X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (54284) 54284
Newspaper Article (134) 134
Book Chapter (31) 31
Publication (23) 23
Dissertation (18) 18
Conference Proceeding (12) 12
Magazine Article (7) 7
Book / eBook (2) 2
Newsletter (1) 1
Reference (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (36986) 36986
animals (24179) 24179
mice (13644) 13644
cell cycle (13557) 13557
female (13187) 13187
cell biology (13025) 13025
oncology (11593) 11593
biochemistry & molecular biology (11426) 11426
phosphorylation (11153) 11153
apoptosis (10797) 10797
expression (10648) 10648
male (10492) 10492
cell line, tumor (10006) 10006
cancer (9250) 9250
proteins (7864) 7864
cell proliferation (7799) 7799
cyclin d1 (6497) 6497
gene expression (6467) 6467
signal transduction (6389) 6389
cyclins - metabolism (6222) 6222
cyclin d1 - metabolism (5577) 5577
middle aged (5483) 5483
kinases (5403) 5403
cyclin-dependent kinases (5279) 5279
cyclin-dependent kinases - metabolism (5266) 5266
research (5237) 5237
immunohistochemistry (5226) 5226
cells, cultured (5144) 5144
cyclin-dependent kinase inhibitor p21 (5144) 5144
activation (5133) 5133
tumor suppressor protein p53 - metabolism (5112) 5112
proliferation (4909) 4909
cell line (4828) 4828
cell proliferation - drug effects (4750) 4750
apoptosis - drug effects (4572) 4572
cyclin-dependent kinase inhibitor p21 - metabolism (4544) 4544
cell cycle proteins - metabolism (4461) 4461
adult (4406) 4406
aged (4352) 4352
rats (4352) 4352
mutation (4266) 4266
gene expression regulation, neoplastic (4237) 4237
blotting, western (4210) 4210
protein (4196) 4196
p53 (4174) 4174
growth (4066) 4066
research article (3900) 3900
tumor cells, cultured (3859) 3859
analysis (3726) 3726
transfection (3616) 3616
molecular sequence data (3598) 3598
cell cycle - drug effects (3557) 3557
multidisciplinary sciences (3512) 3512
gene (3491) 3491
cell-cycle (3483) 3483
tumors (3475) 3475
genetic aspects (3441) 3441
protein binding (3394) 3394
cyclins - genetics (3382) 3382
gene-expression (3342) 3342
cell division (3262) 3262
rna, messenger - metabolism (3248) 3248
down-regulation (3236) 3236
biology (3215) 3215
protein-serine-threonine kinases - metabolism (3143) 3143
genetics & heredity (3063) 3063
cell growth (3014) 3014
cyclin d1 - genetics (3008) 3008
antineoplastic agents - pharmacology (2949) 2949
cyclin-dependent kinase inhibitor p16 - metabolism (2918) 2918
prognosis (2912) 2912
inhibition (2880) 2880
cells (2876) 2876
time factors (2853) 2853
p21 (2845) 2845
differentiation (2816) 2816
genes (2806) 2806
flow cytometry (2781) 2781
physiological aspects (2754) 2754
cyclin-dependent kinase inhibitor p27 (2748) 2748
amino acid sequence (2745) 2745
transcription factors - metabolism (2743) 2743
cell cycle - physiology (2713) 2713
gene expression regulation (2633) 2633
medicine (2622) 2622
mitosis (2606) 2606
reverse transcriptase polymerase chain reaction (2596) 2596
retinoblastoma protein - metabolism (2595) 2595
cyclin-dependent kinase inhibitor p21 - genetics (2590) 2590
pathology (2572) 2572
rna, messenger - genetics (2527) 2527
tumor suppressor protein p53 - genetics (2524) 2524
up-regulation (2489) 2489
transcription (2486) 2486
signal transduction - drug effects (2471) 2471
base sequence (2460) 2460
breast cancer (2429) 2429
cell cycle proteins - genetics (2413) 2413
hela cells (2313) 2313
in-vitro (2312) 2312
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (53960) 53960
Chinese (907) 907
Japanese (314) 314
Russian (67) 67
German (55) 55
French (42) 42
Polish (25) 25
Portuguese (22) 22
Spanish (21) 21
Korean (10) 10
Czech (7) 7
Norwegian (5) 5
Croatian (4) 4
Dutch (4) 4
Hungarian (4) 4
Italian (4) 4
Ukrainian (4) 4
Bulgarian (2) 2
Danish (2) 2
Romanian (2) 2
Serbian (2) 2
Swedish (2) 2
Turkish (2) 2
Finnish (1) 1
Hebrew (1) 1
Persian (1) 1
Slovak (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Journal of gastroenterology and hepatology, ISSN 0815-9319, 03/2009, Volume 24, Issue 3, pp. 443 - 452
Background and Aims:  We examined extrinsic and intrinsic (endogenous) mitochondrial apoptosis pathways in experimental non‐alcoholic steatohepatitis (NASH).... 
