclin-Dependent Kinase 2 - metabolism | Antineoplastic Agents, Hormonal - pharmacology | Cell Cycle Proteins - metabolism | Cyclin-Dependent Kinase 2 - genetics | Proto-Oncogene Proteins pp60(c-src) - antagonists & inhibitors | Cell Transformation, Neoplastic - metabolism | Down-Regulation - genetics | Drug Resistance, Neoplasm - genetics | Tyrosine - metabolism | Breast Neoplasms - genetics | Proto-Oncogene Proteins pp60(c-src) - genetics | Proto-Oncogene Proteins pp60(c-src) - metabolism | Feedback, Physiological - genetics | Tamoxifen - pharmacology | Cell Line, Tumor | Carcinoma - genetics | Carcinoma - therapy | Carcinoma - metabolism | Cyclin E - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Index Medicus
Journal Article
Molecular Endocrinology, ISSN 0888-8809, 12/2012, Volume 26, Issue 12, pp. 2016 - 2030
During each menstrual cycle, the human uterus undergoes a unique transformation, known as decidualization, which involves endometrial stromal proliferation and... 
CROSS-TALK | FORKHEAD TRANSCRIPTION FACTOR | IMPLANTATION FAILURE | KINASE-A | ENDOCRINOLOGY & METABOLISM | LEUKEMIA INHIBITORY FACTOR | CELL-CYCLE | PROGESTERONE-RECEPTOR | MICE LACKING | BINDING-PROTEIN-BETA | MAMMARY-GLAND | Phosphorylation | Cell Proliferation | Humans | Menstrual Cycle - metabolism | Uterus - metabolism | Embryo Implantation - genetics | Gene Expression Profiling | RNA, Messenger - metabolism | Receptors, Interleukin-11 - genetics | Retinoblastoma Protein - biosynthesis | Chromatin Immunoprecipitation | RNA Interference | Female | Transcription, Genetic | Cell Differentiation | Endometrium - cytology | Estrogens - metabolism | Cyclic AMP - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Endometrium - metabolism | Cyclin-Dependent Kinase 2 - metabolism | Embryo Implantation - physiology | CCAAT-Enhancer-Binding Protein-beta - genetics | RNA, Messenger - genetics | Retinoblastoma Protein - metabolism | Stromal Cells - metabolism | Cells, Cultured | Signal Transduction - genetics | E2F1 Transcription Factor - metabolism | CCAAT-Enhancer-Binding Protein-beta - metabolism | Cell Cycle | Receptors, Interleukin-11 - metabolism | Progesterone - metabolism | E2F1 Transcription Factor - biosynthesis | RNA, Small Interfering | Cyclin E - metabolism | Stromal Cells - cytology | Index Medicus | Original Research
Journal Article
Endocrinology, ISSN 0013-7227, 02/2016, Volume 157, Issue 2, pp. 883 - 899
TGFβ has been implicated in preeclampsia, but its intracellular signaling via phosphorylated mothers against decapentaplegic (SMADs) and SMAD-independent... 
HUMAN PLACENTAL TROPHOBLAST | IN-VITRO | BEWO CELLS | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | TRANSFORMING-GROWTH-FACTOR | EPITHELIAL-MESENCHYMAL TRANSITIONS | E-CADHERIN | CELL-LINES | TRANSCRIPTION FACTOR | SYNCYTIAL FUSION | Pre-Eclampsia - genetics | Oncogene Proteins - genetics | Phosphorylation | Cyclin-Dependent Kinase 4 - genetics | Humans | Pregnancy Proteins - genetics | Male | rhoA GTP-Binding Protein - metabolism | Pregnancy Proteins - metabolism | rhoA GTP-Binding Protein - genetics | RNA, Messenger - metabolism | Cyclin E - genetics | Case-Control Studies | Gene Products, env - metabolism | Young Adult | Cell Differentiation - genetics | Smad2 Protein - genetics | Gene Products, env - genetics | Adult | Female | Pre-Eclampsia - metabolism | Nuclear Proteins - genetics | Smad7 Protein - genetics | Real-Time Polymerase Chain Reaction | Infant, Newborn | Smad7 Protein - metabolism | Cell Proliferation - genetics | Trophoblasts - metabolism | Signal Transduction | Oncogene Proteins - metabolism | Smad2 Protein - metabolism | Ubiquitin-Protein Ligases - metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | Cyclin-Dependent Kinase 4 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Gestational Age | Placenta - metabolism | Pregnancy | Transcription Factors - metabolism | Transforming Growth Factor beta - genetics | Adaptor Proteins, Signal Transducing - genetics | Adolescent | Cyclin E - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Ubiquitin-Protein Ligases - genetics | Transforming Growth Factor beta - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 06/2017, Volume 8, Issue 1, pp. 15916 - 15916
Deregulation of the cell cycle machinery is a hallmark of cancer. While CDK4/6 inhibitors are FDA approved (palbociclib) for treating advanced estrogen... 
