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Nature, ISSN 0028-0836, 05/2016, Volume 534, Issue 7605, pp. 55 - 62
Somatic mutations have been extensively characterized in breast cancer, but the effects of these genetic alterations on the proteomic landscape remain poorly... 
PATHWAYS | HETEROGENEITY | PIK3CA MUTATIONS | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | GENES | BIOLOGY | RECEPTOR | EXPRESSION | SIGNATURE | REVEALS | Protein Kinases - metabolism | Focal Adhesion Kinase 1 - genetics | Receptor, Epidermal Growth Factor - genetics | Protein Kinases - genetics | Cyclin-Dependent Kinases - metabolism | Receptor, ErbB-2 - genetics | Receptors, G-Protein-Coupled - metabolism | Genomics | Humans | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Phosphoproteins - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - genetics | Tumor Suppressor Protein p53 - genetics | Breast Neoplasms - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - metabolism | Breast Neoplasms - enzymology | Receptor, Epidermal Growth Factor - metabolism | Phosphoproteins - analysis | Mass Spectrometry | src-Family Kinases - metabolism | Female | Cyclin-Dependent Kinases - genetics | Focal Adhesion Kinase 1 - metabolism | Chromosomes, Human, Pair 5 - genetics | Breast Neoplasms - classification | Chromosome Deletion | p21-Activated Kinases - genetics | Signal Transduction | Molecular Sequence Annotation | Calcium-Binding Proteins - deficiency | Phosphoproteins - genetics | Mutation - genetics | S-Phase Kinase-Associated Proteins - metabolism | p21-Activated Kinases - metabolism | Phosphatidylinositol 3-Kinases - genetics | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Proteomics | S-Phase Kinase-Associated Proteins - genetics | Receptors, G-Protein-Coupled - genetics | src-Family Kinases - genetics | Calcium-Binding Proteins - genetics | Breast cancer | Genetic aspects | Research | Oncology, Experimental | Cancer | Physiological aspects | Methods | Mutation (Biology) | Proteins | Gene amplification | Peptides | Protein expression | Genomes | Mutation | Kinases | Deoxyribonucleic acid--DNA | Tumors
Journal Article
1997, Cancer surveys, ISBN 0879695188, Volume 29., vi, 363
Book
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2008, Volume 118, Issue 1, pp. 217 - 228
Loss of the tumor suppressor gene von Hippel-Lindau (VHL) plays a key role in the oncogenesis of clear cell renal cell carcinoma (CCRCC). The loss leads to... 
MEDICINE, RESEARCH & EXPERIMENTAL | HIF-1 | TUMOR SUPPRESSION | CYCLIN D1 | PATHWAY | GENE ALTERATIONS | VHL | KIDNEY | FATE | EXPRESSION | CANCER | Neoplasm Transplantation | RNA, Small Interfering - genetics | Humans | Carcinoma, Renal Cell - genetics | Male | Intracellular Signaling Peptides and Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Basic Helix-Loop-Helix Transcription Factors - metabolism | Cell Transformation, Neoplastic - genetics | Serrate-Jagged Proteins | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Female | Membrane Proteins - metabolism | Von Hippel-Lindau Tumor Suppressor Protein - genetics | Carcinoma, Renal Cell - drug therapy | Intracellular Signaling Peptides and Proteins - genetics | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Carcinoma, Renal Cell - pathology | Membrane Proteins - genetics | RNA, Small Interfering - pharmacology | Intercellular Signaling Peptides and Proteins - genetics | Signal Transduction - genetics | Receptor, Notch1 - metabolism | Cell Transformation, Neoplastic - metabolism | Cyclin-Dependent Kinase Inhibitor p27 | Carcinoma, Renal Cell - metabolism | Animals | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Mice | Cell Transformation, Neoplastic - drug effects | Receptor, Notch1 - antagonists & inhibitors | Cell Transformation, Neoplastic - pathology | Receptor, Notch1 - genetics | Calcium-Binding Proteins - genetics | Care and treatment | Enzyme inhibitors | Tumor suppressor genes | Dosage and administration | Genetic aspects | Carcinoma, Renal cell | Research | Health aspects | RNA; Small Interfering/genetics/pharmacology | Membrane Proteins/genetics/metabolism | Mice; Nude | Signal Transduction/drug effects/genetics | Intracellular Signaling Peptides and Proteins/genetics/metabolism | Basic Helix-Loop-Helix Transcription Factors/genetics/metabolism | Receptor; Notch1/antagonists & inhibitors/genetics/metabolism | Von Hippel-Lindau Tumor Suppressor Protein/genetics/metabolism | Cell Transformation; Neoplastic/drug effects/genetics/metabolism/pathology | Cyclin-Dependent Kinase Inhibitor p21/genetics/metabolism | Cell Line; Tumor | Intercellular Signaling Peptides and Proteins/genetics/metabolism | Calcium-Binding Proteins/genetics/metabolism | Carcinoma; Renal Cell/drug therapy/genetics/metabolism/pathology
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2013, Volume 110, Issue 31, pp. 12655 - 12660
The positive transcription elongation factor b (P-TEFb) is involved in physiological and pathological events including inflammation, cancer, AIDS, and cardiac... 