mitochondria | methionine and choline deficiency | insulin‐like growth factor‐1 | TRAIL‐R killer/DR5 | TNF receptors | cell death pathways | p53 | Cell death pathways | TRAIL-R killer/DR5 | Methionine and choline deficiency | Mitochondria | Insulin-like growth factor-1 | P53 | HEPATOCYTE APOPTOSIS | ACIDS | DR5 | insulin-like growth factor-1 | NONALCOHOLIC STEATOHEPATITIS | LIVER-DISEASE | PATHOGENESIS | NECROSIS | TNF-ALPHA | LIGAND | GASTROENTEROLOGY & HEPATOLOGY | TRAIL-R killer | NF-KAPPA-B | HEPATIC STEATOSIS | Liver - pathology | Liver - enzymology | Fatty Liver - pathology | Mitochondria, Liver - metabolism | Caspase 3 - metabolism | Choline Deficiency - complications | Male | Alanine Transaminase - blood | Receptors, TNF-Related Apoptosis-Inducing Ligand - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | RNA, Messenger - metabolism | fas Receptor - metabolism | Tumor Suppressor Protein p53 - genetics | Time Factors | Receptors, Tumor Necrosis Factor, Type II - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Nutritional Status | Receptors, TNF-Related Apoptosis-Inducing Ligand - genetics | BH3 Interacting Domain Death Agonist Protein - metabolism | Disease Models, Animal | Methionine - deficiency | Receptors, Tumor Necrosis Factor - metabolism | Fatty Liver - metabolism | Mitochondria, Liver - pathology | Liver - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Tumor Suppressor Protein p53 - metabolism | Mitochondria, Liver - enzymology | Animals | Mice | bcl-X Protein - metabolism | Insulin-Like Growth Factor I - metabolism | Apoptosis | Fatty Liver - etiology | BH3 Interacting Domain Death Agonist Protein, metabolism | Receptors, Tumor Necrosis Factor, Type II, metabolism | Fatty Liver, pathology | Mitochondria, Liver, metabolism | Receptors, Tumor Necrosis Factor, metabolism | Cyclin-Dependent Kinase Inhibitor p21, metabolism | Insulin-Like Growth Factor I, metabolism | Caspase 3, metabolism | Antigens, CD95, metabolism | RNA, Messenger, metabolism | Tumor Suppressor Protein p53, metabolism | Methionine, deficiency | Choline Deficiency, complications | Tumor Suppressor Protein p53, genetics | bcl-X Protein, metabolism | Mitochondria, Liver, pathology | Liver, pathology | Alanine Transaminase, blood | Liver, metabolism | Fatty Liver, metabolism | Receptors, TNF-Related Apoptosis-Inducing Ligand, metabolism | Liver, enzymology | Fatty Liver, etiology | Mitochondria, Liver, enzymology | Receptors, Tumor Necrosis Factor, Type I, metabolism | Receptors, TNF-Related Apoptosis-Inducing Ligand, genetics | Messenger RNA | Choline | Methionine | Mitochondrial DNA | Tumor proteins | Peptide hormones | Growth factors
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 7/2013, Volume 110, Issue 31, pp. 12655 - 12660
The positive transcription elongation factor b (P-TEFb) is involved in physiological and pathological events including inflammation, cancer, AIDS, and cardiac... 