PALBOCICLIB | APOPTOSIS | CELLS | OXIDATIVE STRESS | ADVANCED BREAST-CANCER | MULTIDISCIPLINARY SCIENCES | REGULATES AUTOPHAGY | LETROZOLE | G1 ARREST | DEPENDENT KINASE INHIBITOR | ANTITUMOR-ACTIVITY | Cyclin-Dependent Kinase 6 - antagonists & inhibitors | Piperazines - administration & dosage | Cyclin-Dependent Kinase 4 - genetics | Humans | Cellular Senescence - drug effects | Cytoplasm - metabolism | Antineoplastic Agents - administration & dosage | Breast Neoplasms - physiopathology | Cyclin E - genetics | Autophagy - drug effects | Breast Neoplasms - metabolism | Female | Cytoplasm - genetics | Cyclin-Dependent Kinase 4 - antagonists & inhibitors | Pyridines - administration & dosage | Retinoblastoma Protein - metabolism | Cyclin-Dependent Kinase 6 - genetics | Cyclin-Dependent Kinase 6 - metabolism | Cyclin-Dependent Kinase 4 - metabolism | Breast Neoplasms - drug therapy | Drug Synergism | Protein Kinase Inhibitors - administration & dosage | Animals | Breast Neoplasms - genetics | Mice, Nude | Retinoblastoma Protein - genetics | Cell Line, Tumor | Mice | Cyclin E - metabolism | Cell Cycle - drug effects | Cytoplasm - drug effects | Senescence | Deregulation | Estrogens | Estrogen | Estrogen receptors | Breast cancer | In vitro testing | Autophagy | Machinery | Cyclin-dependent kinase 4 | Inhibitors | Cell cycle | Xenografts | Biomarkers | Breast | Cyclin E | Bioindicators | Inhibition | Phagocytosis | Tumors | Cancer | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2005, Volume 7, Issue 8, pp. 831 - 836
The cyclin-dependent kinase inhibitor p27Kip1 is known as a negative regulator of cell-cycle progression and as a tumour suppressor. Cdk2 is the main target of... 
CYCLIN-E | ENTRY | CDK2 | GROWTH | MOUSE | DEPENDENT-KINASE INHIBITOR | HYPERPLASIA | MAMMALIAN-CELLS | MICE LACKING | P27(KIP1) | CELL BIOLOGY | Cell Proliferation | Cyclin-Dependent Kinases - metabolism | Ovarian Neoplasms - pathology | Gonads - pathology | Male | Mitosis - genetics | Body Weight - genetics | CDC2 Protein Kinase - metabolism | Ovarian Neoplasms - genetics | Cyclin-Dependent Kinase 2 | Cyclins - metabolism | 2-Aminopurine - analogs & derivatives | Cyclin-Dependent Kinases - antagonists & inhibitors | Thymus Gland - metabolism | Fibroblasts - metabolism | Tumor Suppressor Proteins - metabolism | Pituitary Neoplasms - genetics | Cell Cycle Proteins - metabolism | Enzyme Inhibitors - pharmacology | CDC2-CDC28 Kinases - metabolism | Mice, Knockout | Fibroblasts - drug effects | Fibroblasts - cytology | Mice | Interphase - genetics | Interphase - physiology | Infertility - genetics | Transfection | Tumor Suppressor Proteins - genetics | Thymus Gland - pathology | Cell Cycle Proteins - genetics | Female | G1 Phase - genetics | CDC2 Protein Kinase - genetics | Mice, Inbred C57BL | Pituitary Neoplasms - pathology | 2-Aminopurine - pharmacology | G1 Phase - physiology | S Phase - genetics | Cyclin-Dependent Kinase Inhibitor p27 | Animals | Spleen - metabolism | RNA, Double-Stranded - genetics | Sex Factors | Protein Binding | S Phase - physiology | S Phase - drug effects | Cyclin E - metabolism | CDC2-CDC28 Kinases - genetics | Interphase - drug effects | Crosses, Genetic | Physiological aspects | Tumor suppressor genes | Control | Cellular signal transduction | Research | Cell cycle | Index Medicus
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 04/2017, Volume 486, Issue 2, pp. 385 - 390
TEA domain transcription factor 4 (TEAD4), which has critical functions in the process of embryonic development, is expressed in various cancers. However, the... 
