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Genes and Development, ISSN 0890-9369, 05/2013, Volume 27, Issue 10, pp. 1101 - 1114
Tumorigenesis results from dysregulation of oncogenes and tumor suppressors that influence cellular proliferation, differentiation, apoptosis, and/or... 
Huwe1 | c-Myc | Mule | Ras | p21 | Miz1 | DNA-DAMAGE | KERATINOCYTE GROWTH | DEVELOPMENTAL BIOLOGY | CELL BIOLOGY | BASE EXCISION-REPAIR | HUWE1 UBIQUITIN LIGASE | NEGATIVE REGULATION | GENETICS & HEREDITY | ARF TUMOR-SUPPRESSOR | DIFFERENTIATION | MIZ-1 | HUMAN CANCER | Protein Inhibitors of Activated STAT - deficiency | Tetradecanoylphorbol Acetate - pharmacology | 9,10-Dimethyl-1,2-benzanthracene - pharmacology | Male | Protein Inhibitors of Activated STAT - metabolism | Cyclin-Dependent Kinase Inhibitor p15 - biosynthesis | Oncogene Protein p21(ras) - metabolism | Cyclin-Dependent Kinase Inhibitor p16 | Oncogene Protein p21(ras) - antagonists & inhibitors | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cell Transformation, Neoplastic - genetics | Cyclin-Dependent Kinase Inhibitor p15 - genetics | Nuclear Proteins - deficiency | Protein Inhibitors of Activated STAT - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Female | Nuclear Proteins - genetics | Protein Inhibitors of Activated STAT - antagonists & inhibitors | Skin Neoplasms - pathology | Cyclin-Dependent Kinase Inhibitor p21 - biosynthesis | Signal Transduction | Down-Regulation | Cells, Cultured | Ubiquitin-Protein Ligases - metabolism | Nuclear Proteins - metabolism | Skin Neoplasms - chemically induced | Proto-Oncogene Proteins c-myc - metabolism | Mice, Knockout | Skin Neoplasms - metabolism | Keratinocytes - pathology | Animals | Tumor Suppressor Protein p53 | Keratinocytes - drug effects | Keratinocytes - metabolism | Nuclear Proteins - antagonists & inhibitors | Proto-Oncogene Proteins c-myc - deficiency | Skin Neoplasms - genetics | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Ubiquitin-Protein Ligases - deficiency | Mice | Proto-Oncogene Proteins c-myc - genetics | Cyclin-Dependent Kinase Inhibitor p15 - metabolism | Genes, ras | Ubiquitin-Protein Ligases - genetics | Oncogene Protein p21(ras) - genetics | Carcinogenesis | Ras genes | Analysis | Index Medicus | Research Paper
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, pp. e0122679 - e0122679
Journal Article
Cancer Research, ISSN 0008-5472, 11/2011, Volume 71, Issue 21, pp. 6718 - 6727
Journal Article
Cancer Research, ISSN 0008-5472, 12/2010, Volume 70, Issue 24, pp. 10310 - 10320
Journal Article
Cancer Research, ISSN 0008-5472, 12/2008, Volume 68, Issue 24, pp. 10094 - 10104
Journal Article
Oncogene, ISSN 0950-9232, 01/2009, Volume 28, Issue 2, pp. 243 - 256
Cancer cells have complex, unique characteristics that distinguish them from normal cells, such as increased growth rates and evasion of anti-proliferative... 