T lymphocytes | Pathology | RNA | HEK293 cells | Genes | Cell lines | Antibodies | Small nuclear RNA | Gene expression regulation | HIV 1 | 7SK SNRNA | TRANSCRIPTION FACTOR BCL11B | ELONGATION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | POLYMERASE | HIV-1 TAT | MICROGLIAL CELLS | T-CELL LINEAGE | BROMODOMAIN PROTEIN BRD4 | RNA-Binding Proteins - genetics | Humans | Myosin Heavy Chains - genetics | Cardiomegaly - pathology | Positive Transcriptional Elongation Factor B - metabolism | Positive Transcriptional Elongation Factor B - genetics | Myosin Heavy Chains - metabolism | RNA, Small Nuclear - genetics | Tumor Suppressor Proteins - genetics | HEK293 Cells | Cardiac Myosins - metabolism | Repressor Proteins - metabolism | Promoter Regions, Genetic | Tumor Suppressor Proteins - metabolism | Protein Structure, Secondary | Cyclin-Dependent Kinase 9 - genetics | Cardiac Myosins - genetics | Repressor Proteins - genetics | Transcription Factors - genetics | Transcription Factors - metabolism | Animals | Cyclin-Dependent Kinase 9 - metabolism | Mice | RNA, Small Nuclear - metabolism | Cardiomegaly - genetics | RNA-Binding Proteins - metabolism | Cardiomegaly - metabolism | Physiological aspects | Transcription factors | Genetic aspects | Heart enlargement | Health aspects | Myosin | Life Sciences | Neurons and Cognition | Biological Sciences
Journal Article
Nature (London), ISSN 1476-4687, 2018, Volume 562, Issue 7725, pp. 69 - 75
Primary liver cancer represents a major health problem. It comprises hepatocellular carcinoma (HCC) and intrahepatic cholangiocarcinoma (ICC), which differ... 
PATHOGENESIS | HEPATOCELLULAR-CARCINOMA | READ ALIGNMENT | HEPATOCYTES | DNA | INFLAMMATION | PACKAGE | MULTIDISCIPLINARY SCIENCES | FRAMEWORK | BINDING | DISCOVERY | Humans | Tumor Microenvironment | Apoptosis - genetics | Hepatocytes - pathology | Male | Gene Expression Profiling | Genes, myc | Hepatocytes - metabolism | Proto-Oncogene Proteins c-akt - genetics | DNA-Binding Proteins - metabolism | Carcinoma, Hepatocellular - genetics | DNA Transposable Elements - genetics | Female | Liver Neoplasms - pathology | Cell Differentiation | Cell Lineage - genetics | Disease Models, Animal | Cyclin-Dependent Kinase Inhibitor p16 - deficiency | Cytokines - metabolism | Liver Neoplasms - genetics | Carcinogenesis - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Transcription Factors - metabolism | Cholangiocarcinoma - pathology | Animals | Epigenesis, Genetic - genetics | Carcinoma, Hepatocellular - pathology | Cholangiocarcinoma - genetics | Mosaicism | Mice | Necrosis - genetics | Genes, ras | Liver cancer | Research | Carcinogenesis | Oncology, Experimental | Apoptosis | Cancer | Animal models | Cytokines | Liver | Hepatocellular carcinoma | Genomes | Metastasis | Risk analysis | Gene expression | Risk factors | Metastases | Hepatocytes | Morphology | DNA methylation | Tumorigenesis | Bioinformatics | Cholangiocarcinoma | Deoxyribonucleic acid--DNA | Tumors | T-Box Domain Proteins/metabolism | Hepatocytes/pathology | DNA-Binding Proteins/metabolism | Life Sciences | Cholangiocarcinoma/pathology | Transcription Factors/metabolism | Necrosis/genetics | Cytokines/metabolism | Epigenesis, Genetic/genetics | Cyclin-Dependent Kinase Inhibitor p16/deficiency | Carcinoma, Hepatocellular/pathology | DNA Transposable Elements/genetics | Cell Lineage/genetics | T-Box Domain Proteins/genetics | Transcription Factors/genetics | Apoptosis/genetics | Proto-Oncogene Proteins c-akt/genetics | Liver Neoplasms/genetics | Cholangiocarcinoma/genetics | Liver Neoplasms/pathology | DNA-Binding Proteins/genetics | Hepatocytes/metabolism | Carcinoma, Hepatocellular/genetics | Carcinogenesis/genetics
Journal Article
Nature (London), ISSN 1476-4687, 2016, Volume 534, Issue 7605, pp. 55 - 62
Somatic mutations have been extensively characterized in breast cancer, but the effects of these genetic alterations on the proteomic landscape remain poorly... 