TEA domain transcription factor 4 | Human oral squamous cell carcinoma | Yes-associated protein (YAP) | TARGET | BIOMARKER | BIOCHEMISTRY & MOLECULAR BIOLOGY | HIPPO PATHWAY | YAP | FAMILY | BIOPHYSICS | DNA | PROGNOSTIC MARKER | EXPRESSION | RNA, Small Interfering - genetics | Cell Proliferation | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Male | Cyclin E - genetics | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Cell Nucleus - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Muscle Proteins - antagonists & inhibitors | Transcription, Genetic | Mouth Neoplasms - genetics | Cyclin-Dependent Kinase 2 - metabolism | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction | Cyclin-Dependent Kinase 2 - genetics | Cyclin-Dependent Kinase 6 - metabolism | Cyclin-Dependent Kinase 4 - metabolism | Muscle Proteins - genetics | Cell Nucleus - genetics | Cyclin D1 - genetics | Mouth Neoplasms - surgery | Cell Line, Tumor | RNA, Small Interfering - metabolism | Cyclin D1 - metabolism | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Cyclin-Dependent Kinase 4 - genetics | Gene Expression Regulation, Neoplastic | Carcinoma, Squamous Cell - surgery | Mouth Neoplasms - metabolism | Phosphoproteins - metabolism | DNA-Binding Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Muscle Proteins - metabolism | G1 Phase Cell Cycle Checkpoints | Female | Cyclin-Dependent Kinase 6 - genetics | Transcription Factors - antagonists & inhibitors | Phosphoproteins - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Transcription Factors - metabolism | Adaptor Proteins, Signal Transducing - genetics | Mouth Neoplasms - pathology | Aged | Cyclin E - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Immunohistochemistry | Medical colleges | Embryonic development | Squamous cell carcinoma | Growth | Analysis | DNA polymerases | Genetic transcription | Health aspects | Development and progression | Index Medicus | TRANSCRIPTION FACTORS | CHAIN REACTIONS | BIOLOGICAL MARKERS | CELL PROLIFERATION | CELL CYCLE | INTERACTIONS | 60 APPLIED LIFE SCIENCES | PLANT GROWTH
Journal Article
The EMBO Journal, ISSN 0261-4189, 05/2004, Volume 23, Issue 10, pp. 2116 - 2125
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 3/2008, Volume 180, Issue 5, pp. 915 - 929
Cyclin-dependent kinases (Cdks) fulfill key functions in many cellular processes, including cell cycle progression and cytoskeletal dynamics. A limited number... 
Growth cones | Phosphorylation | Neurites | HEK293 cells | Neurons | Microtubules | Cell cycle | Cell adhesion | Histones | Cyclins | NEURONAL MIGRATION | PROTEIN | NEURITE OUTGROWTH | GENE | PHOSPHORYLATION | TUBULIN DEACETYLASE | IN-VIVO | VERTEBRATE CELLS | CDK COMPLEXES | HISTONE DEACETYLASE | CELL BIOLOGY | Oncogene Proteins - genetics | Cyclin-Dependent Kinases - metabolism | Humans | Protein Processing, Post-Translational - genetics | Cyclin A - genetics | Cyclin E - genetics | Cell Movement - genetics | Cyclin-Dependent Kinase 5 - genetics | Cell Differentiation - genetics | Microtubules - metabolism | Sirtuin 2 | Microtubules - ultrastructure | Hippocampus - ultrastructure | Cyclin-Dependent Kinases - genetics | Sirtuins - genetics | Amino Acid Sequence | Cell Line | Cyclin-Dependent Kinase 2 - metabolism | Catalytic Domain | Hippocampus - embryology | Cyclin A - metabolism | Oncogene Proteins - metabolism | Cyclin-Dependent Kinase 2 - genetics | Nerve Tissue Proteins - genetics | Serine - metabolism | Cyclin-Dependent Kinase 5 - metabolism | Nerve Tissue Proteins - metabolism | Hippocampus - metabolism | Animals | Growth Cones - metabolism | Growth Cones - ultrastructure | Mice | Cyclin E - metabolism | HeLa Cells | Sirtuins - metabolism | Evaluation | Motility | Cell physiology | Research | Properties | Genetic regulation | Phosphotransferases | Cells | Cell division | Cytoskeleton | Biochemistry | Cyclin-dependent kinases | Index Medicus
Journal Article