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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2016, Volume 311, Issue 4, pp. H871 - H880
We previously reported that endoplasmic reticulum (ER) stress is induced in the subfornical organ (SFO) and the hypothalamic paraventricular nucleus (PVN) of... 
Heart failure | Brain | Sympathetic activity | Hypothalamic paraventricular nucleus | Mitogen-activated protein kinase | Subfornical organ | Endoplasmic reticulum stress | heart failure | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | INDUCED PHOSPHORYLATION | MYOCARDIAL-INFARCTION | ER STRESS | subfornical organ | KINASE | RATS | sympathetic activity | KAPPA-B | brain | endoplasmic reticulum stress | hypothalamic paraventricular nucleus | mitogen-activated protein kinase | UNFOLDED PROTEIN RESPONSE | PERIPHERAL VASCULAR DISEASE | UP-REGULATION | Cholagogues and Choleretics - pharmacology | Tumor Necrosis Factor-alpha - genetics | Heart Failure - physiopathology | Male | NF-KappaB Inhibitor alpha - genetics | Peptidyl-Dipeptidase A - drug effects | Interleukin-1beta - genetics | Sympathetic Nervous System - physiopathology | RNA, Messenger - metabolism | Activating Transcription Factor 6 - genetics | Subfornical Organ - drug effects | Brain - metabolism | Heat-Shock Proteins - genetics | Inflammation - metabolism | Receptor, Angiotensin, Type 1 - genetics | Cyclooxygenase 2 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Real-Time Polymerase Chain Reaction | Echocardiography | Signal Transduction | Rats | Cyclooxygenase 2 - drug effects | Heart Failure - metabolism | Rats, Sprague-Dawley | Blotting, Western | Brain - drug effects | Tumor Necrosis Factor-alpha - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Endoplasmic Reticulum Stress | Paraventricular Hypothalamic Nucleus - metabolism | Infusions, Intraventricular | Mitogen-Activated Protein Kinases - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | Interleukin-1beta - drug effects | Sympathetic Nervous System - drug effects | Mitogen-Activated Protein Kinase 1 - drug effects | X-Box Binding Protein 1 - drug effects | Sympathetic Nervous System - metabolism | Transcription Factor RelA - genetics | Activating Transcription Factor 6 - drug effects | Mitogen-Activated Protein Kinase 3 - drug effects | Taurochenodeoxycholic Acid - pharmacology | Peptidyl-Dipeptidase A - genetics | Renin-Angiotensin System | Receptor, Angiotensin, Type 1 - drug effects | RNA, Messenger - drug effects | Subfornical Organ - metabolism | Heat-Shock Proteins - drug effects | Activating Transcription Factor 4 - genetics | Activating Transcription Factor 4 - drug effects | NF-KappaB Inhibitor alpha - drug effects | Paraventricular Hypothalamic Nucleus - drug effects | p38 Mitogen-Activated Protein Kinases - drug effects | Animals | Transcription Factor RelA - drug effects | X-Box Binding Protein 1 - genetics | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Cellular signal transduction | Endoplasmic reticulum | Health aspects | Mitogen-activated protein kinases | Cardiovascular Neurohormonal Regulation
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2014, Volume 45, pp. 233 - 244
Highlights • TLR4 participates in the neuroinflammation induced by ethanol during adolescence. • TLR4 participates in myelin and synaptic damage caused by... 
Psychiatry | Allergy and Immunology | Adolescence | Cognitive behavior | Myelin alterations | TLR4 | Synaptic dysfunction | Binge ethanol treatment | ACTIVATION | SUBSTANCE USE | DRUG-USE | PSYCHIATRY | RECEPTOR | HMGB1 | IMMUNOLOGY | CHILDHOOD | NEUROSCIENCES | PREFRONTAL CORTEX | ALCOHOL-USE | NEUROINFLAMMATION | BRAIN MATURATION | Central Nervous System Depressants - adverse effects | Cognition Disorders - chemically induced | Mitogen-Activated Protein Kinase Kinases - immunology | NF-kappa B - immunology | Myelin Sheath - drug effects | HMGB1 Protein - immunology | Synapses - genetics | Alcohol-Related Disorders - genetics | Myelin Proteins - drug effects | Central Nervous System Depressants - pharmacology | Nitric Oxide Synthase Type II - immunology | Ethanol - adverse effects | Cyclooxygenase 2 - immunology | Synapses - drug effects | Nitric Oxide Synthase Type II - drug effects | Toll-Like Receptor 4 - genetics | Synapses - ultrastructure | Cognition Disorders - genetics | Cyclooxygenase 2 - drug effects | HMGB1 Protein - drug effects | Cognition - drug effects | Mice, Knockout | Ethanol - pharmacology | Animals | Signal Transduction - drug effects | Myelin Sheath - genetics | Mitogen-Activated Protein Kinase Kinases - drug effects | Mice | Myelin Sheath - ultrastructure | Myelin Proteins - metabolism | NF-kappa B - drug effects | Alcohol | Alcohol, Denatured | Cognition disorders | Index Medicus
Journal Article
International Journal of Cancer, ISSN 0020-7136, 02/2014, Volume 134, Issue 3, pp. 519 - 529
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 06/2010, Volume 110, Issue 3, pp. 697 - 705
Journal Article
International Journal of Cancer, ISSN 0020-7136, 12/2007, Volume 121, Issue 11, pp. 2357 - 2363
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 2007, Volume 569, Issue 3, pp. 228 - 236
Journal Article
Journal Article
Inflammopharmacology, ISSN 0925-4692, 6/2013, Volume 21, Issue 3, pp. 201 - 232
Paracetamol is used worldwide for its analgesic and antipyretic actions. It has a spectrum of action similar to that of NSAIDs and resembles particularly the... 
Prostaglandin | COX-2 | Dermatology | Diabetes, rhabdomyolysis | Rheumatology | COX-3 | Inflammation | Prostacyclin | MIF | Allergology | Analgesia | Cyclooxygenase, COX-1 | Paracetamol | Biomedicine | Immunology | Pain | Acetaminophen | Gastroenterology | Atherosclerosis | Resveratrol | Myeloperoxidase | Peroxidase | Thromboxane | Pharmacology/Toxicology | ACETYLSALICYLIC-ACID | INHIBITORY FACTOR MIF | ACETAMINOPHEN PARACETAMOL | RENAL-FUNCTION | IMMUNOLOGY | SPINAL PROSTAGLANDIN E-2 | CYCLOOXYGENASE-3 PUZZLE | TOXICOLOGY | ANALGESIC EFFICACY | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | DOUBLE-BLIND | SELECTIVE INHIBITORS | Acetaminophen - adverse effects | Inflammation - pathology | Humans | Analgesics, Non-Narcotic - pharmacology | Cyclooxygenase 2 - drug effects | Analgesics, Non-Narcotic - therapeutic use | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Cyclooxygenase Inhibitors - pharmacology | Animals | Anti-Inflammatory Agents, Non-Steroidal - adverse effects | Acetaminophen - pharmacology | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Inflammation - drug therapy | Analgesics, Non-Narcotic - adverse effects | Cyclooxygenase 2 - metabolism | Cyclooxygenase 1 - drug effects | Acetaminophen - therapeutic use | Cyclooxygenase 1 - metabolism | Cyclooxygenase Inhibitors - adverse effects | Cyclooxygenase Inhibitors - therapeutic use | COX-2 inhibitors | Rheumatoid factor | Unsaturated fatty acids | Prostaglandins | Isoenzymes | Physiological aspects | Pharmacology | Health aspects | Index Medicus
Journal Article