PATHWAYS | HETEROGENEITY | PIK3CA MUTATIONS | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | GENES | BIOLOGY | RECEPTOR | EXPRESSION | SIGNATURE | REVEALS | Protein Kinases - metabolism | Focal Adhesion Kinase 1 - genetics | Receptor, Epidermal Growth Factor - genetics | Protein Kinases - genetics | Cyclin-Dependent Kinases - metabolism | Receptor, ErbB-2 - genetics | Receptors, G-Protein-Coupled - metabolism | Genomics | Humans | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Phosphoproteins - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - genetics | Tumor Suppressor Protein p53 - genetics | Breast Neoplasms - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - metabolism | Breast Neoplasms - enzymology | Receptor, Epidermal Growth Factor - metabolism | Phosphoproteins - analysis | Mass Spectrometry | src-Family Kinases - metabolism | Female | Cyclin-Dependent Kinases - genetics | Focal Adhesion Kinase 1 - metabolism | Chromosomes, Human, Pair 5 - genetics | Breast Neoplasms - classification | Chromosome Deletion | p21-Activated Kinases - genetics | Signal Transduction | Molecular Sequence Annotation | Calcium-Binding Proteins - deficiency | Phosphoproteins - genetics | Mutation - genetics | S-Phase Kinase-Associated Proteins - metabolism | p21-Activated Kinases - metabolism | Phosphatidylinositol 3-Kinases - genetics | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Proteomics | S-Phase Kinase-Associated Proteins - genetics | Receptors, G-Protein-Coupled - genetics | src-Family Kinases - genetics | Calcium-Binding Proteins - genetics | Breast cancer | Genetic aspects | Research | Oncology, Experimental | Cancer | Physiological aspects | Methods | Mutation (Biology) | Proteins | Gene amplification | Peptides | Protein expression | Genomes | Mutation | Kinases | Deoxyribonucleic acid--DNA | Tumors
Journal Article
Journal of Virology, ISSN 0022-538X, 12/2004, Volume 78, Issue 24, pp. 13522 - 13533
Article Usage Stats Services JVI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
RNA-POLYMERASE-II | CYCLIN-DEPENDENT KINASES | ACTIVATION | VIROLOGY | TAT-ASSOCIATED KINASE | IN-VIVO | CARBOXYL-TERMINAL DOMAIN | HIGH-AFFINITY | HIV-1 TAT | PROTEINS SPECIFICALLY ASSOCIATE | ELONGATION-FACTOR | tat Gene Products, Human Immunodeficiency Virus | Gene Products, tat - genetics | HIV-1 - metabolism | Phosphorylation | Chromosomal Proteins, Non-Histone - metabolism | Humans | Transcriptional Activation | Cells, Cultured | Cyclin-Dependent Kinase 9 - antagonists & inhibitors | Positive Transcriptional Elongation Factor B - metabolism | RNA Polymerase II - metabolism | HIV-1 - genetics | Spodoptera | Transcriptional Elongation Factors - metabolism | Gene Products, tat - metabolism | DNA Methylation | Animals | Piperidines - pharmacology | Trans-Activators - metabolism | Transcription, Genetic | Cyclin-Dependent Kinase 9 - metabolism | Flavonoids - pharmacology | HeLa Cells | Histones - metabolism | Trans-Activators/metabolism | Flavonoids/pharmacology | Histones/metabolism | Cyclin-Dependent Kinase 9/metabolism | Life Sciences | Chromosomal Proteins, Non-Histone/metabolism | HIV-1/genetics | Gene Products, tat/metabolism | Transcriptional Elongation Factors/metabolism | HIV-1/metabolism | Virology | Microbiology and Parasitology | Human health and pathology | Infectious diseases | Positive Transcriptional Elongation Factor B/metabolism | Cyclin-Dependent Kinase 9/antagonists & inhibitors | RNA Polymerase II/metabolism | Piperidines/pharmacology | Gene Products, tat/genetics
Journal Article
Reproduction, Fertility and Development, ISSN 1031-3613, 07/2009, Volume 21, Issue 6, pp. 805 - 815
In the present study, we investigated the effects of the Sonic hedgehog (Shh) protein on porcine oocyte maturation and early embryo development.... 