T lymphocytes | Pathology | RNA | HEK293 cells | Genes | Cell lines | Antibodies | Small nuclear RNA | Gene expression regulation | HIV 1 | 7SK SNRNA | TRANSCRIPTION FACTOR BCL11B | ELONGATION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | POLYMERASE | HIV-1 TAT | MICROGLIAL CELLS | T-CELL LINEAGE | BROMODOMAIN PROTEIN BRD4 | RNA-Binding Proteins - genetics | Humans | Myosin Heavy Chains - genetics | Cardiomegaly - pathology | Positive Transcriptional Elongation Factor B - metabolism | Positive Transcriptional Elongation Factor B - genetics | Myosin Heavy Chains - metabolism | RNA, Small Nuclear - genetics | Tumor Suppressor Proteins - genetics | HEK293 Cells | Cardiac Myosins - metabolism | Repressor Proteins - metabolism | Promoter Regions, Genetic | Tumor Suppressor Proteins - metabolism | Protein Structure, Secondary | Cyclin-Dependent Kinase 9 - genetics | Cardiac Myosins - genetics | Repressor Proteins - genetics | Transcription Factors - genetics | Transcription Factors - metabolism | Animals | Cyclin-Dependent Kinase 9 - metabolism | Mice | RNA, Small Nuclear - metabolism | Cardiomegaly - genetics | RNA-Binding Proteins - metabolism | Cardiomegaly - metabolism | Physiological aspects | Transcription factors | Genetic aspects | Heart enlargement | Health aspects | Myosin | Life Sciences | Neurons and Cognition | Biological Sciences
Journal Article
Cancer, ISSN 0008-543X, 06/2017, Volume 123, Issue S11, pp. 2104 - 2117
Journal Article
Cancer, ISSN 0008-543X, 01/2018, Volume 124, Issue 1, pp. 84 - 94
BACKGROUND Human immunodeficiency virus–infected individuals (HIVIIs) have a higher incidence of head and neck squamous cell carcinoma (HNSCC), and clinical... 
human papillomavirus (HPV) | mutation | head and neck cancer | human immunodeficiency virus (HIV) | TP53 gene | UNITED-STATES | HIV/AIDS | POPULATION | HUMAN-PAPILLOMAVIRUS | HIV PATIENTS | P53 | P53-MEDIATED APOPTOSIS | BREAST-CANCER | CIGARETTE-SMOKING | ONCOLOGY | INFECTION | Receptors, Transforming Growth Factor beta - genetics | Class I Phosphatidylinositol 3-Kinases - genetics | Proto-Oncogene Proteins p21(ras) - genetics | Carcinoma, Squamous Cell - genetics | Humans | Middle Aged | ErbB Receptors - genetics | Male | Receptor, Notch2 - genetics | Case-Control Studies | Papillomaviridae - genetics | Tumor Suppressor Protein p53 - genetics | Caspase 8 - genetics | Kelch-Like ECH-Associated Protein 1 - genetics | F-Box-WD Repeat-Containing Protein 7 - genetics | Squamous Cell Carcinoma of Head and Neck | In Situ Hybridization | Receptor, Transforming Growth Factor-beta Type II | Tumor Suppressor Proteins - genetics | Adult | Female | NF-E2-Related Factor 2 - genetics | Cadherins - genetics | Nuclear Proteins - genetics | Intracellular Signaling Peptides and Proteins - genetics | Papillomavirus Infections - complications | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - genetics | Cyclin-Dependent Kinase Inhibitor p18 - genetics | Cyclin D1 - genetics | Carcinoma, Squamous Cell - complications | HIV Infections - complications | HLA-A Antigens - genetics | Head and Neck Neoplasms - complications | Head and Neck Neoplasms - genetics | LIM Domain Proteins - genetics | Aged | Receptor, Notch1 - genetics | Multiplexing | Pathogenesis | Exons | p53 Protein | Genes | Viruses | Genomes | Infections | Cdc4 protein | Gene sequencing | Biological effects | Human papillomavirus | Human immunodeficiency virus--HIV | Deoxyribonucleic acid--DNA | Head | Squamous cell carcinoma | Nucleotide sequence | Epidermal growth factor receptors | Data processing | Patients | Polymerase chain reaction | Head and neck cancer | Histocompatibility antigen HLA | Mutation | Tumors | DNA sequencing | Cancer