WAF1/CIP1 GENE PROMOTER | TRANSCRIPTIONAL ACTIVATION | SUBEROYLANILIDE HYDROXAMIC ACID | BIOCHEMISTRY & MOLECULAR BIOLOGY | G/M PROMOTERS | siRNA | p21(WAF1/Cip1) | histone deacetylase | SP1 SITES | CELL BIOLOGY | P21/WAF1/CIP1 GENE | ONCOLOGY | Sp1 | HUMAN COLON-CANCER | IN-VIVO | GENETICS & HEREDITY | TUMOR-SUPPRESSOR PROTEIN | cancer | HISTONE DEACETYLASE INHIBITORS | Tumor Suppressor Protein p53 - antagonists & inhibitors | Epigenesis, Genetic | Humans | Neoplasm Proteins - physiology | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | HeLa Cells - metabolism | Neoplasm Proteins - antagonists & inhibitors | Bone Neoplasms - pathology | Repressor Proteins - antagonists & inhibitors | Gene Knockdown Techniques | Repressor Proteins - physiology | Tumor Suppressor Protein p53 - physiology | RNA, Messenger - biosynthesis | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Acetylation | Sp1 Transcription Factor - physiology | Binding Sites | Cell Line, Tumor - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - biosynthesis | Brain Neoplasms - drug therapy | Chick Embryo | HeLa Cells - drug effects | Animals | RNA, Neoplasm - biosynthesis | Glioblastoma - pathology | Histone Deacetylase Inhibitors | Protein Processing, Post-Translational | Histone Deacetylases - physiology | Histones - metabolism | Glioblastoma - drug therapy | Osteosarcoma - pathology | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 02/2014, Volume 4, Issue 1, pp. 4012 - 4012
The quiescent (G0) phase of the cell cycle is the reversible phase from which the cells exit from the cell cycle. Due to the difficulty of defining the G0... 
PROGENITOR CELLS | SKELETAL-MUSCLE | MUSCLE REGENERATION | CDK INHIBITORS | S-PHASE ENTRY | MULTIDISCIPLINARY SCIENCES | HEMATOPOIETIC STEM-CELLS | NICHE | SATELLITE CELLS | P27(KIP1) | G-G TRANSITION | RNA-Binding Proteins - genetics | Selenium-Binding Proteins - genetics | Tumor Suppressor Protein p53 - biosynthesis | Stem Cells - cytology | Retinoblastoma Protein - biosynthesis | RNA-Binding Proteins - biosynthesis | Selenium-Binding Proteins - biosynthesis | Apoptosis Regulatory Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p16 - biosynthesis | G1 Phase - genetics | Apoptosis Regulatory Proteins - biosynthesis | Recombinant Fusion Proteins - biosynthesis | Cell Line | Cyclin-Dependent Kinase Inhibitor p21 - biosynthesis | Gene Expression | Carrier Proteins - biosynthesis | Bacterial Proteins - genetics | Mice, Transgenic | MicroRNAs - biosynthesis | Carrier Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p57 - biosynthesis | Animals | Resting Phase, Cell Cycle - genetics | Recombinant Fusion Proteins - genetics | Luminescent Proteins - genetics | Mice | 3T3 Cells | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Cyclin-dependent kinase | Rodents | Stem cells | Cell cycle | Defective mutant | Transgenic mice | Stem cell transplantation | Cyclin-dependent kinase inhibitor p27 | Fusion protein | Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 10/2010, Volume 29, Issue 40, pp. 5464 - 5474
PBK/TOPK (PDZ-binding kinase, T-LAK-cell-originated protein kinase) is a serine-threonine kinase that is overexpressed in a variety of tumor cells but its role... 
tumor suppressor p53 | PDZ-binding kinase | cyclin-dependent kinase inhibitor p21WAF1 | PBK/TOPK | GLIOMA-CELLS | WILD-TYPE | APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ORIGINATED PROTEIN-KINASE | DNA-DAMAGE | MITOTIC KINASE | CELL BIOLOGY | TOPK | ONCOLOGY | GENETICS & HEREDITY | MUTANT P53 | HUMAN HOMOLOG | Cell Cycle - genetics | Immunoprecipitation | Oligonucleotide Array Sequence Analysis | Humans | Gene Expression Regulation, Neoplastic | Apoptosis - genetics | Tumor Suppressor Protein p53 - genetics | Gene Knockdown Techniques | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Flow Cytometry | Mitogen-Activated Protein Kinase Kinases | Chromatin Immunoprecipitation | Transcription, Genetic | Protein Interaction Domains and Motifs | Protein-Serine-Threonine Kinases - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - biosynthesis | Gene Expression | Signal Transduction | Cell Separation | HCT116 Cells | Protein-Serine-Threonine Kinases - genetics | Tumor Suppressor Protein p53 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Two-Hybrid System Techniques | Enzyme inhibitors | Physiological aspects | Tumor suppressor genes | Dosage and administration | Genetic transcription | Binding proteins | Properties | Genetics | Biochemistry | Mutation | Kinases | Colorectal cancer | Index Medicus
Journal Article