mitogen-activated protein kinase | cyclin B1 | calcium | Calcium | Mitogen-activated protein kinase | Cyclin B1 | ACTIVATED PROTEIN-KINASE | PIG OOCYTES | INTRACELLULAR CALCIUM | DEVELOPMENTAL BIOLOGY | MEIOTIC MATURATION | INDIAN HEDGEHOG | REPRODUCTIVE BIOLOGY | INOSITOL 1,4,5-TRISPHOSPHATE | ZOOLOGY | GENE-EXPRESSION | CUMULUS CELLS | IN-VITRO FERTILIZATION | MOUSE OVARY | Phosphorylation | Receptors, G-Protein-Coupled - metabolism | Calcium - metabolism | Hedgehog Proteins - metabolism | Embryo Culture Techniques | Ovarian Follicle - drug effects | Swine | Veratrum Alkaloids - pharmacology | Oocytes - drug effects | Female | Patched Receptors | Ovarian Follicle - metabolism | Recombinant Proteins - metabolism | Oocytes - metabolism | Blastocyst - metabolism | Cells, Cultured | Oncogene Proteins - metabolism | Receptors, Cell Surface - metabolism | Follicular Fluid - metabolism | Embryonic Development | Hedgehog Proteins - antagonists & inhibitors | Animals | Fertilization in Vitro | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Cyclin B - metabolism | Trans-Activators - metabolism | Mice | Zinc Finger Protein GLI1 | Patched-1 Receptor | Mitogen-Activated Protein Kinase 1 - metabolism | Calcium, metabolism | Signal Transduction, drug effects | Hedgehog Proteins, metabolism | Hedgehog Proteins, antagonists and inhibitors | Recombinant Proteins, metabolism | Trans-Activators, metabolism | Follicular Fluid, metabolism | Veratrum Alkaloids, pharmacology | Oocytes, drug effects | Receptors, G-Protein-Coupled, metabolism | Mitogen-Activated Protein Kinase 3, metabolism | Signal Transduction | Cyclin B, metabolism | Ovarian Follicle, metabolism | Oncogene Proteins, metabolism | Oocytes, metabolism | Blastocyst, metabolism | Ovarian Follicle, drug effects | Mitogen-Activated Protein Kinase 1, metabolism | Receptors, Cell Surface, metabolism
Journal Article
Fish Physiology and Biochemistry, ISSN 0920-1742, 9/2011, Volume 37, Issue 3, pp. 619 - 647
The molecular mechanisms controlling sex determination and differentiation in zebrafish (Danio rerio) are largely unknown. A genome-wide analysis may provide... 
Life Sciences | Testis | Animal Physiology | Ovary | Animal Biochemistry | Sex differentiation | Zoology | Proteomics | Freshwater & Marine Ecology | Zebrafish | Animal Anatomy / Morphology / Histology | Multidimensional protein identification technology (MudPIT) | PHYSIOLOGY | BIOCHEMISTRY & MOLECULAR BIOLOGY | WD40 REPEAT PROTEIN | ENDOPLASMIC-RETICULUM STRESS | PRIMORDIAL GERM-CELLS | LASER-CAPTURE MICRODISSECTION | MIGRATION-INHIBITORY FACTOR | TO-AUTOINTEGRATION FACTOR | FISHERIES | MESSENGER-RNA | SEX DETERMINATION | GENE-EXPRESSION | GROWTH-FACTOR-I | Egg Proteins - metabolism | Phosphorylation | Testis - metabolism | Cyclin-Dependent Kinases - metabolism | Membrane Glycoproteins - metabolism | Male | RNA - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Cell Cycle Proteins - genetics | Female | Septins - metabolism | Cyclin-Dependent Kinases - genetics | Cytokines - genetics | Ovary - metabolism | RNA - metabolism | Dyneins - metabolism | Zona Pellucida Glycoproteins | Septins - genetics | Vitellogenins - metabolism | Cytokines - metabolism | Cell Cycle Proteins - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Gene Expression Regulation - physiology | Receptors, Cell Surface - metabolism | Vitellogenins - genetics | DNA - metabolism | Sex Characteristics | Membrane Glycoproteins - genetics | DNA - genetics | Animals | Zebrafish - metabolism | SOX Transcription Factors - metabolism | SOX Transcription Factors - genetics | Signal Transduction - physiology | Dyneins - genetics | Egg Proteins - genetics | Apoptosis | Receptors, Cell Surface - genetics | Proteins | Anopheles | Messenger RNA | Analysis | Genomics | Index Medicus
Journal Article
Cell (Cambridge), ISSN 0092-8674, 2018, Volume 173, Issue 1, pp. 104 - 116.e12
Human diseases are often caused by loss of somatic cells that are incapable of re-entering the cell cycle for regenerative repair. Here, we report a... 