Journal Article
by Curtis, Bruce A and Curtis, Bruce A and Tanifuji, Goro and Tanifuji, Goro and Burki, Fabien and Maruyama, Shinichiro and Gruber, Ansgar and Gile, Gillian H and Irimia, Manuel and Hopkins, Julia F and Maruyama, Shinichiro and Eveleigh, Robert J. M and Arias, Maria C and Nakayama, Takuro and Ball, Steven G and Malik, Shehre-Banoo and Onodera, Naoko T and Gile, Gillian H and Slamovits, Claudio H and Hirakawa, Yoshihisa and Spencer, David F and Hopkins, Julia F and Lane, Christopher E and Kuo, Alan and Gray, Michael W and Rensing, Stefan A and Archibald, John M and Schmutz, Jeremy and Symeonidi, Aikaterini and Burki, Fabien and Hirakawa, Yoshihisa and Elias, Marek and Reyes-Prieto, Adrian and Eveleigh, Robert J. M and Herman, Emily K and Keeling, Patrick J and Fast, Naomi M and Klute, Mary J and Green, Beverley R and Nakayama, Takuro and Oborník, Miroslav and Grisdale, Cameron J and Gruber, Ansgar and Reyes-Prieto, Adrian and Kroth, Peter G and Virginia Armbrust, E and Aves, Stephen J and Irimia, Manuel and Arias, Maria C and Beiko, Robert G and Coutinho, Pedro and Ball, Steven G and Dacks, Joel B and Kuo, Alan and Durnford, Dion G and Schmutz, Jeremy and Grimwood, Jane and Fast, Naomi M and Green, Beverley R and Lindquist, Erika and Grisdale, Cameron J and Lucas, Susan and Hempel, Franziska and Salamov, Asaf and Henrissat, Bernard and Grigoriev, Igor V and Höppner, Marc P and Rensing, Stefan A and Ishida, Ken-Ichiro and Symeonidi, Aikaterini and Kim, Eunsoo and Elias, Marek and Herman, Emily K and Kořený, Luděk and Kroth, Peter G and Klute, Mary J and Dacks, Joel B and Liu, Yuan and Oborník, Miroslav and Malik, Shehre-Banoo and Kořený, Luděk and Maier, Uwe G and McRose, Darcy and Durnford, Dion G and Mock, Thomas and Neilson, Jonathan A. D and Armbrust, E. Virginia and Neilson, Jonathan A. D and Rocap, Gabrielle and Onodera, Naoko T and Poole, Anthony M and Aves, Stephen J and Liu, Yuan and Pritham, Ellen J and Richards, Thomas A and Beiko, Robert G and Rocap, Gabrielle and Coutinho, Pedro and Henrissat, Bernard and Roy, Scott W and ... and Joint Genome Institute (JGI) and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Institutionen för medicinsk biokemi och mikrobiologi and Uppsala universitet
Nature, ISSN 0028-0836, 12/2012, Volume 491, Issue 7427, pp. 59 - 65
Journal Article
NATURE, ISSN 0028-0836, 02/2011, Volume 470, Issue 7333, pp. 264 - 264
Journal Article
Dermatology, ISSN 1018-8665, 06/2017, Volume 233, Issue 1, pp. 1 - 15
Human pigmentation characteristics play an important role in the effects of sun exposure, skin cancer induction and disease outcomes. Several of the genes most... 
Acquired melanocytic naevus | Melanogenesis | Pigmentation | Melanoma | Skin cancer | OCULOCUTANEOUS ALBINISM