heart failure | cyclin | proliferation | cell cycle | regeneration | cytokinesis | cardiomyocyte | cell division | CDK | heart | HYPERTROPHY | INFARCTION | EXPRESSION ANALYSIS | AMPLIFICATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MICE | ZEBRAFISH HEART REGENERATION | FAILURE | CANCER | FIBROBLASTS | PROGRAM | CELL BIOLOGY | Cyclin D1 - metabolism | Cell Proliferation | Protein-Tyrosine Kinases - metabolism | Cyclin-Dependent Kinase 4 - genetics | Heart - physiology | Humans | Myosin Heavy Chains - genetics | Cyclin B1 - metabolism | CDC2 Protein Kinase - metabolism | Cell Cycle Proteins - antagonists & inhibitors | Myocardial Infarction - pathology | Transforming Growth Factor beta - antagonists & inhibitors | Myocardial Infarction - veterinary | Induced Pluripotent Stem Cells - cytology | Induced Pluripotent Stem Cells - metabolism | CDC2 Protein Kinase - genetics | Myocytes, Cardiac - cytology | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Cyclin B1 - genetics | Rats | Mice, Transgenic | Nuclear Proteins - metabolism | Myocardial Infarction - metabolism | Cyclin-Dependent Kinase 4 - metabolism | Cytokinesis | Regeneration | Animals | Cyclin D1 - genetics | Nuclear Proteins - antagonists & inhibitors | Myocytes, Cardiac - metabolism | Mice | Transforming Growth Factor beta - metabolism | Protein-Tyrosine Kinases - antagonists & inhibitors | Heart | Bone morphogenetic proteins | Transforming growth factors | Stem cells | Cell cycle | Cell Cycle | Cardiomyocyte | Cell Division | Cyclin
Journal Article
Biochemical and biophysical research communications, ISSN 0006-291X, 04/2017, Volume 486, Issue 2, pp. 385 - 390
TEA domain transcription factor 4 (TEAD4), which has critical functions in the process of embryonic development, is expressed in various cancers. However, the... 
TEA domain transcription factor 4 | Human oral squamous cell carcinoma | Yes-associated protein (YAP) | TARGET | BIOMARKER | BIOCHEMISTRY & MOLECULAR BIOLOGY | HIPPO PATHWAY | YAP | FAMILY | BIOPHYSICS | DNA | PROGNOSTIC MARKER | EXPRESSION | RNA, Small Interfering - genetics | Cell Proliferation | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Male | Cyclin E - genetics | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Cell Nucleus - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Muscle Proteins - antagonists & inhibitors | Transcription, Genetic | Mouth Neoplasms - genetics | Cyclin-Dependent Kinase 2 - metabolism | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction | Cyclin-Dependent Kinase 2 - genetics | Cyclin-Dependent Kinase 6 - metabolism | Cyclin-Dependent Kinase 4 - metabolism | Muscle Proteins - genetics | Cell Nucleus - genetics | Cyclin D1 - genetics | Mouth Neoplasms - surgery | Cell Line, Tumor | RNA, Small Interfering - metabolism | Cyclin D1 - metabolism | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Cyclin-Dependent Kinase 4 - genetics | Gene Expression Regulation, Neoplastic | Carcinoma, Squamous Cell - surgery | Mouth Neoplasms - metabolism | Phosphoproteins - metabolism | DNA-Binding Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Muscle Proteins - metabolism | G1 Phase Cell Cycle Checkpoints | Female | Cyclin-Dependent Kinase 6 - genetics | Transcription Factors - antagonists & inhibitors | Phosphoproteins - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Transcription Factors - metabolism | Adaptor Proteins, Signal Transducing - genetics | Mouth Neoplasms - pathology | Aged | Cyclin E - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Immunohistochemistry | Medical colleges | Embryonic development | Squamous cell carcinoma | Growth | Analysis | DNA polymerases | Genetic transcription | Health aspects | Development and progression | TRANSCRIPTION FACTORS | CHAIN REACTIONS | BIOLOGICAL MARKERS | CELL PROLIFERATION | CELL CYCLE | INTERACTIONS | 60 APPLIED LIFE SCIENCES | PLANT GROWTH
Journal Article